Oral Cavity and Esophageal Problems Review
Care of Patients with Oral Cavity and Esophageal Problems
Core Concepts
Priority Concepts: Tissue integrity, Nutrition
Interrelated Concepts: Gas Exchange, Pain
Learning Outcomes (Goals for Collaborative Care)
Plan interprofessional collaborative care to promote tissue integrity and nutrition in patients with oral and esophageal problems.
Educate adults on reducing the risk for oral cavity and esophageal problems.
Instruct patients and caregivers on common drugs and management strategies for oral and esophageal problems.
Develop patient- and family-centered nursing interventions to minimize the psychosocial impact of living with these conditions.
Apply anatomical, physiological, and pathophysiological knowledge to provide evidence-based care affecting tissue integrity and nutrition.
Analyze assessment and diagnostic findings to generate solutions and prioritize nursing care.
Organize care coordination and transition management for patients.
Use clinical judgment for evidence-based nursing care to promote tissue integrity, nutrition, and prevent complications.
Incorporate factors affecting health equity into the plan of care.
A Healthy Oral Cavity: Fundamental Linkages
Tissue integrity, nutrition status, and gas exchange are significantly impacted by oral and esophageal problems.
Patients in this category often present with high-risk factors for various complications.
Stomatitis: Oral Cavity Inflammation
Pathophysiology
Inflammation within the oral cavity, leading to painful, inflamed ulcerations.
Etiology and Genetic Risk
Primary Stomatitis: Often includes aphthous stomatitis (canker sores), herpes simplex stomatitis, and traumatic ulcers.
Secondary Stomatitis: Results from infection by opportunistic pathogens (e.g., Candida albicans), or is a side effect of drugs (e.g., chemotherapy) or radiation therapy.
Incidence and Prevalence
Recurrent Aphthous Stomatitis (RAS): Affects approximately 20\% of the general population.
Incidence is higher in females.
Health Promotion/Disease Prevention
Maintain proper oral hygiene.
Consume a well-balanced diet and ensure adequate hydration.
Implement stress management techniques.
Perform weekly self-examinations of the mouth and report any changing conditions promptly.
Ensure dentures are in good repair and fit properly.
Brush and floss teeth twice daily.
Avoid alcohol-based mouthwashes.
Minimize or avoid drugs known to increase inflammation.
Schedule regular dental check-ups.
Assessment (Recognizing Cues)
History Taking: Inquire about recent infections, changes in nutrition, oral hygiene habits, any oral trauma, and presence of stress.
Physical Assessment: Observe for signs and symptoms such as lesions, inflammation, pain, and difficulty eating.
Psychosocial Assessment: Evaluate the psychological and social impact of the condition, including effects on eating, communication, and self-esteem.
Analysis and Prioritization (Analyze Cues & Prioritize Hypotheses)
Key Issues:
Impaired tissue integrity due to oral and/or esophageal lesions.
Pain due to oral and/or esophageal lesions.
Planning and Implementation (Generate Solutions & Take Action)
Focus on preserving tissue integrity.
Implement strategies for minimizing pain.
Care Coordination and Transition Management
Educate on the importance of taking medication as prescribed.
Discuss potential side effects of prescribed pain medication.
Provide guidance on appropriate dietary choices to minimize irritation and promote healing.
Evaluation of Outcomes
Patient achieves healthy oral mucosa without inflammation or infection.
Patient experiences minimized discomfort or absence of pain.
Oral Tumors: Premalignant Lesions
Erythroplakia
Considered precancerous.
Appears as red, velvety mucosal lesions.
Common locations include the floor of the mouth, tongue, palate, and mandibular mucosa.
Leukoplakia
Characterized by thickened, white, firmly attached patches on the oral mucosa.
These patches cannot be easily scraped off.
Appear as slightly raised and sharply rounded lesions.
Most lesions are benign.
However, lesions located on the lips or tongue have a higher potential to progress to cancer.
Tobacco use is a significant risk factor for developing leukoplakia.
