Occurs beneath the coraco-acromial arch.
Vulnerable structures:
Greater tuberosity
Supraspinatus tendon
Long head of biceps
Major site of compression is slightly anterior to the angle of the acromion.
Also referred to as painful arc syndrome.
Complete or partial rupture of rotator cuff.
Supraspinatus tendinitis.
Calcific deposits.
Subacromial bursitis.
Subdeltoid bursitis.
Periarthritis.
Bicipital tenosynovitis.
Fracture of greater trochanter.
Common symptoms:
Pain,
Swelling,
Limitation of shoulder movements,
Muscle atrophy,
Tenderness over the greater tuberosity.
Types of shoulder pain:
Chronic: Diffuse posterior shoulder girdle pain, seen in throwing athletes with internal impingement. Pain increases during the late-cocking phase of throwing when the arm is in 90° abduction and full external rotation.
Acute: Non-throwing athletes report acute shoulder pain post-injury.
Functional impairments:
Decrease in throwing velocity or loss of control in overhead athletes.
"Dead arm" sensation post-throwing with symptoms of weakness and shoulder slipping.
Muscular asymmetry between dominant and nondominant shoulder, often noted in overhead athletes.
Muscular/Neuromuscular Imbalance:
Commonly seen in the shoulder complex due to improper neuromuscular control.
Increased Laxity:
May present with global laxity or isolated anterior laxity in the dominant shoulder.
Anterior Instability:
Patients may report instability symptoms such as apprehension or sensation of subluxation in abduction and external rotation positions.
Rotator Cuff Pathology:
Symptoms may mimic other rotator cuff pathologies. In young athletes, suspicion for internal impingement should be raised due to overlap of symptoms.
Signs of muscle balance disruption among the four rotator cuff muscles (infraspinatus, supraspinatus, teres minor, and subscapularis).
Grade I:
Affects young adults/athletes (ages 18-30).
Caused by overstress and repeated overhead activity.
Supraspinatus inflammation leading to painful arc during shoulder abduction (40 to 120°).
Pain occurs in late-cocking and early acceleration phases of throwing; daily activities are usually pain-free.
Grade II:
Affects individuals aged 40-45.
Associated with supraspinatus tendinitis or subacromial bursitis.
Overuse or degeneration, with localized pain in the posterior shoulder in throwing phases.
Grade III:
Occurs in those over 45 years of age.
Linked with occupational overuse, falls, or sudden activity increases leading to degenerative changes in rotator cuff.
Basic Examination:
Palpation of the shoulder complex.
Observation of muscle symmetry between shoulders.
Gross strength testing of shoulder and rotator cuff muscles.
Assessment of joint accessory mobility across GH/ST/AC/SC joints.
Flexibility tests for shoulder, thoracic, and cervical spine.
Range of motion assessments for the GH/scapulothoracic joints and cervical/thoracic spine, noting dysfunction can affect shoulder performance.
Tenderness to palpation in the posterior shoulder/joint line.
Involved shoulder may show increased muscle bulk and altered position compared to the unaffected side.
Abnormal scapulothoracic rhythm and movements.
Weakness in rotator cuff, middle/lower traps, rhomboids, and serratus anterior muscles.
Decreased dorsal glenohumeral glide and tightness in posterior capsule.
Muscular imbalances in pectoralis muscles, latissimus dorsi, and upper trap.
Decreased internal rotation and increased external rotation at GH joint typically noted.
SICK Scapula: Scapular protraction, malposition, coracoid pain, and dyskinesis identified during examination.
Plain X-rays are less useful.
MRI and arthrography provide reliable diagnostic information.
Conservative treatment:
NSAIDs, hydrocortisone injections, subacromial steroid injections, active and passive exercises, temporary immobilization, and massage.
High recovery rate (90%) with conservative measures.
Strengthening:
Closed kinetic chain exercises for rotator cuff stabilization.
Addressing GIRD (glenohumeral internal rotation deficit) and posterior capsule strengthening.
Phase One Rehabilitation:
Soft tissue mobilization, scapula setting, joint mobilization, and improving shoulder ROM.
Neuromuscular re-education for recurrence prevention and restoring muscle balance.
Phase Two Rehabilitation:
Focus on dynamic stability and muscle balance restoration with complex exercises.
Start introducing eccentric and open kinetic chain exercises tailored to athletic movements.
Phase Three Rehabilitation:
Functional rehab plans for athletes focusing on returning to full activities, progressing plyometric exercises with controlled movements.
