AC

Dental Office Medical Emergencies – Comprehensive Bullet-Point Study Notes

Asthma & Bronchospasm

  • Pathophysiology
    • Acute episode = spasm of smooth muscle around bronchi/bronchioles → marked luminal narrowing.
    • Consequence: inadequate O₂ in / CO₂ out → potential fatal hypoxia / hypercarbia.

  • Pre-appointment history checklist
    • "What type of asthma do you have?" – \text{Allergic} vs \text{Non-allergic}.
    • Non-allergic often precipitated by fear / anxiety / inadequate pain control.
    • Ask: "What are you allergic to?" (aspirin common trigger).
    • Current meds: rescue bronchodilator (e.g., albuterol) ± daily inhaled corticosteroid.
    • Frequency & triggers of attacks; recent attacks in dental chairs? fear of dentistry = red flag.
    • History of attacks that "did not stop" or hospitalization → determines need to pre-alert 911.

  • Preventive office strategies
    • Advise pt during confirmation call to bring own inhaler (rescue med).
    • Consider prophylactic sedation if stress-induced attacks likely.

  • In-office management of an attack
    • Immediately sit pt upright (easier diaphragmatic excursion).
    • Encourage self-administration of rescue inhaler placed on bracket tray.
    • Typical dose: 2 puffs; reassess after 5 min; may repeat.
    • If pt, provider, or situation remains uncomfortable → terminate procedure, reschedule, treat underlying fear before resuming care.
    • Office backup: own bronchodilator if pt’s fails.

Airway Obstruction (Choking)

  • Most common cause: food; pt initially conscious but mute; universal choking sign.
  • Critical window: if airway not reopened within 15–20 s → loss of consciousness.
  • Management sequence
    • Abdominal thrusts (Heimlich) on conscious pt.
    • Activate EMS, assess need for medical evaluation even after object expelled.
  • Dental-specific prevention
    • Tie dental floss to any small object placed intra-orally (rubber dam clamp, implant screw, cotton roll etc.) – "little things save lives".

Allergic Reactions & Anaphylaxis

  • Immunology crash-course
    • Allergen binds to sensitized mast-cell receptors → degranulation → release of chemical mediators (primarily histamine).
    • Histamine actions: itching, urticaria, rash, bronchospasm, vasodilation, edema.
    • Severity depends on speed + site of mediator release:
    – Skin-only = pruritus / rash (non-life-threatening).
    – Lungs = bronchospasm.
    – CVS = vasodilation ↓BP + edema → anaphylaxis.

  • Classification by onset
    • Late (delayed) onset: signs ≥ 1 h post-exposure; usually cutaneous; e.g., pt who took prophylactic penicillin developed mild rash.
    • Immediate onset: seconds–minutes; involves skin + airway and/or CVS = true anaphylaxis.

  • Management algorithm (PABCD)
    • P – Position: unconscious → supine; conscious → whatever eases breathing.
    • A/B/C – assess, support airway, breathing, circulation; instruct staff to call 911.
    • D – Definitive drug: Epinephrine 0.3–0.5 mg IM mid-thigh ASAP; repeat q 5 min until recovery or ALS arrives.
    – Benefits: potent bronchodilation + vasopressor (restores BP).
    – No absolute contraindication when anaphylaxis suspected.
    • After epi & once pt stable → administer antihistamine (e.g., diphenhydramine) to blunt residual histamine.

Local Anesthetic (LA) Overdose

  • Error chain
    • Wrong treatment plan: four-quadrant dentistry in one visit on child.
    • Wrong drug: long-acting LA w/ vasoconstrictor when short-acting sufficient.
    • Injecting every quadrant sequentially.
    • Using full cartridge 1.8\,\text{mL} per site when fraction would suffice.

  • Toxicity timeline
    • Peak blood level ≈ 5 min post-injection; first manifestation often generalized seizure <60 s.
    • If airway not maintained post-seizure → hypoxia + hypercarbia ↓ seizure threshold → second, longer seizure 2–3 min later.

  • Management
    • Secure airway, deliver \ge 5\,\text{L/min} O₂.
    • Prevent hypoxia / hypercarbia to avoid recurrence.
    • Monitor vitals until drug redistributes & pt recovers.

Oral / Inhalation Sedatives & Monitoring

  • Premed given ≈ 60 min pre-procedure; pt must remain under continuous observation.
  • Once in chair:
    • Apply pulse oximeter – real-time SpO₂ & HR.
    • Low-SpO₂ alarm → immediate airway check, reposition, supplemental O₂, consider reversal agents if narcotics/benzodiazepines involved.

Coronary Artery Disease (CAD) & Acute Coronary Syndromes

  • Pathogenesis
    • Atherosclerotic plaque buildup in coronary walls → chronic inflammation → luminal narrowing.
    • Stress/effort ↑ myocardial O₂ demand; narrowed lumen can’t augment supply → ischemia.

  • Angina Pectoris
    • Ischemic chest discomfort: tight/heavy; pt may clench fist over sternum (Levine sign).
    • Usually known dx; relieved by vasodilator (nitroglycerin).

  • Myocardial Infarction (MI)
    • Plaque rupture → platelet aggregation → thrombus occludes artery → downstream myocardium ischemic → necrosis.
    • Ischemic tissue electrically unstable → arrhythmias → sudden cardiac arrest.
    • "Golden window": reperfusion (balloon angioplasty or thrombolytics) within 2 h → minimal damage.

  • Chair-side differentiation & first aid
    • Persistent pain after 3 doses nitro (q 5 min) OR first-ever chest pain → treat as MI.
    • Immediate steps (MONA):
    – Morphine IV (or 50\% N₂O/O₂ mix if available) for pain & anxiety.
    – Oxygen 5 L/min via mask or cannula.
    – Nitroglycerin sub-lingual or spray up to 3 doses unless hypotensive.
    – Aspirin 325 mg chew & swallow if no contraindication (allergy, GI bleed, anticoag tx).
    • Always activate EMS early.
    • Typical MI pain scale: 10/10; may radiate to epigastrium, L-arm (pinky tingling), L-neck, L-mandible; pt pale, diaphoretic, anxious but conscious.

Cardiac Arrest, CPR & AED

  • Only signs: sudden unresponsiveness + apnea + no carotid pulse.
  • Provider sequence
    • Stop dental work; shout for help.
    • Check responsiveness; call/name; no response.
    • Open airway (head-tilt/chin-lift), look–listen–feel; if no breathing → 2 rescue breaths with O₂.
    • Palpate carotid 10 s; if pulseless start high-quality chest compressions.
    • Delegate: "Activate 911, bring emergency kit & AED!"
    • Continue CPR at 100–120 compressions/min, depth \approx 2 in (adult) with minimal interruptions.
    • Apply AED ASAP; follow prompts – defibrillate if advised.
  • Complications
    • Rib fractures common, especially elderly / osteoporotic – legally acceptable outcome when CPR done correctly.
    • Multiple shocks cause skin burns; continue care until ROSC or ALS arrival.

Infection-Control Nuggets Identified in Video

  • Observed lapses: contaminated gloves touching cabinets/phones, jewelry under gloves, etc.
  • Reminder: maintain high standards (hand hygiene, PPE removal before touching non-clinical surfaces, no jewelry) – clinical reality often falls short; vigilance protects patients & providers.