The Skeletal System: Bone Tissue (Chapter 6)
Bone as an Organ and the Skeletal Framework
- Bone = an organ composed of multiple tissues
- Osseous tissue (compact & spongy)
- Cartilage
- Dense connective tissue (periosteum, ligaments)
- Adipose tissue (yellow marrow)
- Nervous tissue (sensory fibers accompanying blood vessels)
- Skeleton = bones + cartilages ➜ complete supportive framework
- Division of skeleton
- Axial (skull, vertebral column, ribs, sternum, hyoid)
- Appendicular (upper & lower limbs, pectoral & pelvic girdles)
Functions of the Skeletal System
- Support: structural scaffolding for body
- Protection: encases vital organs (brain, heart, lungs, spinal cord, reproductive organs)
- Movement assistance: levers for muscle contraction
- Mineral homeostasis
- Major reservoir for Ca and P
- Releases minerals on demand
- Hemopoiesis: red marrow forms RBCs, WBCs, platelets
- Triglyceride storage: yellow marrow (medullary cavities) stores energy
Classification of Bones
- Long: length > width; mostly compact bone
- Humerus, radius, ulna, femur, tibia, fibula, metacarpals/metatarsals, phalanges
- Short: nearly equal dimensions; mostly spongy bone
- Flat: thin, two parallel plates of compact bone enclosing spongy
- Cranial bones, sternum, ribs, scapulae
- Irregular: complex shapes
- Vertebrae, many facial bones
- Sesamoid: seed-like, develop in tendons under friction/ stress
- Patellae (largest), variable small sesamoids in hands/feet
Structure of a Typical Long Bone
- Diaphysis: shaft; thick compact bone surrounding medullary cavity
- Epiphyses (2): proximal & distal ends; mostly spongy bone with red marrow; articular cartilage (hyaline) covers joint surfaces
- Metaphyses (2): transition regions; in growing bone house epiphyseal (growth) plate ➜ becomes epiphyseal line after fusion
- Periosteum: double-layered membrane around diaphysis
- Outer fibrous dense irregular CT ➜ tendon/ligament anchor via Sharpey’s fibers
- Inner osteogenic layer ➜ osteoprogenitors/osteoblasts for growth & repair
- Medullary cavity: hollow space; yellow marrow in adults
- Endosteum: thin membrane lining cavity & trabeculae; contains bone-forming cells
Histology of Bone
- Extracellular matrix (ECM)
- \approx 15\% water
- 30\% collagen fibers ➜ flexibility & tensile strength
- 55\% crystallized mineral salts (hydroxyapatite = Ca3(PO4)2 · Ca(OH)2) ➜ hardness
- Why “inorganic”? ➜ mineral crystals lack carbon; responsible for rigidity
Cellular components
- Osteoprogenitor cells: mitotic stem cells in periosteum/endosteum; derive from mesenchyme
- Osteoblasts (“blast = build”): synthesize & secrete bone matrix (collagen, organic components); initiate calcification
- Osteocytes: mature cells trapped in lacunae; maintain metabolism, communicate via canaliculi (“cytes maintain the site”)
- Osteoclasts (“clast = crack”): large multinucleated cells from WBC lineage; ruffled border secretes acids & lysosomal enzymes for resorption
Tissue Types
- Compact (cortical) bone
- Strong, dense outer layer of all bones
- Predominates diaphyses of long bones
- Resists stresses of weight & movement
- Spongy (cancellous, trabecular) bone
- Lattice of trabeculae; spaces filled with red marrow
- Forms interiors of short, flat, irregular, sesamoid bones and epiphyses of long bones
- Lighter weight ➜ reduces overall mass; supports hemopoiesis
Compact Bone Micro-architecture
- Osteon (Haversian system) = repeating unit aligned along stress lines
- Central (Haversian) canal: blood/lymph vessels, nerves
- Concentric lamellae around canal
- Lacunae between lamellae house osteocytes
- Canaliculi interconnect lacunae & central canal for nutrient/waste diffusion
- Interosteonic (Volkmann’s) canals: transverse channels connecting periosteum, medullary cavity, and central canals
- Circumferential lamellae: external & internal layers encircling entire bone
- Interstitial lamellae: remnants of old osteons between new ones
Spongy Bone Micro-architecture
- Trabeculae arranged along lines of stress, containing concentric lamellae, lacunae, canaliculi
- No osteons; nutrients diffuse from vessels in marrow spaces
- Osteoblasts & osteoclasts line trabecular surfaces under endosteum
- Marrow spaces ➜ red marrow in adults (site of hemopoiesis)
Vascular & Neural Supply
- Bone = highly vascular & innervated
- Periosteal arteries/veins: enter/exit diaphysis via perforating (Volkmann’s) canals
- Nutrient artery/vein: large vessel through nutrient foramen into medullary cavity; branches proximally & distally
- Metaphyseal & epiphyseal vessels: supply respective regions
