Presentation by: Jessie Seeck, STLCC Wildwood
Major Groups:
Congenital Heart Disease: Defects present at birth.
Acquired Cardiac Disorders: Develop during or after birth.
Impact: Both types can cause varying degrees of heart failure.
History Collection:
Maternal health history (diabetes, lupus, substance use, infections like rubella, teratogenic drugs- DILANTIN).
Family health history.
Physical Assessment Techniques:
Inspection
Palpation
Percussion
Auscultation
Physical Exam of Child with cardio dysfunction
Failure to thrive
Cyanosis
Respiratory excursions
Clubbing of fingers
Abdomen: Hepatomegaly or splenomegaly may be present.
Peripheral pulses: rates regularity and amplitude may reveal discrepancies.
Auscultation of heart: tachy, brady, irregular rhythm, murmurs.
Additional Evaluations:
Diagnostic evaluations like EKG, echocardiogram, chest X-ray, MRI, CBC, ABGs, hyperoxia test.
Heart catheterizations for diagnostics and treatment.
PRE/POST OP
Preprocedural Care:
Complete nursing assessment including allergies, signs of infection, and vital signs.
Postprocedural Care:
Monitor for complications, including vital signs, extremities checks at access point, bleeding from dressings, and fluid intake.
Closure of Fetal Shunts:
Umbilical Cord Clamping: Triggers functional closure of the umbilical vein and arteries, and foramen ovale.
Foramen Ovale: Closes as left atrial pressure exceeds right atrial pressure.
Ductus Arteriosus: Begins to close when oxygen levels increase.
Important structures include:
Superior vena cava, inferior vena cava, pulmonary artery, aorta, and valves.
Tools Used:
EKG
Holter Monitor
Echocardiogram
Chest X-ray
MRI
CBC and ABGs
Hyperoxia test
Heart catheterizations
Pre-Procedure:
Assess and mark pulses, baseline O2 saturation, explain procedures to parents and children.
NPO for 6-8 hours before the procedure.
Post-Procedure Monitoring:
Monitor heart rhythm with heart monitor, oxygen saturation, and check for signs of bleeding or infection.
pulse, temp, check extremitiesand assess capillary refill to ensure adequate circulation and recovery.
FULL 1 MIN PULSE COUNT
BP- monitor for hypotension = BLEEDING
Dressing- check for bleeding.
Fluid intake- at risk for hypovolemia/dehydration
Blood glucose- infants are at risk for HYPOGYLCEMIA= IV FLUID DEXTROSE
Strict bed rest, particularly for diaper-wearing patients to prevent infection.
Teaching and Guidance:
Daily bandage changes for two days, keeping the site clean and dry.
Avoid baths and swimming for one week; sponge baths are acceptable.
Regular diet intake, use of acetaminophen for pain.
Discuss return to activities with the healthcare provider.\
Keep follow up visits.
Educate the patient and family about signs of complications, such as increased fatigue or difficulty breathing.
If you apply 02 to a baby with an acyanotic defect it will do NOTHING.
Categories:
Increased Pulmonary Blood Flow: blood shifts from the left to right side of heart through a hold. Acyanotic defects, e.g.,
Ventricular Septal Defect (VSD)- can close spontaneously, loud harsh murmur, S/S of heart failure.
Atrial Septal Defect (ASD)- can be asymptomatic, loud murmur, s/s of heart failure (fluid overload,dyspnea, edema)
Patent Ductus Arteriosus (PDA).
Obstruction to Blood Flow: Obstructive defects like pulmonary and aortic stenosis.
Decreased Pulmonary Blood Flow: Right to left shunts, e.g., Tetralogy of Fallot, Tricuspid Atresia.
Mixed Blood Flow: Transposition of Great Vessels, Truncus Arteriosus, Hypoplastic Left Heart Syndrome.
Ventricular Septal Defect:
Characteristics: loud murmur, possible asymptomatic, signs of heart failure.
Treatment: device closure, surgical repair depending on the size.
Pulmonary Stenosis:
Diagnosis and management through catheterization or surgical procedures based on age and severity.
Aortic Stenosis:
Requires careful monitoring of vital signs and potential interventions for severe cases.
General Characteristics:
Present simultaneously with other issues like ASD or VSD, leading to cyanosis and signs of hypoxia.
Tetralogy of Fallot:
Significant nursing considerations include high calorie nutrition and preparing for hypercyanotic episodes.
Goals of Treatment:
Improve cardiac function, remove excess fluid and sodium, decrease cardiac demands, and improve tissue oxygenation.
Medications:
Digoxin for contractility, ACE inhibitors, and diuretics for fluid management.
Monitor for side effects and adjust care based on patient's needs.
Clinical Manifestations: High fever, edema, erythema, peeling skin, strawberry tongue, rash, lymphadenopathy.
Nursing Management: IVIG, aspirin treatment, monitoring heart function and providing symptomatic support.
Blood Pressure: Infant SBP 70+; Age 10+ SBP 90+
Pulse Rates and Respirations varied across age groups, with detailed ranges provided for newborns to adolescents.
What to do in case of a hypercyanotic spell?
Signs and symptoms of digoxin toxicity in children?
Key manifestations and treatment options for Kawasaki Disease?