Chapter 12:
Intellectual Disability (intellectual development disorder)
Historical Labels vs Current Terminology
The 5th edition of DSM replaced mental retardation with intellectual disability
AAMR now AAIDD
IQ two standard deviations below the mean (70 or below)
Adaptive behavior deficits–two standard deviations below the mean
Before age 18–it must be present during the developmental period
Assessed by levels of needed supports
Must be deficits in intellectual and adaptive functioning (especially adaptive)
The context/culture is important
DSM Approach
Must occur during the intellectual period of childhood
Adaptive behavior deficits in atleast two areas
Onset before 18
Categorized in levels of mild, moderate, severe, profound
About 85% of all cases of mild form
Intelligence & adaptive behavior
Knowledge, ability to think or learn, capacity to adapt
The testing movement
Binet
Psychometric approach - g factor
Mental age (MA), chronological age (CA)
Intelligence complex, malleable, influenced by environment
Terman
Stanford-Binet
IQ ratio (quotient)
Eugenics: there is a huge bias and many tests aren’t catered toward many people of color
Stability and validity of IQ tests
Stability over time - test-retest
Scores fairly stable
Lower scores more stable
Some debate about credibility of IQ scores
Reasonably good predictors of school grades, but not everything
Concerns about cultural bias
Flynn effect: IQ scores systematically improve over time, bc the population scores are based on averages
Adaptive Functioning
What people do to take care of themselves
Areas often assessed:
Socialization skills
Practical skills
Community living/sociological context
Communication
Behavior can vary across cultures
Children with ID can experience varying levels of adaptive behavior
Is it situational?
Description
Variability even within classification groups
Increasing medical problems as level of ID increases
Lifespan has increased, but still below average
Specific deficits in working memory, attention, organization,
Co-occurring disorders:
ADHD
Self-injury
Aggression
autism
Variability in the severity of these disorders
Prevalence rates vary
Diagnostic overshadowing: failure to diagnose or recognize these disorders when compared to ID
Epidemiology
1-3% of the general pop.
More severe cases noticed earlier
Dramatic shift when children enter school
More prevalent in males (may be due to reporting bias, environment)
More prevalent in low SES group, especially mild cases
Developmental course
For mild ID, early intervention and training can result in a child no longer meeting the criteria for diagnosis
The rate of intellectual development can increase or regress
Intelligence tends to develop in a sequential way
Etiology:
Organic vs cultural-familial
Organic influences vs multigenic
Psychosocial influences
Multifactor causation
Genetic Syndromes
Behavioral types:
Down syndrome
Most common single disorder
Caused by trisomy 21: occurs randomly & not inherited
Decrease in brain size
Higher risk in maternal age
Alzheimer's
Moderate to severe ID
Delayed speech, verbal–short-term memory, and auditory processing deficits
Deficits in developmental skills during childhood
Fragile X
Most commonly inherited form fractured X chromosome
More common in boys– also more severe forms
Long faces, prominent jaws, large ears (males)
Visual-spatial, sequential processing, motor coordination & executive function deficits
Social impairments
Likely to have intellectual disabilities
Long-term memory is a strength
Williams Syndrome
Rare disorder, random
Deletions on chromosome 7
Cardiac and kidney problems, sound sensitivity, depth perception weaknesses
Mild to moderate ID
General knowledge & visual-spatial deficits
Relative strengths in language
