Post-Traumatic Stress Disorder
Trauma exposure
Continued re-experiencing
(e.g., memories, nightmares, flashbacks)
Avoidance
Emotional numbing
Reckless or self-destructive behavior
Interpersonal problems
Refers to problems that persist for more than one month after the trauma
Acute stress disorder assigned for posttraumatic symptoms lasting less than a month
Statistics
6.8% (life); 3.5% (year)
Prevalence varies
Most people who undergo traumatic events do not develop PTSD
Type of trauma
E.g., experiencing repeated sexual assault makes an individual 2 to 3 times as likely to develop PTSD
Proximity – more likely to develop PTSD if closer to the trauma
Trauma intensity – PTSD more likely with severe trauma
Generalized biological vulnerability
Twin studies
Reciprocal gene-environment interactions
Generalized psychological vulnerability
Beliefs about uncontrollability and unpredictability of threatening situations
Poor social support = greater risk
Exposure to actual or threatened event
Presence of one or more intrusional symptoms
Persistent avoidance of stimuli associated with traumatic event
Negative alterations in cognitions and mood associated with traumatic event
Marked alterations in arousal and activity associated with the traumatic event
Sleep disturbance
Significant distress
Not attributable to substance use
Threatening cues activate CRF system
CRF system activates fear and anxiety areas
Amygdala (central nucleus)
Increased HPA axis activation
Cortisol
Cognitive-behavioral treatment
Imaginal exposure to memories of traumatic event
Graduated or massed
Increase positive coping skills
Increase social support
Highly effective
Psychoanalytic therapy: catharsis = reliving emotional trauma to relieve suffering
Medications
SSRIs can be helpful
Relieve heightened anxiety and panic attacks common to PTSD
Trauma exposure
Continued re-experiencing
(e.g., memories, nightmares, flashbacks)
Avoidance
Emotional numbing
Reckless or self-destructive behavior
Interpersonal problems
Refers to problems that persist for more than one month after the trauma
Acute stress disorder assigned for posttraumatic symptoms lasting less than a month
Statistics
6.8% (life); 3.5% (year)
Prevalence varies
Most people who undergo traumatic events do not develop PTSD
Type of trauma
E.g., experiencing repeated sexual assault makes an individual 2 to 3 times as likely to develop PTSD
Proximity – more likely to develop PTSD if closer to the trauma
Trauma intensity – PTSD more likely with severe trauma
Generalized biological vulnerability
Twin studies
Reciprocal gene-environment interactions
Generalized psychological vulnerability
Beliefs about uncontrollability and unpredictability of threatening situations
Poor social support = greater risk
Exposure to actual or threatened event
Presence of one or more intrusional symptoms
Persistent avoidance of stimuli associated with traumatic event
Negative alterations in cognitions and mood associated with traumatic event
Marked alterations in arousal and activity associated with the traumatic event
Sleep disturbance
Significant distress
Not attributable to substance use
Threatening cues activate CRF system
CRF system activates fear and anxiety areas
Amygdala (central nucleus)
Increased HPA axis activation
Cortisol
Cognitive-behavioral treatment
Imaginal exposure to memories of traumatic event
Graduated or massed
Increase positive coping skills
Increase social support
Highly effective
Psychoanalytic therapy: catharsis = reliving emotional trauma to relieve suffering
Medications
SSRIs can be helpful
Relieve heightened anxiety and panic attacks common to PTSD