Hairy Leukoplakia (Special Type)
Manifests as white plaques on the tongue.
Often serves as one of the first visible signs of human immunodeficiency virus (HIV) infection.
Oral Cancer
Prevention and Risk Factors
Regular dental visits (twice a year) are crucial for professional dental hygiene and oral cancer screening.
Occupational exposure to certain chemicals or substances can increase risk.
Types of Oral Cancer
Squamous Cell Carcinomas: Most common type, frequently found on the lips, tongue, buccal mucosa, and oropharynx. (Visual example provided in transcript depicts squamous cell carcinoma on the tongue).
Basal Cell Carcinoma: Primarily affects the lips.
Kaposi’s Sarcoma: Can manifest on the hard palate, gums, tongue, and tonsils, often associated with immunocompromised states (e.g., HIV).
Assessment (Recognize Cues)
Assess the patient's oral hygiene regimen thoroughly.
Perform a comprehensive inspection of the oral cavity for any suspicious lesions.
Diagnostic Procedures: Biopsy is essential for definitive diagnosis. CT (Computed Tomography) or MRI (Magnetic Resonance Imaging) may be needed for staging and assessing metastasis.
Interventions (Take Action)
Nonsurgical Management: May include radiation therapy, chemotherapy, or targeted therapy depending on the type and stage of cancer.
Surgical Management: Involves removal of the tumor, which can range from local excision to more extensive resections, potentially requiring reconstructive surgery.
Care Coordination and Transition Management
Nutrition Therapies: Often necessary due to difficulty eating, requiring a dietary consult (e.g., with a Registered Dietitian Nutritionist).
Educate the patient to report any symptoms of infection promptly.
Sialadenitis: Salivary Gland Inflammation
Definition
Inflammation of a salivary gland.
Complications
If left untreated, an abscess can develop within the gland.
Etiology
Often occurs after ionizing radiation to the head or neck area.
Treatment
Prioritize patient hydration.
Apply moist heat to the affected gland.
Perform massage of the gland to promote drainage.
Administer NSAIDs (Nonsteroidal Anti-Inflammatory Drugs) for pain and inflammation.
Prescribe antibiotics if bacterial infection is suspected or confirmed.
Gastroesophageal Reflux Disease (GERD)
Pathophysiology
The most common upper GI disorder in the U.S.
Occurs due to the backward flow of stomach contents into the esophagus.
This reflux can damage the esophageal mucosa.
Etiology and Genetic Risk
There is no single causative agent for GERD; it's often multifactorial.
Presence of a nasogastric (NG) tube can compromise lower esophageal sphincter (LES) function.
Genetic predisposition or connection is observed in some cases.
Certain lifestyle factors significantly contribute (e.g., obesity, diet, smoking).
Barrett Esophagus (Barrett Epithelium): A complication where the normal esophageal lining is replaced by abnormal columnar epithelium, increasing the risk of esophageal cancer.
Incidence and Prevalence
18-28\% of adults in North America experience reflux disease.
Health Promotion/Disease Prevention
Patients may initially be asymptomatic, emphasizing the importance of preventive measures.
Adopt healthy eating habits, limiting fried, fatty, spicy foods, and caffeine.
Remain in an upright position for at least one hour after eating to aid digestion and prevent reflux.
Assessment (Recognize Cues)
History Taking: Inquire about heartburn, morning hoarseness, and nocturnal coughing or wheezing.
Physical Assessment/Signs and Symptoms: Look for dyspepsia (indigestion), and auscultate lungs for crackles, which may indicate aspiration.
Psychosocial Assessment: Evaluate the impact on quality of life, sleep, and anxiety levels.
**Diagnostic Assessment:
Upper Endoscopy (Esophagogastroduodenoscopy - EGD): Allows direct visualization of the esophagus, stomach, and duodenum, and tissue biopsy.
Ambulatory Esophageal pH Monitoring: Measures esophageal acid exposure over 24-48 hours to correlate symptoms with reflux episodes.
