Endocrine System
Selective forces:
Endocrine System is an integrative system that controls an animal’s activities
Animals must coordinate activities
Many responses coordinated through the use of hormones
Chemical coordination
Endocrine glands: small (smaller than other organs), well-vascularized ductless glands
Pituitary, Thyroid, pancreas, adrenals, testes, ovaries
Hormones: a chemical messenger that initiates a physiological response.
Can be local or circulating
Local: working in a very defined, small space
Released in small amounts
Does not enter the circulatory system, never enters into the blood vessels
Effective for minutes
Target cell uses up the small amount of hormone really quickly
Circulating: has to go to some distant location
Released in large amounts
Enters the CVS (cardiovascular system/circulatory)
Effective until removed/used up, can hang around longer
Target cell uses it, but whatever it doesn’t use up gets removed by the kidneys
Target cells: cells affected by the release of a hormone
Has ability to interact with many hormones
Any given cell may have receptors for a wide variety of hormones
Receptor: integral proteins present on the surface of a target cell
“Lock and key” interaction- the specific interaction between a hormone and receptor. The shape has to match in order to bind
Up regulation: adding active receptors to a target cell surface when hormone levels drop. Adds receptors so that the hormone is more likely to bind to ensure that the process takes place
Down regulation: removing active receptors from a target cell surface when hormone levels are high. Decreasing rate of contact to maintain optimal rate of binding (prevent burn out of the cell).
What are the effects of hormones?
Change enzymatic activity/alter cellular metabolism
Like trophic cascades, called tropic cascade
Change membrane permeability.
Any hormone involved in water conservation or secretion
Cause other glands to release their own hormones
Differences between the nervous and endocrine systems
Response time:
NS: can generate immediate change, incredibly rapid signal
ES: hormone must diffuse, time lag, takes longer for change to be instigated
Duration of effect:
NS: lasts as long as the action potential (AP) (nerve impulse)
ES: lasts as long as the hormone is present, could be hours or days
Mechanisms of hormone action:
Chemical Classes of hormones
Water-soluble hormones
Protein based and produced in the RER (hormone is a protein)
Too large or too polar to diffuse through membrane, stuck on the outside where it binds to the receptor
Must bind to transmembrane protein that acts as receptor
Hormone and receptor trigger a cascade of events
Second-messenger concept: the hormone must rely on a second molecule to alter target cell activity
Hormone is the 1st messenger and activates the 2nd messenger system in the cytoplasm
Typically the 2nd messenger is cAMP (cyclic adenosine monophosphate). Once it is activated, there is a cascade effect
Steps: Protein (hormone) travels through the blood vessel, leaves vessel, protein binds to the receptor, activates cAMP, cAMP goes around activation other protein kinases which does other mechanisms (transcription, translation, phosphorylation) which changes activities of the cell
Lipid-soluble hormones
Cholesterol-based and produced in the SER
Must bind to a transport protein to travel through blood, cannot move through the plasma by itself because it is hydrophobic. ex) of transport protein: albumin
Transported protein brings the hormone to the target cell where it binds to the receptor
Single-messenger concept: the hormone is directly involved in altering the target cell’s activity
Hormone diffuses through the membrane and binds to nuclear receptors (to alter transcription, translation, phosphorylation, etc.)
Control of secretion rates
Negative feedback: a physiological change initiates the release of a hormone that causes a change in the opposite direction. Brought back to homeostasis after you deviated. Most common mechanism in living things. Controlling minor fluctuations in homeostasis.
Relies on antagonistic hormones: one works to increase value, other works to bring it back down, work together to maintain the range
Any deviation in a set point is immediately corrected. Any deviation results in disease in that organism.
