Video Flashcards: Psychology Concepts (Heredity, Environment, and Brain)
Module 1.1 Interaction of Heredity & Environment
Psychology: Scientific study of the human mind and its functions.
Nature vs. nurture debate: Nature = biological/genetic factors (e.g., eye color, hair color); Nurture = environmental influences.
Darwin's Origin of Species ideas related to psychology:
Evolutionary psychology: how human thoughts and behaviors developed to help survive.
Natural selection: traits that aid survival are more likely to be passed down.
Behavior genetics: genetic and environmental influences on behavior.
How evolutionary psychologists explain behavior tendencies using natural selection:
Behaviors that enhance survival are more likely to be inherited.
Adaptation: a species becomes fitted to its environment via natural selection.
Mutation: errors in gene replication that lead to change.
How behavior genetics explains individual differences:
Result of complex interactions between genes and environment.
Key terms:
Heredity: genetic transfer of characteristics from parents to offspring.
Genome: instructions for making an organism.
Genes: DNA that stores and passes genetic information between generations.
Twin and adoption studies: used to disentangle genetic and environmental contributions; suggest that all human behaviors reflect influences of physical and psychological predispositions that helped ancestors survive.
Monozygotic vs. dizygotic twins:
Monozygotic (identical): same egg; splits.
Dizygotic (fraternal): different eggs; regular siblings.
How heredity and environment work together:
Genes influence traits, but the environment can trigger gene expression.
Epigenetics: environment influences how genes are expressed without changing the DNA sequence.
Module 12 Overview of the Nervous System
Nervous system: body’s network of cells and nerves that carry messages to and from the brain.
Central nervous system (CNS): processes sensory information and controls higher functions.
Peripheral nervous system (PNS): connects CNS to the rest of the body.
Autonomic nervous system (ANS): division that controls involuntary functions (e.g., heart rate, digestion, respiration).
Somatic nervous system: division of the PNS that controls skeletal muscles and transmits sensory information to the CNS.
Afferent neurons: sensory neurons that transmit information toward the CNS.
Efferent neurons: motor commands from the CNS to muscles/glands.
Interneurons: connect afferent and efferent neurons; act as intermediaries.
Reflexes: involuntary, rapid, automatic responses that do not require conscious thought.
Sympathetic nervous system: prepares the body for stressful or emergency situations (fight or flight).
Parasympathetic nervous system: promotes rest and digest; conserves energy.
Module 1.1 ongoing: Key terms relating to genes and environment
Epigenetics: environment influences how genes are expressed without changing the DNA sequence; environment can up-regulate or down-regulate gene expression.
Module 13a The Neuron & Neural Firing: Neural Communication & the Endocrine System
Neuron: a fundamental unit of the nervous system; a specialized cell that transmits electrical and chemical signals throughout the body.
Structure of a neuron (labeling and function):
Dendrite: receive signals from other neurons; transmit toward the cell body.
Soma (cell body): contains nucleus; integrates incoming signals and generates nerve impulses.
Axon: carries nerve impulses away from the cell body to other neurons, muscles, and glands.
Axon terminals / Terminal buttons: release neurotransmitters into the synaptic cleft.
Myelin sheath: fatty layer around the axon; speeds transmission.
Nodes of Ranvier: gaps in the myelin where impulses jump to speed conduction.
Schwann cells: produce the myelin sheath in the peripheral nervous system.
Glial cells: support neurons; provide nourishment, insulation, and waste removal; contribute to various other functions.
How information passes through the neuron:
Through electrochemical processes involving the action potential and neurotransmitters.
Key aspects of neural transmission:
Threshold: level of stimulus required to trigger an action potential.
Action potential: the neural impulse that travels along the axon.
Resting potential: neuron at rest maintains a negative charge inside relative to the outside.
Refractory period: brief period after an action potential during which the neuron is less responsive to stimuli.
All-or-none response: an action potential either fires at full strength or not at all, regardless of stimulus strength above threshold.
Neurotransmitters: chemical messengers released from the presynaptic neuron into the synaptic cleft; bind to receptors on the postsynaptic neuron to transmit the signal.
Synapse / synaptic cleft: junction between two neurons where communication occurs.
Reuptake: neurotransmitters are reabsorbed by the presynaptic neuron, helping terminate the signal.
Excitatory neurotransmitters: increase the likelihood that the postsynaptic neuron will fire an action potential.
Inhibitory neurotransmitters: decrease the likelihood that the postsynaptic neuron will fire an action potential.
Neurotransmitters (summary chart):
Acetylcholine (ACh): learning, memory, muscle contraction, attention.
Dopamine: reward/motivation; motor control; implicated in Parkinson's disease; schizophrenia.
Serotonin: mood regulation, sleep, appetite, digestion, memory; linked to depression, anxiety, OCD.
Norepinephrine: fight-or-flight response; arousal; anxiety; ADHD.
Glutamate: major excitatory neurotransmitter; involved in learning and memory; excitotoxicity if in excess.
GABA: major inhibitory neurotransmitter; reduces neuronal excitability; anxiety reduction; memory effects.
Endorphins: natural pain relief; euphoria; stress reduction.
Substance P: signals for pain.
Note: Malfunctions of these neurotransmitters can contribute to various disorders (e.g., Alzheimer’s, Parkinson’s, depression, anxiety, schizophrenia, ADHD, etc.).
Module 13b The Neuron & Neural Firing: Substance Use Disorders & Psychoactive Drugs
How drugs and other chemicals alter neurotransmitters:
By mimicking or enhancing neurotransmitter effects or by blocking neurotransmitter effects.
Agonists vs. antagonists:
Agonists: bind to receptors and activate them, mimicking natural ligands (e.g., morphine).
