Genetics, Altered Immune Responses & Transplantation
Genetics, Altered Immune Responses & Transplantation
Course and Unit Information
Course Code: NUR 229
Semester: Fall 25
Unit Objectives
Upon completion of this unit of study, students will be able to:
Describe the etiology, clinical manifestations, diagnostic findings, drug therapy, dietary needs, and nursing care for the following types of immune disorders:
Hypersensitivity reactions:
Type I
Type II
Type III
Type IV
Allergic disorders
Autoimmunity
Organ transplantation
Graft versus host disease
Describe cultural considerations related to disorders that affect the immune system.
Discuss concepts of caring when providing nursing care to a patient with complications related to the immune system.
Discuss the utilization of critical thinking in the care of patients with an altered immune response.
Describe the use of therapeutic communication while interacting with patients with disorders of the immune system.
Develop a teaching plan for the patient with an alteration of the immune system.
Discuss legal and ethical considerations related to disorders of the immune system.
Preparation Requirements
Required Reading:
Lewis, 12th edition, Chapter 14: pgs. 213 - 235
Immunity Overview
Definition of Immunity
Immunity: The body’s ability to resist infection or disease; works in three ways:
Defense
Homeostasis
Surveillance
Importance of Immune Response
The immune response protects against invasion by microorganisms and prevents infection by attacking foreign antigens and pathogens.
Immune processes must function properly to maintain health; problems arise when the immune response is altered.
Related to concepts of inflammation, infection, and tissue integrity.
Key Concepts in Immune Function
Balance: Prevents excessive or harmful immune responses.
Continuous mutation recognition: Cells are recognized as foreign and destroyed.
Types of Immunity
Definitions
Innate Immunity:
Present at birth; primary role is first-line defense against pathogens.
Involves a nonspecific response – responds within minutes to pathogens without prior exposure.
Key WBCs involved: Neutrophils and monocytes.
Acquired Immunity:
Develops after exposure to antigens.
Two subtypes:
Active Acquired Immunity:
Body develops antibodies after a disease or immunization; long-lasting.
Passive Acquired Immunity:
Host receives antibodies rather than producing them (e.g., maternal antibodies; gamma globulin injections).
Immediate effect but short-lived; no memory cell production.
The Immune Response Process
Key Components
Lymphoid Organs:
Central: Bone marrow and thymus.
Peripheral: Lymph nodes, tonsils, spleen, lymphoid tissue.
Mononuclear Phagocytes:
Consists of monocytes (WBC in blood) and macrophages (found throughout the body).
Macrophages capture, process, and present antigens to lymphocytes, activating the immune response.
Lymphocytes:
Types include:
B Lymphocytes: Differentiate into plasma cells that make antibodies.
T Lymphocytes:
T Cytotoxic Cells (CD8): Attack foreign pathogen antigens.
T Helper Cells (CD4): Regulate immune responses by assisting B cells and cytotoxic T cells.
Natural Killer Cells: Attack tumors and virally infected cells.
Cytokines:
Chemical messengers that instruct cells to alter their activity in the immune process.
Types of cytokines include:
Interleukins (ILs): Immunomodulatory factors.
Colony-Stimulating Factors: Growth-regulating factors for hematopoietic cells.
Interferons: Antiviral and immunomodulatory agents.
Immune Response Dynamics
Viral Invasion:
A virus invades through breaks in the skin.
Antigen Recognition:
Macrophages digest the virus and display antigens.
T Helper Cell Activation:
Upon recognizing the antigen, T helper cells produce cytokines to regulate the response.
B Cell Activation:
Cytokines stimulate B cells to produce antibodies.
Cytotoxic Responses:
T cytotoxic cells and natural killer cells destroy infected cells.
Memory Cell Formation:
Memory B and T cells remain for quicker response upon reinfection.
Humoral vs. Cell-Mediated Immunity
Humoral Immunity
Involves antibodies in bodily fluids; mediated by B cells.
