subjective; signs that only the pregnant person can perceive
most obvious: absence of menstruation
bonus if accompanied by consistent nausea, fatigue, breast tenderness, and urinary frequency
these symptoms (above) can be caused by conditions other than pregnancy
fetal movements are also known as_________
quickening
Presumptive Signs | When to expect them |
Breast tenderness | 3 to 4 weeks |
Amenorrhea | 4 weeks |
N/V | 4 to 14 weeks |
Breast enlargement and tenderness | 6 weeks |
Urinary frequency (without dysuria) | 6 to 12 weeks |
Fatigue (excessive) | 12 weeks |
Fetal movements (quickening) felt by mom | 18 to 20 weeks |
objective; can be detected on physical examination by HCP
these symptoms can be caused by conditions other than pregnancy, including elevated hCG levels
elevated hCG levels can be caused by ovarian cancer, choriocarcinoma, hydatidiform mole
Common signs:
Goodell sign: softening of the cervix (5 weeks)
Chadwick sign: bluish-purple coloration of the vaginal mucosa and cervix (6-8 weeks)
Hegar sign: softening of the lower uterine segment or isthmus (~16 weeks)
Other signs:
Positive pregnancy test (4–12 weeks)
changes in the size & shape of the uterus; increased abdominal girth from uterine enlargement (14 weeks)
Hyperpigmentation of the skin (16 weeks)
Ballottement: HCP pushes against the patient’s cervix during a pelvic exam and feels a rebound from the floating fetus (16–28 weeks)
Braxton Hicks contractions (16-28 weeks)
Palpable fetal outline
Serum and urine tests measure the presence of human chorionic gonadotropin (hCG)
a glycoprotein
earliest biochemical marker for pregnancy
hCG <5 mIU/mL = negative
hCG >25 mIU/mL = positive
levels should double every 29-53 hours during the first 30 days after implantation and peak ~8-10 weeks gestation
this has been used by HCP’s to differentiate normal from abnormal gestations
low levels = ectopic pregnancy
higher-than-normal levels = multiple-gestation pregnancy
corresponds with morning sickness period (6-12 weeks)
At-home tests:
highly accurate but a + result should be confirmed with Doppler fetal heart sounds, ultrasound, physical examination, or a serum pregnancy test
major concern = possibility of false negatives if performed too early (hCG hasnt reached detectable levels)
recommendation: if absence of menses continues, retest in 1 week
these confirm that a fetus is growing in the uterus
Common signs:
Ultrasound verification (4-6 weeks)
FHR seen on ultrasound (5–6 weeks)
Auscultation of fetal heart tones via Doppler (10 to 12 weeks)
Palpation of fetal movement felt by experienced clinician (20 weeks)
if pregnancy is confirmed, the HCP should:
include an estimated gestational age
give pts info about normal s&s and instruct them to report concerns
set up a schedule of prenatal visits to assess mom and baby throughout the entire pregnancy
decreased uterine vascularity and muscle tone
increase in blood flow
uterine growth:
stimulated by estrogen
occurs as a result of both hyperplasia and hypertrophy of the myometrial cells
Hyperplasia: increase in # of cells
Hypertrophy: increase in size of existing cells
uterine walls thin
Blood vessels elongate, enlarge, dilate, and sprout new branches
the diameter of the main uterine artery doubles in size to accommodate the increased blood volume needed to supply the placenta
Increased strength and elasticity allow uterus to contract and expel fetus during birth
Braxton Hicks contractions: spontaneous, irregular, and painless contractions beginning in the 1st trimester
continue throughout pregnancy
more noticeable in the last month as they thin/efface the cervix before birth
the isthmus (lower portion of the uterus) progressively thins as pregnancy progresses, thereby forming the lower uterine segment
positive Hegar sign (softening and compressibility of the lower uterine segment) results in exaggerated uterine anteflexion (tilting or curving of the uterus tilts toward the bladder) during early months of pregnancy
uterus remains in pelvic cavity for the first 3 months of pregnancy, after which it progressively ascends into the abdomen
growing uterus presses on the bladder → increased urinary frequency
supine hypotensive syndrome: uterus falls backwards against the inferior vena cava when lying down → vena cava compression → reduced venous return and decreased CO & BP → orthostatic stress
occurs from lying to sitting to standing
symptoms experienced: weakness, lightheadedness, nausea, dizziness, or syncope
solution: side-lying position which displaces the uterus to the left and off the vena cava