Recommend initial immobilization for three weeks.
Encourage active movements in elbow, hand, and wrist early on.
Begin strong isometric deltoid exercises after ten days, maintaining conservative post-operative measures.
Q1: What is Anterior Impingement Syndrome?A1: Anterior Impingement Syndrome occurs beneath the coraco-acromial arch, affecting vulnerable structures like the greater tuberosity, supraspinatus tendon, and long head of biceps, with compression most prevalent just anterior to the angle of the acromion.
Q2: What are the primary causes of Impingement Syndrome?A2: Causes include:
Complete or partial rupture of the rotator cuff
Supraspinatus tendinitis
Calcific deposits
Subacromial and subdeltoid bursitis
Periarthritis
Bicipital tenosynovitis
Fracture of the greater trochanter
Q3: What are the common clinical features or symptoms of this syndrome?A3: Common symptoms are pain, swelling, limited shoulder movements, muscle atrophy, and tenderness over the greater tuberosity. There are different types of shoulder pain:
Chronic: Diffuse posterior shoulder girdle pain, typical in throwing athletes with internal impingement, with pain increasing in the late-cocking phase of throwing.
Acute: Reported by non-throwing athletes post-injury.
Q4: How does Anterior Impingement Syndrome affect athletic performance?A4: Functional impairments include decreased throwing velocity, loss of control in overhead athletes, and a "dead arm" sensation post-throwing, with symptoms of weakness and perceived shoulder instability.
Q5: What is the pathophysiology behind Anterior Impingement Syndrome?A5: Key aspects include:
Muscular/Neuromuscular Imbalance: Commonly due to improper control in the shoulder complex.
Increased Laxity: Present as global or isolated anterior laxity.
Anterior Instability: Symptoms such as apprehension or feelings of subluxation in specific arm positions.
Rotator Cuff Pathology: Overlap of symptoms with other rotator cuff issues, especially in younger athletes.
Q6: How is Anterior Impingement Syndrome graded or staged?A6: The grading system includes:
Grade I: Affects young adults (18-30); caused by overstress, leading to supraspinatus inflammation and painful arc during abduction (40° to 120°).
Grade II: Affects those 40-45 years; associated with supraspinatus tendinitis or subacromial bursitis.
Grade III: Occurs in individuals over 45; linked to degenerative changes from overuse or falls.
No
Q7: What examination techniques are used to assess Anterior Impingement Syndrome?A7: Basic techniques involve palpation, muscle symmetry observation, strength testing, and range of motion assessments for relevant joints. Additional tests include flexibility tests and assessing joint accessory mobility.
Q8: What findings might be observed during the examination of a patient with this syndrome?A8: Possible findings include tenderness in the posterior shoulder, increased muscle bulk on one side, abnormal scapulothoracic movements, and weakness in specific muscle groups.
Q9: What additional considerations should be made during assessment?A9: Consider muscular imbalances in pectoralis muscles, latissimus dorsi, and upper trap, as well as altered internal and external rotation in the glenohumeral joint.
Q10: Which imaging investigations are useful for diagnosing Anterior Impingement Syndrome?A10: Plain X-rays are generally less useful, while MRI and arthrography provide reliable diagnostic information.
Q11: What are the management strategies for Anterior Impingement Syndrome?A11: Conservative treatment may include NSAIDs, steroid injections, exercises, and temporary immobilization. Recovery rates are high (90%) with these measures. Rehabilitation is phased:
Phase One: Soft tissue mobilization, scapula setting, joint mobilization.
Phase Two: Dynamic stability and muscle balance restoration exercises.
Phase Three: Functional rehabilitation focusing on returning athletes to full activities.
Q12: What post-surgery physiotherapy is recommended?A12: Initial immobilization for three weeks, early active movement in elbow, hand, and wrist, followed by strong isometric deltoid exercises after ten days, while maintaining conservative measures post-operatively.
Profile: 22-year-old male baseball pitcher.
Symptoms: Reports a painful arc during shoulder abduction (40° to 120°), primarily during practice. Experiences pain in the late-cocking phase of throwing.
Evaluation: Mild supraspinatus inflammation noted during examination, with tenderness around the greater tuberosity.
Management: Treated with NSAIDs, physiotherapy focused on soft tissue mobilization and scapula setting, with emphasis on shoulder ROM exercises.
Outcome: After 6 weeks of rehabilitation, the athlete returned to pitching with no pain.
Profile: 43-year-old female recreational tennis player.