Can be engaged with others but may have a hard time distinguishing good ppl (facial expressions)
“Elfinlike” appearance – may look a lot older than they are
Prader-Willi Syndrome
70% of cases result from paternal deletion of genes on chromosome 15
IQ ranges from borderline to moderate impairment
Hyperphagia and food hoarding
Other compulsions, such as skin-picking
Strengths and weaknesses may vary depending on the cause
Family
Adjustment to a lifelong process
Stressors
Diagnostic
Medical
Parental distress (stigma, guilt, blame)
Unique decisions
Today, families viewed in a more normative way
Factors affecting coping
Behavioral issues
Marital interaction
Parental IQ
Social class variables
Ethnic differences
Support–care and services
Siblings
Research findings mixed
Rewards and satisfactions
Many families do well
Balance of challenging
Assessment
Developmental tests
Bayley–most common
Both child and parent report
Catered to detect more severe presentations of ID
Intelligence tests
Stanford Binet
Weschler tests
WPPSI-IV
WISC-V
Kauffman battery
KABC-II
Adaptive behavior
Vineland adaptive behavior
Scales
Interviews and rating scales for parents and caregivers, as well as teachers
Communication
Daily living
Socialization
Motor skills
Maladaptive behavior
AAIDD adaptive behavioral scales
Guidelines for support
needs in 87 areas
Intervention for IDs:
Changing views
Normalization
Decreased institutionalization
Most live at home
Prevention:
Prenatal care and diet
Education on the impact of toxins
Early intervention programs
Educational services
IDEA:
Inclusion
Academic vs functional curriculum
Transition
Behavioral intervention
Discrete trial learning
Naturalistic or incidental training (non-formal)
Parent training
Enhancing adaptive behavior
Reducing challenging behavioral
Self-injurious behavior
Positive behavioral support
Relies on behavioral principles, functional assessment
Environment modification
Behavioral consequences
Functional analysis
Other interventions
Pharmacological
Medications should be used with caution
Antipsychotic meds
Employed when a child exhibits behavioral problems
Limited evidence for efficacy
Psychotherapy
Talk therapies not widely employed or researched
Chapter 3:
Autism Spectrum and Schizophrenia
History:
Bleuler – schizophrenia
Attempted to use the term schizophrenia as hearing voices that are not here
Childhood schizophrenia was considered any similar disorder
Kanner – autism
Dsm-5 recognizes: scizophrenia & autism spectrum disorders (ASD) as separate disorders
Autism spectrum disorders:
Characterized by:
Deficits in social communication and interaction
Restricted, repetitive, stereotyped behavior and interests
Hand flapping, ritualized behavior, fixation of things w/ abnormal focus
Symptoms must occur at an early age
Primary symptoms:
Symptoms vary from child to child, including the severity
3 categories ranging from low support to very high support needed
Social interaction
Deficits in joint attention behavior
Poor eye contact
Poor processing of social stimuli (lack of recognizing faces/facial expressions)
Difficulties with social subtleties (social cues)
Communication
Mutism or echolalia (repeating back a word) and pronoun reversals (referring to others as “I” or “me”)
Poor pragmatics
Deficits in verbal communication
Hyperlexia: being able to identify a word/passage yet not comprehend it
Restricted behavior and interests (not necessarily abnormal, but the frequency of when they do it)
Repetitive movements
Rocking, hand flapping, self-injurious behaviors
Persists through childhood
Insistence on sameness
Fixation on objects, tasks, hobbies, order of things
Is it getting in the way of school, ability to do other things, or impacting functioning?
