GI Disorders MCN Notes
Anatomy and Physiology Comparison
Identify differences between pediatric and adult gastrointestinal (GI) anatomy and physiology.
Medications and Treatments
Discuss common medications and treatments for managing pediatric GI disorders.
Nursing Assessments and Interventions
Identify assessments and interventions for children with GI illnesses.
Common GI Illnesses
Distinguish between cleft lip and palate, dehydration, hypertrophic pyloric stenosis, intussusception, gastroesophageal reflux, appendicitis, Hirschsprung disease, and celiac disease.
Mouth
Lower Esophageal Sphincter
Esophagus
Stomach
Intestines
Evaluate growth patterns.
Address dietary concerns.
Review toilet training and bowel patterns.
Collect patient history and family history.
Esophageal pH probe
Ultrasound (abdominal)
KUB
Barium studies
Stool studies
Inspection & Observation
Assess abdominal size and shape.
Auscultation
Percussion
Palpation
Occurs more readily in infants and young children due to:
Greater proportion of body water (up to 2 years of age).
Infants have higher insensible fluid losses and a higher body surface area (BSA) to body mass ratio.
The renal system of infants does not concentrate urine like adults.
Goal: Restore fluid volume and prevent hypovolemia.
Physical Signs
Sunken eyes.
Reduced level of consciousness.
Dry mucous membranes and tissue turgor.
Tachypnea and tachycardia.
Oliguria and hypotension.
Sudden weight loss.
Reduced capillary refill time.
Mild to Moderate Dehydration:
Oral rehydration: 50-100 mL/kg with solutions like Pedialyte.
Severe Dehydration:
IV fluid management: 20 mL/kg with Lactated Ringer or Normal Saline.
Risk Factors
Health history and physical exam.
Family education.
C: Calming techniques
L: Lie on back
E: Evaluate airway
F: Feeding techniques
T: Teaching
L: Lip protection (Logan bow)
I: Infection
P: Parent bonding
P: Pain management
A: Airway management
L: Liquid diet
A: Avoid hard foods and objects in the mouth
T: Avoid hot, spicy, and citrusy foods.
E: Educate parents on infection signs, use of arm restraints, and diet advancement.
Pylorus becomes hypertrophied leading to obstruction.
Health History & Symptoms
Non-bilious emesis 30-60 minutes after feeding.
Signs of hunger despite feeding.
Physical Exam:
“Olive shaped” mass in the upper abdomen.
Progressive dehydration.
Manage fluid and electrolyte levels.
Provide parental education.
Administer pre and post-operative care.
Resume feedings post-op.
Bowel telescopes into a distal segment, leading to edema, impaired blood circulation, and obstruction.
Health History & Symptoms
Sudden onset of intermittent abdominal pain.
Bilious emesis and “currant jelly” stools.
Physical Exam:
Lethargy and “sausage”-shaped abdominal mass.
Barium or air enema.
Surgical repair if necessary.
Pre and post-operative care.
Obstruction leads to increased appendix pressure, edema, bacterial overgrowth, and potential perforation.
Health History
Right lower quadrant pain, nausea, vomiting, fever.
Assessment Techniques
McBurney’s Point and rebound tenderness.
Labs:
CT scan or ultrasound.
Perforated (Complicated):
Appendectomy, peritoneal drainage, IV antibiotics.
Non-Perforated (Uncomplicated):
Appendectomy with pre-op antibiotics and fluids.
Studied: Antibiotics only (provider preference).
Failure to thrive, respiratory problems, irritability, “wet burps” or vomiting
Specific symptoms in infants and older children.
Alter feeding techniques:
Small, frequent feedings; upright positioning; thickened formula.
Medications:
Histamine 2 blockers, proton pump inhibitors, prokinetics.
Consider Nissen fundoplication.
Lack of ganglionic cells in the bowel resulting in inadequate motility.
Not passing meconium within the first 24 hours, male.
Abdominal distension, pain, constipation, vomiting, slow growth.
Surgical resection of bowel with reanastomosis; "pull-through" procedure.
Autoimmune disorder impairing the ability to digest gluten, leading to villous damage in the small intestine.
Implement a strict gluten-free diet.
Health history and symptoms indicating malnutrition: anemia, steatorrhea, poor weight gain.
Screening:
Blood test for Tissue Transglutaminase Antibodies (tTG-IgA) yields positive results in most cases.
Definitive Diagnosis:
Small bowel biopsy showing villi atrophy.
Genetic Testing:
Testing for HLA-DQ2 and HLA-DQ8 genes.
