Health, United States Spotlight: Racial and Ethnic Disparities in Heart Disease
Health, United States Spotlight: Racial and Ethnic Disparities in Heart Disease (April 2019)
Overview of Heart Disease and Disparities
- Heart disease is the leading cause of death in the United States.
- The risk of death from heart disease varies significantly between different racial and ethnic groups.
- The spotlight focuses on:
- Deaths
- Reported prevalence
- Risk factors associated with heart disease.
- Four clinical risk factors discussed include:
- Hypertension
- Obesity
- Diabetes
- High total cholesterol
- Behavioral risk factors such as smoking and physical inactivity also exhibit disparities by race and ethnicity.
Heart Disease Topic Areas
Deaths
- Data Source: National Center for Health Statistics (NCHS), National Vital Statistics System (NVSS).
- Notes:
- Reporting for racial and ethnic groups other than non-Hispanic White and non-Hispanic Black is often inconsistent on death certificates.
- Despite this inconsistency, misclassification is minor for Hispanic and non-Hispanic Asian or Pacific Islander groups.
Prevalence
- Data Source: NCHS, National Health Interview Survey (NHIS).
- Notes:
- Prevalence figures are self-reported; respondents indicate if health professionals had diagnosed them with conditions like:
- Coronary heart disease
- Angina
- Heart attack
- Other heart conditions.
Demographics of Death Rates
Age-adjusted death rates for heart disease (1999-2017):
- Metrics: Deaths per 100,000 persons
- Trends indicate:
- From 1999 through 2017, heart disease death rates declined for all groups; however, the rate of decrease has slowed recently.
- Non-Hispanic Black individuals were over twice as likely to die from heart disease compared to non-Hispanic Asian or Pacific Islander individuals in both 1999 and 2017.
- Death rates:
- 2017:
- Black, not Hispanic: 337.4
- White, not Hispanic: 168.9
- Hispanic: 114.1
- Asian or Pacific Islander, not Hispanic: 85.5
- Trend Analysis:
- Non-Hispanic White adults aged 18 and over:
- 2017: 11.5% had heart disease (age-adjusted).
- 1999: Significantly different from other racial groups.
- Non-Hispanic Black adults aged 18 and over:
- 2017: 9.5% had heart disease (age-adjusted).
- 1999: Also significantly different from other groups (stable trend).
- Hispanic adults aged 18 and over:
- 2017: 6.0% had heart disease (age-adjusted), stabilized since 1999.
Learning Objectives
- Explain the relationships between different lipids and chronic diseases such as saturated fats, trans fats, cholesterol, unsaturated fats, and essential fatty acids.
- List current dietary recommendations for lipid intake.
- Post-Prandial State (After a Meal):
- Increased Insulin, decreased Glucagon.
- Insulin encourages lipogenesis (conversion of excess energy sources into fat, hierarchy: fat > carbohydrates > proteins) and triglyceride storage.
- Post-Absorptive State (Between Meals):
- Decreased Insulin, increased Glucagon.
- Glucagon promotes lipolysis (fat breakdown).
- Main storage site for lipids is in lipid droplets across different classes of adipocytes (fat storage cells).
Exogenous Lipid Pathway
- Post-Prandial State:
- Begins with dietary lipids.
- Digestion (~75% occurs in the small intestine via pancreatic lipase).
- Free Fatty Acids (FFA) and glycerol are absorbed by enterocytes, re-esterified, and packed into chylomicrons (CM) along with cholesterol.
- CM released into lymph capillaries then into bloodstream.
- In circulation, Lipoprotein Lipase (LPL) removes fatty acids from Triglycerides (TAGs) inside CM, allowing FFA uptake by extrahepatic tissues (e.g., adipose and skeletal tissues) for energy or storage.
- CM remnants are then absorbed by the liver.
Endogenous Lipid Pathway
- Post-Absorptive State:
- Starts at the liver.
- Newly synthesized FAs or those from extrahepatic tissues are re-esterified into TAGs and packed into Very-Low-Density Lipoprotein (VLDL).
- In circulation, LPL removes FAs from VLDL, making FFA available to peripheral tissues.
- FAs can be utilized as energy or undergo oxidation (FFA → Acetyl-CoA).
- VLDL is converted into Intermediate-Density Lipoprotein (IDL);
- 2/3 of IDL is taken by the liver, and 1/3 is further metabolized into Low-Density Lipoprotein (LDL).
- LDL is rich in cholesterol esters, taken up by peripheral tissues via LDL receptors, and can be degraded by^ macrophages or taken back by the liver.
Reverse Cholesterol Pathway
- High-Density Lipoprotein (HDL):
- Forms in both liver and small intestines.
- Transports cholesterol esters from peripheral tissues to the liver, converting them into bile which is the primary excretory route for cholesterol.
- HDL serves as a repository for transferable apoproteins.
Atherosclerosis and Cardiovascular Disease (CVD)
- Altered ratios of LDL, HDL, cholesterol, and TAGs are associated with increased risk of atherosclerosis, the primary cause of CVD.
- LDL (bad cholesterol) delivers cholesterol esters to peripheral tissues.
- HDL (good cholesterol) collects excess cholesterol esters to return to the liver.
Healthy Blood Lipid Profile
- Total cholesterol: < 200 mg/dL
- LDL-cholesterol (LDL-C): < 100 mg/dL
- HDL-cholesterol (HDL-C): > 60 mg/dL
- Triglycerides: < 150 mg/dL
Impact of Atherosclerosis
- Atherosclerosis restricts blood flow:
- Blocks flow to the brain (causing stroke).
- Blocks flow to the heart (causing heart attack).
Cardiovascular Disease (CVD)
- A broad term covering conditions affecting the heart and blood vessels, including:
- Blood vessel diseases (e.g., coronary artery disease [CAD])
- Arrhythmias (irregular heart rhythms)
- Congenital heart defects
- Heart valve diseases
- Heart infections
- Leading global causes of death in 2021, affecting men and women across racial and ethnic groups.
Heart Disease Statistics (2023)
- Heart disease is the leading cause of death, accounting for
- 919,032 deaths (1 in every 3 deaths).
- Cost estimates: ~ $417.9 billion (2020-2021).
- Coronary heart disease is the most common type, resulting in 371,506 deaths in 2022.
- Highest prevalence in southern US; increases with age, highest in males.
Risk Factors for Coronary Artery Disease (CAD)
- Signs/Symptoms include:
- Shortness of breath
- Chest pain
- Fatigue
- Dizziness
- Dietary factors influencing CAD include:
- Role of nutrition may improve or worsen CAD depending on dietary choices.
Dietary and Lifestyle Recommendations
- Avoid trans fats.
- Keep saturated fat < 10% of total energy intake, and increase mono-/polyunsaturated fats.
- Maintain a healthy weight.
- Avoid smoking.
- Exercise regularly.