Can serve as self-stimulation
Restricted, fixated interests
Associated Impairments
Sensory /perceptual
over/undersensitivity
Overselectivity: focuses on something random than what’s in front of them
Intellectual performance
Higher IQ associated with better prognosis
Performance scores better than verbal scores
Splinter skills–an ability that is much higher than expected
Savant skills–skills are strikingly better than expected
Adaptive behavior deficits:
Deficits in the function of daily life
Adaptive deficits increase with age
Underlying Cognitive Deficits
Theory of mind: the ability to understand the mental states of others and oneself and how it impacts how individuals act
Around 5 or 6
Sally-Anne task: (first-order task) where a child thinks what someone else is thinking
(second-order) when a child is able to guess what another person is thinking of based on another person
Kids with autism usually fail the first-order task
Faux pas test: saying something that would harm someone else; can they recognize that it was wrong to say that word or statement
Weak Central Coherence
The inability to get the gist of the conversation/information
Too focused on the details and not the big picture
Executive functions
Underlies many autism behaviors
Tend to perform poor in executive functioning, but varies
Also seen in ADHD
Must take a multidefict framework in order to represent the variability in these symptoms
Physical & other features
Higher than normal physical abnormalities
Some graceful, but others exhibit poor balance, uncoordinated gait, impaired gross motor skills
Co-occurrence
Determining comorbidity is challenging because of language and cognitive problems of ASD hinder communication
Simple phobias, social phobias, generalized anxiety
Effects of ID are often not separated from those of ASD
Epidemiology
Prevalence rate has been increasing
May be due to better identification
Earlier diagnosis
Broadened criteria
More prevalent in males
Girls are more diagnosed later in life and may display similar symptoms as boys yet not diagnosed
Unrelated to social class; biases in ethnicity
Developmental course
3 patterns of onset
1st–occurs in most; abnormalities become apparent by age 1 or soon after
2nd–involves regression (of ASD children)
3rd–mild delays until about age 2 and then developmental arrest and plateau
6 trajectories
Better prognosis with higher-functioning youths
Etiology:
Environment & social interaction
3-component function
No demonstrated vaccine link–ASD was once associated with vaccines
Govt. & professional groups in the US haven't found support for the theory that MMR vaccine causes the disorder
Assessment for ASD:
Requires a broad-based assessment
Median diagnoses is 4-5 years
Interviews
Observations
Standardized checklists for autistic behaviors
M-CHAT
CARS 2
ADI-R (actual autism diagnoses)
Can be used on anyone 2 years and older
ADOS
Assesses children using direct observation
Can be used 12 months—into adulthood
Intellectual assessment–can be helpful for co-occurring ID, but not necessary
Prevention of ASD:
Prenatal care
Improvement in environmental qualities
Early intervention
Behavioral approaches
Social skills like joint attention, imitation, language skills, behavioral regulation, & cognitive development
Not all children benefit from these interventions
Pharmacological treatment
Atypical antipsychotics used to improve behavior
Risperidone: doesn't target all
Stimulant meds, but some appear to have negative side effects–may exacerbate other symptoms
Behavioral interventions (more empirical support)
Young autism project:
Pivotal response training:
TEACCH:
Home adjustment
Community
Falls into 2 approaches: Targeted vs Comprehensive
Programs should:
Be intensive, start early, be carefully controlled/systematic, promote generalization, involve parents, be flexible
Chapter 11:
Communication and learning disorders
History:
Curiosity about discrepant, or unexpected, lack of abilities within individuals can be traced to work in Europe in the 1800s
Created a more universal definition, operationalized
Definitional concerns
Learning disabilities
Rely on Individuals with Disabilities Education Act for definition
Much debate
Psychological processes not defined
No criteria for identifying disabilities
Definition
IQ-achievement discrepancy
Look for discrepancy between IQ and achievement (typically 2 standard deviations)
Has been critisized, no longer a requirement of diagnosing learning disabilities anymore
A flaw is that intelligence may be an underestimation of correctly diagnosing a child, other important things to consider
The IQ range low/high may indicate different things
Can fail to consider many years of issues with one subject