- Sensory nerves accompany vessels; periosteum richly innervated ➜ pain with damage
- Occurs in four situations
- Embryonic/fetal development
- Childhood/adolescent growth
- Remodeling throughout life
- Fracture repair
Two embryonic mechanisms
- Intramembranous ossification
- Bone develops directly within mesenchyme sheets ➜ flat bones of skull, mandible, clavicle
- Endochondral ossification
- Hyaline cartilage model first, replaced by bone
- Forms most of skeleton; continues at epiphyseal plates for length growth (interstitial)
Growth in Length (Interstitial)
- Epiphyseal plate zones (from epiphysis → diaphysis)
- Resting cartilage
- Proliferating cartilage
- Hypertrophic cartilage
- Calcified cartilage (osteoclasts dissolve; osteoblasts lay bone)
- Balance of cartilage growth on epiphyseal side & replacement on diaphyseal side ➜ elongation
- Closure ➜ epiphyseal line; length growth ceases \approx ages 18–21
Growth in Diameter (Appositional)
- Periosteal osteoblasts secrete new circumferential lamellae ➜ external growth
- Endosteal osteoclasts enlarge medullary cavity maintaining cortical thickness
- Cooperative cycle continues through life (thickening/widening)
Bone Remodeling & Wolff’s Law
- Remodeling: coordinated osteoclast resorption + osteoblast deposition
- Renews tissue before deterioration
- Heals micro-damage
- Adapts to mechanical stress; redistributes matrix along stress lines
- Wolff’s Law: bone architecture reflects demands
- ↑ mechanical load (bodybuilder) ➜ ↑ bone mass
- ↓ load (astronaut, bedridden) ➜ bone loss
Fractures & Repair
- Fracture = break in bone; types: open (compound), comminuted, greenstick, impacted, Pott, Colles, etc.
- Healing phases (3 phases, 4 steps)
- Reactive: fracture hematoma (inflammatory) forms
- Reparative: fibrocartilaginous (soft) callus ➜ bony (hard) callus
- Remodeling: callus remodeled; original shape restored
Bone & Calcium Homeostasis
- Bones store \approx 99\% of body Ca$^{2+}$
- Hypocalcemia stimulus ➜ parathyroid glands release PTH
- PTH actions
- Stimulate osteoclasts ➜ ↑ resorption, Ca$^{2+}$ release
- Kidneys retain Ca$^{2+}$, excrete phosphate, produce calcitriol (active Vitamin D)
- Calcitriol + dietary Vitamin D ➜ ↑ intestinal Ca$^{2+}$ absorption
- Hypercalcemia stimulus ➜ thyroid C-cells release calcitonin
- Calcitonin actions: inhibit osteoclasts & stimulate osteoblasts ➜ ↓ blood Ca$^{2+}$
Exercise & Aging Effects
- Weight-bearing exercise ➜ osteoblast stimulation, ↑ bone density, retard age-related loss
- Aging
- Until adolescence: deposition > resorption
- Early–middle adulthood: equilibrium
- Older age / post-menopause: resorption > deposition ➜ osteoporosis risk (esp. females due to ↓ estrogen)
Factors Affecting Bone Growth
Minerals
- Ca & P: hardness of ECM
- Mg: cofactor for ECM formation
- F: strengthens ECM
- Mn: enzyme co-factor for ECM synthesis
Vitamins
- Vitamin A: osteoblast activity; deficiency stunts growth
- Vitamin C: collagen synthesis; deficiency ⇒ scurvy-like bone issues
- Vitamin D (calcitriol): ↑ Ca absorption; deficiency ⇒ rickets (children), osteomalacia (adults); toxicity possible
- Vitamins K & B12: bone protein synthesis; deficiency ⇒ low density
Hormones
- GH (anterior pituitary): via IGFs ➜ general growth, osteoblast stimulation
- IGFs (liver & tissues): protein synthesis, osteoblast activity
- Thyroid hormones (T3, T4): stimulate osteoblasts, normal growth
- Insulin: ↑ bone protein synthesis
- Sex hormones (estrogen, testosterone)
- Teenage growth spurt
- Epiphyseal plate closure \approx 18–21
- Adult remodeling: ↓ osteoclast, ↑ osteoblast activity
- PTH: ↑ resorption, ↑ calcitriol
- Calcitonin: ↓ resorption
Lifestyle & Aging
- Exercise: weight-bearing maintains mass
- Aging/menopause: ↓ sex hormones ➜ bone loss & osteoporosis
Homeostatic Imbalances & Diagnostics
- Bone Scan: nuclear medicine to detect areas of altered metabolism (dark “hot spots” ⇒ ↑ turnover; light “cold spots” ⇒ ↓ blood flow)
- Osteoporosis: porous bone; post-menopausal women, Caucasians, small frame, low Ca/Vit D, sedentary, smoking/alcohol, family history ➜ preventive weight-bearing exercise, Ca & Vit D intake, meds (bisphosphonates, SERMs)
- Rickets: Vitamin D deficiency in children ➜ improper calcification, bowed legs
- Osteomalacia: adult counterpart; soft bones
Ethical & Practical Implications
- Importance of nutritional education (Vit D, Ca) across life stages
- Public health policies for osteoporosis screening (DEXA) & fall-prevention in elderly
- Ethical duty in sports/space travel to mitigate bone loss via training protocols