Labor Complications
Risk factors
biophysical
may affect development/functioning
originates with pregnant women/fetus
psychosocial
maternal behavior/adverse lifestyle have negative effect on health
risks include emotional distress, hx depression, intimate partner violence, substance use
sociodemographic
lack of prenatal care, low income, single marital status, ethnicity
environmental
hazards in workplace/general environment
include environmental chemicals, anesthetic gases, radiation
Antepartum assessment
ultrasonography during pregnancy
first trimester
confirm pregnancy and viability
determine gestational age
rule out ectopic pregnancy
detect vaginal bleeding, maternal abnormalities
determine multiple gestation
second trimester
establish/confirm dates
confirm viability
detect poly/oligohydramnios
detect congenital anomalies/IUGR
detect placental location
evaluate for preterm labor
third trimester
confirm gestational age/viability
detect macrosomia, IUGR, congenital anomalies, placental abruption
determine fetal position
AFI assessment
detect placental maturity/preterm birth
BPP
at least one episode of fetal breathing movement of at least 30 sec in a 30 min observation
at least 3 trunk/limb movements in 30 min
at least one episode of active extension and flexion/opening and closing of hand
deepest vertical pocket >2 cm
reactive NST
BPP Score | Interpretation | Management |
10 | normal, low risk for chronic asphyxia | repeat testing at weekly to twice weekly intervals |
8 | normal, low risk for chronic asphyxia | repeat testing at weekly to twice weekly intervals |
6 | suspect chronic asphyxia | consider birth if ≥36-37 with positive testing for fetal pulmonary maturity repeat BPP in 4-6 hrs if negative testing deliver if oligohydramnios |
4 | suspect chronic asphyxia | proceed to birth if ≥36 weeks repeat score of <32 weeks |
0-2 | strongly suspect chronic asphyxia | extending testing time to 120 min proceed to birth if score is ≤4 |
Fetal Positioning
lie
the position of the long axis of the fetus
described as longitudinal, transverse or oblique
presentation
part of fetus that lies closest to/has entered the pelvis
described as cephalic, breech, shoulder
breech can cause lack of engagement of the cervix, umbilical cord can come out before baby
position
relationship of presenting part to maternal pelvis
described as anterior, posterior, transverse, right, left
attitude
relationship of fetal parts to each other
described as flexion or extension
Preterm labor and birth
spontaneous
75% of PTB
definitive factor=infection
congenital structural abnormalities of uterus
placental cause
maternal/fetal stress
decreased progesterone
indicated
25% of PTB
preeclampsia
multiple gestation
placental cause
maternal code blue
fetal fibronectin test (fFN)
used to predict who will not go into preterm labor
negative test shows less than 1% chance of giving birth within two weeks
cannot have anything in vagina for 24 hours
cervical insufficiency
passive/painful dilation of cervix leading to recurrent PTB during second trimester
can be caused by abnormally short cervix
risk factors include collagen disorder, uterine anomalies, hx cervical trauma, prior cervical surgery
tx with cervical cerclage placement
teach pelvic rest (nothing goes in the vagina)
monitor for any types of contractions
restrict activity but no bedrest
give tocolytic meds or nifedipine to suppress uterine activity
give antenatal glucocorticoids (betamethasone) to reduce respiratory distress syndrome, hemorrhage, necrotizing enterocolitis, death
Prelabor rupture of membranes (PROM)
SROM/leakage of fluid prior to onset of labor at any gestational age
often preceded by infection (chorioamnionitis)
bacterial infection of amniotic cavity
maternal fever, tachycardia, uterine tenderness, foul odor
administer magnesium sulfate for 24 hrs for fetal neuroprotection
Post term pregnancy
≥42 weeks of gestation
risk for perineal injury, maternal morbidity, macrosomia, meconium aspiration, shoulder dystocia
more frequent fetal assessment needed (NST or BPP)
Dystocia
lack of progress in labor
long, difficult, abnormal
may need c-section
causes
pelvic dystocia
contracture of pelvic diameter than reduce capacity of bony pelvis, inlet, midpelvis, oulet
soft-tissue dystocia
obstruction of birth passage by anatomic abnormality