Doesn't consider environmental factors
Below-average achievement – grade or age
Child performs below grade level in at least one academic area
Compare performance to same-age peers on standardized tests–child scores 2 SDs below mean
The degree to which an individual is compared to their peers
Response to Intervention (RTI):
Exposes children to a valid intervention before diagnosing
Moves children through phases, levels depending on how the child is reacting to the intervention
Unresolved issues about the approach
Basic Components of Language:
Phonology
Morphology
Syntax
Semantics
Pragmatics
Receptive language
Expressive language
After age 5, it gets harder to obtain a language and learn it
As children pass 2 years language increases dramatically
By age 7 we have many of the language skills that we need in adulthood
DSM Classification & Diagnosis:
Speech-Sound Disorder
Fails to display developmentally appropriate speech sounds
Language Disorder
Deficits in vocabulary, grammar, comprehension, other aspect of language output
Deficits in both expression & reception language
Children would speak in a very short manner, with unusual word order, low vocab
Epidemiology & course:
Rates of 3–4%
More prevalent in boys
More prevalent in low SES (may be skewed due to dialectal differences)
Phonological problems tend to remit by age 6
Children w/ receptive language deficits at a higher risk for learning disabilities
Cognitive Deficits, theories:
Limited information-processing capacity
A delayed reaction time to information
Deficits in auditory processing
Deficits in verbal short-term and working memory
Specific learning disorders
A specific neurological disorder in:
Reading
Written expression
Mathematics
IQ-achievement discrepancy
Significant disturbance in achievement and daily living
Not due to sensory deficit
Have persisted 6 months +
Must begin during school-age years
SLD with impairment in reading
Word-level reading problems (dyslexia):
Involve problems in accurate or fluent word recognition, poor spelling abilities
Text-level reading problems (problems of comprehension)
Can decode words but can’t understand what they read
Prevalence–5 to 15 % of US school-age children
Impairments in reading is the most common type
May be more prevalent in boys (may be referral bias)
Disorder tends to persist into adolescence, adulthood
Mathew effect: abilities can worsen over time
SLD with impairment in written expression:
Errors in writing–quality
Hard to decipher
Disorganized content
Transcription–putting ideas into written form
Poor punctuation, capitalization, word placement–handwriting & spelling central
Text generation (composition)
Creation of meaning in written form: poor word memory, sentence structure, topic expansion
Epidemiology:
6–14% of school-age children
Usually apparent by 2nd grade, referrals spike in 4th grade
Prevalent in kids with ADHD
SLD with Impairment in Mathematics
Learn counting procedures and strategies more slowly, struggle to apply them
Struggle to learn math terms (multiplication tables), spatial organization
Approx. 7% of school-age children show some form of impairment
Figures likely lower when cases w/ occurring reading disorders excluded
No gender differences found
Chapter 10:
Attention-Deficit/Hyperactivity Disorder
History:
Early account–George Still, English physician, described a group of boys with a “defect in moral control”
Encephalitis epidemic
Terms hyperkinessis, hyperkinetic syndrome, and hyperactive child syndrome used
Three diagnostic presentations
Predominately inattentive (ADHD-PI)
Predominately Hyperactive/Impulsive (ADHD-PHI)
Combine type (ADHD-C)
Must have some symptoms before age 12
Display symptoms for at least 6 months
Impaired social, academic, occupational functioning
Seen in at least 2 settings (at home/school)
Symptoms can’t be explained by another disorder
Inattention:
Cannot focus consistently (sustained attention)
Child may play video games for hours
Difficult tasks increase problems
Usually daydream
Can be hyperfocused on certain tasks and can't focus at all on some tasks (boring/difficult)
Has trouble managing simultaneous stimuli or switching from one task to another
May focus on unimportant information
Easily distracted
The central problem is control and self-regulation rather than simply inattention
Hyperactivity & impulsivity
Always on the run
Restless fidgety
Can't sit still
Accident prone
Disorganized
More apparent in structured situations (context matters)
Impulsivity:
Uninhibited
Acts without thinking
Interrupts others
Cuts in line
Engages in dangerous behaviors
May be described as
Careless, immature, rude
Secondary Features
Motor skills
Clumsiness, delays in milestones, poor performance in sports
