Behavioral Genetics & Development – Lecture 2 Vocabulary
Lecture Scope & Recap
- Builds directly on Lecture 2 (Tinbergen’s Four Questions, proximate vs. ultimate explanations, hypothesis construction & testing)
- Today’s focus: developmental and genetic bases of behavior; explicit goal to relate proximate questions to ultimate ones in every example
Learning Objectives (Stated)
- Identify & define key genetic/developmental terms that influence behavior
- Contrast and evaluate alternative hypotheses for any behavioral phenomenon
- Understand methodological pipelines: observation → hypothesis → prediction → experimental/quantitative test → inference
- Prepare for Canvas quiz covering this material (date: July 31, 2025)
Core Terminology & Concepts
- Genotype = complete DNA sequence inherited from parents (fixed across lifespan)
- Environment = all external (abiotic & biotic) and internal (social, hormonal) conditions influencing gene expression
- G \times E interaction = phenotype results from non-additive interplay of genotype and environment
- Ontogeny = organism’s developmental trajectory from early stage onward
- Phenotypic plasticity = capacity of one genotype to express >1 phenotype depending on cues
- Polyphenism = discrete, discontinuous plasticity (e.g., queen vs. worker)
- Maternal effect = offspring phenotype change caused by mother’s phenotype/physiology independent of alleles inherited
- Epigenetics = heritable changes in gene activity without DNA-sequence alteration (DNA methylation, histone modification, non-coding RNA)
- Supergene = linked block of genes inherited as one unit due to suppressed recombination; often generated by chromosomal inversions and underlies fixed polymorphisms
Molecular Foundation – Central Dogma
- DNA regions (genes) are transcribed → mRNA (nucleus) → translated by ribosomes → protein
- Standard schematic: \text{DNA} \xrightarrow{\text{transcription}} \text{mRNA} \xrightarrow{\text{translation}} \text{Protein}
- Poll check-in: students click the cellular location where translation occurs (ribosome in cytoplasm/RER)
Hypothesis Logic in Animal Behavior
- Hypotheses must be mutually exclusive; poll used to test student recognition of this principle
- Development of alternative hypotheses and clear predictions critical before data collection
- Ways we test hypotheses:
- Controlled lab manipulations (e.g., RNAi knockdown in bees)
- Field manipulations (e.g., hormone implants in squirrels)
- Natural experiments/comparative studies (e.g., burrowing in Peromyscus spp.)
Genotype vs. Environment Thought Exercise
- Slide prompt “Genotype OR Environment – Behavior 1/2” used to emphasize false dichotomy; modern view: behaviors arise through G \times E synergism
Case Study 1 – Honey Bee Worker Ontogeny
- Eusocial caste system: workers (sterile females), queens (fertile females), drones (haploid males)
- Age-polyethism in workers (cleaning → nursing → food packing → foraging)
- Probability curve of task performance vs. age provided (Fig 3.1)
- Gene-expression profiling (Fig 3.2):
- Young (Y) vs. Old (O); Nurses (N) vs. Foragers (F)
- Differentially expressed genes correlate with behavioral state more than chronological age ⇒ behavior-linked transcriptional program
Royal Jelly & Caste Determination
- All larvae receive some royal jelly; future queens receive it throughout larval life, workers switched to honey/pollen
- Proteins in royal jelly:
- Silence Dnmt3 (DNA methyltransferase) → global hypomethylation
- Initiate epigenetic cascade: altered DNA accessibility, histone tail modifications, small ncRNA action
- RNAi Experiment (Fig 3.4):
- Silenced Dnmt3 in larvae + normal worker diet
- Outcome: queen phenotype frequency ≈ larvae fed full royal jelly ⇒ supports hypothesis that DNA methylation state, not nutrient calories, directs caste
- Additional hypothesis: diet-specific phytochemicals in honey/pollen may actively suppress queen development; suggests dual regulatory model (promotion + suppression)
Case Study 2 – Genetically Based Burrowing & Migration
- Peromyscus mice (Fig 3.6):
- Oldfield mice build long, complex burrows early (precocious)
- Deer mice build short/simple burrows later (delayed)
- Trait persists in lab ⇒ strong genetic component
- Black vs. common redstart:
- Species-specific night “migratory restlessness” timing (days 0–225 post-hatch chart) indicating innate migratory program
Evo-Devo Approaches
- Forward genetics (phenotype → gene): e.g., search for loci controlling mouse burrow length
- Reverse genetics (gene → phenotype): e.g., CRISPR/RNAi knockdown of Dnmt3 or vasopressin receptors in voles
- Vole example: prairie vole (monogamous) vs. montane vole (polygynous); differing vasopressin receptor distribution identified via reverse genetics
Early-Life Developmental Hypotheses
- Developmental Constraint: poor early environment → lifelong fitness cost (no predictive match)
- Predictive Adaptive Response (PAR): early cues tune phenotype to expected adult environment
Empirical Tests
- Amboseli baboons:
- Females born during drought show reduced fertility if adult drought recurs ⇒ supports constraint, not PAR
- Discussion prompt: what result would support PAR? (Higher fertility in drought for drought-born)
- North-American red squirrels (Dantzer 2013):
- High cone years → population density ↑ → maternal cortisol ↑
- Elevated maternal cortisol experimentally increases offspring growth rate
- Outcomes: faster growth ⇒ higher first-winter survival, but reduced adult lifespan ⇒ adaptive trade-off; qualifies as maternal effect possibly favored in fluctuating density environments
Phenotypic Plasticity & Polyphenism
- Three distribution patterns (Fig 3.28):
- High variance continuous trait (plastic or genetic)
- Low variance continuous trait (canalized)
- Discontinuous multi-modal trait (polyphenism)
- Types of polyphenism:
- Density-dependent (locust solitary ↔ gregarious)
- Switch initiated by social tactile cues; maintained via epigenetic + ongoing social input (Ernst 2015)
- Predator-induced, food-induced, socially-induced in fishes & amphibians
- Social-status plasticity in cichlid Astatotilapia burtoni:
- Removal of dominant male → subordinate male escalates aggression within minutes
- Surge in egr\text{-}1 gene activity in preoptic brain region; returns to baseline after dominance established (Fig 3.15)
Moving Beyond Plasticity – Behavioral Polymorphism
- Behavioral polymorphism = fixed alternative strategies; plasticity lost
- Often underpinned by supergenes (chromosomal inversions blocking recombination)
Ruff (Philomachus pugnax) Example (Fig 3.17)
- Three male morphs: Independent (territorial, dark plumage), Satellite (non-territorial, light), Faeder (female-mimic)
- Each satellite & faeder morph controlled by distinct supergenes (~100 linked genes) influencing plumage + reproductive physiology
White-Throated Sparrow Supergene
- Tan-striped (TS) vs. White-striped (WS) morphs; obligate disassortative mating (TS×WS only)
- WS possess rearranged chromosome 2 (ZAL2m) locked with ancestral ZAL2 → supergene
- Key genes within inversion:
- ESR1 (estrogen receptor-α) — modulates aggression/parental care
- VIP (vasoactive intestinal peptide) — affects prolactin & social behaviors
- WS show higher ESR1 & VIP expression in aggression centers → behavioral dimorphism maintained genetically
Genetic Basis of Diet Choice – Garter Snakes
- Coastal California neonates prefer banana slugs, inland neonates avoid
- Slug-cube assay on lab-born offspring:
- Coastal genetic line → tongue-flick response high
- Inland line → low response
- Supports heritable chemosensory preference; discussion on allelic variation vs. gene-by-experience effects
- “Define gene.” → consensus: DNA segment coding for protein/RNA
- True/False: “Organisms are set and cannot change phenotype throughout life.” → class overwhelmingly answered False (reinforces plasticity concept)
- Snowball Q-A exercise: students anonymously contribute one question + one biology fact; promotes engagement & identifies misconceptions
Ethical, Philosophical & Practical Considerations
- Epigenetic inheritance blurs line between genetic determinism and environmental influence; societal debates on trans-generational effects (nutrition, toxins)
- Understanding maternal effects crucial for wildlife management under climate change (e.g., squirrel density fluctuations)
- Supergene research informs conservation genetics because suppressed recombination may limit adaptive potential
- Bee caste manipulation via diet/epigenetics underpins apicultural practices and raises questions on pesticide impact on developmental pathways