Complexity
Can cause distress/impairment in many areas of daily functioning
Chronic, relatively intense, associated with role impairment, cause stress on self or others
Prone to perceiving threat
concerned/worried when there is no threat/situation is ambiguous
Causes
Biological
Genetics: tense, uptight, anxious - inherited, some predisposed to experience anxiety/panic more than others
neuroanatomy/neurotransmitters: CRF system; limbic system, behavioural inhibition system, fight/flight system, GABA/serotonin etc.
Psychological
Behaviour factors: anxiety learned through classical conditioning, operant condition, modeling
Cognitive factors: Beck’s Triad
Interpersonal factors: parenting style/attachment, sense of control/uncontrollability
Anxiety sensitivity
Social
biological/psychological predispositions for anxiety/panic triggered by stressful events (stress, trauma, environment etc.)
Integrated approach (triple vulnerability)
Combines biological, psychological, sociological factors explain development of anxiety disorders
Summary
Generalized anxiety disorder: uncontrollable worrying about minor things
Panic disorders/agoraphobia: unexpected recurrent panic attacks with physiological symptoms (dizziness, rapid heart etc.)
Social anxiety disorder: irrational fear/avoidance of social situations
Specific phobia: fear/avoidance of objects that are not real dangers
Separation anxiety disorder: unrealistic worry something will happen to important people when they leave
Generalized anxiety disorder
intense/unfocused worry about minor events
Feeling a catastrophe after event is over
Cannot stop worry-anxiety style
Ongoing more days/period of at least six months
Symptoms: muscle tensions, irritability, sleep disturbance, fatigue
Causes
Biological
Runs in family
Kendler (1992) found risk of GAD is greater for identical female twins than fraternal female twins
Heritability of anxiety sensitivity (AS) rather than GAD
AS: fear of anxiety symptoms (dizziness, shakiness) due to belief that these are signs of harmful outcomes
Psychological
Dugas and Ladouceur (1998) developed model containing 4 distinct cognitive characteristics of GAD:
Intolerance of uncertainty
Positive beliefs about worry
Poor problem orientation
Cognitive avoidance
Treatment for anxiety attacks
Drugs: benzodiazepines for short term, antidepressants/antipsychotics
Psychological
CBT
Patients confront worry-evoking images/thoughts; goal is for patient to obtain coping mechanisms
Training in relaxation skills
learning to substitute calming/adaptive thoughts for intrusive ones
Learning skills of de-catastrophizing
Mindfulness-based therapy
Panic attacks
Panic: abrupt experiencer of fear/acute discomfort-associated with sympathetic nervous system
Two types:
cued/expected-always occurring in specific situation
uncued/unexpected-without warning
Panic disorder: repeated episodes of unexpected attacks
Agoraphobia
Intense fear/avoidance of situations that may be difficult to escape
Agora derives from Greek word “fear of the marketplace”
Interoceptive avoidance: removing self from activities that produce physiological arousal-similar to panic attacks
Marked fear/anxiety for ⅖ of the following
Using public transport
Being in open spaces
Being in enclosed spaces
Standing in line/crowd
Being outside of home alone
Panic disorder causes
Biological
Some are genetically predisposed
Psychological
Conditioning Theory of Panic Attacks
Attacks = classical conditioning response
Treatment for panic attacks
Medication
Prozac and Paxil (SSRIs)
60% patients are free of panic if drug use continues
Relapse rates high post-medication (50%)
Psychological intervention
Panic control treatment (PCT)
Form of CBT - patients exposed to cluster of bodily sensations
Cog therapy/relax techniques are practiced
Integrated approach
More effective to use sequential strategy than combining treatments
Phobic disorders
Phobia: irrational/exaggerated fear of specific object that is inconsistent to real dangers/threats
Specific phobias
Irritations fear of particular object/situation
Phobic item usually provokes fear/anxiety
Impact of specific phobia depends on person's ability to avoid
Prevalence varies from one culture to another
Specific types of phobias
Forming phobia name comes from Greek word phobia meaning “fear of”
5 major subtypes
Blood-injury-injection ie. needles
Animal ie. spiders
Natural environment ie. thunderstorms
Situational ie. flying