fetal causes
anomaly
cephalopelvic disproportion
malposition/malpresentation
fix with external cephalic version
turn fetus from breech/shoulder presentation to vertex before birth
contraindications
uterine anomaly
third trimester bleeding
multiple gestation
oligohydramnios
previous c-section/uterine surgery
obvious CPD
multifetal pregnancy
Obstetric vaginal birth
increases risk for subgaleal hemorrhage in neonate
scalp pulled away from bony calvaria, vessels torn and blood collects in subgaleal space
increased risk to perineal trauma
forceps-assisted
piper forceps used to assist with delivery of head in breech birth
vacuum-assisted
discouraged for gestational age <34 weeks
using forceps after failed vacuum attempt increases newborn complications
Cesarean birth
birth through transabdominal incision of uterus
vertical skin incision
advantages
quicker to perform
better uterine visualization
can extend upward for more visualization if need (usually for obese woman)
disadvantages
visible scarring when healed
greater chance of dehiscence and hernia formation
pfannenstiel skin incision
advantages
less visibility when healed
low chance of dehiscence and hernia formation
disadvantages
less visualization of uterus
cannot be done quickly in emergency
cannot be extended easily
another c-section will take more time
low vertical incision
advantages
can be extended upward to make larger incision
disadvantages
more likely to rupture
tear may extend incision downward
low transverse incision
advantages
unlikely to rupture during subsequent birth
VBAC possible for another birth
less blood loss
easier repair
less adhesions
disadvantages
limited ability to enlarge incision
classical incision
advantages
may be only choice for:
placental implantation on lower anterior uterine wall
dense adhesions from previous surgery
transverse lie of large fetus with impacted shoulder
disadvantages
likely uterine incision to rupture during another birth
no VBAC option for subsequent birth
OB Emergencies
Trial of labor after cesarean (TOLAC)
TOLAC considered high risk for uterine rupture (highest for previous classical/T-shaped incision, prior uterine rupture, transfundal uterine surgery)
anxiety increases catecholamine and inhibits oxytocin release
delays progress of labor, can lead to repeat c-section
encourage VBAC for later pregnancy instead of another c-section
uterine rupture
symptomatic disruption and separation of layers of uterus/previous scar
mostly occur because of scarred uterus from previous c-section
can cause ejection of fetal parts/entire fetus into peritoneal cavity
risk factors
c-section
prior uterine rupture
trauma
abortion
multiparity
uterine overdistention
malpresentation
signs
abnormal FHR tracing
abrupt decrease in FHR
late/variable decelerations
absent baseline variability
tachy/bradycardia
loss of fetal station/no fetal descent
sudden sharp abdominal pain/ripping or tearing sensation
bright red vaginal bleeding/signs of hypovolemic shock
palpable fetal parts through abdomen
nursing role
start IV fluids
transfuse blood products
administer oxygen
prepare for immediate surgery
support family and provide info about tx during emergency
prognosis determined by whether placental abruption occurs and degree of maternal hemorrhage and hypovolemia
Meconium-stained amniotic fluid
green, can be thin or thick
result of:
hypoxia-induced peristalsis
sphincter relaxation
cord compression-induced vagal stimulation
risk for meconium aspiration syndrome in newborn
cause severe aspiration pneumonia
before birth
assess amniotic fluid for presence of meconium after ROM
gather equipment and supplies for neonatal resuscitation
immediately after birth
assess newborn respiratory effort, HR, muscle tone
suction only mouth and nose if WDL vital signs
Shoulder dystocia
head is born but anterior shoulder cannot pass under pubic arch
r/t excessive fetal size or maternal pelvic abnormality
cannot be predicted
risk factors:
hx shoulder dystocia
maternal diabetes
prolonged second stage of labor
signs
slow progress of second stage of labor
fetal head retraction following emergence (turtle sign)
no external rotation
interventions
assume McRoberts maneuver (pull mother’s knees to ears)
suprapubic pressure
clavicle fracture
