Evaluation of Milner (1966): The Case Study of HM (Bullet-Point Format)
Introduction
Memory: A cognitive process involving encoding, storage, and retrieval of information.
Hippocampus: A medial temporal lobe structure responsible for consolidating short-term memory into long-term memory.
Approach relevance:
Supports the cognitive approach through insights into memory models.
Supports the biological approach by linking brain structures to behavior.
Essay focus: Evaluation of Milner’s (1966) study using the TEACUP framework: Testability, Empirical support, Applications, Construct validity, Unbiasedness, and Predictive validity.
Theory
Supports the Multi-Store Model of Memory (Atkinson & Shiffrin, 1968): distinct memory stores (STM and LTM).
Demonstrates localization of function: the hippocampus is crucial for forming new long-term memories.
Differentiates between explicit (declarative) and implicit (procedural) memory systems.
Evidence
Aim: Investigate the role of the hippocampus in memory formation.
Method:
Longitudinal case study (50+ years).
Method triangulation: IQ tests, interviews, memory tasks, MRI scans.
Findings:
HM had anterograde amnesia: unable to form new explicit long-term memories.
Retained short-term memory and procedural memory (e.g., mirror drawing).
MRI scans (Corkin, 1992 & 2003) revealed hippocampal and medial temporal lobe damage.
Application
Demonstrated memory is not unitary: different brain systems for different types of memory.
Informed treatments for amnesia and neurodegenerative conditions like Alzheimer’s.
Encouraged use of neuroimaging techniques (e.g., MRI) in cognitive neuroscience.
Criticism
Low generalizability: Findings based on one individual.
Retrospective limitations: Lack of pre-surgical baseline data.
Confounding variable: Anti-epileptic medication may have influenced cognitive function.
Unanswered Questions
To what extent can neuroplasticity allow compensation by other brain areas?
How are semantic and episodic memory differently affected by hippocampal damage?
Practical Use
Influenced neurosurgical practices: highlighted risks of removing medial temporal structures.
Supported rehabilitation strategies: focused on preserved memory systems (e.g., implicit learning).
Counterarguments
Clive Wearing: Similar case reinforces HM’s findings—damage to hippocampus leads to profound amnesia.
Alternative models:
Working Memory Model (Baddeley & Hitch, 1974) suggests a more dynamic view of memory than the linear Multi-Store Model.
Conclusion
HM’s case provides strong empirical support for the role of the hippocampus in memory consolidation.
Despite methodological limitations, it remains highly influential in both cognitive and biological psychology.
Future research should explore interactions between memory systems and intervention strategies for memory disorders.