Process:
Antigen entry activates the primary immune response with initial production of IgM antibodies, progressing to production of IgG in subsequent exposures.
Key characteristics of immunoglobulins:
IgG: Major antibody in secondary immune response; crosses the placenta for passive immunity.
IgA: Found in mucosal surfaces and breast milk; provides passive immunity to infants.
IgM: First antibody produced during an immune response.
IgD: Present on lymphocyte surfaces.
IgE: Associated with allergic reactions.
Cell-Mediated Immunity
Initiated by T cells and involves T cells, macrophages, and NK cells.
Important for immunity against viruses, tumors, and fungal infections; involved in transplant rejection.
Immune Response to Allergens
Hypersensitivity Reactions
Types of Hypersensitivity:
Type I: IgE-mediated (e.g., allergies).
Type II: Cytotoxic reactions (e.g., transfusion reactions).
Type III: Immune complex reactions (e.g., lupus).
Type IV: Delayed hypersensitivity (e.g., contact dermatitis).
Type I - IgE Mediated Reactions
Involves mast cells and basophils; releases chemical mediators like histamine leading to allergic symptoms.
Clinical Examples:
Allergic rhinitis, asthma, atopic dermatitis, anaphylaxis.
Anaphylaxis
Can be life-threatening and presents with bronchial constriction, affecting multiple organ systems.
Initial symptoms include itching, leading to potential shock without treatment.
Type II - Cytotoxic/Cytolytic Reactions
Involve the binding of IgG or IgM to antigens on cell surfaces, leading to complement system activation.
Targets include blood cells (e.g., in transfusion reactions, autoimmune hemolytic anemia).
Type III - Immune Complex Reactions
Result from the deposition of antigen-antibody complexes in tissues or blood vessels, causing inflammation and tissue destruction.
Common in autoimmune conditions such as rheumatoid arthritis.
Type IV - Delayed Hypersensitivity Reactions
A T cell-mediated response occurs 24-48 hours following exposure.
Common examples include contact dermatitis and organ transplant rejection.
Allergy Assessment and Diagnostics
Subjective Data Collection
Gather history of past allergies, family history, and environmental exposures (e.g., pets, pollen).
Objective Data Collection
Signs of allergic reactions:
Rhinitis, conjunctivitis, skin reactions, asthma.
Diagnostic Tests:
Complete Blood Count (CBC) with differential, skin tests, and serum IgE testing for eosinophils.
Treatment Modalities for Allergies
Pharmacological Interventions
Antihistamines: e.g., diphenhydramine.
Corticosteroids: for inflammation control.
Mast Cell Stabilizers and Leukotriene Receptor Antagonists: e.g., montelukast for asthma.
Immunotherapy: Gradual exposure to allergens for symptom control.
Latex Allergies
Overview and Nursing Care
Common in healthcare workers; two types:
Type I: Immediate reaction to latex proteins.
Type IV: Delayed reaction due to chemicals in latex products.
Patient Education
Hazards of latex exposure and cross-sensitivities with certain foods (e.g., banana, avocado).
Graft-Versus-Host Disease (GVHD)
Occurs when immunocompetent cells from a donor attack host tissues.
Common symptoms: skin rash, jaundice, gastrointestinal disturbance.
Treatment involves immunosuppressive therapy.
Transplantation Overview
Types of Transplants
Commonly transplanted organs: kidneys, heart, liver, etc.
Matching Donors and Recipients
HLA Typing: Critical for minimizing rejection risk.
Crossmatching: Tests for pre-existing antibodies against donor HLAs.
Immunosuppressive Therapy
Goals and Regimens
To prevent transplant rejection while maintaining enough immunity for protection against infections.
Typically includes a regimen of calcineurin inhibitors, corticosteroids, and mycophenolate mofetil.
Concerns and Complications
Risks of infections and malignancies due to immune suppression.
Ethical and Legal Considerations
Importance of informed consent, patient rights regarding organ donation, and ethical considerations in transplantation.