Antagonists: bind to receptors but do not activate them; block the action of agonists or natural ligands (e.g., naloxone).
Endocrine-nervous system interaction:
The endocrine system transmits information and interacts with the nervous system through a complex interplay of hormones and neural signals.
Endocrine system chart (hormones, release sites, functions):
Adrenaline (epinephrine): released by adrenal glands; increases heart rate, blood pressure, energy supply; “fight or flight.”
Cortisol: released by adrenal glands; regulates metabolism; involved in stress response.
Oxytocin: release site: posterior pituitary; promotes social bonding; roles in reproduction.
Melatonin (pineal gland): regulates sleep-wake cycles.
Other endocrine components include the pituitary gland and hypothalamus as regulators of hormone release.
Psychoactive drugs and their impact:
Substances that alter brain function and result in changes in mood and cognition.
Substance use disorder: a complex disorder characterized by a maladaptive pattern of substance use leading to clinically significant impairment or distress.
Chart of psychoactive drugs (categories and examples):
Depressants: e.g., alcohol, barbiturates; slow brain activity; can impair judgment and coordination; physical and psychological dependence.
Stimulants: e.g., cocaine, caffeine, amphetamines, MDMA (ecstasy); increase alertness and energy; can cause increased heart rate, anxiety, addiction.
Hallucinogens: e.g., LSD, psilocybin (magic mushrooms), mescaline; alter perception and mood; can cause hallucinations and unpredictable effects.
Marijuana: sometimes categorized as a depressant or hallucinogen depending on effects; various subjective effects.
Additional terms:
Near-death experience: altered state of consciousness reported after close brush with death.
Tolerance: body becomes used to a drug; larger doses needed for the same effect.
Withdrawal: unpleasant physical and psychological symptoms when stopping drug use.
When does drug use become addiction?
Addiction is when drug use becomes dependent and individuals continue using despite harmful consequences and show cravings, tolerance, and withdrawal.
Potential consequences of excessive/prolonged drug use:
Physical and mental health problems; addiction and dependence; relationship, financial, and legal problems; social consequences.
Module 14a The Brain: Neuroplasticity & Tools of Discovery
Biological psychologists focus on the relationship between brain, behavior, and mental processes.
Biopsychosocial approach: health and illness result from the interplay of biological, psychological, and social factors.
How biology and experience influence the brain:
Neurogenesis: generation of new neurons in the brain.
Neuroplasticity: brain’s ability to change its structure and function in response to experience.
Brain imaging and measurement tools:
Electroencephalograph (EEG): measures brain electrical activity via scalp electrodes.
Magnetoencephalography (MEG): measures magnetic fields produced by neural activity.
Positron Emission Tomography (PET): uses radioactive tracers to visualize brain metabolism.
Magnetic Resonance Imaging (MRI): uses magnetic fields to produce detailed anatomical images.
Functional MRI (fMRI): measures brain activity by detecting blood flow changes.
Computed Tomography (CT): uses X-rays to create cross-sectional images of the brain.
Module 1.4b The Brain: Brain Regions & Structures
Explain how the brain has evolved over time:
Hindbrain: essential for vital functions (breathing, heart rate, balance); reflexes; survival.
Midbrain: processes sensory information and controls movement; a step up in complexity.
Forebrain: largest part; supports thought, language, memory, voluntary action.
(continued) Brain imaging modalities and brain function mapping (brief reference): EEG, MEG, PET, MRI, fMRI, CT (see above).
Location, purpose/function, and effects of damage for each brain structure (overview):
Brainstem: vital automatic functions (breathing, heart rate, blood pressure); damage can be life-threatening.
Thalamus: relays sensory and motor signals to cerebral cortex; damage affects consciousness and sensory/motor integration.
Medulla: controls vital functions like breathing and heart rate; damage can be fatal.
Pons: relays signals between cerebrum and cerebellum; involved in sleep and arousal; damage can cause sleep disturbances and cranial nerve issues.
Reticular formation: regulates sleep-wake cycles, consciousness, and arousal.
Cerebrum / Cerebral cortex: higher-order cognitive functions; motor and sensory processing; language; emotion.
Hypothalamus: regulates eating, thirst, body temperature; controls autonomic and endocrine functions.
Amygdala: processes emotions (fear, aggression); involved in emotional memory.
Hippocampus: memory formation and spatial navigation.
Corpus callosum: connects left and right hemispheres; its integrity supports interhemispheric communication.
Pituitary gland: master endocrine gland; secretes hormones that regulate other glands.
Pineal gland: secretes melatonin; regulates sleep-wake cycles.
Regions of the cerebral cortex (drawn/labelled):
Frontal lobes: Prefrontal cortex (personality, planning, decision making);
Motor cortex: controls voluntary movements;
Broca’s area: speech production and language processing (dominant hemisphere);
Parietal lobes: Somatosensory cortex; processes touch, temperature, body awareness; angular gyrus involved in written language and number processing;
Temporal lobes: Auditory cortex; Wernicke’s area for language comprehension.
Occipital lobes: Visual cortex; processes visual information.
Association areas: regions not directly involved in primary sensory or motor functions; integrate information across modalities (e.g., planning, interpretation).
Phineas Gage case: famous neuropsychology case showing frontal lobe involvement in personality and behavior; rod injury led to personality changes, illustrating the frontal lobe's role in executive function and social behavior.
Split-brain: a procedure where the corpus callosum is severed to treat severe epilepsy; reduces cross-hemispheric communication and has been used to study hemispheric specialization.
Module 1.4c The Brain: Damage Response & Brain Hemispheres
Brief reference: the split-brain procedure demonstrates how the two hemispheres can operate somewhat independently when their connections are severed; lateralization of language, spatial processing, and other functions.