Symptoms: Complains of localized pain in the posterior shoulder, especially while serving or performing overhead motions. Limited shoulder mobility observed.
Evaluation: Examination reveals tenderness over subacromial space and signs of subacromial bursitis.
Management: Injections of corticosteroids, in combination with a structured physiotherapy program focused on strengthening and improving shoulder stability.
Outcome: Improved shoulder function and reduced pain after 8 weeks; able to return to recreational play without significant discomfort.
Profile: 58-year-old male construction worker.
Symptoms: Experiences severe shoulder pain after a fall, with acute limitation in shoulder mobility. Reports difficulty lifting objects and persistent “dead arm” sensation.
Evaluation: Examination confirms significant weakness in rotator cuff muscles and radiologic imaging shows changes consistent with degenerative rotator cuff disease.
Management: Surgery indicated due to severe symptoms and structural changes. Post-operative management included a progressive rehabilitation program emphasizing strength restoration and functional activities.
Outcome: After 3 months of rehabilitation, the patient reports significant improvement and is able to perform work-related activities with minimal pain.
Assessment Diagnosis: Physical examination reveals mild supraspinatus inflammation, tenderness around the greater tuberosity, and a painful arc in shoulder abduction (40° to 120°).
Physiotherapy Management:
Focus on soft tissue mobilization and scapula setting.
Progress shoulder range of motion (ROM) exercises to alleviate symptoms.
Strengthening exercises once pain decreases, with adjustments based on tolerance.
Assessment Diagnosis: Examination shows tenderness over the subacromial space and limited shoulder mobility, indicating subacromial bursitis.
Physiotherapy Management:
Initiate with corticosteroid injections for inflammation control.
Structured physiotherapy program focusing on strengthening rotator cuff and scapular stabilizers.
Emphasis on functional movements related to overhead activities, restoring mobility and reducing pain.
Assessment Diagnosis: Significant weakness in rotator cuff muscles confirmed via examination; radiologic imaging displays degenerative changes linked to recurrent shoulder pain after a fall.
Physiotherapy Management:
Post-operative rehabilitation focusing on strength restoration and gradual return to functional activities.
Emphasize progressive exercises, including concentric and eccentric strengthening of rotator cuff, along with scapular stabilization.
Prioritize pain management and gradual reintroduction to work-related activities.
file (1)
Occurs beneath the coraco-acromial arch.
Vulnerable structures:
Greater tuberosity
Supraspinatus tendon
Long head of biceps
Major site of compression is slightly anterior to the angle of the acromion.
Also referred to as painful arc syndrome.
Complete or partial rupture of rotator cuff.
Supraspinatus tendinitis.
Calcific deposits.
Subacromial bursitis.
Subdeltoid bursitis.
Periarthritis.
Bicipital tenosynovitis.
Fracture of greater trochanter.
Common symptoms:
Pain,
Swelling,
Limitation of shoulder movements,
Muscle atrophy,
Tenderness over the greater tuberosity.
Types of shoulder pain:
Chronic: Diffuse posterior shoulder girdle pain, seen in throwing athletes with internal impingement. Pain increases during the late-cocking phase of throwing when the arm is in 90° abduction and full external rotation.
Acute: Non-throwing athletes report acute shoulder pain post-injury.
Functional impairments:
Decrease in throwing velocity or loss of control in overhead athletes.
"Dead arm" sensation post-throwing with symptoms of weakness and shoulder slipping.
Muscular asymmetry between dominant and nondominant shoulder, often noted in overhead athletes.
Muscular/Neuromuscular Imbalance:
Commonly seen in the shoulder complex due to improper neuromuscular control.
Increased Laxity:
May present with global laxity or isolated anterior laxity in the dominant shoulder.
Anterior Instability:
Patients may report instability symptoms such as apprehension or sensation of subluxation in abduction and external rotation positions.
Rotator Cuff Pathology:
Symptoms may mimic other rotator cuff pathologies. In young athletes, suspicion for internal impingement should be raised due to overlap of symptoms.
Signs of muscle balance disruption among the four rotator cuff muscles (infraspinatus, supraspinatus, teres minor, and subscapularis).
Grade I:
Affects young adults/athletes (ages 18-30).
Caused by overstress and repeated overhead activity.
Supraspinatus inflammation leading to painful arc during shoulder abduction (40 to 120°).
Pain occurs in late-cocking and early acceleration phases of throwing; daily activities are usually pain-free.
Grade II:
Affects individuals aged 40-45.
Associated with supraspinatus tendinitis or subacromial bursitis.
