COMPLICATIONS OF PERINATAL PERIOD
multiple gestation
post date
atypical presentation
dystocia
augmentation
alterations in FHR
shoulder dystocia
cord prolapse
c section
hemmorage post partum
disseminated intravascular coagulopathy
amniotic fluid embolism
perinatal loss
MULTIPLE GESTATION RISK FACTORS
hyperemesis
anemia
preterm labour
gestational hy[ertension
placental abruption
poly/oligohydramnious
prolapsed cord
uterine dysfunction
cardiovascular complications
hemmorage post partum
FETAL RISKS MULTIPLE GESTATION
stillbirth
preterm
low birth weight
intrauterine growth restrictin
congenital abnormalities
cord accident
abnormal placentas
POST TERM PREGNANCY
more common in primagravidas
fetal mortality rate doubles at 42 weeks
placental function deterioration
oligohydramnious, poor fetal nutrition, oxygenation
RISK OF POST TERM
meconium aspiration
induction of labour
large for gestational age
tissue trauma
c section
NURSING ASSESMENTS FOR POST TERM
antepartum
fetal surveillance
amniotic fluid volume
intrapartum
induction
pain management
ATYPICAL PRESENTATION
3 main presentation
cephalic (head first)
breech presentation (buttocks, feet, or both feet)
shoulder presentation
internal or external version can be performed
nurses role is to monitor fetus and support mom
obtain maternal vital signs and assess comfort
UTERINE DYSTOCIA
defined as abnormally slow progress in labour
caused by various conditions related to 5 ps
hypotonic, uncoordinated infrequent uterine contractions or ineffective bearing down efforts (power is most common cause)
alterations of pelvic structure (passageway)
fetal cause, abnormal presentation of position, anomalies, excessive size and number os fetuses (passenger)
maternal position during labour
psychological response of mother is related to experiences, and support system
OTHER FACTORS INCREASE RISK FOR UTERINE DYSTOCIA
over weight, short stature, advanced maternal age, infertility difficulties, prior cephalic external version, uterine abnormalities (congenital malformations, over distension with polyhydramnious)
malpresentation and position of fetus
cephalopelvic disproportion (CPD)
uterine overstimulation with oxytocin
maternal fatigue, dehydration and electrolyte imbalance
LABOUR DYSTOCIA
inadequate progression of labour
physician dx, CPD, medications, and nursing actions required
DYSTOCIA PREVENTION
prenatal education
management of labour
ambulation
continuous emotional support
amniotomy
augmentation with oxytocin
AUGMENTATION
stimulation of uterine contractions after labour has stopped
implemented of hypotonic uterine dysfunction resulting in slowing of labour
augmentation methods are oxytocin infusion and amniotomy
non invasive methods
bladder emptying, ambulation, position changes, relaxation measures, hydration
freidman’s curve: graph that documents hours in labour are plotted against cervical dilation. measured in cm, increasing dilation indicated progress of fetus as it moves in birth canal. the curve is S shaped
OXYTOCIN INDICATIONS FOR INDUCTION OR AUGMENTATION OF LABOUR
suspected fetal distress (IUGR)
inadequate uterine contractions (dystocia)
pre labour rupture of membranes
post term pregnancy
chorioamniomitis
renal disease, diabetes, severe Rh isoimmunization
preeclampsia, eclampsia
fetal death
CONTRAINDICATIONS TO OXYTOCIN STIMULATION IN LABOUR
prolapsed cord, cephalic disproportion
abrominal fetal heart rate
placenta previa/vasa previa
prior classic uterine incision or uterine surgery
genital infection
cancer of cervix
previous uterine rupture
NURSING CONSIDERATIONS FOR CONTRAINDICATIONS FOR OXYTOCIN
oxytocin will be decreased or not given if tachystole resulting in abnormal fetal heart rate or if a pattern occurs
the Friedman graph again is used to determine normal length and pace of labour. If cervix does not dilate accordingly, will be sent in for c section
INDUCTION METHODS
membrane sweeping
medications
cervidil
prostaglandin gel
misoprostol
oxytocin
ballon catheters