Complex movements and sequencing most affected
Intelligence and academic achievement
May perform somewhat lower on intelligence tests, but a range is possible
Academic failure is common
56% need tutoring
30% may repeat a grade
30-40% may receive special education placement
10-35% may fail to graduate high school
Executive functions
Difficulties with the processes needed for goal-directed behavior, planning, organizing, and self-regulating
Working memory
Verbal self-regulation
Self-monitoring
Inhibition of behavior
Emotional regulation
Motor control
Adaptive behavior
Immature
Deficits in self-care, independence, and the ability to perform everyday skills
Social behavior and relationships
Social problems are very common due to loud, disruptive, aggressive, and impulsive behaviors
Aggressive, negative style of interaction
Physical and verbal aggression toward others
ADHD-C may have stable, positive self-bias and be unaware of impact on others
Peer and teacher relationships
ADHD children are frequently disliked, rejected by peers
Teachers are directive and controlling toward other children with ADHD
Family relations
Parents less rewarding, more negative and directive
Certain parent and family characteristics can make the situation worse
Mother-child interaction may be less adept than father-child relationships
Parental reactions interact with the child’s behavior and vice versa
Health, sleep, accidents
Sleep problems common
Trouble falling asleep, night awakening, involuntary movement
May be related to co-occurring symptoms–anxiety/depression
May be related to medications
Higher risks for accidents
Bone fractures
Accidental poisonings
Car accidents/speeding tickets
Subtypes
Most research is on ADHD-C (combined) type
Research on ADHD-PHI (hyperactive-impulsive) type is scant
ADHD-PI (inattentive) type may be missed
Sometimes categorized as “ sluggish cognitive tempo”
Lethargic
Prone to daydreams
Confused
Socially withdrawn
Usually 6 symptoms for each subtype
Debates about subtypes
Co-occurring Disorders
ADHD-C often occurs with other disorders
Learning disabilities 15-40%
May be a result of cognitive deficits associated with ADHD or behavioral difficulties
Epidemiology
5 to 9% of school age children
Statistics may be higher due to resources, parental/teacher report
Gender imbalance
Boys consistently display higher rates of the disorder (3:1 ratio to girls)
Girls possibly underdiagnosed
A bias towards reporting boys, expressed behaviors
Social class, race/ethnicity, and culture
ADHD occurs in all social classes, but may be more prevalent in lower SES
Low but increasing among Hispanic youths vs non-Hispanic youths
Some studies find higher rates in AA vs white children, but others show reverse
Developmental course
Individuals do not outgrow ADHD
Infancy and preschool
Difficult temperament
High activity level
Less cooperative/manageable
Poor emotional regulation
Symptom presentation can change throughout the years & varies from child to child
Childhood
Most ADHD cases are diagnosed during elementary school
Self-regulation and organizational deficits
Peer rejection, poor academic achievement
ODD
Adolescence and adulthood
Symptoms may diminish/change
Longer-term problems may be associated with co-occurring diagnoses
Etiology:
Neuropsychological theories of ADHD
Executive functions, inhibition
Barkley’s Model
Behavioral inhibition
Inhibit prepotent responses: responses that are likely to be reinforced
Interrupt prepotent responses
Protect form interference
4 executive functions:
Nonverbal working memory
Internalization of speech (verbal working memory)
Self-regulation
reconstitution
Sensitivity to reward
High preference for immediate reward
Perform poorly in situations with low incentive
Temporal processing, aversing
Temporal processing
Delay aversion: children tend to dislike/avoid the delay of time rather than the reward itself later
Multiple-pathway models
Dual pathways model
Executive function deficits
Delay aversion
Temporal processing a third pathway
Multiple deficit models
Neurobiological abnormalities
Small brain volume associated with and severity of ADHD symptoms
Prefrontal lobe and striatal area associated with the core symptoms of ADHD
Inhibition, working memory, and other executive functions
Decreased blood flow and glucose utilization, slow brain waves
Abnormalities in frontal, striatal, and cerebellar structures and their networks play an important role
Underarousal of brain
Dopamine and norepinephrine deficiency
Other brain regions are also implicated
A heterogeneous disorder
Etiology:
Genetic influences:
Runs in families
Twin studies average heritability rate of .80, .90
DRD4, DAT1 genes
Gene-environment interactions