Autonomic nervous system: signals generated by the ANS either increases or decreases hormone production
Sympathetic: increase production of the hormone
Parasympathetic: decrease production of the hormone
Tropic cascade: the release of a hormone from one gland triggers the release of another hormone from a second gland
ex) hypothalamus
Positive feedback: a physiological change in the body causes the release of hormones that amplifies that change. Going away from homeostasis on purpose
Requires a shutoff mechanism
Potentially dangerous
ex) childbirth (not a lot of other examples): oxytocin targets the uterus to contract, baby’s head hits receptors which causes the pituitary gland to release more oxytocin. Only way to shut off the mechanism is when the baby is actually born. Dangerous if mom loses too much blood
Hypothalamus
Master program, center for homeostasis, knows what is normal for every aspect of the body and is continuously checking to make sure it is in that range
Major integrating center: takes in multiple stimuli and puts it together to make an appropriate response
Contains neurons that receives/interprets sensory info. Called a neuroendocrine organ
Part of the thalamus, right next to pituitary gland
Secretes releasing (prompts the release of something else) or inhibiting (inhibits the activity of that gland) hormones
Pituitary gland
Part of the diencephalon (part of the brain)
Controlled by the hypothalamus (either directly or by the hormones)
Connected to the hypothalamus by the infundibulum (little bridge connecting them)
“Master gland”- it produces a ton of hormones that regulate a lot of different aspects of the anatomy and physiology
Anterior pituitary gland
Largest portion, 75% of the mass of the pituitary gland, produces more hormones than the posterior
Endocrine tissue, very vascular capillary bed
Under tropic control of hypothalamus (releasing and inhibiting hormones target this gland)
Lacks a true connection to the brain (all endocrine tissue)
Large blood supply (so all hormones can easily diffuse into the capillaries and enter the circulatory system)
Posterior Pituitary gland
Under direct control of hypothalamus by ANS
Dominated by nervous tissue, direct connection of neurons from the hypothalamus
Doesn’t make its own hormones, stores the hormones that the hypothalamus makes
Hormones of the anterior pituitary gland (released from pituitary gland but stimulated by hypothalamus)
Thyroid-stimulating hormone (TSH)
Target: thyroid gland
Action: stimulate production of thyroid hormones
TSH and the thyroid hormones has negative feedback
Follicle-stimulating hormone (FSH)
Target: gonads (in both males and females)
Action:
Females: stimulated egg production, causes maturation of oocyte within the follicle
Males: stimulates sperm production, plays a role in spermatogenesis
Luteinizing hormone (LH)
Target: gonads
Action:
Females: stimulates/promotes ovulation, corpus luteum production, and secretion of female sex steroids (estrogen and progesterone). Related to menstrual cycle and early fetal development
Males: stimulates/promotes production of testosterone
Adrenocorticotropic hormone (ACTH)
Target: adrenal cortex
Action: increases production of glucocorticoids (all about carbohydrate metabolism). Plays important role in mobilizing resources when needed (fight or flight)
Growth hormone (GH), aka somatotropin
Target: cartilage, bone, skeletal muscle, liver
Action: stimulates metabolism and cell division directly in the cartilage, bone, and skeletal muscle
Stimulates secretion of insulin-like growth factor (when it targets the liver) which promotes resources being dumped into the bloodstream, like glycogen which breaks down into glucose to be released in the bloodstream which also stimulates metabolism and cell division. Liver stuff fuels the growth of the other things
Prolactin (released of it is from oxytocin initially)
Target: mammary glands
Action: promotes and maintains lactation
In fishes it plays a role in mucus secretion and osmoregulation
In amphibians is plays a role in metamorphosis and development
In birds is plays a role in brooding behavior
Melanocyte-stimulating hormone (MSH)
Target: melanocytes
Action:
Fish, amphibians, nonavian reptiles: promotes dispersion of pigment (darkens skin)
Birds and mammals: physiological function unclear
Hormones of the posterior pituitary gland
Oxytocin
Target: smooth muscle of the uterus (in all verts), mammary glands (mammals)
Action:
Stimulates uterine contractions
Eject milk in response to suckling with prolactin (mammals), but once baby is out of body, prolactin takes over
Antidiuretic Hormone (ADH), aka vasopressin
Target: kidneys (loop of henle and collecting duct)
Action: increase water reabsorption (makes it so that you don’t pee as much)
Concentrated urine/restricted flow
Pineal Gland
Part of the diencephalon (neuroendocrine) in the thalamus
Activity regulated by light levels
In ectothermic verts- acts as photoreceptive sensory organ
Referred to as “third eye” in some (parietal foramen)
Can be homologous to the lens of an eye in some species
Birds and mammals - entirely glandular structure, evolutionarily has changed
Melatonin
Targets: global, targets every single cell in your body (every cell has receptors for it)
Action: regulates sleep cycle
When light levels are high (during the day), there are low levels of melatonin and vice versa
Nonmammalian verts: pineal gland maintains circadian rhythms
Mammals: circadian rhythms regulated by part of the hypothalamus but the pineal gland/melatonin reinforces it. Why we are able to do things like night shifts
Birds and mammals have photoperiod which regulates seasonal rhythms in reproduction because days get shorter in the winter
Thyroid gland
Large endocrine gland in the neck of all verts
Highly vascular
Divided into halves (in most verts) with a small connection point in between them
Tissue composed of sphere-like units (follicles)
Follicles produce the hormones
Triiodothyronine (T3)
Target: global
Action:
Promote growth/development of NS
Regulate metabolism by regulating cell’s ability to use oxygen
Iodine is a very significant structure in thyroid gland
Thyroxine (T4)
Target: global
Action:
Promote growth/development of NS
Regulate metabolism by regulating cell’s ability to use oxygen
Synergist with T3
Thyroid gland contains ½ of the iodine in the body, iodine deficiencies cause metabolism problems
Under secretion of thyroid hormones
Hypothyroidism: slows metabolic activities
In fish, birds, mammals under secretion drastically impairs growth and nervous system development
Malfunction from a very early age causes cretinism in humans (truncated growth)
Weight gain if acquired later in age
Oversecretion of thyroid hormones
Hyperthyroidism: increases metabolic activities
Causes precocious development in all verts, ramping development up, growth and development takes place too quickly, early maturation, goes through puberty too soon
Effect most prominent in frogs and fish
Thyroid hormones and adaptation
Thyroid gland promotes adaptation to cold climates (due to heat production)
Cold adapted animals eat more food in winter than summer
T3 and T4 drop during hibernation (can be 40% drop) to slow metabolism to make sure the food lasts longer.
