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resistance training

Resistance Training

Principles of Training

  • Overload

    • Training effect occurs when a physiological system is exercised at a level beyond which it is normally accustomed.
  • Specificity

    • Training effect is specific to:
    • Muscle fibers recruited during exercise
    • Energy system involved (aerobic vs. anaerobic)
    • Velocity of contraction
    • Type of contraction (eccentric, concentric, isometric)
  • Reversibility

    • Gains are lost when overload is removed.

Physiological Effects of Strength Training

  • Muscular strength

    • Definition: The maximal force a muscle or muscle group can generate.
    • Measured as 1 repetition maximum (1-RM).
  • Muscular endurance

    • Definition: The ability to make repeated contractions against a submaximal load.
  • Strength training types:

    • High-resistance training (2–10 RM):
    • Gains in strength.
    • Low-resistance training (20+ RM):
    • Gains in endurance.

Muscle Adaptations to Anaerobic Exercise Training

  • Anaerobic exercise

    • Definition: Short-duration (i.e., 10-30 seconds) all-out effort.
    • Muscle fibers involved: Both type I and II muscle fibers.
    • Energy supply sources:
    • Exercise lasting 10 seconds or less mainly supplied by the ATP-PC system.
    • Exercise lasting 20-30 seconds: 80% of energy provided anaerobically and 20% aerobically.
  • Performance increases

    • 4-10 weeks of anaerobic training can increase peak anaerobic power by 3-28% across individuals.
  • Muscle buffering capacity improvements

    • Achieved by increasing both intracellular buffers and hydrogen ion transporters.
  • Hypertrophy of type II muscle fibers

    • Increases in enzymes involved in both the ATP-PC system and glycolysis.
    • High-intensity interval training (>30 seconds) promotes mitochondrial biogenesis at or above VO2 max.

Resistance Training

  • Neural adaptations responsible for early gains in strength occur within the first 8–20 weeks and include:

    • Increased ability to recruit motor units.
    • Altered motor neuron firing rates.
    • Enhanced motor unit synchronization.
    • Removal of neural inhibition.
  • Hyperplasia

    • Definition: Increase in muscle fiber number.
    • Limited evidence in humans, with most studies indicating that 90–95% of muscle enlargement is due to hypertrophy.
  • Hypertrophy specifics:

    • Both type I and II fibers experience hypertrophy, with a greater degree in type II fibers.
    • Increases in myofibrillar proteins lead to:
    • Increased number of cross-bridges.
    • Increased ability to generate force.
  • mTOR (Mechanistic target of rapamycin):

    • A protein kinase that increases ribosome production and accelerates protein synthesis.
    • Leucine and BCAA supplementation provide a small increase in mTOR activation but are ineffective in untrained individuals.

Muscle Fiber Changes

  • Fiber type shifts from type IIx to IIa:
    • Change of 5-11% following 20 weeks of resistance training.
    • Small increases in type I fibers occur; however, resistance training-induced fast-to-slow shifts in fiber type are less prominent compared to endurance training.

Concurrent Strength and Endurance Training

  • Interference potential:

    • Strength training increases muscle fiber size, while endurance training does not.
    • Degree of interference depends on the intensity, volume, and frequency of the training.
  • Impairment of strength gains through concurrent training:

    • Combining strength and endurance training can impair strength gains compared to strength training alone, with the extent of interference depending on workout specifics (intensity, volume, frequency).
  • Neural factors affecting strength development:

    • Impaired motor unit recruitment.
    • Limited evidence supports this concept; low muscle glycogen content due to successive bouts of endurance exercise may result in impaired ability to perform subsequent resistance training bouts.
  • Overtraining:

    • No direct evidence to prove that overtraining causes impairments in strength gains during concurrent training.
    • Depressed protein synthesis may occur due to endurance training adaptations that interfere (↓mTOR).

Detraining Effects

  • Loss of muscle strength and fiber size:

    • A slow decrease in strength of 31% following 30 weeks of detraining, associated with small changes in fiber size:
    • Type I fiber size: -2%
    • Type IIa fiber size: -10%
    • Type IIx fiber size: -14%
    • Changes attributed primarily to nervous system changes.
  • Retraining:

    • Rapid regain of strength and muscle size can occur within 6 weeks after resuming training.
    • Strength maintenance is possible with reduced training up to 12 weeks.

Aging, Strength, and Training

  • Decline in strength after age 50 due to:

    • Loss of muscle mass (sarcopenia).
    • Loss of both type I and II fibers, including atrophy of type II fibers.
    • Loss of intramuscular fat and connective tissue.
    • Loss of motor units and reorganization of motor units.
  • Progressive resistance training:

    • Important for causing muscle hypertrophy and strength gains, which are vital for activities of daily living, maintaining balance, and reducing the risk of falls.

Study Guide Questions

  • How does concurrent training impact gains in strength and endurance?
  • What is mTOR and why is it crucial in resistance training?
  • How do muscle fibers adapt following resistance training?
  • What is the difference between hypertrophy and hyperplasia, and which is more relied upon during resistance training?
  • What physiological changes occur during the first 8 weeks of resistance training?
  • How do strength and muscle size change after periods of detraining, and how do they respond to retraining?
  • How is strength affected by aging?