other types ie. vomiting
Phobia causes
Two-factor model of phobia development based on classical/operant conditioning
Develops from classical: neutral objects/situations gain ability to evoke fear-pairs with noxious/aversive stimuli
Person learns to reduce conditions fear by avoiding CS (operant)
Negative reinforcement
Observational learning - modeling and receiving negative info
Prepared learning (non-associative model) - some fears reflect classical conditioning but only stimuli where organism is physiologically prepared for sensitivity
Treating Phobias (learning/exposure based)
Gradual exposure: patient exposed gradually to increase fearful stimuli
Systematic desensitization: similar to gradual, patient exposed to more progressively fearful stimuli and being taught relaxation techniques
Modeling therapy: patient exposed to others filmed/lived demonstrations of others interacting
Virtual reality therapy: exposed to computer-simulated fear situation, can be gradual
Social anxiety disorder (social phobia)
Is excessive fear/anxiety of social situations due to possibility of being scrutinized by others
Individual fears acting in away that causes negative reactions from peers
Thoughts of being judged/embarrassed/humiliated/rejected
Social anxiety is a mental health disorder, shyness is a personality trait
Causes of social anxiety
Biological
Genetic heritability
Behavioural inhibition
People with SAD react to angry faces-activation of amygdala
Neurotransmitters
Psychological
Conditioning theory of panic attacks can be applied to explain SAD
Treating social anxiety
Drug therapies
Antidepressants (SSRIs) - Paxil and Zoloft
Some suggest combining psychological and drug treatments
Cognitive behavioural group therapy (CBGT)
Involves group role play of feared situation
Social mishap exposures
Patients encouraged to confront social phobic situations
Patients try to become aware of irrational thoughts
Trauma-Related and OCD-week 9
Acute stress disorder
Traumatic event - person exposed to actual/threatened death, serious injury, sexual violence
Most who encounter trauma experience stress-this is normal
If stress causes impairment to social functioning and lasts for less than a month, Acute Stress Disorder is diagnosed
Longer than a month is PTSD diagnosed
Symptoms start shortly after, or a month-year
Nearly half acute stress disorder cases become PTSD
Post-Traumatic Stress Disorder (PTSD)
Extreme response to stress-increases anxiety, stimuli avoidance, numbing of emotional responses
Fear of re-experiencing
nightmares/flashbacks
Avoidance of feelings of event
Often experienced by first responders
Chronic or delayed-onset
Clusters of symptoms in ASD and PTSD
Re-experiencing
Avoidance of stimuli
Reduction of responsiveness
Symptoms increase arousal
PTSD causes/risks
Direct exposure; intensity; severity
Gender (more in females)
Perceived life threat
Family history
Dissociative symptoms
Coping with stress-focus on emotions
Anxiety sensitivity
Causal theories
Psychological-learning theories
PTSD comes from classical conditioning
Avoidance builds up
Cognitive-biological theories
Constant involuntary recollection of event
Abnormal activity of cortisol and norepinephrine
Constant biochemical arousal can damage key brain areas (hippocampus, amygdala)
Social and cultural factors
No to little support system
Coping skills
PTSD treatments
Exposure therapy - prolonged exposure
Combined CBT: involved step-by-step process of being exposed to imagery reflecting trauma
Exposure accompanied by changing thoughts
Virtual exposure therapy
MDMA
OCD/related disorders
Characterized by patterns of repetitive behaviours associated with significant personal distress
Disorders include
Obsessive-compulsive
Body dysmorphic
Hoarding
Trichotillomania (hair pulling)
Excoriation (skin picking)
OCD
Uncontrollable thoughts to repeat certain acts over and over
Obsessions
Persistent thoughts/ideas that invade consciousness
Fears contamination; expressing sexual impulse; hypochondriac fears of bodily dysfunction
Compulsions
Repetitive behaviour a person feels need to perform
Often occurs in response to obsessive thoughts, frequent, interfere with daily life
Two categories: checking rituals & cleaning rituals
OCD causes
Biological: low levels of serotonin activity; brain regions (orbitofrontal cortex, caudate nuclei) these convert sensory info to thoughts and actions