Fundal height best correlates with gestational age between 18-32 weeks
Obesity, hydramnios, and uterine fibroids may interfere with accuracy
20 weeks : at the level of the umbilicus and measures 20 cm
36 weeks: at the xiphoid process (its highest level)
38-40 weeks: drops due to lightening (see next bullet)
lightening: when the fundal height drops as the fetus begins to descend and engage into the pelvis
first pregnancy: usually occurs ~2 weeks before onset of labor
multigravida (second or subsequent pregnancy): usually occurs at the onset of labor
6-8 weeks of pregnancy: vasocongestion (tissues swell due to increased blood flow) and the influence of estrogen → Goodell sign (softening of cervix)
endocervical glands: increase in size and number & produce more cervical mucus
progesterone forms a thick mucus plug that blocks the cervical os and protects the opening from bacterial invasion
increased cervical vascularization → Chadwick sign (cyanosis or bluish-purple discoloration of cervix and vaginal mucosa)
increased estrogen and changes in the solubility of collagen → Cervical ripening (softening, effacement, increased distensibility)
begins ~4 weeks before birth
cervical connective tissues change elasticity and strength
Increases in mass, water content, and vascularization
Changes from a relatively rigid to a soft, distensible structure that allows the fetus to be expelled
estrogen increases vaginal vascularity → pelvic congestion (tissues swell due to increased blood flow) and vaginal hypertrophy (size increase) in preparation for the distention needed for birth
vaginal mucosa thickens
connective tissue begins to loosen
smooth muscle begins to hypertrophy (size increase)
vaginal vault (upper part of the vagina) begins to stretch and lengthen in preparation for expansion during birth
leukorrhea (whitish/yellowish vaginal discharge) increases during the second trimester
helps prevent bacterial infections
Vaginal pH increases → increased risk for candidiasis
can be transmitted at birth
neonates develop thrush
treated with local antifungal agents
the increased blood supply enlarges them until ~12-14 weeks gestation
elevated estrogen and progesterone levels cease ovulation and block secretion of follicle-stimulating hormone (FSH) and luteinizing hormone (LH) from the anterior pituitary
weeks 6-7: the ovaries stop producing hormones (the corpus luteum regresses) and the placenta takes over the major production of progesterone
increase in fullness (size), become tender, and grow larger due to estrogen and progesterone
become highly vascular; veins become visible under the skin
nipples become larger and more erect
nipples and areola become deeply pigmented
tubercles of Montgomery (sebaceous glands) enlarge and become prominent and keep the nipples lubricated for breastfeeding
breast growth and thinning of the skin → striae (stretch marks)
begin as pink to purple color, then eventually fade to a silver color, but never completely disappear
colostrum (creamy, yellowish breast fluid) can be expressed from the breast, if squeezed, by the 3rd trimester
motility specifically is most effected, not secretory nor absorptive functions
intra-abdominal portion of the esophagus may be displaced into the thorax
increased progesterone → relaxation of the lower esophageal sphincter and a decrease in gastric tone → dyspepsia
increased estrogen and proliferation of blood vessels and circulation to the mouth → gums become hyperemic (increased blood flow), swollen, and fragile and tend to bleed easily
Taste perception often changes
saliva produced in the mouth becomes more acidic
decreased unconscious swallowing when nauseated → excessive salivation (ptyalism or sialorrhea)
typically resolves spontaneously during the 2nd trimester, although it sometimes endures throughout the pregnancy
relief interventions: gum chewing, frequent sips of water, sucking on hard candies, or antiemetics
increased dental plaque, calculus, and debris deposits → periodontal disease (gingivitis and periodontitis)
Increased hormone production boosts vascular permeability and tissue edema, promoting the development of gingivitis and periodontitis
Elevated progesterone levels → smooth muscle relaxation → delayed gastric emptying and decreased peristalsis → more water than normal is reabsorbed → bloating and constipation
Constipation can also result from low-fiber food choices, reduced fluid intake, iron supplements, decreased activity levels, and intestinal displacement secondary to a growing uterus
Constipation, increased venous pressure, and the pressure of the uterus contribute to the formation of hemorrhoids.