symphsiotomy- incision in cartilage between pubic bones to enlarge pelvic opening
Prolapsed umbilical cord
cord lies below presenting part of fetus
risk factors
long cord
malpresentation (breech or transverse lie)
preterm labor
polyhydramnios
external cephalic version procedure
induction using balloon catheter
signs
visible/palpable after ROM
sudden onset variable/prolonged decels or bradycardia
report of feeling cord in vagina
management
recognize ASAP as hypoxia from cord compression can cause CNS damage/death
relieve pressure off cord
hold presenting part off umbilical cord
assist into lateral position, trendelenburg, knee-chest
Breech vaginal delivery
risks
umbilical cord prolapse
trapping of fetal head
trauma from fetal head extension/fetal arm positioned around neck
apply suprapubic pressure to keep head flexed until delivery
Uterine inversion
fundus collapses into uterine cavity after birth (turns inside out)
caused by:
fundal pressure when uterus is not well contracted
excessive umbilical cord traction when placenta is high in uterus
treatment
reinsertion of uterus
prepare for hemorrhage
terbutaline to stop contraction
magnesium sulfate, general anesthetic, nitroglycerin to relax smooth muscle
Hypertensive Disorders
CONTROLLING BLOOD PRESSURE IS THE OPTIMAL INTERVENTION TO PREVENT DEATH FROM STROKE/PREECLAMPSIA
gestational hypertension | development of HTN after week 20 of pregnancy in a woman with previously normal BP |
preeclampsia | HTN and proteinuria after 20 weeks of gestation who previously had neither condition in absence of proteinuria: new-onset HTN with new onset of ANY OF THESE—thrombocytopenia, renal sufficiency, impaired liver function, pulmonary edema, cerebral/visual symptoms |
chronic hypertension | present HTN before pregnancy/diagnosed before 20 weeks gestation |
superimposed preeclampsia | chronic HTN associated with preeclampsia |
Gestational HTN management
defined as systolic BP of >140 or diastolic of >90
frequent BP and weight measurement
report promptly if:
increase in BP
persistent headache
visual change
rapid weight gain
decreased fetal movement
uterine contractions
Preeclampsia
HTN and proteinuria after 20 weeks of gestation who previously had neither condition
can also develop in postpartum period
in absence of proteinuria, preeclampsia may be defined as HTN along with:
thrombocytopenia
renal sufficiency
impaired liver function
pulmonary edema
cerebral/visual symptoms (blinking stars/dots)
risk factors include:
preeclampsia hx
multifetal gestation
chronic HTN
pregestational/gestational diabetes
SLE
obstructive sleep apnea
nulliparity
BMI >30
AMA
thrombophilia
assisted reproductive technology
pathophysiology
placenta is root cause
begins to resolve after placenta expelled
spinal arteries fail to get larger and thicker
decreased placental perfusion and endothelial dysfunction= preeclampsia
placental ischemia=endothelial cell dysfunction
generalized vasospasm=poor tissue perfusion in organ system
fetal changes
impaired uteroplacental blood flow can cause:
IUGR
oligohydramnios
placental abruption
nonreassuring fetal status
preterm labor
preeclampsia with severe features
thrombocytopenia <100
renal insufficiency with elevated serum creatinine >1.1 mg/dl/doubling
pulmonary edema
headache unresponsive to medication
visual disturbances
systolic BP ≥160 or diastolic BP ≥110 at least twice 4 hrs apart
gestational age
onset prior to 34 weeks is most often severe
manage at facility with resources for management of serious maternal/neonatal complications
induction at 37 weeks indicated for preeclampsia without severe features
management
assess BP and edema
assess deep tendon reflex/hyperactive reflex (clonus)
assess PCR
evaluate for these s/s:
severe frontal headache
epigastric pain (heartburn)
right upper quadrant tenderness
visual disturbance
Eclampsia
onset of seizure activity/coma in preeclamptic patient
no hx of preexisting pathology
higher in multifetal gestation and women who did not receive prenatal care
caused by cerebral edema
watch for headache or visual disturbance
Chronic hypertension w/ superimposed preeclampsia
dx based on
sudden increase in BP that was previously well controlled
new-onset/sudden and sustained increase in proteinuria in woman known to have proteinuria before conception/early pregnancy
Magnesium Sulfate