Cognitive: exaggerate risk of unfortunate events to prevent them; comes from trying too hard to control thinking
Treatment - exposure and response (ritual) preventing (ERP)
Therapists achieve results in treating OCD with ERP
Exposure involves patients placing themselves in situations that start compulsive rituals
People with OCD learn to tolerate anxiety triggered of obsessive thoughts while prevents from performing rituals
Obsessive-compulsive-related disorders
Body dysmorphic
Imagined physical defect in appearance causing some to feel ugly/disfigure
Become obsessed with perceived defect/engage in fixing
Co-occurs with OCD
Exposure therapy
Hoarding disorder
Need to retain useless possessions causing distress or making it difficult to maintain safe living space
Starts early and gets worse
CBT often given
Trichotillomania (hair-pulling)
Has severe social consequences
1-5% college students-more so females
Excoriation (skin-picking)
1-5% of general population
Scabs, scars, open wounds
Treatment: habit reversal training
schizophrenia-week 11
Other psychotic disorders
Schizophreniform: people experiencing symptoms of schizophrenia for less than 6 months
Schizoaffective: people who have schizophrenia symptoms/characteristics of mood disorders
Delusional disorder: people with persistent belief contrary to reality without other schizo characteristics
Brief psychotic disorder: people with 1 or more positive symptoms (delusions, hallucinations etc.) in less than a month
Attenuated psychosis syndrome: one or more schizo symptoms but person is aware
Schizotypal personality: characteristics similar to those who experience schizo but less severe
Schizophrenia
Psychotic disorder with major disturbances of thought, emotion, behaviour
Characterized by: disordered thinking-not logically related, faulty perception/attention, flat/inappropriate affect, bizarre disturbances in motor activity
Prevalence and comorbidity
In canada: 1% general population lives with schizophrenia
Half treated in community
10% commit suicide
Mortality rate 3x higher than canadians w/o schizo
50% suffer from comorbid disorder
Substance abuse (37%)
Depression (40%)
Higher in Canada, worldwide prevalence levels declining
Most given to immigration rates/high latitude
Greater prevalence found:
Among those who immigrate
Countries with high latitudes with less sunlight and reduced vit. D
Course of schizophrenia
Sometimes begins childhood; appears late adolescence or early childhood
Have number of acute episodes of symptoms
Often less severe but debilitating symptoms between episodes
Most treated in community; hospitalization sometimes necessary
Common phases:
Prodromal - period of gradual deterioration
Acute - characterized by delusions, hallucinations, illogical thinking
Residual - follows acute phase; characterized by return to prodromal level
Children with Schizophrenia-week 11
Clinical description
Schizophrenia spectrum disorders: recognized by those in field of schizophrenia
DSM-5-TR includes dimensional assessment of symptoms on 0-4 scale
0 = no symptoms
1 = equivocal evidence
2 = present but mild
3 = present and moderate
4 = present and severe
Symptoms
Positive symptoms: active presentation of problematic symptoms
excesses/bizarre distortions to one's thoughts, emotions, behaviours
60-80% experience hallucinations; 70% experience delusions
Negative symptoms: absence/insufficiency of behaviours
Alogia: negative thought disorder; absence of speech; poverty of speech
Avolition: inability to initiate activities; apathy; loss of motivation; ambivalence
blunted/flat affect: show less outward emotions (blunted); show almost no outward emotions (flat); immobile/expressionless
Anhedonia: inability to experience pleasure
Asociality: lack of interest in socializing; social withdrawal; attend to own ideas; breakdown of social skills
Disorganized symptoms - rare
Inappropriate affect: emotional responses out of context; shift rapidly from one emotion to another for no reason
Disorganized behaviour: awkward movements; repealed grimaces; odd gestures
Catatonia: rigidity; posturing; excitement
Delusions
Beliefs held but not contrary to reality
Common delusions
Persecution
Reference
Grandeur
Control
Cotard’s syndrome
Capgras syndrome
Found in more than half with schizo
Hallucinations and heightened perceptions
Perception intensifies
Can develop years before actual onset
Hallucinations
Sensory perception absent of stimulation from environment
Distortions