Calcitonin
Target: osteoclasts (bone carving cells, break down bone)
Action: decreases blood Ca2+
Shuts the osteoclasts down, which activates the osteoblasts (bone building). The osteoblasts grab/remove all the calcium in the bloodstream (decreases calcium levels)
Parathyroid glands
Closely associated with the thyroid gland (may even be within it)
Number of them is highly variable (humans have 2, some reptiles have 1, birds have 4, 2 is most common though)
In birds and mammals- removal of gland rapidly decreases blood calcium
Parathyroid Hormone (PTH)
Fish are the only verts that do not secrete PTH
Target: osteoclasts
Action: increases blood Ca2+ levels (opposite of calcitonin)
Promotes osteoclast activity, inhibits osteoblasts, osteoclasts release calcium into the bloodstream
Antagonist to thyroid gland to maintain calcium homeostasis, negative feedback
Adrenal gland
Paired glands that is composed of unrelated tissue types in mammals
Unrelated tissue types: have cortex and inner medulla
Sit right on top of the kidneys
Cortisol (glucocorticoid)- in the adrenal cortex
Target: global
Action:
Regulate food metabolism (glucocorticoid mobilizes glucose)
Reduces inflammation
Helps body store fat (promotes fat storage)
Primarily mammalian and aves hormone
Aldosterone (mineralocorticoid)- in the adrenal cortex
Target: kidneys
Action: regulates water balance/urinary output by regulating sodium levels (mineralocorticoid metabolisms minerals (sodium in this case))
Promotes the reabsorption of sodium and plays a role in urination because water diffuses with the increase of sodium concentration
Androgens- in the adrenal cortex
Target: hair follicles
Action: stimulates hair growth, associated with sexual maturation
Epinephrine and norepinephrine - in the adrenal medulla
Target: global
Action: activates flight or fight response
Pancreas
Endocrine and exocrine gland
Contains endocrine cells (islets of langerhans)- little hubs of hormone production, have different cells within them (but that is beyond our scope)
Insulin
Target: liver, skeletal muscle, adipose tissue
Action: stimulates cells to take in glucose (by glycogen) and store it, works to reduce blood glucose levels
Glucagon
Target: liver, adipose (NEVER skeletal muscle)
Action: stimulates cells to breakdown glycogen and release glucose into the blood
Antagonistic hormones because it does the opposite of insulin
Hormones and the reproductive cycle
Typically seasonal or cyclic because they have an estrous cycle
Females are only sexually receptive during brief periods of “heat” (estrus)
Endometrium (inner epithelial layer of the uterus) is not shed, instead it reverts to original state (in nonoriginal state it swells, fills with blood so that it can support an implanted zygote)
Menstrual cycle
Characteristic of anthropoids (primarily) and elephant shrews and some bats
Females are receptive throughout cycle
Ends with the breakdown and discharge of the endometrium
Menopause is the permanent cessation of ovulation, due to a decline in estrogen and progesterone
Only occurs in a few groups of mammals
Usually because it caps lifetime fitness- so the “grandmother hypothesis” is one possible explanation for its evolution
Grandmothers help raise grandchildren, helping daughters raise grandchildren indirectly helps fitness, kin selection
ex) orcas, japanese macaques, humans, etc.
Fight or flight response
Aka: “General adaptation syndrome”
Activated when exposed to extreme or long term stress
What it does:
Prepares the body to deal with a stressor (fighting or getting away)
Allows the body to work outside of homeostasis for a short period
Stage 1: alarm reaction
Something horrifying just happened
Initiated by hypothalamus when stress is perceived
Sympathetic NS is activated:
Adrenal medulla releases epinephrine (causes increase in blood glucose)
Pancreas
Increases glucagon and decreases insulin to increase blood glucose
Heart contractions increase, vasodilation in coronary and skeletal muscles, vasoconstriction in the kidneys
All non-essential organ system functions are inhibited by vasoconstriction (kidneys, digestive system, etc.)
Increases resources to the brain, heart, and muscles. Increases oxygen and glucose
Stage 2: resistance reaction
Extended releases of epinephrine causes release of cortisol. For sustained stimulus
Stimulates glucose metabolism to continue powering the flight and use additional resources
Pain receptors suppressed
Inflammation reduced (to allow animal to continue fighting by preventing stressors that would prevent the animal from doing the task)
Stage 3: exhaustion
Resistance reaction cannot be maintained any longer (resources have been depleted)
Prolonged exposure to cortisol causes
Wasting of muscles
Suppression of immune system
Storage of fat (adipose cells) why weight gain is a common response to stress