mag sulfate for seizure prophylaxis is indicated for:
preeclampsia with severe features
severe gestational HTN
all cases of severe HTN regardless of classification
high Mg levels can causes relaxation of smooth muscle
initiate when diagnosed with preeclampsia
continue until 24 hrs post delivery or 24 hrs after last seizure if eclamptic
dose: 4-6gm loading dose over 20-30 min, then 2gm/hr
side effects
flushing/warm
drowsiness
sweating
N/V
HoTN
dizziness
headaches
slurred speech
visual disturbance
muscle weakness/decreased DTR
toxicity
absence/change in DTR
decreased LOC
SOB
decreased respiratory rate
persistent HoTN
chest pain
bradycardia/cardiac arrest
Mg greater than 8 mg/dL
give 1 g IV calcium gluconate over 3 min for toxicity
Meds for chronic HTN during pregnancy | ||
labetalol | 200-2400 mg | first-line in pregnancy, does not reduce uterine blood flow |
nifedipine | 30-90 mg daily | use with caution with mg sulfate |
methyldopa | 500-2000 mg | not effective when taken less than three times a day |
hydrochlorothiazide | 12.5-25mg daily | may be continued if taken before conception but not started as new med in pregnancy risk for thrombocytopenia for newborn |
medications to avoid | |
angiotensin-converting enzyme inhibitors (captopril) angiotensin II receptor antagonist (losartan) | assoc. with birth defects and impaired fetal renal fx |
atenolol | assoc. with growth restriction avoid use in early pregnancy, caution in late pregnancy |
HELLP Syndrome
can lead to:
liver hematoma/rupture
ARDS
sepsis
hypoxic encephalopathy
fetal/maternal death
preterm delivery
recurrent preeclampsia
treatment
induction regardless of gestational age
monitor CBC and liver enzymes Q6H
magnesium infusion
BP control
early epidural placement
Long-term Risks after hypertensive disorders of pregnancy
increased risk for pulmonary edema and cardiomyopathy
counsel on increased risk of future cardiovascular disease
evaluate/tx BNP, EKG, cardio echo, for low O2 sat, SOB, dyspnea
Hemorrhagic Disorders
Risk factors
multiparity
multiple gestation
assisted reproductive techniques
hx uterine surgery
STIs
hemorrhagic condition during previous pregnancy
alcohol
caffeine
cigarette smoking
Antepartum hemorrhagic disorders
antepartum hemorrhage
bleeding in pregnancy jeopardizes maternal/fetal well-being
blood loss decreases oxygen carrying capacity, increases risk for:
hypovolemia
anemia
infection
preterm labor
impaired O2 to fetus
hypoxemia of fetus
hypoxia of fetus
spontaneous abortion (miscarriage)
occurs before 20 weeks
loss of fetus weighing ≤500g
management
assess pregnancy hx, vital signs, pain, bleeding, labs, emotional status
administer misoprostol (cytotec)
dilation and curettage
emphasize rest
ectopic pregnancy
fertilized ovum implants outside uterine cavity
manifestations
dull, lower quadrant pain on one side
delayed menses
abnormal vaginal bleeding (spotting)
management
assess b-hCG level and transvaginal UA exam
administer methotrexate
refrain from taking folate
surgery
ruptured ectopic pregnancy
abdominal discomfort to colicy pain when tube stretches to sharp, stabbing pain
referred shoulder pain
management
pain meds
assess for s/s shock (faintness, dizziness)
surgery
hydatidiform mole
placental trophoblast growth in which chorionic villi develop into edematous, cystic, avascular transparent vesicles that hang in grape-like cluster
complete
no embryonic/fetal parts
sperm fertilizes empty egg
partial
often have embryonic/fetal parts and amniotic sac
abnormal placenta forms, two sperm fertilize one egg
generally can’t survive
manifestations
anemia from blood loss
hyperemesis gravidarum
abdominal cramps
large fundal height
preeclampsia
management
transvaginal UA
monitor b-hCG levels weekly for 3 weeks then monthly for 6-12 months
suction curettage
instruct to not get pregnant for 12 months
can increase risk for cancer
placenta previa
placenta implanted in lower uterine segment near/over internal cervical os
risk factors
previous c-section
AMA
multiparity
hx suction curettage
maternal cocaine use
smoking
low-lying
placenta in lower segment of uterus
does not reach opening of cervix
marginal
placenta next to cervix but does not cover
partial
placenta covers part of opening
complete
placenta covers entire opening
manifestations
painless bright red vaginal bleeding during second/third trimester
