## Summary of Key Biological Concepts
Here's a structured summary of the biological concepts you provided, organized for clarity and emphasis on key points:
**1. Thyroid Gland & Metabolism:**
* **Hormones:** Produces T4 (thyroxine) and T3 (triiodothyronine) to regulate metabolism.
* **Cellular Impact:** Thyroid hormones stimulate cellular activity, affecting protein synthesis and oxygen use.
* **Homeostasis:** Maintains balanced bodily functions like heart rate, temperature, and energy expenditure.
* **Disorders:**
* Hypothyroidism (underactive): Slows metabolism, leading to fatigue, weight gain, and cold intolerance.
* Hyperthyroidism (overactive): Speeds metabolism, causing weight loss, increased heart rate, and heat intolerance.
**2. Adrenal Glands & Stress Response:**
* **Hormones:** Secretes adrenaline (epinephrine), noradrenaline (norepinephrine), and cortisol.
* **Stress Response:** Prepares the body for "fight or flight" by increasing heart rate, blood pressure, and energy availability.
* **HPA Axis:** Part of the hypothalamic-pituitary-adrenal axis, involved in cortisol production.
* **Chronic Stress:** Prolonged stress leads to elevated cortisol, negatively impacting the immune system, metabolism, and mental health.
**3. Female Reproductive System:**
* **Ovaries:** Produce eggs (ova) and hormones (estrogen, progesterone).
* **Fallopian Tubes:** Transport eggs to the uterus; site of fertilization.
* **Uterus:** Where a fertilized egg implants and develops during pregnancy.
* **Key Processes:**
* Ovulation: Egg release from ovary.
* Endometrium: Uterine lining that thickens for implantation.
* Cervix: Uterine opening connecting to the vagina.
* **Fertilization:** Typically occurs in the fallopian tube. Cilia help move the egg.
**4. Menstrual Cycle:**
* **Phases:**
* Menstrual: Bleeding (shedding of uterine lining).
* Follicular: Egg maturation. FSH (follicle-stimulating hormone) is key.
* Ovulation: Egg release (triggered by LH - luteinizing hormone).
* Luteal: Uterine lining thickening (progesterone from corpus luteum).
* **Hormones:** FSH, LH, estrogen, and progesterone play crucial roles.
**5. Nervous System & Homeostasis:**
* **Sensory Input:** Detects changes via receptors.
* **Signal Transmission:** Electrical impulses travel along neurons to the CNS (brain and spinal cord).
* **Integration & Response:** Brain processes input and signals effectors (muscles/glands).
* **Negative Feedback:** Counteracts changes to maintain stability (set point).
* **Examples:** Temperature regulation, blood pressure control, blood glucose control.
* **Key Terms:** Homeostasis, feedback mechanism, receptor, effector.
**6. DNA Replication:**
* **Key Enzymes:**
* DNA Helicase: Unwinds DNA.
* DNA Primase: Synthesizes RNA primers.
* DNA Polymerase: Builds new DNA strand.
* DNA Ligase: Joins DNA fragments.
* Single-Stranded Binding Proteins: Stabilize single DNA strands.
**7. RNA Types:**
* **mRNA (messenger RNA):** Carries protein coding sequences from DNA to ribosomes. Contains codons.
* **tRNA (transfer RNA):** Carries amino acids to ribosomes during protein synthesis.
* **rRNA (ribosomal RNA):** Forms the core of ribosomes and catalyzes peptide bond formation.
**8. DNA Replication Errors (Mutations):**
* **Types:** Insertions, deletions, point mutations, double-strand breaks, repeat expansion/contraction.
* **Causes:** Deoxyribonucleotides, free radicals, purines, oxygen.
* **Repair Mechanisms:** Proofreading, base excision repair (BER), homologous/non-homologous end-joining repair.
**9. Central Dogma of Molecular Biology:**
* **Flow of Information:** DNA → RNA → Protein.
* **Processes:** Replication, transcription, translation.
**10. Types of Mutations:**
* **Point Mutations:** Missense (amino acid change), silent (no change), nonsense (stop codon).
* **Frameshift Mutations:** Insertion/deletion altering the reading frame.
* **Chromosomal Alterations:** Translocations, duplications, deletions.
**11. Effects of Mutations:**
* **Range:** No effect (silent) to significant changes in protein function (disease, lethality).