slowed gastric emptying + relaxation of the cardiac sphincter → reflux → heartburn (pyrosis)
relieved with OYTC antacids
Cholelithiasis (gallstone disease) is more likely to occur
The emptying time of the gallbladder is prolonged secondary to the smooth muscle relaxation from progesterone
Hypercholesterolemia can increase the risk of gallstone formation
Other risk factors: high BMI, multiparity (more than 1 previous viable pregnancy)
N/V symptoms usually last from 6-20 weeks but may continue
usually occurs in the morning but can last all day
highest incidence is 6-12 weeks
linked to high levels of hCG and circulating estrogen and progesterone, gastric acidity, GERD, genetic factors, and lowered tone and motility of the digestive tract
increased: HR, CO, plasma volume & blood volume (the most striking cardiac alteration)
decreased: total peripheral resistance, systemic vascular resistance, BP
Blood volume:
increases beginning at the 10-12th week, peaks at the 30-34th week, and decreases slightly by the 40th week
this rise correlates directly with fetal weight
the increase is mostly made up of plasma (75%) with an increasing but small amount of RBCs (refer to purple text under “blood components” within this heading)
leads to “physiologic anemia of pregnancy”
lower h&h
normal adaptation of pregnancy
increase is needed to provide:
adequate hydration to fetal and maternal tissues
blood flow to perfuse the uterus
a reserve to compensate for blood loss at birth and during the postpartum period
increased perfusion to other organs, especially mom’s kidneys since they are excreting waste products for herself and baby
CO and HR:
peaks at 20-24 weeks and declines slightly thereafter
CO increases with greater venous return, increased HR, and right heart output (to the lungs), especially when lying on the left side after 20 weeks
HR increases by 10-30bpm until term
slight hypertrophy (size increase) of the heart
accommodates the increase in blood volume and CO
when blood volume peaks, someone with preexisting heart disease may have worsened symptoms as the heart begins to decompensate (cant keep up anymore)
Close monitoring is warranted at 28-35 weeks
BP:
1st trimester = remains at prepregnancy level
2nd trimester = BP decreases 5-10 mmHg
progesterone → peripheral vasodilation → decreased BP, especially diastolic pressure
3rd trimester = returns to 1st trimester levels
high BMI = high BP
Blood components:
RBC count increases, depending on amount of iron available
necessary to transport the additional oxygen required during pregnancy.
RBC count increases, but plasma volume rises even more due to hormones and sodium & water retention (refer to purple text under “blood volume” within this heading)
mainly due to increased circulating erythropoietin
demands of growing fetus + increase in maternal blood volume = increased iron requirements
fetal tissues prevail over mom’s tissues with respect to use of iron stores
fibrin and plasma fibrinogen levels increase, plus various blood clotting factors which make pregnancy a hypercoagulable state
contribute to slowed venous return, pooling, and dependent edema which increase the risk for venous thrombosis
oxygen consumption increases due to the increased oxygen requirements of the developing fetus, placenta, and maternal organs
causes faster, deeper breathing
decreased space for lungs as the diaphragm shifts upward due to pressure from the uterus
increased: diaphragmatic excursion, chest circumference, transverse diameter
increases tidal volume (volume of air inhaled) causing deeper, diaphragmatic breathing
causes hyperventilation and hypocapnia (low co2 blood levels)
can cause dyspnea in later pregnancy
thoracic muscles & cartilage relax → chest broadens, thoracic breathing → increased air volume per minute
increased estrogen → increased blood flow in the respiratory tract → congestion (more blood flow = swelling of tissues)
increased sex hormones & heightened allergy sensitivity → possible epistaxis and rhinitis
increased CO and relaxin → decreases efferent and afferent resistance (carries blood to and from the kidneys) → increases blood volume and blood flow to kidneys → increases GFR → increases urine flow and volume, substances delivered to the kidneys, and perfusion, filtration (hyperfiltration) & excretion of protein, amino acids, and glucose
