AS

innate immunity part 1

First Line Defenses

  • Definition and Importance

    • Always present defenses not turned on or off.

    • Skin is the largest immune organ and provides a physical barrier.

  • Skin Characteristics

    • Composed of multiple compact cell layers; outer layer consists of dead cells filled with keratin.

    • Keratin imparts toughness and waterproof qualities, preventing microbial penetration.

    • Some parasitic worms can burrow through the skin (exceptions).

  • Mucous Membranes

    • Internal surfaces exposed to the environment: gastrointestinal tract, respiratory tract, urogenital tract.

    • Continuous production of mucus by goblet cells provides a physical barrier that traps microorganisms.

    • Ciliary action in the respiratory tract helps move mucus loaded with microbes up to the mouth for swallowing.

Antimicrobial Factors

  • Saliva and Other Secretions

    • Contains antimicrobial substances like lysozyme, peroxidase, and lactoferrin.

    • Lysozyme breaks down bacterial cell walls; peroxidase generates toxic radicals; lactoferrin sequesters iron to inhibit microbial growth.

  • Stomach Defenses

    • Acidic pH (low) helps kill most microorganisms that survive the mouth.

    • Rapid pH change and normal microbiota further protect the gastrointestinal tract.

Normal Microbiota Role

  • Occupied space and consumed nutrients, preventing pathogen attachment.

  • Produced antimicrobial factors, adding to the protective effect.

  • Peristalsis: Continuous movement of food in the digestive tract helps flush out pathogens.

Urinary Tract Defense Mechanisms

  • Regular flushing mechanism prevents urinary tract infections.

  • Normal microbiota in the vagina further protect against infections.

Respiratory Tract Defense Mechanisms

  • Nasal passages trap microbes in mucus that is moved by cilia.

  • Mucociliary escalator carries microbes to the mouth for swallowing.

  • Macrophages present in the lungs can engulf any microbes that reach this depth.

Innate Immune Response Overview

  • Detection of Invaders

    • Sensors like complement proteins detect foreign cells and damage.

  • Innate Immune Cells

    • Understanding innate immune cells is key; these include neutrophils, macrophages, and dendritic cells.

Hematopoiesis and Immune Cells

  • Hematopoiesis

    • Occurs in bone marrow; progenitor cells differentiate into myeloid and lymphoid lines.

  • Key Immune Cells

    • Myeloid lineage: include red blood cells, platelets, neutrophils, monocytes, macrophages, dendritic cells.

Immune Response Recognition and Communication

  • Pattern Recognition Receptors (PRRs)

    • Recognize microbial patterns on pathogens or damage patterns on host cells.

    • Two groups of PRRs:

      • MAPs (Microbe-Associated Molecular Patterns): e.g., LPS, flagellin.

      • DAMPs (Damage-Associated Molecular Patterns): signal cell damage.

  • Cytokines as Communicators

    • Soluble signals released by cells (like macrophages) to inform other cells and initiate responses (e.g., inflammation).

    • Example cytokines: Interleukin-1 (induce fever), TNF (promote inflammation).

Phagocytosis Process

  • Key Phagocytes

    • Neutrophils: First responders that circulate in blood.

    • Macrophages: Tissue residents developed from monocytes.

  • Phagocytosis Steps

    1. Recognition and attachment to pathogens.

    2. Engulfment of pathogens into a membranous vesicle (phagosome).

    3. Fusion of phagosome with lysosome to form a phagolysosome for digestion.

    4. Elimination of microbial components via exocytosis.

Microbial Evasion Mechanisms

  • Some microbes produce substances such as enzymes and capsules to evade phagocytosis.

  • Others can survive within phagosomes and avoid lysosomal destruction.

Eosinophils in Response to Large Parasites

  • Eosinophils: Target larger eukaryotic parasites by attaching and releasing granules containing toxic products.

  • Elevated eosinophil counts in blood can indicate parasitic infections.

Summary

  • Small microbes are eliminated by phagocytosis while larger ones are attacked by eosinophils and other granulocytes. Next topics will cover the complement system and inflammatory responses.