1.2 Cardiovascular System
Pathway of Blood:
1) The walls of the right atrium squeeze and push the blood downwards to the right ventricle
2) The ventricles contract and the pulmonary artery then transports deoxygenated blood to the lungs
3) Blood is oxygenated (due to gas exchange) at the lungs
4) The pulmonary vein transports oxygenated blood back to the left atrium
5) Blood is pushed downwards and collects in the left ventricle
6) Oxygenated blood is the ejected and transported to the body (muscles) through the aorta
7) The deoxygenated blood is returned to the heart through the vena cava
Cardiac Control:
Neural:
Proprioceptors - in muscles, tendons, and joints - informs the CCC that movement has increased
Chemoreceptors - in aorta and carotid arteries- detects a decrease in blood pH due to an increase in lactic acid and CO2
Baroreceptors - in blood vessel walls - informs CCC of increased blood pressure
Intrinsic:
Temperature - affect blood viscosity and and the speed of nerve impulse transmission
Venous Return - affects the stretch in ventricular walls, force of contraction, and so stoke volume
Hormonal:
Adrenaline - released from the adrenal glands and increase SV and HR
Noradrenaline - released from the adrenal glands and decreases SV and HR
Regulation of HR during exercise:
Controlled by the autonomic nervous system (ANS) and determines the frequency of firing of the SA node
Located in the medulla oblongata - brain
Responsible for regulating the heart via motor nerves
Sympathetic nervous system - accelerator nerve - during exercise - increases heart rate
Parasympathetic nervous system - vagus nerve - during recovery - decreases heart rate
Conduction System:
Myogenic - generates its own impulse, which causes the heart to contract
SA Node - generates an electrical impulse, causing atrial systole
SA Node is known as the pacemaker and causes the hearts walls to contract
AV Node - collects the impulse and delays it by 0.1 seconds to allow the atria to finish contracting
Bundle of His - located in the septum, and splits the impulse in 2, and is ready to be distributed to the ventricles
Bundle Branches - carries the impulses to the bases of each ventricles
Purkinje Fibres - distributes the impulse through the ventricle walls, causing them to contract
Cardiac Cycle:
Atrial Systole
Atria contract
Forces blood into the ventricles
Ventricular Systole
Ventricles contract
AV valves close
Semilunar valves open
Blood is forced out the ventricles and into the arteries leaving the heart
Diastole
Relaxation of the atria and ventricles
Blood passively flows through atria and into the ventricles
AV valves are open - blood can move from atria to ventricles
Semilunar valves are closed
Heart Values:
Heart Rate: number of beats per minute
Untrained at rest = 70-72
Trained at rest = 50
Untrained sub-maximal = 100-130
Trained sub-maximal = 95-120
Untrained maximal = 200-age
Trained maximal = 220-age
Stroke Volume: amount of blood ejected from the left ventricle per beat
Untrained at rest = 70ml
Trained at rest = 100ml
Untrained sub-maximal = 100-120ml
Trained sub-maximal = 160-200ml
Untrained maximal = 100-120ml
Trained maximal = 160-200ml
Cardiac Output: amount of blood ejected from the left ventricle per minute
Untrained at rest = 5L/ min
Trained at rest = 5L/ min
Untrained sub-maximal = 10-15L/ min
Trained sub-maximal = 15-20L/ min
Untrained maximal = 20-30L/ min
Trained maximal = 30-40L/ min
Response to Exercise:
Heart Rate
Response to sub-maximal - Anticipatory rise, Rapid increase, Steady plateau, Rapid decrease
Response to maximal - Anticipatory rise, Rapid increase, Slower increase, Rapid decrease, Slower decrease
Stroke Volume:
Increases proportionately to exercise intensity until a plateau is reached at around 40-60% of working capacity
increased venous return
the Frank-Starling mechanism (increased venous return - increased SV - walls stretch more - more forceful contractions)
dependent on venous return
more blood returns to the heart during exercise
causes the atria to stretch more than at rest
forces more blood into the ventricles - they stretch more
recoil produces a larger contraction, forcing more blood out the heart per beat
increases stroke volume
Plateaus because increased HR towards maximal intensity doesn't allow enough time for the ventricles to fully fill during diastole
Cardiac output will increase in line with exercise intensity and plateu during maximal exercise
Recovery
Cardiac Output, Heart Rate and Stroke Volume
SV is maintained during early stages of recovery as HR rapidly decreases
Maintains blood flow and the removal of waste products
Lowers stress and workload on cardiiac muscle
In recovery, there is a rapid decrease of CO followed by a slower decrease back to resting levels
Vascular System
Blood Vessels
Arteries and arterioles
carry oxygenated blood from the heart to organs
high blood pressure
large layer of smooth muscle and elastic tissue
vasoconstrict and vasodilate - control blood flow and pressure
Capillaries:
walls are one cell thick
gas exchange takes place
oxygen passes through the walls and into the tissues
CO2 passes from the tissue into the capillary walls
Veins and venules
carry deoxygenated blood from muscles and organ to the heart
thin walls
smooth layer of muscle - vasoconstrict and vasodilate
blood under low pressure
one way pocket valves - prevent backflow of blood
Venous Return Mechanisms
pocket valves - prevent backflow of blood - located in the veins
smooth muscle - vasoconstricts and pushes blood back towards the heart - vein walls
gravity - gravity aids blood in the upper body
muscle pump - veins are located between skeletal muscle - surrounding muscles squeeze on veins and push blood back to the heart
respiratory pump - returns blood in the thoracic cavity - changes the pressure between high and low during exercise - pushes blood back to the heart
Cardiovascular Drift
occurs during exercise in the heat
sweating
lose fluids
decreased blood plasma volume
blood becomes more viscous
SV decreases
HR increases to maintain CO
Blood Pooling
blood builds up in the pocket valves, and pools - often in lower body
occurs after going from exercise to rest with no cooldown
can be prevented by performing a cooldown after exercise