Types: auditory; visual; tactile (sensations not happening); somatic (things in body); gustatory (tasting things); olfactory (sensing odors)
Attentional deficiencies
Screening out background noises/other stimuli
Attention: ability to focus on relevant stimuli/ignore irrelevant
With schizo people have difficulty filtering out irrelevant stimuli - makes it hard to focus attention, organize thoughts, filter out unimportant info
Etiology of Schizophrenia-week 10
Biological explanations: Genetic
Some inherit; disposition triggered by exposure to extreme stress
Family studies: increased risk of genetic relationship becoming closer; risk of other disorders
Twin studies: concordance for MZ twins (48%), DZ twins (17%)
Adoption studies: children without contact of schizophrenic mothers still likely to develop disorder; good environment reduces risk
Genetic linkage studies: regions on numerous chromosomes
Biological explanations: Biochemical
Dopamine hypothesis
Originally assumed its caused by excess dopamine
Developed from accidental discovery of antipsychotic drugs
Provided by amphetamines and development of amphetamine psychosis - causes release of norepinephrine/dopamine
Newer assumptions: excess/oversensitive dopamine receptors over high level of dopamine play a role
Excessive stimulation of striatal dopamine receptors
Prefrontal dopamine receptor deficiency
Prefrontal activity from glutamate transmission
Biological explanations: Brain Structure
Brain scans show abnormalities in physical structures and brain functioning
Enlarged ventricles found in majority
Frontal lobe abnormalities - hypofrontality
Abnormal functioning/lose of brain tissue in prefrontal cortex
Abnormalities in brain circuitry - connects to prefrontal cortex/lower brain structures
Causes: genetics, poor nutrition, fetal development, immune reactions etc.
Social-cultural explanations
Stress: lead to predisposition
Social economical status: increases rates; found in central city areas inhibited in lowest SES
Socio-genic hypothesis - stress associated with low social class
Social-selection theory - reverses casualty direction between social class and schizo
Family functioning: some high in expressed emotion (EE), increasing relapse risk
Treatment of schizophrenia
APA multi-point treatment course
Antipsychotic medication controls acute psychotic symptoms
Treatment of comorbid disorders include substance abuse and depressive disorders
Use of psychosocial treatment
Biological treatments - drug therapies: First Gen - typical
helps control flagrant patterns (keeps positive symptoms from coming back)
Blocks dopamine receptors
Antipsychotics: phenothiazines, chlorpromazine, thioridazine, trifluoperazine, fluphenazine
Side effects: dizziness, blurred vision, restlessness
Extrapyramidal side effects: nerve tracts dysfunctions descend from brain to spinal motor neurons
Tremors of fingers
Drooling
Dystonia: muscular rigidity
Dyskinesia: abnormal motion of voluntary/involuntary muscles
Akathisia: inability to remain still
Akinesia: expressionless face, slow motor activity
Tardive dyskinesia
Mouth muscles involuntarily make sucking, lip-smacking, chin-wagging motions
Whole body can have involuntary motor movements
Affects 20-50% patients using antipsychotics for long period/not responsive
Biological treatments - drug therapies: Second Gen - atypical
Largely replace earlier gen
Advantage: carries less neurological side effects/lower risk of Tardive dyskinesia
Positive and negative symptoms
Includes: clozapine, risperidone, olanzapine, quetiapine, ziprasidone, aripiprazole
Impact of Schizophrenia on Life
delusions/hallucinations cause distress
Cognitive impairment and avolition makes employment hard
Strange behaviour and social skills can cause loss of friends
High substance abuse rates
Suicide rate is high
Homelessness
Stigmatization
Personality Disorders-week 11
Personality: unique expressed characteristics influencing behaviour, emotion, thought, and interpretation
Personality disorders: persistent patterns of emotions, cognitions, and behaviour resulting in emotional distress
Overly rigid/maladaptive behaviours reflecting variations of personality traits; undue suspiciousness, excessive emotionality, impulsivity
Seen in adolescence or early adulthood
Warning signs in childhood
Ego syntonic: feelings perceived as natural/compatible - most PDs!!!
Ego dystonic: feelings perceived foreign to self-identity
Categories or dimensions?
