Ch 65 SCI
Chapter Overview
Focus on Peripheral Nerve and Spinal Cord issues
Detailed exploration of Spinal Cord Injury (SCI)
Spinal Cord Problems
Definition: Trauma or damage to the spinal cord that results in dysfunction.
Statistics:
17,000 new SCIs annually in the United States
282,000 people currently living with an SCI
Increased mortality and decreased life expectancy
30% rehospitalization rate
Types of Injury
Primary Injury
Caused by direct trauma (blunt/penetrating).
Examples include spinal cord compression from:
Bone displacement
Blood supply interruption
Traction on the cord
Can also be due to penetrating wounds resulting in tearing or transection of the cord.
Secondary Injury
Ongoing damage that follows primary injury, leading to permanent damage.
Pathophysiology:
Begins minutes after injury and continues for months.
Edema occurs, leading to compression and ischemia.
Permanent damage can occur within 24 hours of edema development.
Classification of SCI
Mechanisms of Injury:
Flexion
Flexion-rotation (most unstable)
Hyperextension
Vertical compression
Extension-rotation
Lateral flexion
Degree of Injury:
Complete: Total loss of sensory and motor functions
Incomplete: Mixed loss with some functional preservation
Incomplete Injuries
Cauda Equina Syndrome:
Results from damage to lumbar and sacral nerve roots, causing symptoms like:
Asymmetrical weakness
Flaccid paralysis
Loss of sensation
Areflexic bladder and bowel
Clinical Manifestations
Respiratory System
Complications based on injury level:
Injury above C3: Total loss of respiratory function.
C3-C5: Respiratory insufficiency; requires intubation.
Cervical/thoracic injuries can cause ineffective cough leading to:
Aspiration
Atelectasis
Pneumonia
Cardiovascular System
Injury above T6 can lead to:
Neurogenic shock (bradycardia, hypotension).
Hemorrhagic shock may exacerbate symptoms.
Urinary System
Neurogenic Bladder: Abnormal bladder function leading to:
Overactive detrusor or sphincter muscles
Urinary retention/incontinence.
Gastrointestinal System
Neurogenic Bowel: Loss of voluntary control leading to:
Constipation
Incontinence
Risk of ileus or megacolon.
Integumentary System
Risk of skin breakdown due to:
Decreased sensation
Pressure injuries leading to infection.
Diagnostic Studies
Imaging:
CT scans preferred for injury location.
MRI for soft tissue and neurological conditions.
Comprehensive neurologic assessment essential.
Interprofessional Care
Prehospital Care
Main focus on airway, breathing, circulation (ABCs).
Immediate Goals:
Ensure airway integrity
Circulatory volume maintenance
Acute Care
Emergency management including monitoring vital signs, supporting CB, and assessing injury extent.
Interventions for hemodynamic stability noted for initial care and stabilization.
Rehabilitation
Addressing complications like respiratory dysfunction and cardiovascular instability.
Maximizing nutritional management is crucial for recovery.
Pain Management
Nociceptive Pain
Management includes anti-inflammatory drugs and opioids.
Neuropathic Pain
Occurs post-SCI; management includes:
Antiseizure medications
Relaxation therapy hinges on education about pain triggers.
Skin Care
Risk of pressure injuries necessitates:
Routine skin assessments
Repositioning every 2 hours.
Neurologic Recovery
Reflex returns may complicate rehabilitation, leading to issues like autonomic dysreflexia if spinal shock resolves.
Autonomic Dysreflexia (AD)
A life-threatening condition characterized by:
Severe hypertension
Bradycardia
Triggered primarily by bladder distention or rectal impaction, requiring immediate intervention.
Sexuality and Relationships
Addressing sexual health and function is essential for psychosocial support post-SCI.