Human Anatomy & Physiology Chapter 27 - Metabolism & Nutrition
Metabolism & Nutrition Overview
Focus on Lipid Metabolism, Protein Metabolism, Carbohydrate Metabolism, Cellular Respiration, Metabolic Rate, and Starvation.
Metabolic Rate
Definition: Amount of energy used by the body to support metabolic processes.
Measurement:
Recorded in Calories (actually kilocalories).
Defined as the energy needed to raise 1g of water by 1°C. This is an extremely small amount of energy; hence, Calories are often used (i.e., kilocalories).
Measuring Metabolic Rate
Methods of Measurement:
Can be measured through heat production (calorimetry).
More commonly measured via oxygen consumption.
Energy Expenditures Components
Energy expenditures consist of
Basal Metabolic Rate (BMR):
Represents around 60% of total calories burned.
Comprises metabolic activities needed for maintaining homeostasis (e.g., Na+/K+ pumps, cellular activities).
Includes resting heart rate, resting breathing rate, and resting nervous system activity.
Active Metabolic Rate (AMR):
Involves physical activity of any kind.
Proportion of total energy used varies greatly between individuals.
Specific Dynamic Action (SDA):
Energy used to digest food.
Accounts for 5% to 10% of total calories burned.
Growth:
If BMR + AMR + SDA does not equal calorie intake, difference results in growth (positive or negative).
BMR Conditions
A person is operating at BMR under the following conditions:
Well-rested.
Fasted for about 12 hours.
No recent physical activity (within the last hour).
No physical stress is induced.
At a constant and comfortable temperature.
Energy Balance
Definition: The balance between energy intake and energy expenditure.
Optimal energy homeostasis requires energy intake to match energy expenditure; otherwise, weight is gained or lost.
Components of Energy Intake: Consists of food consumption.
Components of Energy Expenditure: Encompasses metabolic rate, physical activity, and digestion.
Body Mass Index (BMI)
Formula:
ext{BMI} = \frac{\text{Weight (kg)}}{\text{Height (m)}^2}Categories:
Underweight: BMI < 18.5
Normal weight: BMI 18.5 - 24.9
Overweight: BMI 25 - 29.9
Class 1 Obesity: BMI 30 - 34.9 (some risk)
Class 2 Obesity: BMI 35 - 39.9 (moderate risk)
Class 3 Obesity: BMI ≥ 40 (high risk)
Problems with BMI
Not an accurate measure for very tall or short individuals.
Does not differentiate between muscle mass and fat mass.
Some debate about the definitions of the BMI categories.
Obesity
Negative implications include:
Decreased life expectancy.
Increased rates of atherosclerosis, hypertension, diabetes, joint problems, kidney stone formation, and gallstones.
Elevated cancer risk in:
Women: breast, uterine, and liver cancers.
Men: colon, rectal, and prostate cancers.
Appetite Control
Neural Controls:
Control centers located in the hypothalamus help regulate hunger and satiety, influenced by blood nutrient levels.
Hormonal Control:
Insulin and Cholecystokinin (CCK) facilitate satiety.
Glucagon, epinephrine, and ghrelin stimulate appetite.
Leptin:
Hormone produced by adipose tissue that suppresses appetite.
In obese individuals, leptin levels are high, but they often become resistant to its effects, resulting in unregulated appetite levels.
Psychological Factors:
"Comfort eating" may occur, providing stress relief by activating the parasympathetic nervous system, but this behavior is not driven by regular appetite controls.
Starvation
Definition: A severe deficiency in caloric intake due to fasting, anorexia, deprivation, or illness.
Phases of Starvation:
First Phase:
Blood glucose levels are maintained via glycogenolysis for several hours.
After glycogen stores are depleted, fats and proteins are catabolized to maintain glucose levels.
Fatty acid catabolism occurs for most body cells, while glucose from amino acids is utilized for the central nervous system (CNS).
Second Phase:
May last for several days or weeks.
The liver produces ketone bodies from fatty acids; the CNS begins to increasingly rely on these ketone bodies.
Risk of ketoacidosis may develop due to high ketone levels in the blood.
Third Phase:
Initiated once fat reserves are depleted.
Proteins become the primary energy source; this leads to depletion of muscle proteins.
Essential proteins for cellular function become degraded, resulting in severe changes such as extreme weight loss, apathy, listlessness, and heightened susceptibility to infections.
Consequences may include changes in hair color and abdominal edema.