Categorical
Problematic personality are present or absent
Personality disorder either displayed or not displayed
Person suffering from PD is not troubled by personality traits outside disorder
Issues distinguishing PD from other clinical symptoms
Overlapping disorders
Difficulty distinguishing between normal and abnormal behaviour
Labels confused with explanations
Advantages of dimensional model
Some differ from degree than kind
Advantages:
Retains more info
More flexible due to categorical/dimensional differentiation
Avoids arbitrary decision assigning a person to a category
Model considered focuses on continuum of: self (how a person views themself) and interpersonal functioning (intimacy abilities)
Some suggest Big 5 model
Clusters of personality disorders
Cluster A: characterized by odd behaviour (ie. paranoid, schizoid, schizotypal)
Cluster B: characterized by dramatic, emotional, and erratic behaviour (ie. anti-social, borderline, histrionic, narcissistic)
Cluster C: characterized by anxious behaviour (ie. avoidant, dependent, obsessive-compulsive)
Cluster A: Paranoid Personality Disorder (PPD)
Characteristics: suspicious, expectation of mistreatment, won’t confide in others, tendency to blame others, extreme jealousy
Differential diagnosis and comorbidity: hallucinations/full-blown delusions, less impairment in social/occupational functioning, comorbid with schizotypal, avoidant, BPD
Etiology and treatment
Genetics, childhood trauma, schemas, cultural
Cognitive therapy
Cluster A: Schizoid Personality Disorder
Characteristics: no desire for social relationships, appear dull/bland, rarely show emotions, no interest in sex, experience less activities they like, indifferent to praise/criticism, loners
Prevalence and comorbidity:
prevalence <1%
comorbid with schizotypal, avoidant, paranoid disorders
Etiology and treatment
Childhood shyness, abuse, neglect etc.
Social skills training
Cluster A: Schizotypal Personality Disorder
Characteristics: similar interpersonal difficulties, eccentric thinking
Prevalence and comorbidity:
Prevalence <1%
Comorbidity higher than other disorders
Comorbid with borderline, avoidant, paranoid disorders
Etiology and treatment
Genetics, left hemisphere damage
Antipsychotic, medication CBT
Cluster B: BPD
Characteristics: impulsive/instability, feelings towards other vary/swing, erratic, argumentative and sarcastic, cannot tolerate being alone
Prevalence and comorbidity:
Prevalence 1-2%; 20-25% of psychiatric admissions
More common in women
Comorbid with mood disorder, substance abuse, PTSD etc.
Etiology and treatment:
Genetics, related to mood disorders, early trauma
Antipsychotic and antidepressants, dialectical behavioural therapy
Self-mutilation
Engage in impulsive acts like cutting oneself to escape deep/emotional pain
Expression of anger
Intended to counteract feelings of numbness
Cluster B: Histrionic Personality Disorder
Characteristics: overly dramatic/attention-seeking, used physical appearance for attention, emotion displayed extravagantly, self-centered, overly concerned with attractiveness
Prevalence and comorbidity
Prevalence <1%
Common in women
Comorbid with depression and BPD
Etiology and treatment
Sociocultural: co-occurs with antisocial personality disorder
Improve problematic interpersonal relationships
Cluster B: Narcissistic Personality Disorder
Characteristics: grand view of own abilities, preoccupied with fantasies of great success, requires constant attention/admiration, lacks empathy, envious of others, and arrogant/entitled
Prevalence and comorbidity:
Prevalence 1%
Comorbid with BPD
Etiology and treatment:
Failure of empathy
Coping strategies, CBT
Cluster B: Antisocial Personality Disorder
Two main components
Conduct disorder before age of 15
Pattern of antisocial behaviour continuity into adulthood
Characteristics: violation of social norms, lack of long-term goals, impulsive, violent, lacks empathy
Prevalence and comorbidity
Prevalence 1.7-3.7%; 50% in prison
More common in men
Comorbid with substance abuse
Etiology and treatment
Gene-environment, under-arousal of cortex
Multifaceted, CBT, preventing best approach
Psychopathy
Related to APD but emphasizes thoughts/feelings
Lacks remorse, charming, manipulative, thrill seeking; impulsive, arrogant, deficient, early onset
Many psychopaths can have APD but many with don’t meet criteria
Criminality
Tend to think APD as synonymous with criminals
Not all criminals have antisocial disorders/not all with it become criminals
Many are law abiding
Some criminals do not meet the criteria
Cluster C: Avoidant Personality Disorder
Characteristics: fear of social situations, sensitive to criticism/rejection, reluctant to enter relationship
Prevalence and comorbidity
Prevalence 1%
Comorbid with dependent personality disorder, depression, generalized social phobia
Etiology and treatment
Integrated biological/psychosocial influences
Born with difficult temperament, rejection etc.
