Sensation – detection of physical energy (light, sound, pressure, heat, chemical) by specialized receptor cells that convert that energy into neural impulses.
Perception – integration, organization, and interpretation of those sensory impulses, producing conscious experience.
Key distinction: sensation = raw data acquisition; perception = meaningful interpretation (e.g., hearing pressure changes vs. recognizing a melody).
Theme: All sensation begins with physical energy → sense receptor → neural impulse → brain.
Sensory receptors – unique cells in each organ (rods, cones, hair cells, taste-bud cells, etc.).
Transduction – biochemical process that converts physical energy into coded neural signals the CNS can understand.
Pathway summary: Environmental energy → receptor membrane potential change → action potentials → specific sensory pathway → cortex → PERCEPTION.
A stimulus must reach the receptor’s threshold to be detected.
Minimum stimulation needed to consciously detect a stimulus 50\% of the time.
Vision: candle flame at 30 miles on a dark, clear night.
Hearing: watch tick at 20\,ft in quiet.
Taste: 1 tsp sugar in 2 gal water.
Smell: one drop perfume diffused through a 3-room apartment.
Touch: bee’s wing falling 0.5\,in onto cheek.
Smallest change in stimulation required to detect a difference.
Weber’s Law – JND is a constant proportion k of the original stimulus intensity S.
\Delta S = kS or in logarithmic form \Psi = k \log S ((\Psi) = sensation magnitude).
Fechner’s extension integrates all JNDs to describe total perceived intensity; reflects non-linear relationship between physical & psychological magnitude.
Prolonged, constant stimulation ➝ declining receptor response and diminished conscious sensation (e.g., clothes on skin, background hum).
Light = electromagnetic radiation; described by wavelength (distance peak-to-peak) and amplitude.
Perceptual correlates:
Wavelength ➝ Hue (color name).
Purity of wavelength ➝ Saturation.
Amplitude ➝ Brightness.
Cornea – fixed curved surface; begins focusing.
Pupil – adjustable aperture within iris; size varies with light & arousal.
Lens – elastic; performs accommodation (changes curvature) to focus near vs. far objects onto the retina.
Retina – thin neural tissue lining back wall; contains photoreceptors.
Fovea – central pit, all cones, point of greatest acuity.
Optic disk / blind spot – no receptors where optic nerve exits; brain “fills in.”
Rods (~120\text{ million})
Long, thin; extreme light sensitivity; grayscale vision; peripheral & night vision; none in fovea.
Cones (~6\text{ million})
Short, tapered; require more light; encode color; concentrated in fovea ➝ high visual acuity.
Bipolar cells collect signals from rods/cones ➝ ganglion cells.
Each ganglion cell has a receptive field; rod inputs are pooled (many:1) ➝ low detail; cone inputs few:1 ➝ high detail.
Axons of ganglion cells form optic nerve.
Optic nerves meet at optic chiasm: nasal fibers cross, temporal stay ipsilateral ➝ thalamus (LGN) ➝ primary visual cortex (V1).
Cortical segregation:
Ventral stream (“what”) ➝ temporal lobe, object identity.
Dorsal stream (“where/how”) ➝ parietal lobe, spatial location & action.
Blindsight – V1 damage can leave dorsal/ventral sub-paths partially functional ➝ unconscious vision.
Involuntary, consistent blending of senses (e.g., grapheme-color, sound-taste).
fMRI & connectivity studies (Ramachandran, Cytowic) suggest hyper-connectivity among sensory cortical areas.
Trichromatic Theory (Young–Helmholtz)
Three cone types tuned to long (red), medium (green), short (blue) wavelengths.
Explains color mixing & red-green color blindness.
Opponent-Process Theory (Hering)
Post-receptor channels antagonistic: red ↔ green, blue ↔ yellow, black ↔ white.
Explains after-images & color contrast.
Modern view: trichromatic coding at receptor level ➝ opponent processing in ganglion, LGN, and cortex.
Red–green color blindness – missing/abnormal L or M cones; blue perception intact.
Myopia – focus in front of retina; distant blur. Hyperopia – focus behind retina; near blur.
Accommodation declines with age (presbyopia).
Sound wave – variation in air pressure.
Amplitude ➝ Loudness (decibels, \text{dB}).
Frequency ➝ Pitch (Hertz, \text{Hz});
Humans hear 20{-}20{,}000\,\text{Hz}.
Complexity ➝ Timbre (quality; allows piano vs. sax to be distinguished on same note).
0\,\text{dB} – threshold of hearing.
70\,\text{dB} – busy traffic; prolonged exposure begins risk.
90\,\text{dB} – lawn mower; >8 hr unsafe.
120\,\text{dB} – rock concert; immediate damage.
140\,\text{dB} – jet engine/shotgun; any exposure unsafe.
Outer ear – pinna collects waves ➝ auditory canal ➝ eardrum (tympanic membrane).
Middle ear – ossicles: hammer (malleus), anvil (incus), stirrup (stapes) amplify vibration; stirrup pushes on oval window.
Inner ear – cochlea (fluid-filled spiral): oval window vibration ➝ fluid waves ➝ basilar membrane motion ➝ bending of hair-cell cilia against tectorial membrane ➝ transduction.
Neural impulses along auditory nerve ➝ thalamus ➝ primary auditory cortex (temporal lobe).
