SlideDeck_Chapter 4
Page 1: Introduction
Title: CORLSECECHDSYCS Chapter PowerPoint
Source: OpenStax College Psychology
Page 2: What is Consciousness?
Consciousness: Awareness of internal and external stimuli (e.g., feelings of hunger, pain, light).
Sleep: Described as a quiet and mysterious pause in daily life.
Wakefulness: High levels of sensory awareness, thought, and behavior.
Illustrated with an art piece titled "Two Sleeping Girls on the Stove" by Albert Anker.
Page 3: Biological Rhythms
Biological Rhythm: Internal cycle of biological activity.
Includes: Fluctuation of body temperature, menstrual cycles, alertness levels.
Circadian Rhythm: Biological rhythm occurring over approximately 24 hours.
Generated by the Suprachiasmatic Nucleus (SCN).
Sleep-wake cycle linked to natural light-dark cycles.
Hypothalamus: Maintains homeostasis in biological systems.
Page 4: Circadian Rhythms
Illustration showing circadian changes in body temperature over 28 hours.
Body temperature rises during waking hours, peaks in the afternoon, and falls during sleep.
Page 5: Suprachiasmatic Nucleus (SCN)
Location: In the hypothalamus, acting as the brain's clock.
Function: Synchronizes with light information from the retina to align with the external environment.
Page 6: Melatonin and Sleep Regulation
Melatonin: Hormone regulating the sleep-wake cycle; secreted by the pineal gland.
Release stimulated by darkness, inhibited by daylight.
Chronotype: Individual's unique circadian activity pattern.
Page 7: Disruptions of Normal Sleep
Jet Lag: Symptoms due to mismatches in internal circadian cycles and the external environment (fatigue, irritability).
Rotating Shift Work: Changing work hours disrupt normal circadian rhythms, leading to exhaustion and agitation.
Bright light therapy can help align biological clocks.
Page 8: Sleep Deprivation
Negative consequences of persistent sleep deprivation illustrated.
Sleep Debt: Chronic insufficiency in sleep.
Sleep Rebound: Shorter sleep onset after deprivation.
Page 9: What is Sleep?
Sleep Definition: Low physical activity and reduced awareness state.
Controlled by brain areas: thalamus and hypothalamus, pons for REM sleep.
Hormones regulated during sleep: Melatonin, Follicle Stimulating Hormone, Luteinizing Hormone, Growth Hormone.
Page 10: Brain Areas Involved in Sleep
Key Structures: Hypothalamus, Thalamus, Pineal Gland, Suprachiasmatic Nucleus (SCN), Pituitary Gland, Pons.
Pineal and Pituitary glands secrete multiple hormones during sleep.
Page 11: Why Do We Sleep?
Adaptive Function: Restores resources exhausted during the day, responds to darkness-related predation risks.
Cognitive Function: Crucial for cognitive processes and memory formation; deprivation leads to cognitive deficits.
Benefits: Vital for maintaining weight, lowering stress, improving mood, and enhancing cognition and memory.
Page 12: Stages of Sleep
Describes various stages of sleep: Stage 1, Stage 2, Stage 3, Stage 4, Stage 5.
Page 13: Brainwaves During Sleep
Brainwave activity alters significantly across sleep stages; observed via EEG.
Types: Alpha (low, high amplitude), Theta (low frequency, low amplitude), Delta (low frequency, high amplitude).
Page 14: Stages 1 and 2
Stage 1: Transitional phase; respiration and heartbeat slow, muscle tension decreases.
Stage 2: Deep relaxation; characterized by Theta waves, sleep spindles, K-complexes.
Page 15: Stages 3 and 4
Stages 3 and 4: Known as slow-wave sleep; characterized by Delta waves,
Further decrease in respiration and heart rate.
Page 16: Rapid Eye Movement (REM)
Characterized by rapid eye movements, paralysis of voluntary muscles, frequent dreaming.
Brainwave activity resembles that of wakefulness.
Page 17: Hypnogram of Sleep Stages
Hypnogram: Visual representation of different stages of sleep throughout a sleep cycle.