Links to social anxiety
CBT, systematic desensitization, anxiety meds
Cluster C: Dependent Personality Disorder
Characteristics: lacks self-reliance, overly dependent, intense care needs, uncomfortable alone
Prevalence and comorbidity:
Comorbid with bipolar disorder, depression, anxiety, bulimia
Etiology and treatment:
Genetic; disruptions in early childhood
Develop confidence
Cluster C: OCPD
Characteristics: perfectionist, preoccupied with details/rules etc., serious and rigid, can’t discard useless items
Differential diagnosis
Obsessive compulsive personality disorder (OCPD) differs from OCD
Prevalence and comorbidity:
Prevalence 2% of population
Comorbid with OCD (20%), panic disorder, depression, GAD, avoidant personality disorder
Etiology and treatment
Genetics
Relaxation techniques; CBT
Sexual Dysfunctions and Paraphilic Disorders-week 12
Sexual dysfunctions
Persistent problems with sexual interest, arousal, or response
Affects 40-45% of adult women
Affects 20-30% of adult men
Lifetime disorder vs acquired disorder
Situational disorder vs generalized disorder
Categories of sexual dysfunctions
Sexual desire disorders
Male hypoactive
Absent sexual urges
Little-no interest
20-30% general adult pop.
Female sexual interest/arousal
Low sex drive to become aroused
20% prevalence
In both desire and arousal phases
Sexual arousal disorders
Male erectile disorder
Problems becoming aroused
Have sexual urges
7-70% prevalence
Age dependent - common in aging populations
Orgasmic disorders
Female disorder
Absence of orgasm after normal sexual excitement
16-46% prevalence
10% women never experience an orgasm
Male disorder
Delayed ejaculation: infrequency, 2-8% prevalence
Premature ejaculation: recurrent pattern with minimal sex drive, affects 40% of men
Sexual pain disorders
Genito-pelvic pain/penetration disorder
Vaginismus: involuntary spasm of outer third of vagina that makes sex harder, normal arousal possible with oral/manual stimulation
Dyspareunia (genito-pelvic pain): recurrent pain during sex, associated with low sexual desire
Assessing sexual behaviour
Interviews
Talking to clinician about issues
Can include questionnaire
Medical exam
Helps rule out medical cause
Psychophysiological
Assesses ability to become aroused
Ie. penile strain gauge, vaginal photoplethysmography (VPG)
Biological explanations
Hormone imbalances
Deficient testosterone/estrogen production and over/underactivity of thyroid
Medical conditions
Temporal physical conditions, nerve damage, diabetes, lung disorder etc.
Medications
Antihypertensive meds, antiandrogens, antidepressants, anti anxiety meds
Illicit drugs
Psychological explanations
Cognitive interference
Irrational beliefs and attitudes
Performance anxiety: excessive concern about performance abilities
Mental health
Depression, anxiety, stress
Poor body image
Traumatic sexual experiences
Communication and relationship issues
marital/relationship issues
Resentments and conflicts
Sociocultural explanations
Sexual scripts theory
Guidelines for appropriate behaviours
Gendered
Men expected to be enthusiastic more so than women
Stereotypes that sexual pleasure is mostly for men
Sociocultural beliefs ie. religion
Sexual taboos ie. paraphilia
Early traumatic sexual events
Current life/relationship difficulties
Psychosocial treatments
Human sexual inadequacy
Anxiety reduction
Directed masturbation
Change attitudes
Skill and communication training
Couples therapy
Sexual dysfunction treatments
Medication: testosterone gel patch, sildenafil (viagra), birth control, vasodilator injection, SSRIs
Physical: treat underlying physical issues (ie. STI, UTI)
Surgery: penile prosthesis/vacuum devices, vaginal dilators, unblocking of blood vessels
Paraphilic disorders
Para: deviance; philia: attraction = bad attraction
Conditions involving sexual attraction to atypical objects/unusual sexual behaviours
Involve urges, preferred way of seeking gratification
At least 6 months
Must cause significant distress or behaviour places person/others at harm
Paraphilia vs paraphilic disorder
4 distinguishing features
uncommon /atypical from statistical and sociocultural perspectives
Intense interest in sexual activities that do not involve mature, consenting, or human partners
