Shock, also known as hypoperfusion, is defined as inadequate cellular perfusion.
Any compromise in perfusion can lead to cellular injury or death.
In the early stages of shock, the body attempts to maintain homeostasis.
Pathophysiology
Diffusion
Diffusion is a passive process where molecules move from an area of high concentration to an area of low concentration.
Oxygen and carbon dioxide move across the walls of the alveoli through diffusion.
Impaired Perfusion
In cases of poor perfusion (shock), the transportation of carbon dioxide out of tissues is impaired, leading to a buildup of waste products and potential cellular damage.
Cardiovascular System Failure
Shock is a state of collapse and failure of the cardiovascular system, resulting in inadequate circulation.
Early recognition and rapid treatment are crucial for saving lives.
Components of the Cardiovascular System
The cardiovascular system consists of three main parts:
Pump (heart)
Set of pipes (blood vessels or arteries)
Contents (the blood)
Perfusion Triangle
The heart, blood vessels, and blood represent the three parts of perfusion, often referred to as the "perfusion triangle."
When a patient is in shock, one or more of these components is not working correctly.
Blood Pressure
Blood pressure is the pressure of blood within the vessels.
Systolic pressure: Peak arterial pressure.
Diastolic pressure: Pressure in the arteries when the heart rests between beats.
Pulse Pressure
Pulse pressure is the difference between systolic and diastolic pressures.
It signifies the force the heart generates with each contraction.
A pulse pressure less than 25 mm Hg may be seen in patients with shock.
Capillary Beds
Blood flow through the capillary beds is regulated by capillary sphincters, controlled by the autonomic nervous system.
Regulation of blood flow is determined by cellular needs.
Requirements for Adequate Perfusion
Perfusion requires adequate:
Oxygen exchange in the lungs.
Nutrients, particularly glucose, in the blood.
Waste removal, primarily through the lungs.
Support Mechanisms
Mechanisms are in place to support the respiratory and cardiovascular systems when the need for perfusion of vital organs increases. These include the autonomic nervous system and hormones.
Hormonal Response
Hormones are triggered when the body senses pressure falling, leading to:
Increased heart rate.
Increased strength of cardiac contractions.
Peripheral vasoconstriction.
This response causes the signs and symptoms of shock.
Causes of Shock
Many different types of shock result from three basic causes:
Pump failure.
Poor vessel function.
Low fluid volume.
Cardiogenic Shock
Caused by inadequate function of the heart.
A major effect is the backup of blood into the pulmonary vessels, resulting in pulmonary edema.
Cardiogenic shock develops when the heart cannot maintain sufficient output to meet the body's demands.
Cardiac output depends on:
Contractility of the heart muscle.
Amount of blood to pump (preload).
Resistance to flow in peripheral circulation (afterload).
Patients in cardiogenic shock should not receive nitroglycerin because they are hypotensive.
Patients usually have low blood pressure, a weak/irregular pulse, cyanosis, anxiety, and nausea.
Obstructive Shock
Caused by a mechanical obstruction that prevents adequate blood volume from filling the heart chambers.
Common examples include:
Cardiac tamponade.
Tension pneumothorax.
Pulmonary embolism.
Cardiac Tamponade
Collection of fluid between the pericardial sac and the myocardium (pericardial effusion) becomes large enough to prevent ventricles from filling with blood.
Caused by blunt or penetrating trauma. Signs and symptoms may be referred to as Beck Triad.
Tension Pneumothorax
Caused by damage to lung tissue.
Air normally held within the lung escapes into the chest cavity, collapsing the lung and applying pressure to organs, including the heart and great vessels.
Pulmonary Embolism
A blood clot blocks blood flow through pulmonary vessels.
If massive, it can result in complete backup of blood in the right ventricle, leading to catastrophic obstructive shock and complete pump failure.
Distributive Shock
Results from widespread dilation of small arterioles, small venules, or both.
Circulating blood volume pools in the expanded vascular beds, decreasing tissue perfusion.
Septic Shock
Occurs due to severe infections where toxins are generated by bacteria or infected body tissues.
Vessel walls leak and are unable to contract well, leading to widespread dilation and plasma loss, resulting in shock.
Neurogenic Shock
Usually results from high spinal cord injury.
Nerve impulses to blood vessels below the injury are blocked.
Vessels cut off from nerve impulses dilate, causing blood to pool.
Anaphylactic Shock
Occurs when a person reacts violently to a substance to which they have been sensitized.
Sensitization means becoming sensitive to a substance that did not initially cause a reaction.
Subsequent exposures tend to produce more severe reactions.
Psychogenic Shock
Caused by a sudden reaction of the nervous system, producing temporary, generalized vascular dilation that results in fainting (syncope).