Page 18: Dreams
Varied theories regarding dream meanings, including:
Freud: Dreams access the unconscious (manifest and latent content).
Carl Jung: Dreams tap into the collective unconscious; symbols reflect universal archetypes.
Research suggests dreams reflect life events or provide a mental prototype.
Lucid Dreams: Awareness during dreaming.
Page 19: Insomnia
Defined by difficulty in falling/staying asleep at least 3 times weekly for a month.
Common sleep disorder; may be linked with depression.
Contributing Factors: Age, drug use, exercise, mental status, bedtime routines.
Treatment: Stress management techniques, behavioral changes, cognitive-behavioral therapy.
Page 20: Parasomnia
Parasomnias: Unwanted motor behaviors or experiences during sleep.
Includes sleepwalking, REM sleep behavior disorder, restless leg syndrome, night terrors.
Page 21: Sleep Apnea
Sleep Apnea: Breathing stop during sleep for 10-20 seconds.
Leads to fatigue; more common in overweight individuals.
Types: Obstructive and Central sleep apnea; treated with CPAP.
Page 22: Sudden Infant Death Syndrome (SIDS)
SIDS: Sudden cessation of breathing in infants; highest risk below 12 months.
Contributing Factors: Prematurity, household smoking, hyperthermia.
Safe to Sleep campaign aims to reduce risk factors.
Page 23: Narcolepsy
Narcolepsy: Irresistible sleep urge during wakefulness, often triggered by stress or arousal.
Shares features with REM sleep: cataplexy, hypnagogic hallucinations.
Treatment: Psychomotor stimulant drugs.
Page 24: Substance Use Disorders
Substance Use Disorder: Compulsive drug use despite negative consequences.
Involves physical and psychological dependence:
Physiological Dependence: Normal bodily function changes, withdrawal upon cessation.
Psychological Dependence: Emotional need for drug.
Tolerance: Increased quantity needed for effects; linked with physiological dependence.
Withdrawal: Negative effects from discontinued use.
Page 25: Drug Categories
Overview of various classes of drugs including:
Antipsychotics: Haldol, Risperdal, Seroquel.
Stimulants: Cocaine, Amphetamines.
Depressants: Alcohol, Barbiturates.
Narcotic Analgesics: Morphine, Heroin.
Hallucinogens: LSD, Mescaline.
Page 26: Depressants
Depressants: Drugs that suppress CNS activity.
Commonly GABA agonists with a quieting effect.
Examples: Alcohol (impacts reaction time and alertness), Barbiturates (anticonvulsants).
Can lead to loss of consciousness.
Page 27: GABA
GABA-gated chloride channel's role in neuron activity; binding of depressants reduces neuronal firing through chloride entry.
Page 28: Stimulants
Stimulants: Increase levels of neural activity; typically dopamine agonists.
Highly addictive due to effects on dopamine reuptake.
Examples: Cocaine, Amphetamines.
Page 29: Dopamine Agonists
Mechanism: Block dopamine reuptake, increasing synaptic levels of dopamine.
Page 30: Nicotine and Caffeine
Caffeine: Increases alertness by antagonizing adenosine; associated with arousal.
Nicotine: Highly addictive; interacts with acetylcholine for arousal and reward.
Page 31: Opioids
Opioids: Decrease pain through effects on the opioid neurotransmitter system; highly addictive.
Includes: Heroin, Morphine, Methadone, Codeine.
Page 32: Hallucinogens
Hallucinogens: Cause changes in perception and sensory experiences; often result in vivid hallucinations.
Mechanisms variable, affecting different neurotransmitter systems (e.g., Serotonin, NMDA receptors).
Page 33: Other States of Consciousness: Hypnosis
Hypnosis: Extreme self-focus with suggested behavior changes; differs from media portrayals.
Can aid in memory recall, pain management, depression treatment, smoking cessation, weight loss.
Page 34: Other States of Consciousness: Meditation
Meditation: Focus on a single target (e.g., breath) to achieve a modified state of consciousness.
Benefits include stress management, improved sleep, pain management, and mood enhancement.