Overview by Nancy Cox, PTA, MS PTAS.
References: Manske & Shankman; chapters 2, 8-12.
Indicates assessment criteria and general principles of healing in orthopedic conditions.
Acute Inflammatory Response: Initial response mechanism to injury.
Repair & Regeneration (Proliferation): Involves the building of new tissue.
Remodeling & Maturation: Ongoing adjustments to tissue organization.
Additional resource: Video on Phases of Healing.
Definition: Immunological defense mechanism against injury/infection. Characterized by:
Increased blood flow
Migration of white blood cells
Release of chemical mediators.
Importance: Triggers tissue healing, but chronic inflammation can lead to permanent changes.
Signs:
Pain
Localized tissue edema
Loss of function
Heat, redness, tenderness, swelling
Physiological Events:
Increased capillary permeability
Releases systemic responses like fever.
Acute Inflammation: Usually lasts 4-6 days post-injury, with signs persisting up to 2 weeks.
Subacute Inflammation: Lasts greater than 4 weeks.
Chronic Inflammation: Extends beyond 6 months.
Pain:
Pain before end of range of motion: acute inflammation.
Pain at end range: subacute inflammation.
Stretching sensation limit: chronic inflammation.
Localized Edema Measurement:
Use circumferential or figure-8 techniques; compare affected vs. non-affected sides.
Pitting Edema: Sustained indentation when pressed, common in CHF patients.
Brawny Edema: Hard, leathery texture, usually found in venous disease.
Pain is subjective and must be treated seriously.
Use standardized scales to evaluate pain levels thoroughly.
Document patient responses, such as body language and facial expressions.
PTA should halt intervention if red flag symptoms arise and report to PT or MD.
Referred Pain: Felt in a location distant from the actual injury.
Visceral Pain: Associated with internal organs, poorly localized and may involve autonomic symptoms.
Trigger Points: Small areas within muscles that cause localized pain upon palpation.
Apoptosis: Programmed cell death without inflammation.
Hypoxia and Necrosis: Related to insufficient blood flow and results in inflammation.
Involves collagen fibril cross-linking and remodeling with factors including endothelial and fibroblast activity.
Injured tissue may regenerate through cytokine activation and can vary from labile to permanent cell types.
Ligaments: High elastin content, receive blood supply at origins.
Tendons: Transmit muscle forces, often covered by synovial sheaths for nutrition and movement facilitation.
Common causes include rupture of collagen fibers and insertion at bony sites.
Classification of Injury:
Grade I: Mild sprain.
Grade II: Moderate sprain.
Grade III: Complete ligament tear.
Ligaments generally heal slower, with scar replacement instead of actual regeneration.
Optimal conditions for heal include proper alignment, consistent stress application, and protection from excessive forces.
Include age, blood supply, immobilization, smoking, and existing medical conditions like diabetes.
Can cause joint stiffness, atrophy, and reduced hydration; 20% weight loss observed after 8 weeks of immobilization.
Introducing motion can enhance healing and strength. Continuous passive motion (CPM) is recommended to maintain tissue health and minimize complications.
Timeline from injury through gradual recovery phases, each with specific treatment recommendations.
Risk of injuries increase with age and overuse; tendinosis may occur in absence of clear inflammation.
Classification into first, second, and third-degree strains based on the extent of fiber damage.
Leads to decreased muscle mass, loss of endurance, and increased fall risk.
Key approaches include promoting motion in unaffected joints and safe muscle contractions for the immobilized area.
Types of nerve injuries (Neuropraxia, Axonotmesis, Neurotmesis) and their implications on recovery and treatment.
Importance of protecting repair sites and considerations for sensory reeducation are crucial during rehabilitation.
Essential processes include inflammation and remodeling, monitoring for signs such as pulselessness and hematoma.
Importance of anticoagulants and early mobilization; risk factors include surgery and immobility.
A condition that requires immediate attention characterized by severe pain, swelling, and potential muscle and nerve damage.
Atherosclerosis can lead to intermittent claudication, emphasizing the need for early diagnosis and management.
Comprehensive understanding of tissue healing is essential for effective orthopedic intervention and rehabilitation.