Analysis and Prioritization (Analyze Cues & Prioritize Hypotheses)
Key Issues:
Potential for compromised nutrition status due to dietary selection (to avoid triggering symptoms) and discomfort.
Acute pain due to reflux of gastric contents irritating the esophageal lining.
Planning and Implementation (Generate Solutions & Take Action)
Priorities: Balancing nutrition and minimizing pain.
**Nonsurgical Management:
Lifestyle Changes: Elevate the head of the bed, avoid large meals, stop smoking, avoid tight clothing.
Drug Therapy: Includes antacids, H_2-receptor antagonists (e.g., famotidine), proton pump inhibitors (PPIs) (e.g., omeprazole), and prokinetic agents.
Endoscopic Therapies: Such as Stretta procedure or EsophyX procedure to improve LES function.
Surgical Management: Fundoplication (e.g., Nissen fundoplication) to reinforce the LES.
Care Coordination and Transition Management
Educate on making appropriate dietary selections. (e.g., trigger foods).
Emphasize adherence to prescribed drug therapy.
Teach patients the signs of esophageal stricture (difficulty swallowing) and Barrett esophagus (changes in esophageal lining) to report.
Referral to a Registered Dietitian Nutritionist is often beneficial.
Suggest local support groups for ongoing management and emotional support.
Evaluation of Outcomes
Patient adheres to appropriate diet, medication therapy, and lifestyle modifications.
Patient experiences minimized or absence of pain related to GERD.
Hiatal Hernias (Diaphragmatic Hernias)
Pathophysiology
Also known as diaphragmatic hernias.
Involves the protrusion of a portion of the stomach through the esophageal hiatus of the diaphragm into the chest cavity.
Types: Classified into Types I through IV.
Sliding Hiatal Hernia (Type I): The gastroesophageal junction and part of the stomach slide up into the chest through the hiatus.
Paraesophageal (Rolling) Hiatal Hernia (Type II): The fundus of the stomach rolls up through the hiatus, alongside the esophagus, while the gastroesophageal junction remains in its normal position.
Assessment (Recognize Cues)
Many individuals are asymptomatic.
Others experience symptoms similar to GERD.
Symptoms often intensify after a meal or when lying supine.
**Diagnostic Assessment:
Barium Swallow Study with Fluoroscopy: Visualizes the stomach and esophagus as the patient swallows a contrast agent.
EGD: Direct visualization.
High-Resolution Manometry with Esophageal Pressure Topography: Measures pressure changes in the esophagus and LES function.
Interventions (Take Action)
**Nonsurgical Management:
Drug Therapy: Similar to GERD management (e.g., antacids, PPIs).
Nutrition Therapy: Small, frequent meals; avoid trigger foods.
Lifestyle Changes: Elevating the head of the bed, avoiding bending over, weight management.
**Surgical Management:
Patients may need to lose weight prior to surgery to minimize risks.
Laparoscopic Nissen Fundoplication: A minimally invasive surgical procedure to repair the hernia and reinforce the LES.
Care Coordination and Transition Management
Provide detailed instructions on nutrition modification post-surgery.
Recommend stool softeners or bulk laxatives to prevent straining, which could compromise the surgical repair.
Teach daily incisional inspection for signs of infection or complications.
Educate on specific signs to report to the health care provider immediately (e.g., severe pain, fever, difficulty swallowing, drainage).
Advise avoidance of individuals who are ill to prevent respiratory infections that could cause coughing and strain.
Esophageal Tumors
Pathophysiology
Some esophageal tumors can be benign, but the majority are malignant.
More than half of malignant esophageal tumors metastasize by the time of diagnosis.
Primary Risk Factors
Chronic alcohol intake.
Poor diet (e.g., low in fruits and vegetables, high in processed foods).
Obesity.
Smoking (both active and passive).
Untreated or long-standing GERD, which can lead to Barrett esophagus.
Assessment (Recognize Cues)
Assess for the presence of known risk factors.
Esophageal tumors are often referred to as