Life-threatening causes include irregular heartbeat and brain aneurysm.
Non-life-threatening events include receiving bad news or experiencing fear or unpleasant sights.
Hypovolemic Shock
Results from an inadequate amount of fluid or volume in the circulatory system.
Can be caused by hemorrhagic (bleeding) and nonhemorrhagic (dehydration, burns) factors.
Occurs with severe thermal burns.
Progression of Shock
Stages in the progression of shock:
Compensated shock: early stage when the body can still compensate for blood loss.
Decompensated shock: late stage when blood pressure is falling.
It is impossible to assess when effects are irreversible, so early recognition and treatment are essential.
Blood pressure may be the last measurable factor to change, especially in infants and children where a drop in blood pressure indicates they are close to death.
Also expect shock if a patient has multiple severe fractures, abdominal or chest injury, spinal injury, a severe infection, a major heart attack, or anaphylaxis.
Patient Assessment
Scene Size-Up
Be alert to potential hazards.
Use gloves and eye protection for trauma scenes or if bleeding is suspected.
Consider the mechanism of injury/nature of illness.
Primary Assessment
Perform a rapid exam.
Determine the level of consciousness.
Identify and manage life-threatening concerns.
Determine the priority of the patient and transport.
Provide high-flow oxygen to assist in perfusion.
Treat aggressively for hypoperfusion and provide rapid transport.
Request advanced life support (ALS) as necessary.
Increased respiratory rate is often an early sign of impending shock.
A rapid pulse suggests compensated shock.
In shock or compensated shock, the skin may be cool, clammy, or ashen.
Assess for and identify any life-threatening bleeding and treat it immediately.
Trauma patients with shock or a suspicious MOI generally should go to a trauma center.
History Taking
Determine the chief complaint.
Obtain a SAMPLE history.
Secondary Assessment
Repeat the primary assessment, followed by a focused assessment.
Treat any immediate life-threatening problems.
Obtain a complete set of baseline vital signs.
Use monitoring devices.
Reassessment
Reassess the patient’s:
Vital signs
Interventions
Chief complaint
ABCs (Airway, Breathing, Circulation)
Mental status
Determine what interventions are needed.
Focus on supporting the cardiovascular system.
Treat for shock early and aggressively by:
Providing oxygen
Keeping the patient warm
Emergency Medical Care for Shock
As soon as you recognize shock, begin treatment.
Follow standard precautions.
Control all obvious bleeding.
Make sure the patient has an open airway.
Maintain manual in-line stabilization if necessary, and check breathing and pulse.
Comfort, calm, and reassure the patient.
Never allow patients to eat or drink anything.
If spinal immobilization is indicated, splint the patient on a backboard.
Provide oxygen and monitor the patient’s breathing.
Place blankets under and over the patient.
Consider the need for ALS.
Accurately record the patient’s vital signs approximately every 5 minutes throughout treatment and transport.
Treating Specific Types of Shock
Cardiogenic Shock
Patient cannot generate the necessary contraction to pump blood.
Patients may present with chest pain.
Place the patient in a position that eases breathing.
Assist ventilations as necessary.
Provide prompt transport.
Consider meeting ALS en route to hospital.
Obstructive Shock
Cardiac tamponade: Increasing cardiac output is the priority. Apply high-flow oxygen. Surgery is the only definitive treatment.
Tension pneumothorax: Apply high-flow oxygen to prevent hypoxia. Chest decompression is required. Request ALS early in call if available, but do not delay transport.
Septic Shock
Hospital management is required.
Use standard precautions and transport.
Administer high-flow oxygen; ventilatory support may be necessary.
Use blankets to conserve body heat.
Notify “sepsis team” if available.
Neurogenic Shock
Emergency treatment includes:
Obtaining and maintaining a proper airway.
Providing spinal immobilization.
Assisting inadequate breathing.
Conserving body heat.
Ensuring the most effective circulation.
Transporting promptly.
Anaphylactic Shock
Administer epinephrine.
Promptly transport the patient.
Provide high-flow oxygen and ventilatory assistance en route.
A mild reaction may worsen suddenly.
Consider requesting ALS backup, if available.
Psychogenic Shock
In an uncomplicated case of fainting, circulation to the brain is restored once the patient collapses.
Psychogenic shock can worsen other types of shock.
If the patient reports being unable to walk, suspect another problem.
Transport the patient promptly.
Hypovolemic Shock
Control all obvious external bleeding.
Keep the patient warm.
Recognize internal bleeding and provide aggressive support.
Secure and maintain an airway and provide respiratory support.
Transport as rapidly as possible.
Treating Shock in Older Patients
Older patients have more serious complications than younger ones.
Illness is not just a part of aging.
Many older patients take medications that mask or mimic signs of shock.