* **Influencing Factors:** Gene function, mutation location, environment.
* **Examples:** Sickle cell anemia, cystic fibrosis, antibiotic resistance.
**12. UV Radiation & Mutations:**
* **Mechanism:** Damages DNA by forming pyrimidine dimers.
* **Lesions:** Cyclobutane pyrimidine dimers (CPDs), 6-4 photoproducts (6-4PPs).
* **Consequences:** Replication errors, mutations, potentially cancer.
**13. Antibiotic Resistance:**
* **Causes:** Mutations in target genes, silent mutations, loss-of-function mutations in regulators.
* **Measurement:** Mutation rate (frequency of mutants).
* **Influencing Factors:** Antibiotic treatment, mutator strains.
**14. Fossils & Evolution:**
* **Evidence:** Show how life has changed, relationships between organisms, simple to complex life progression.
* **Usage:** Radiometric dating, categorization, comparison.
**15. Homologous vs. Analogous Structures:**
* **Homologous:** Similar form and genetics due to common ancestry (divergent evolution). Example: Bird and bat wings (bones).
* **Analogous:** Similar function, different structure, independent evolution (convergent evolution). Example: Bird and bat wings (overall structure for flight).
**16. DNA & Amino Acid Sequences:**
* **Codons:** Three nucleotides coding for one amino acid.
* **Processes:** Transcription (DNA → mRNA), translation (mRNA → protein).
**17. Embryological Development:**
* **Stages:** Blastocyst, embryo, fetus.
* **Organ System Development:** Mesoderm, ectoderm, endoderm.
* **Hormones:** hCG.
**18. Puberty Hormones (Males):**
* **Testosterone:** Primary male sex hormone, secondary sex characteristics.
* **FSH:** Stimulates spermatogenesis.
* **LH:** Stimulates testosterone production.
* **DHT:** Initiates puberty.
* **Estrogen:** Affects reproductive health.
**19. Pregnancy Hormones:**
* **hCG:** Thickens uterine lining.
* **Estrogen:** Fetal growth, breast preparation.
* **Progesterone:** Uterine lining support, labor preparation.
* **Relaxin:** Loosens ligaments.
* **Oxytocin:** Uterine contractions, bonding.
* **Prolactin:** Breast milk production.
* **Adrenaline/Noradrenaline:** Stress response.
* **Beta-endorphins:** Pain relief.
**20. Pancreas & Diabetes:**
* **Hormones:** Insulin (lowers blood sugar), glucagon (raises blood sugar).
* **Diabetes Types:**
* Type 1: Pancreas doesn't produce enough insulin.
* Type 2: Insulin resistance.
* **Other Factors:** Pancreatitis, obesity, pituitary tumors, hyperaldosteronism.
**21. Menstrual Cycle Phases (Detailed):**
* **Menstruation:** Shedding of uterine lining.
* **Follicular:** Egg maturation.
* **Ovulation:** Egg release.
* **Luteal:** Corpus luteum produces progesterone.
**22. LH & Ovulation:**
* **Role:** Triggers ovulation, supports pregnancy.
* **Other Effects:** Puberty, testosterone production (males), menstrual cycle control (females).
**23. Progesterone's Function:**
* **Primary Role:** Prepares uterine lining for implantation.
* **Luteal Phase:** Levels rise after ovulation.
* **Pregnancy:** Supports early pregnancy.
* **Menstruation:** Drop in levels triggers shedding.
**24. Irregular Menstrual Periods:**
* **Causes:** Hormonal changes (puberty, menopause, thyroid disorders, PCOS), pregnancy, stress, lifestyle factors, infections, uterine fibroids/polyps, endometriosis, medications.
**25. Complementary Base Pairing:**
* **DNA:** A pairs with T, G pairs with C.
* **RNA:** A pairs with U, G pairs with C.
* **Importance:** Accurate replication, transcription, translation, DNA structure.
**26. Mutations & Base Pairing:**
* **Mutations:** Changes in DNA sequence (substitution, insertion, deletion).
* **Base Pairing's Role:** Ensures accurate replication and minimizes mutations.
**27. DNA as Evidence for Evolution:**
* **Shared Genetic Code:** Universal code suggests common origin.
* **Homologous Genes:** Similar genes in different species indicate shared ancestry.
* **DNA Comparisons:** Used to determine evolutionary relationships.