Recurrent and persistent for 6 months
Sexual activity is persons preferred/primary way of obtaining gratification
More common in men 3-5% than woman 1-6%
High comorbidity
Some referred to as Courtship Disorders (ie. Voyeurism, Exhibitionism, and Frotteurism)
Paraphilic disorders in DSM-5
Sexual sadism
Inflicting pain/humiliation
Practiced with parties (BDSM)
Coercive paraphilic disorder
Sexual masochism
Arousal from experiencing pain/humiliation
Common in women
Infibulation: harming one’s body for sexual pleasure
Transvestism
Arousal from cross-dressing
Must be for sexual pleasure
Fetishism
Excitement in response to non-sexual object
May include body parts (feet), objects (shoes), body features (tattoos), materials (latex), costumes (furries), or stimulation
Exhibitionism
Arousal from exposing self/performing sexual acts for those not consenting
Flasher vs stripper
Voyeurism
Arousal from non-consenting watching of others naked/engaed in sexual acts
Peeping Tom vs. porn
Most common
Frotteurism
Arousal from rubbing/touching one’s private parts against those not consenting
Common within males
Pedophilia
Attraction to prepubescent children
physical/emotional
Pedophilia vs hebephilai (no longer in DSM)
Not an act but a deviant sexual orientation
Not everyone who offends a child is a pedo
Pedophiles vs child molesters
Many never act on attraction
High suicide rates
Other paraphilic disorders
Somnophilia: sexual urge to have sex with someone who is sleeping
Necrophilia: sexual urge to have sex with a dead body
Zoophilia: sexual attraction to animals (bestiality)
Telephone scatologia: sexual urge to make obscene phone calls
Urophilia: sexual excitement of urine
Coprophilia: sexual excitement from feces
Causes of paraphilic disorders
behaviour/cognitive perspectives
Multifactorial causes: childhood sexual abuse, disturbed family, insecure attachments
Inadequate social skills
Early sexual experiences
Learning theories: classical/operant conditioning; modeling
biological/neurological perspectives
Possible role of Androgen (male hormone-principle)
High sex drive - hypersexual
Hormonal disturbances in fetal development
Altered brain activity within frontal/temporal areas
Diagnosing paraphilic disorders
DSM-5 differentiates harmless and harm
DSM-4 paraphilia vs DSM-5 paraphilic disorder
Two prong assessment
For paraphilia
Self-report, indirect measures, physiological measures, vaginal photoplethysmography
distress/non-consenting
Paraphilia treatments
Medication
Antiandrogens
SSRIs
Psychosocial
Focus on changing thoughts/behaviours
Specific techniques to eliminate these behaviours
Aversion therapy
Masturbatory station
Orgasmic reorientation
Relapse prevention
CBT
Stigma can affect those to ask for help
CAMH talking for change
Gender Dysphoria-week 12
Gender identity: ones psychological sense of gender
Gender dysphoria: psychological disorder where people experience personal distress between their assigned birth sex and gender identity
Transgender identity: whose gender identity/expression differs from assigned birth sex, due to social/cultural expectations
Not the same as transvestic disorder
Client patterns of gender
Cisgender: people who identify with assigned birth gender
Female-to-male GD (trans male): assigned as female at birth but identify as male
Male-to-female GD (trans female): assigned male at birth but identify as female
Possible explanation of gender dysphoria
Biological
Genetic: runs in families, concordance found in identical twins
Hormonal: testosterone and estrogen at critical periods of fetal development can genderize the fetus
Structural: brain regions help with human sexuality and consciousness- blood flow and size
Psychological
Cultural
Treatment options
Supports trans individuals who choose hormonally/surgically changes to body shell and matches internal identity
Hormone treatments
Might involve puberty/hormone blockers
Assigned male at birth-given estrogen
Assigned female at birth-given testosterone
Facial/speech surgery
Gender confirming surgery/affirming surgery/sexual reassignment surgery
Male-assigned: partial penis removal; structure into clitoris and vagina
Female- assigned” chest masculinization and hysterectomy; silicone prosthetic penis; phalloplasty