Prenatal influences & birth complications
Some indications that maternal smoking and alcohol use linked to ADHD
Birth comp linked to higher rates
Diet and lead
Diet doesn't play a role in ADHD
Exposure to lead associated to ADHD
Psychosocial influences
Maternal depression
Paternal childhood ADHD
Paternal warmth & sensitivity can mediate risk
Parent behavior, child’s temperament can interact and set the stage for more negative outcomes
Classroom environment, teacher management of behavior can affect a child’s presentation of ADHD
Assessment
Interview
Strengths and weaknesses–medical history, academic history
Parent-child interactions
Child
Teacher interview
School experiences
Should be developmentally accurate (age-based)
Rating scales
–Can allow for the comparison to normative scores
Conners
Parent
Child
Teacher
Direct observation
–natural-based settings
Home and school
Not always practical
Others
Conners Continuous Performance Test II
Medical
Intelligence
Achievement
Interventions:
Pharmacological:
Stimulant medication
Affect dopamine and norepineprine (taken once a day)
Helps in 75% of cases
Can reduce co-occurring behaviors, either directly or indirectly
May affect academic achievement
Concerns:
Stimulants don’t work for 10-20% of children
Side effects:
Sleep problems
Reduced appetite
Headaches, stomach aches
Irritability
Jitteriness
Growth suppression in height & weight
Vocal motor tics
Risk for later substance abuse
Too easily prescribed & an increase in medicated children in the past decades
Can be seen as a “quick fix” and used w/o additional symptomology of ADHD
Behavioral interventions:
Parent training
Classroom management
Contracting
Daily report card
Educational methods
ADHD-I (inattentive) may need help with a slow work style
Organizational skills
On task time
Transitions
Reduce peer reinforcement of inappropriate behavior
Teachers may not be able to spend their time focused on one child, helpful to support the teacher
Multi-modal study — MTA study
Examined effects of medication, behavioral, combined, and community care treatment
Follow-ups
Children who received medication and combined treatment still showed improvement with ADHD
Although children with co-occurring ADHD and anxiety benefited from just behavioral treatment alone
In order for it to be beneficial intervention must be throughout life over an extended period of time
Prevention
Better prenatal care, healthy environment, optimal family life
Parent training
Social interactions training
Chapter 9: Conduct Problems
Externalizing: problems with others
Aggression
Oppositional behaviors
Disruptive/antisocial behaviors
Delinquency: associated with the legal system
Separate from other disorders
Children will usually be bullies, disruptive, well beyond the normal degree
Intermittent Explosive Disorder:
Not intended for children under the age of 6
Outbursts are generally impulsive and are not premeditated or committed to achieve a tangible objective
Antisocial Personality Disorder (APD):
Diagnosed after age 18
Characterized by aggressive antisocial behaviors that begin by age 15 and continue into adulthood
Pattern of traits: lack of empathy, deceitful, arrogant, manipulative interpersonal style, impulsive and irresponsible behavioral style
Oppositional Defiant Disorder:
Pattern of negativistic, hostile, defiant behavior lasting over six months (at least 4 symptoms per week):
Loses temper
Argues with adults (authority figures)
Touchy or easily annoyed by others
Vindictiveness
Actively defies or refuses to comply with adults' requests or rules
Deliberately annoys people
Blames others for his/her mistakes
Mild presentation: displayed frequently in one setting
Moderate: 2 settings
Severe: displayed in 3 or more settings
Conduct Disorder
Persistent pattern of behavior that violates the rights of others and age-appropriate social norms
3 or more behaviors for at least 12 months
Behavior causes impairment
Childhood-onset (before age 10) or adolescent onset (over the age of 10)
Childhood onset more likely to persist in the future
Aggression: bullying, cruel to animals, sexual activity
Decietfulness (theft): lying, breaking into homes/cars, stealing
Serious violation of rules (in an adolescent context): staying out past curfew, running away from home,
Can be mild, moderate, or severe based on the number of symptoms
Presentation of the disorder can exhibit a lack of empathy, callous-unemotional traits
Aggressive vs rule-breaking syndromes
Gender differences:
Boys exhibit significantly higher levels of physical aggression than girls do
Girls more likely to engage in relation aggression
Manipulative, coercive actions
Other issues:
Fire setting:
Factors:
aggressive youngsters