soft, relaxed, nontender abdomen
outcomes
major complication is hemorrhage
morbidly adherent placenta
surgery-related trauma
preterm birth/IUGR
late decels, absent/variable variability
management
no vaginal exams/internal monitors EXCEPT transvaginal ultrasound determining placental location
c-section is only option
cut right through placenta
come to hospital if any bleeding occurs
placental abruption
detachment of all/part of placenta after 20 weeks of gestation
risk factors
maternal HTN
cocaine/methamphetamine use
penetrating/blunt external abdominal trauma
smoking
hx abruption
PPROM
manifestations
painful, tight belly
wave-like contractions
vasa previa
fetal vessels lie over cervical os
vessels implanted into fetal membranes rather than into placenta
velamentous insertion
cord vessels branch at membranes and onto placenta
succenturiate placenta
placenta divided into 2+ lobes
battledore insertion of cord increases risk of fetal hemorrhage
Intrapartum hemorrhagic disorders
disseminated intravascular coagulation (DIC)
acquired secondary complication resulting in formation of clots in microsystem
clotting occurs throughout entire circulation
clotting factors are consumed, unable to keep up with demand
fibrinolysis occurs
fibrin split product damage RBC, cause hemolysis
damage endothelial lining of vessels, capillaries in lungs, platelets
leads to ARDS, pulmonary edema
lab findings
low fibrinogen <100
PT prolonged before PTT
management
administer fresh frozen plasma (FFP)
Postpartum hemorrhagic disorders
postpartum hemorrhage
cumulative blood loss ≥1000 ml OR bleeding associated with s/s of hypovolemia within 24 hrs of birth
primary PPH (early/acute)
occurs within 24 hrs of birth
uterine atony/rupture/inversion
coagulopathy
genital laceration
retained/invasive placenta
secondary PPH (late)
occurs more than 24 hrs to 12 weeks after birth
infection
retained placenta
coagulopathy
subinvolution of uterus
prolonged lochial discharge
irregular/excessive bleeding
sometimes hemorrhage
hemorrhagic (hypovolemic) shock
results of hemorrhage, death may occur
assessment
respirations
pulse
BP
skin
UO
LOC
characteristics
cool, pale, clammy
rapid, shallow respirations
rapid, weak, irregular pulse
lethargy, anxiety
management
fluid/blood replacement therapy
O2 delivery/maintain CO
restore blood volume
Hemorrhage Safety
have hemorrhage cart ready at all times
recognize risk assessment and QBL
low risk
no hx uterine incision
singleton
≤ 4 previous vaginal birth
no bleeding disorder
no hx PPH
medium risk
prior c-section/uterine surgery
multiple gestation
chorioamnionitis
>4 previous vaginal birth
hx PPH
uterine fibroids
high risk
placenta previa
placenta accreta, percreta, increta
Hct <30
PT <100,000
active bleeding
coagulopathy
overdistended uterus
prolonged labor
know massive transfusion protocol (MTP)
stage 1
stage 2
stage 3
3 units blood in 1 hr/ 10 units over 24 hrs
keep puting blood in until blood stops coming out
avoid LETHAL TRIAD
coagulopathy
hypothermia
acidosis
first priority is to replace PRBC and FFP
acidosis
develops from poor perfusion
delayed production of fibrin alters structures and breaks down
hypothermia
affects how platelets work to control bleeding
give warm blood/fluids
management
fundal massage
pack uterus
postpartum balloon
arterial embolization
hysterectomy
assess four Ts
tone
uterine atony associated with high parity, hydramnios, macrosomia, obesity, multiple gestation
tissue
retained placenta
uterine atony
trapped/fragmented placenta
placenta accreta—slight penetration on myometrium
placenta increta—deep penetration of myometrium
placenta percreta—perforation of myometrium and uterine serosa
HOW TO REMEMBER: ATTACHMENT SEVERITY BASED ON ALPHABETICAL ORDER
trauma
vaginal lacerations
first degree—limited to vaginal mucosa
second degree—involves perineal skin, mucous membranes, underlying fasciae, muscles
third degree—
fourth degree
hematoma
surgical complications
thrombin
DIC
idiopathic thrombocytopenia
von Willebrand disease
PPH medications
pitocin (oxytocin)
IM or IV
10-30 MU
misoprostol (cytotec)
rectal/vaginal
800-1000 mcg
methergine (methylergonvine)
PO or IM
0.2 mg
contraindicated in HTN
hemabate (carboprost)
IM
250 mcg
contraindicated in asthma
tranexamic acid
IV
1 gm
treat lacerations