Overuse or degeneration, with localized pain in the posterior shoulder in throwing phases.
Grade III:
Occurs in those over 45 years of age.
Linked with occupational overuse, falls, or sudden activity increases leading to degenerative changes in rotator cuff.
Basic Examination:
Palpation of the shoulder complex.
Observation of muscle symmetry between shoulders.
Gross strength testing of shoulder and rotator cuff muscles.
Assessment of joint accessory mobility across GH/ST/AC/SC joints.
Flexibility tests for shoulder, thoracic, and cervical spine.
Range of motion assessments for the GH/scapulothoracic joints and cervical/thoracic spine, noting dysfunction can affect shoulder performance.
Tenderness to palpation in the posterior shoulder/joint line.
Involved shoulder may show increased muscle bulk and altered position compared to the unaffected side.
Abnormal scapulothoracic rhythm and movements.
Weakness in rotator cuff, middle/lower traps, rhomboids, and serratus anterior muscles.
Decreased dorsal glenohumeral glide and tightness in posterior capsule.
Muscular imbalances in pectoralis muscles, latissimus dorsi, and upper trap.
Decreased internal rotation and increased external rotation at GH joint typically noted.
SICK Scapula: Scapular protraction, malposition, coracoid pain, and dyskinesis identified during examination.
Plain X-rays are less useful.
MRI and arthrography provide reliable diagnostic information.
Conservative treatment:
NSAIDs, hydrocortisone injections, subacromial steroid injections, active and passive exercises, temporary immobilization, and massage.
High recovery rate (90%) with conservative measures.
Strengthening:
Closed kinetic chain exercises for rotator cuff stabilization.
Addressing GIRD (glenohumeral internal rotation deficit) and posterior capsule strengthening.
Phase One Rehabilitation:
Soft tissue mobilization, scapula setting, joint mobilization, and improving shoulder ROM.
Neuromuscular re-education for recurrence prevention and restoring muscle balance.
Phase Two Rehabilitation:
Focus on dynamic stability and muscle balance restoration with complex exercises.
Start introducing eccentric and open kinetic chain exercises tailored to athletic movements.
Phase Three Rehabilitation:
Functional rehab plans for athletes focusing on returning to full activities, progressing plyometric exercises with controlled movements.
Recommend initial immobilization for three weeks.
Encourage active movements in elbow, hand, and wrist early on.
Begin strong isometric deltoid exercises after ten days, maintaining conservative post-operative measures.
Q1: What is Anterior Impingement Syndrome?A1: Anterior Impingement Syndrome occurs beneath the coraco-acromial arch, affecting vulnerable structures like the greater tuberosity, supraspinatus tendon, and long head of biceps, with compression most prevalent just anterior to the angle of the acromion.
Q2: What are the primary causes of Impingement Syndrome?A2: Causes include:
Complete or partial rupture of the rotator cuff
Supraspinatus tendinitis
Calcific deposits
Subacromial and subdeltoid bursitis
Periarthritis
Bicipital tenosynovitis
Fracture of the greater trochanter
Q3: What are the common clinical features or symptoms of this syndrome?A3: Common symptoms are pain, swelling, limited shoulder movements, muscle atrophy, and tenderness over the greater tuberosity. There are different types of shoulder pain:
Chronic: Diffuse posterior shoulder girdle pain, typical in throwing athletes with internal impingement, with pain increasing in the late-cocking phase of throwing.
Acute: Reported by non-throwing athletes post-injury.
Q4: How does Anterior Impingement Syndrome affect athletic performance?A4: Functional impairments include decreased throwing velocity, loss of control in overhead athletes, and a "dead arm" sensation post-throwing, with symptoms of weakness and perceived shoulder instability.
Q5: What is the pathophysiology behind Anterior Impingement Syndrome?A5: Key aspects include:
Muscular/Neuromuscular Imbalance: Commonly due to improper control in the shoulder complex.
Increased Laxity: Present as global or isolated anterior laxity.
Anterior Instability: Symptoms such as apprehension or feelings of subluxation in specific arm positions.
Rotator Cuff Pathology: Overlap of symptoms with other rotator cuff issues, especially in younger athletes.
Q6: How is Anterior Impingement Syndrome graded or staged?A6: The grading system includes:
Grade I: Affects young adults (18-30); caused by overstress, leading to supraspinatus inflammation and painful arc during abduction (40° to 120°).
Grade II: Affects those 40-45 years; associated with supraspinatus tendinitis or subacromial bursitis.