Round window flexes to relieve fluid pressure.
Frequency Theory – entire basilar membrane vibrates at same frequency; hair cells fire volleys; explains low pitches (<\approx 1000\,\text{Hz}).
Volley Principle – groups of neurons alternate firing to reach higher combined rates.
Place Theory – specific location of maximal membrane displacement encodes frequency; best for high pitches (>\approx 5000\,\text{Hz}); cochlea is tonotopic (base = high, apex = low).
Intermediate frequencies use combination of both.
Conduction deafness – ossicle or eardrum damage; hearing aid helps.
Nerve (sensorineural) deafness – hair cell damage (e.g., loud noise); cochlear implant bypasses receptor.
Odorant molecules bind to receptors in nasal epithelium ➝ axons form olfactory nerve ➝ olfactory bulb (brain’s front) ➝ projections to temporal lobe (conscious ID) & limbic system (emotion/memory).
Direct cortical route (no thalamic relay); function declines with age & disease.
Taste buds (~10{,}000) embedded in tongue papillae; each bud ≈ 50 receptor cells with microvilli projecting into taste pore.
Five primary tastes ➝ sweet, sour, salty, bitter, umami (MSG/protein).
“Tongue map” is myth; all regions can detect all tastes but with differing densities.
Skin houses multiple receptors (pressure, vibration, stretch, temperature).
Distribution uneven; areas needing fine control (fingertips, lips) have high receptor density.
Nociceptors – free nerve endings in skin, muscle, organs.
Two fiber systems:
Fast (A-delta) – sharp, localized pain; myelinated.
Slow (C) – dull, throbbing, emotional pain; unmyelinated.
Gate-Control Theory (Melzack & Wall) – spinal “gate” modulates pain signals; inputs from brain (attention, emotion, endorphins) and competing touch signals can open/close gate.
Sensitization – chronic activation increases pathway responsiveness (opposite of adaptation) ➝ chronic pain, phantom limb.
Pain perception routes: sensory (somatosensory cortex) + affective (limbic) components.
Intensify: anxiety, fear, helplessness, negative mood, social contagion.
Reduce: control, positive mood, relaxation, endogenous opioids (endorphin, enkephalin), supportive context.
Proprioceptors in muscles/joints signal limb position & movement.
Vestibular organs (semicircular canals & vestibular sacs in inner ear) detect head rotation, linear acceleration, gravity ➝ balance & spatial orientation.
Bottom-up – data-driven; build perception from sensory elements (feature detectors).
Top-down – concept-driven; expectations, knowledge, culture shape interpretation (e.g., reading messy handwriting, cultural focus on context vs. object).
Chua et al.: U.S. students fixate on focal object; Chinese students scan background ➝ holistic vs. analytic perception.
Figure–Ground – automatic segregation of figure (focus) & background; reversible figures illustrate ambiguity.
Grouping rules (illustrated by dots/lines icons):
Similarity – alike items grouped.
Proximity – close items grouped.
Continuity – smooth paths preferred.
Closure – fill gaps to see whole.
Symmetry – balanced forms favored.
Relative size, Overlap (interposition), Aerial perspective (haze), Texture gradient, Linear perspective, Relative height, Motion parallax (near objects move faster across retina).
Convergence – eye muscle strain; more rotation = closer object.
Binocular (retinal) disparity – positional difference of images; greater disparity = nearer.
Expanding retinal image ➝ object approaching; rate of expansion estimates speed.
Specialized cortical neurons code specific directions & speeds.
Size constancy – perceive actual size despite retinal image change.
Shape constancy – door viewed at angle still “rectangular.”
Müller-Lyer – arrows influence size-distance scaling (carpentered-world hypothesis: experience with right angles enhances illusion susceptibility).
Moon illusion – horizon moon appears larger due to perceived distance & context cues.
Expectancy frame guides interpretation; usually aids accuracy but can cause false positives (seeing faces in clouds).
Proposed perception without sensory input: telepathy, clairvoyance, precognition, psychokinesis.
Studied by parapsychology; empirical support weak/inconclusive.
Clinically & self-administered aides: hypnosis, analgesics, biofeedback, acupuncture plus everyday distraction, guided imagery, relaxation, meditation, positive self-talk, cognitive reappraisal.
Hearing – sound waves – ear – hair cells (cochlea)
Vision – light – eye – rods & cones (retina)
Smell – airborne molecules – nose – olfactory cilia
Taste – dissolved chemicals – tongue – taste-bud receptors
Touch – pressure – skin – Pacinian corpuscles, etc.
Pain – tissue damage – widespread – nociceptors
Proprioception – muscle stretch – muscles/joints – proprioceptors
Vestibular – gravity/motion – semicircular canals/sacs – hair-cell receptors
Weber’s Law: \Delta S = k S
Fechner/Log Law: \Psi = k \log S
Hearing range: 20{-}20{,}000\,\text{Hz}
Hazardous sound level begins ≈ 85\,\text{dB}
Visible light wavelength ≈ 400{-}700\,\text{nm}
These bullet-point notes consolidate every major and minor point from Chapter 3’s transcript, blending definitions, mechanisms, theories, numerical values, examples, cultural & clinical implications, and mnemonic comparisons to serve as a stand-alone study guide on Sensation and Perception.