MSU PSY 209 Final Exam
Psy 209 final review session
Divisions of the nervous system
CNS
Forebrain
Telencephalon
Cortex
Basal ganglia
Limbic system
Diencephalon
Thalamus
hypothalamus
Mesencephalon (Midbrain)
Hindbrain (brainstem)
Metencephalon
Cerebellum
Pons
Myelencephalon (medulla)
Resting potential
Reflects balance between diffusion and electrostatic pressure
Diffusion = diffusion of ions through ion channel
Na+
Sodium
K+
Potassium
How do the forces drive the resting potential
Diffusion
The idea that if the situation allows it, ions will tend to go from high concentration to low
Only sodium can diffuse during resting state
During resting potential, certain ions pass more freely than others
Membrane permeability
Membrane of a cell separates the interior from the exterior and can be selectively permeable (ex via ion channels) to chemicals allowing movement of substances in and out
Channels only opened if cell is being stimulated
Leak potassium channels are open, though
Electrostatic pressure = distribution of electrical charges
Repells if they are like, but polarities are opposite then they attract
Mediates resting potential
The machinery that maintains the resting potential is the sodium-pottasium pump = mechanism that pushes sodium out of the cell and potassium into the cell
Potassium can move, sodium cant
Neuron at rest is in negatively charged state
-60 mV relative to the outside of the cell
Extracellular fluid
Feeding and satiety
VMH
Ventromedial hypothalamus
LH
Lateral hypothalamus
If you get rid of this, you get rid of the feeding response
Necessary for feeding
Homeostasis
Need to determine our energy state, respond to that state
Plasticity and learning
Following LTP (long term potentiation) increase in AMPA receptors increases the synaptic sensitivity to glutamate
The greater the number, the more excitation the neuron is feeling (of LTP number)
Can increase as the two neurons communicate, and stays increased so that when the neuron/s fire onto the neuron, the same electrical charge is expected to be present
Before
What is a primary neurotransmitter responsible for in long term potentiaiton
Glutamate is abundant in all of our brains (90% have some sort of glutamatergic signal)
Acts as a stimulatory, excitatory signal (first role)
Acts predominantly as a stimulant, or driving excitatory factor
Gaba is the opposite
Acts as inhibition of brain activity
NMDA receptor
Glutamate can’t do anything to it; magnesium is blocking it
In the before stage and post synaptic neuron, not enough charge for the magnesium to stop blocking it
During
Calcium enters the cell
Activates something called campK2
Tells cell to do stuff; alters chemistry of the cell so that it is more able to fire more strongly in the future
What does it do to AMPA receptors
Calcium told campK2 that they want to push AMPA receptors up to the surface of the postsynaptic membrane
AMPA receptors get pushed back; go from after to more like before (AMPA goes back into the neuron, and doesn’t have same opportunity to show EPSPs), which results in us forgetting information
Before vs after figures differences
Extra AMPA receptor
Allows the next time the presynaptic cell releases glutamate, more excitatory post synaptic potential
Happens with lots of receptors and lots of neurons
Psy 209
Specific receptor that drive hallucinations
Taste systems, one was salt. What are the channels that allow you to detect salty foods
Anatomical terms
Horizontal planes, if you were to cut through the brain in a certain way, what fields would be shown
Inhibitory post synaptic potential
Hyper polarization, neurons are likely going to fire less if they are in a state where they are bombardier with IPSPs bc they’re more negative, how do we get them (bc we get the buildup of negative ions into the cell)
Drugs
Can kill, emphasize from toxicity dose and lethal dose
Lethal dose 50 - telling us basically at this dose 50% of the population will die if they take the drug at this dose
Who coined the term cells that fire together wire together
Colonergic system
Two types of receptors
Muscerinic
Nicotonic
What does the cerebral spinal fluid do
Labeling of the brain
Use of identify in those brain cells the dna sequence of what the cell expressed
If its serotonin expressing, it’ll have gene code for serotonin, use a technique of ISH to look for just that potential genetic signature, and will label the cell and tell us everything we need to know (ex)
ISH uses dna of cell to label cell
Cells aren’t just genetically able to produce it, a serotonin cell has serotonin dna, but a serotonin cell produces serotonin the protien; other approach - doesn’t look for dna itself but the protien
Amino cyto chemistry approach?
Optogenetics
How do you stimulate cells w it
Glutamatergic pyramidal cells
Pyramidal cells in cortex
Phineas gage
Estimated number of synapses in the brain
Tracing techniques
Anterograde tracer
Move foward
Place in amygdala
Retrograde tracer
Moved backwards
Place in pfc
Describing the neuron
Axonal terminal: what does it do
Basal ganglia: what are the areas of the brain that contain it
Major inhibitory neurotransmitter
GABA; allows negatively charged chloride into the cell
Development
How does the process of cell differentiation work
How does a cell become a dopamine cell, ex
Which neurotransmitter system is associated with Parkinson’s
How many chromosomes do humans possess? 23 pairs
The signals in the gut that make us hungry: ghrelin
Leptin makes us feel full/stops us from eating
Epigenetics
DNA methylation and just one modification
Cell death - apoptosis
What limits apoptosis, why do certain cells come to it and others don’t
Related to nutrients the cells receive: neurotropic factors are how they receive
Saltatory conduction
Which drug blocks the transporters for cathacholamines
Dopamine transporter
Illicit stimulant
Fear learning
Model in the lab
Idea of conditioning
What are the brain mechanisms controlling it
Not just amygdala, but two subdivisions
Central nucleus (encoding) and basal lateral (allowing expression)
Cranial nerves that send taste into
7, 9, 10?
What drug did they apply in chanters cognitive attribution model
Epinephrine
What would happen if you disrupt activity of the t1r2 receptor
Can’t taste sweet
Positive symptoms of schizophrenia
Delusions, hallucinations