INDICATIONS FOR INDUCTION
high priority
preeclampsia >37wks
maternal disease not responding to treatment
significant but stable antepartum hemmorage
chorioamnionitis
fetal compromise
pre labour rupture of membranes with GBS colonization
other indications
postdates or post term pregnancy
uncomplicated twin pregnancy
glucose control may dictate urgency
autoimmune disease at or near term
intrauterine growth restriction
oligohydramnious
gestational hypertension >38wks
intrauterine fetal death
PROM near or at term (GBS) negative
intrauterine device prior to pregnancy
ALTERATIONS IN FHR
5 components
baseline
baseline variability
accelerations
decelerations
trends over time
normal: are the characteristics within normal parameters
atypical: further vigilant assessment is required
abnormal: action is required
atypical: involves correction of reversible cause for compromise, intrauterine resuscitation and scalp stimulation or blood sampling
abnormal: review of overall clinical situation, intrauterine resuscitation and operative delivery (vaginal or section) indicated, or if there is evidence of normal oxygenation by scalp PH assessment
MANAGEMENT OF ATYPICAL OR ABNORMAL FETAL HEART RATE PATTERN
intrauterine resuscitation
stop or decrease oxytocin, and change maternal position
improve maternal hydration with IV fluid bolus
perform vaginal examination to assess progress in labour and relieve pressure off cord
can condor using oxygen 0-10L but evidence shows that there is effectiveness on fetal compromise
prolonged maternal oxygen have negative implications of fetal cord blood gases at birth and should only be used in caution as part of intrauterine resusitation
consider amnioinfusion if variable decelerations present
SHOULDER DYSTOCIA
condition where head is born but anterior shoulder cannot pass under pubic arch
turtle sign: involves appearance and retraction of baby’s head, occurs when baby’s shoulder is obstructed by maternal pelvis
mcroberts manœuvre: used to assist in child birth, employed in cases of shoulder dystocia during birth and involved hyper flexing the legs tightly to abdomen
suprapubic pressure: attempt to manually dislodge anterior shoulder from behind symphysis pubic during shoulder dystocia. Performed by making a fist, placing it above maternal pubic bone and pushing fetal shoulder in one direction or another
hands and knees: wood screw maneuver: this maneuver is used in shoulder dystocia, anterior shoulder is pushed towards baby’s chest, and posterior shoulder is pushed towards baby’s back, making the baby’s head face the mothers rectum
FACTORS THAT CAUSE SHOULDER DYSTOCIA
fwtopelvic disproportion caused by excessive fetal size
maternal pelvic abnormalities can be cause
maternal diabetes (macrosomia)
history of shoulder dystocia with pervious birth
FETAL INJURIES SHOULDER DYSTOCIA
asphyxia
trauma to brachial plexus
phrenic nerve injuries
fracture of humerus or clavicle
brachial plexus injury (Erb Palsy)
NURSING ACTIONS FOR SHOULDER DYSTOCIA
mcroberts manœuvre: romans legs are flexed to abdomen, causes sacrum to straighten, and symphysis pulls rotates towards mothers head: angle of pelvic inclination is decreased freeing the shoulder
suprapubic pressure can be applied
mcroberts is preferred methods when mother is receiving epidural anesthesia
positions that can resolve shoulder dystocia
gaskin manouversL hands and knees position
squatting or lateral recumbent position
fundal pressure can be used as a method of relieving shoulder dystocia is avoided, it is associated with neurological complications
when shoulder dystocia is diagnosed, nurse helps patient assume position that facilitate birth of shoulders, and monitor fetal response. Nurse also provides encouragement and support to reduce anxiety and fear
newborn assessment should indicate examination for fracture of clavicle or humorous, brachial plexus injuries and asphyxia
maternal assessmentt should focus on detection of hemmorage and trauma to soft tissue of birth canal