Grade III: Occurs in individuals over 45; linked to degenerative changes from overuse or falls.
No
Q7: What examination techniques are used to assess Anterior Impingement Syndrome?A7: Basic techniques involve palpation, muscle symmetry observation, strength testing, and range of motion assessments for relevant joints. Additional tests include flexibility tests and assessing joint accessory mobility.
Q8: What findings might be observed during the examination of a patient with this syndrome?A8: Possible findings include tenderness in the posterior shoulder, increased muscle bulk on one side, abnormal scapulothoracic movements, and weakness in specific muscle groups.
Q9: What additional considerations should be made during assessment?A9: Consider muscular imbalances in pectoralis muscles, latissimus dorsi, and upper trap, as well as altered internal and external rotation in the glenohumeral joint.
Q10: Which imaging investigations are useful for diagnosing Anterior Impingement Syndrome?A10: Plain X-rays are generally less useful, while MRI and arthrography provide reliable diagnostic information.
Q11: What are the management strategies for Anterior Impingement Syndrome?A11: Conservative treatment may include NSAIDs, steroid injections, exercises, and temporary immobilization. Recovery rates are high (90%) with these measures. Rehabilitation is phased:
Phase One: Soft tissue mobilization, scapula setting, joint mobilization.
Phase Two: Dynamic stability and muscle balance restoration exercises.
Phase Three: Functional rehabilitation focusing on returning athletes to full activities.
Q12: What post-surgery physiotherapy is recommended?A12: Initial immobilization for three weeks, early active movement in elbow, hand, and wrist, followed by strong isometric deltoid exercises after ten days, while maintaining conservative measures post-operatively.
Profile: 22-year-old male baseball pitcher.
Symptoms: Reports a painful arc during shoulder abduction (40° to 120°), primarily during practice. Experiences pain in the late-cocking phase of throwing.
Evaluation: Mild supraspinatus inflammation noted during examination, with tenderness around the greater tuberosity.
Management: Treated with NSAIDs, physiotherapy focused on soft tissue mobilization and scapula setting, with emphasis on shoulder ROM exercises.
Outcome: After 6 weeks of rehabilitation, the athlete returned to pitching with no pain.
Profile: 43-year-old female recreational tennis player.
Symptoms: Complains of localized pain in the posterior shoulder, especially while serving or performing overhead motions. Limited shoulder mobility observed.
Evaluation: Examination reveals tenderness over subacromial space and signs of subacromial bursitis.
Management: Injections of corticosteroids, in combination with a structured physiotherapy program focused on strengthening and improving shoulder stability.
Outcome: Improved shoulder function and reduced pain after 8 weeks; able to return to recreational play without significant discomfort.
Profile: 58-year-old male construction worker.
Symptoms: Experiences severe shoulder pain after a fall, with acute limitation in shoulder mobility. Reports difficulty lifting objects and persistent “dead arm” sensation.
Evaluation: Examination confirms significant weakness in rotator cuff muscles and radiologic imaging shows changes consistent with degenerative rotator cuff disease.
Management: Surgery indicated due to severe symptoms and structural changes. Post-operative management included a progressive rehabilitation program emphasizing strength restoration and functional activities.
Outcome: After 3 months of rehabilitation, the patient reports significant improvement and is able to perform work-related activities with minimal pain.
Assessment Diagnosis: Physical examination reveals mild supraspinatus inflammation, tenderness around the greater tuberosity, and a painful arc in shoulder abduction (40° to 120°).
Physiotherapy Management:
Focus on soft tissue mobilization and scapula setting.
Progress shoulder range of motion (ROM) exercises to alleviate symptoms.
Strengthening exercises once pain decreases, with adjustments based on tolerance.
Assessment Diagnosis: Examination shows tenderness over the subacromial space and limited shoulder mobility, indicating subacromial bursitis.
Physiotherapy Management:
Initiate with corticosteroid injections for inflammation control.
Structured physiotherapy program focusing on strengthening rotator cuff and scapular stabilizers.
Emphasis on functional movements related to overhead activities, restoring mobility and reducing pain.
Assessment Diagnosis: Significant weakness in rotator cuff muscles confirmed via examination; radiologic imaging displays degenerative changes linked to recurrent shoulder pain after a fall.
Physiotherapy Management:
Post-operative rehabilitation focusing on strength restoration and gradual return to functional activities.
Emphasize progressive exercises, including concentric and eccentric strengthening of rotator cuff, along with scapular stabilization.
Prioritize pain management and gradual reintroduction to work-related activities.