Youngsters without supervision
High-conflict families
Higher rate of paternal drinking & abuse
Higher rates of maternal depression
Delinquency:
Legal term: juvenile who has committed an index crime or status offense
Bullying
Deliberate act of hurting someone else
Can begin in preschool
Victims can experience internalizing behaviors
Aggressive to peers and adults
Positive attitude toward violence
Typical victim:
Anxious
Insecure
Nonaggressive
Low self-esteem
Having a close friend can ward off bullying or the effects
Bullying tied to risks of long-term problems
Epidemiology:
Co occurrence:
ODD and CD
DSM indicates that if criteria for both are met, only CD is given
Most kids with CD also meet ODD criteria
ODD/CD and ADHD
35-70% of ADHD children also develop ODD
ADHD tends to precede the development of others
Parent-child relationship has a huge effect
Course:
Stability of problems over time for at least some individuals
Age of onset developmental pattern
Adolescent onset– fewer childhood problems
More common
Less likely to persist
Childhood onset– related to more serious, persistent problems later
Developmental path:
Loeber’s three pathway model:
Overt
covert
Authority conflict
Etiology:
Socioeconomics
Aggression as a learned behavior
Family influences
Parent-child interactions\
Oregon model
Patterson’s coercion theory
Negative reinforcement
Reinforcement trap: a child throws a fit for candy in a candy store and the mother gives it–the short-term gain leads to long-term negatives
Parental discipline, monitoring
Parents are more likely to be harsh and reprimand semi-normal behaviors
Parental psychopathology (parental influence)
Environment/ context matters if you want to change an aggressive child's behavior
The role of media isn’t clear
Family influences, cont.
Extrafamilial influences and parenta;
Psychopathology
Handing-down of poor parenting practices
Antisocial families
Social disadvantage
Parental substances use
Marital discord
Complex relationship
Maltreatment
Peer relations
Cognitive-emotional influences
Hostile attribution bias
Attributing aggression to other individuals
Reactive vs proactive aggression
Reactive: angry response to something
Proactive: Not associated with anger–starting a fight, bullying, teasing
Parental supervision is a mediator
And higher in boys
Biology
Genetics: twin studies evidence for moderate genetic influence, but environmental influences important
Social conditions
Family variables
Certain social learning experiences
Neurophysiological
Behavioral inhibition system (BIS) underactive
behavioral activation system (BAS) overactive
Or both systems underactive as child tries to seek sensation
Reduced threshold for fight or flight in individuals with conduct problems
Neuropsychological
Frontal lobes
Problems with verbal and executive functions
Substance use
Very common among adolescents
Marijuana is the most common illicit drug
Vs licit drugs: over-the-counter
DSM-5 eliminates distinction between abuse and dependence
Focus is on substance abuse disorders, impairment, and distress–problems with using that disorders
Substance-induced disorders: intoxication, withdrawal–caused by substance use, in response to the drug
Tolerance: need more
Withdrawal:
Epidemiology:
Dramatic increase in vaping
Alcohol most widely used substance
Rates of drug use rise and fall, often with rumors of benefits
SES and ethnic differences–studies inconsistent
CD and ODD highly correlated with use
Higher use in males
Use affected by:
Temperament
Self-control
Problem-solving
Emotional regulation
Attitudes, expectations, intentions, beliefs about control
Exposure to trauma, stress
Modeling
Family, peers
Poor academic performance and low involvement in school activities
Availability of substances
Other models of substance use:
Rite of passage
Stress and coping
Assessment
Interviews
Child
Family
School
Behavioral rating scales
Achenbach
BASC
Conners, Eyberg, Sutter-Eyberg, Self-report measures
Observations
Parent-child observations
Home and school settings
Intervention
Parent training
How to give commands
Parenting skills
How to reinforce behaviors
How to discipline
Cognitive problem-solving skills training (PSST)
Community-based
Teaching family model (for more severe presentation)
Delinquets placed in home with trained foster parents
Transfer back home
Relapse common
Multidimensional treatment foster care
Only one or more youth in the foster home
Outside support from a larger program system
Intervention in multiple domains, therapy, etc.
Multisystemic therapy
Involves family, peers, school, neighborhood, and community
More family therapy
Effective in treating antisocial behavior
Prevention
Early intervention with families of oppositional defiant children