Gas Transport and Respiratory Control - Lecture 19
Oxygen Transport
- Oxygen is carried in two forms: dissolved $O_2$ and bound to haemoglobin (Hb) in red blood cells (RBCs).
- Dissolved $O2$ is limited: about 3\ \mathrm{mL\ O2\ per\ L\ blood}; total content is roughly 15\ \mathrm{mL\ O2\ per\ L}. The body needs ~250\ \mathrm{mL\ O2\ min^{-1}}.
- Hb binds up to four oxygen molecules per Hb molecule (four heme units).
- Most oxygen is transported bound to Hb; only a small fraction is dissolved in plasma.
Hemoglobin Binding Curve
- The Hb-O2 saturation curve is sigmoidal (not linear) due to cooperative binding of O2 between heme sites.
- The curve shows how many Hb sites are occupied as alveolar PO2 increases.
- The shape enables efficient loading in the lungs and unloading in tissues that need O2.
- The curve shifts to the right (lower affinity, easier O2 release) when conditions favor delivery to tissues:
- Lower pH (acidic)\n - Higher temperature\n - Higher CO2 (PCO2)\n- Exercise causes these conditions in active tissues, promoting O2 unloading to muscles.
Oxygen Delivery and Tissue Uptake
- The sigmoidal shape and its shifts help deliver O2 preferentially to tissues with high demand (lower local PO2).
Carbon Dioxide Transport
- CO2 is transported in three forms:
- Dissolved in plasma
- Bound to Hb
- Converted to bicarbonate (HCO3-)
- Majority is transported as bicarbonate; a portion is bound to Hb or dissolved.
- CO2 + H2O ⇌ H2CO3 ⇌ HCO3^- + H^+
- This reaction lowers blood pH when CO2 rises (more acidic blood).
Control of Breathing
- Purpose: keep arterial O2 and CO2 within narrow limits for metabolic stability.
- Ventilation is tightly regulated despite variable O2 use and CO2 production.
Central Controller, Sensors, and Effectors
- Sensors: chemoreceptors, baroreceptors, lung stretch receptors, protective reflex receptors.
- Controller: brain regions (pons, medulla, others).
- Effectors: respiratory muscles (drive ventilation via afferent/efferent signals).
Chemoreceptors
- Monitor O2 and CO2 (and pH) levels in blood.
- If O2 drops or CO2 rises, they increase breathing frequency and tidal volume.
- They are the primary regulator of ventilation and can override voluntary control.
- CO2 receptors play the primary role; O2 receptors are secondary.
- Locations: carotid bodies/arteries and the medulla oblongata.
Baroreceptors
- BP sensors located in carotid sinus and aortic arch.
- Provide feedback to respiratory control via afferent signaling (via vagus/X nerve).
- If arterial BP falls, respiratory minute volume tends to rise; if BP rises, ventilation tends to fall.
Inflation/Deflation and Protective Reflexes
- Lung stretch receptors provide input to prevent over-inflation/over-deflation (Hering-Breuer reflex).
- Irritation triggers protective reflexes (sneeze, cough).
Summary (Essentials)
- O2 transport: mainly Hb-bound; a small portion dissolved in plasma.
- Hb binding curve: sigmoidal, enabling efficient O2 delivery; shifts with pH, temperature, CO2 to aid delivery where needed.
- CO2 transport: mainly as bicarbonate; some bound to Hb or dissolved.
- Respiratory control: chemoreceptors, baroreceptors, stretch receptors, and protective reflexes provide afferent input to brain, which regulates ventilation via efferent output.
Quick Exam Focus
- Pulmonary fibrosis (thickened blood–air barrier) tends to reduce arterial O2 due to slower diffusion.