Module 2: Peripheral Vascular Disease
Course Code: PT 512 CP II
Semester: Spring 2025
Date: Monday, 2/24
Location: Discussion classroom (CGS 129)
Content: All of Module 1 (CR and PR)
Format: ExamSoft (multiple choice)
Download: Available for download on Saturday, 2/22
Note: Calculators permitted; personal phone calculators not allowed.
Total Questions: 20
General CR & PR content: 2 cases with 8-10 questions each (1 cardiac rehabilitation case, 1 pulmonary rehabilitation case)
Key Knowledge Areas:
Terminology and abbreviations discussed in class
Quality of Life (QOL) measures and their categories
Functional tests: assessment methods, appropriate use, cutoff scores, and Minimum Clinically Important Differences (MCID)
Gait speed cutoffs for community ambulation, hospitalization risk, and fall risk
Familiarization with GXT protocols (not memorization)
FITT recommendations by diagnosis
Karvonen formula (not HRR)
Digital calculator
Heart failure (HF) decompensation chart
RPE Borg scales
MET chart
Identify health conditions affecting the peripheral vascular system.
Describe signs and symptoms in individuals with vascular disease.
Differentiate between signs and symptoms of peripheral arterial vs. venous diseases.
Explain medical and surgical management of vascular diseases.
Discuss tests/measures for PT examination related to peripheral vascular diseases.
Identify impairments linked to peripheral vascular diseases and effective physical therapy interventions.
Explain how diabetes mellitus and its related pathologies may lead to vascular impairments that can be treated by physical therapy.
Thrombus: Blood clot
Phlebitis: Vein inflammation, increasing thrombosis risk
Thrombophlebitis: Venous inflammation accompanied by thrombus
Embolism: Detached clot traveling in the bloodstream
DVT: Deep vein thrombosis
VTE: Venous thromboembolism
PE: Pulmonary embolism
Blood Flow: From the left heart
Type of Blood: Oxygenated with low CO2
Blood Flow: Returning to the right heart
Type of Blood: De-oxygenated with higher CO2
Structural Characteristics:
Less distinct layers
Thinner walls
Presence of valves to prevent backflow
Structural Characteristics:
Well-defined layers with thicker walls
Supports higher pressure blood flow
Major Arches and Branches:
External iliac, internal iliac, femoral, popliteal
Anterior and posterior tibial, dorsalis pedis, plantar arch
Key Veins:
Femoral vein, great saphenous vein, popliteal vein
Deep and superficial veins, perforating veins
Conditions:
Atherosclerosis
Arterial thromboembolism
Aneurysm
Conditions:
Varicose veins
Thrombophlebitis (including DVT)
Venous thromboembolism
Comprehensive examination of lower extremity (LE) including:
Observation and quantification of skin, temperature, edema, circulatory changes
Screenings for VTE or arterial embolism
Assessment of functional mobility (e.g., gait speed)
Sign and symptom quantification (e.g., claudication)
Safe exercise to promote circulation and improve tissue oxygenation.
Education on PVD signs/symptoms and importance of physical activity.
Foot care in diabetic patients to prevent complications.
Same pathology as Coronary Artery Disease (CAD)
Can lead to cerebrovascular accidents (strokes) from carotid artery involvement.
Smoking
Hypertension (HTN)
Diabetes
High cholesterol
Age >60 years
Causes include:
Hyperglycemia-induced vascular damage
Microvascular dysfunction affecting blood vessels
Neuropathy affecting pain perception and vascular responses
Symptoms include intermittent claudication characterized by:
Pain or cramping during activity
Relief usually within 10 minutes of rest
Progressively worsening condition with increased risk factors (diabetes, smoking)
Ankle-Brachial Index (ABI):
Essential for diagnosing PAD; <0.9 indicates disease.
Measurement procedures for resting and exercise ABI.
Supervised treadmill training (intermittent exercise) for claudication management:
Frequency: 3 times/week
Duration: 30-50 minutes goals
Intensity: 40%-60% of max workload.
Atherosclerosis, A-fib, endocarditis, myocardial infarction, aneurysms.
Diminished or absent pulses, pain, pallor, muscle weakness.
Use of thrombolytics and embolectomy for management.
True and false aneurysms, dissecting aneurysms.
Severe pain, changes in tissue perfusion if ruptured, palpable masses in some cases.
Options include surgical repair, stenting, or clipping.
Causes include vessel wall weakness and disease.
Symptoms include heaviness, engorgement, skin changes.
Monitor for skin integrity and complications, offer education on management strategies.