Anatomy and Physiology Comparison
Identify differences between pediatric and adult gastrointestinal (GI) anatomy and physiology.
Medications and Treatments
Discuss common medications and treatments for managing pediatric GI disorders.
Nursing Assessments and Interventions
Identify assessments and interventions for children with GI illnesses.
Common GI Illnesses
Distinguish between cleft lip and palate, dehydration, hypertrophic pyloric stenosis, intussusception, gastroesophageal reflux, appendicitis, Hirschsprung disease, and celiac disease.
Mouth
Lower Esophageal Sphincter
Esophagus
Stomach
Intestines
Evaluate growth patterns.
Address dietary concerns.
Review toilet training and bowel patterns.
Collect patient history and family history.
Esophageal pH probe
Ultrasound (abdominal)
KUB
Barium studies
Stool studies
Inspection & Observation
Assess abdominal size and shape.
Auscultation
Percussion
Palpation
Occurs more readily in infants and young children due to:
Greater proportion of body water (up to 2 years of age).
Infants have higher insensible fluid losses and a higher body surface area (BSA) to body mass ratio.
The renal system of infants does not concentrate urine like adults.
Goal: Restore fluid volume and prevent hypovolemia.
Physical Signs
Sunken eyes.
Reduced level of consciousness.
Dry mucous membranes and tissue turgor.
Tachypnea and tachycardia.
Oliguria and hypotension.
Sudden weight loss.
Reduced capillary refill time.
Mild to Moderate Dehydration:
Oral rehydration: 50-100 mL/kg with solutions like Pedialyte.
Severe Dehydration:
IV fluid management: 20 mL/kg with Lactated Ringer or Normal Saline.
Risk Factors
Health history and physical exam.
Family education.
C: Calming techniques
L: Lie on back
E: Evaluate airway
F: Feeding techniques
T: Teaching
L: Lip protection (Logan bow)
I: Infection
P: Parent bonding
P: Pain management
A: Airway management
L: Liquid diet
A: Avoid hard foods and objects in the mouth
T: Avoid hot, spicy, and citrusy foods.
E: Educate parents on infection signs, use of arm restraints, and diet advancement.
Pylorus becomes hypertrophied leading to obstruction.
Health History & Symptoms
Non-bilious emesis 30-60 minutes after feeding.
Signs of hunger despite feeding.
Physical Exam:
“Olive shaped” mass in the upper abdomen.
Progressive dehydration.
Manage fluid and electrolyte levels.
Provide parental education.
Administer pre and post-operative care.
Resume feedings post-op.
Bowel telescopes into a distal segment, leading to edema, impaired blood circulation, and obstruction.
Health History & Symptoms
Sudden onset of intermittent abdominal pain.
Bilious emesis and “currant jelly” stools.
Physical Exam:
Lethargy and “sausage”-shaped abdominal mass.
Barium or air enema.
Surgical repair if necessary.
Pre and post-operative care.
Obstruction leads to increased appendix pressure, edema, bacterial overgrowth, and potential perforation.
Health History
Right lower quadrant pain, nausea, vomiting, fever.
Assessment Techniques
McBurney’s Point and rebound tenderness.
Labs:
CT scan or ultrasound.
Perforated (Complicated):
Appendectomy, peritoneal drainage, IV antibiotics.
Non-Perforated (Uncomplicated):
Appendectomy with pre-op antibiotics and fluids.
Studied: Antibiotics only (provider preference).
Failure to thrive, respiratory problems, irritability, “wet burps” or vomiting
Specific symptoms in infants and older children.
Alter feeding techniques:
Small, frequent feedings; upright positioning; thickened formula.
Medications:
Histamine 2 blockers, proton pump inhibitors, prokinetics.
Consider Nissen fundoplication.
Lack of ganglionic cells in the bowel resulting in inadequate motility.
Not passing meconium within the first 24 hours, male.
Abdominal distension, pain, constipation, vomiting, slow growth.
Surgical resection of bowel with reanastomosis; "pull-through" procedure.
Autoimmune disorder impairing the ability to digest gluten, leading to villous damage in the small intestine.
Implement a strict gluten-free diet.
Health history and symptoms indicating malnutrition: anemia, steatorrhea, poor weight gain.
Screening:
Blood test for Tissue Transglutaminase Antibodies (tTG-IgA) yields positive results in most cases.
Definitive Diagnosis:
Small bowel biopsy showing villi atrophy.
Genetic Testing:
Testing for HLA-DQ2 and HLA-DQ8 genes.