drugs cleared by the kidneys may need higher and more frequent doses for therapeutic blood levels
kidneys must adapt to increased maternal and fetal waste
renal changes result from hormonal influences of estrogen and progesterone, pressure from the enlarging uterus, and increased maternal blood volume
Kidney and ureter dilation raises the risk of urinary stasis and infection
ureters elongate, widen, and become more curved above the pelvic rim by the 2nd trimester due to progesterone
Hormones (progesterone) cause the kidneys to enlarge by increasing firmness and slowing smooth muscle movement
progesterone causes renal pelvis to dilate
kidney activity normally increases when lying down and decreases upon standing
possibly why a pregnant person may need to urinate frequently while trying to sleep
late in the pregnancy, increased kidney activity is even greater when the person lies on their side rather than their back
characterized by posture and walking changes
increased lordosis (inward curve of lower back) & cervicodorsal (neck & upper back) extension to compensate for enlarging abdomen
caused by: center of gravity shifting forward, progesterone & relaxin on pelvic joints, and the increasing weight and position of the growing fetus
progesterone and relaxin cause ligaments around the sacroiliac joints and pubic symphysis to soften and stretch (relaxation), making the joints wider and more flexible
peaks by beginning of 3rd trimester
increases pelvic cavity size and makes delivery easier
causes the characteristic “waddle” gait
postural changes (see 1st bullet) plus with the loosening of the sacroiliac joints (see 2nd bullet) may result in lower back pain
common issues: low back pain, disk disease, pelvic girdle & hip pain, leg & foot pain/cramps, and hand & wrist pain
pelvic girdle: bones that form the base of the spine and connect the spine to the legs
changes in pigment, vascular supply, connective skin tissue, hair growth, nail structure, and gland functions
many disappear but not all, some only fade
common: hyperpigmentation (blotchy, brownish), typically localized & mild
due to elevated estrogen, progesterone, and melanocyte-stimulating hormone levels
mainly seen on the nipples, areola, umbilicus, perineum, and axilla, and the forehead and cheeks of dark-haired people
may develop on the face, aka facial melasma or “mask of pregnancy”
exacerbated by sun exposure
tends to recur in subsequent pregnancies
linea nigra: pigmented line down the middle of the abdomen, extending from the umbilicus to the pubic area
Striae gravidarum (stretch marks): irregular reddish streaks appearing most often on the abdomen, breasts, and thighs, but also on the lower back, buttocks, and upper arm
most prominent by 6-7 months
caused by: genetics, hormone changes, high BMI, reduced connective tissue strength resulting from elevated adrenal steroid levels, tension on the skin, and stretching of the structures secondary from fetal growth
more common in younger people & those carrying larger infants
Vascular-related skin changes:
Varicose veins: form due to long periods of sitting or standing (causing distention, instability, and poor circulation), and pressure from the growing uterus which slows blood flow
preventative measures:
elevate legs when sitting or lying down
Avoiding prolonged standing or sitting
Resting in left lateral position
walk daily for exercise
Avoid tight clothing or knee-high hosiery
Wear compression socks if this is a preexisting condition before pregnancy
vascular spiders: small blood vessels caused by vascular changes and high estrogen levels
may appear on the neck, thorax, face, and arms
typically disappears after childbirth
Palmar erythema: a well-defined pinkish area on the palms of the hands
related to elevated estrogen levels and other factors that cause vascular changes (vascular distention, instability, proliferation of blood vessels)
Hair and nails:
hair growth may decrease
After delivery, the body catches up with subsequent hair loss for several months
Nails typically grow faster
may experience increased brittleness, distal separation of the nail bed, whitish discoloration, and transverse grooves on the nails
most of these conditions resolve postpartum
Thyroid Gland:
enlarges slightly
becomes more active during pregnancy due to increased vascularity and hyperplasia (increased number of cells)
maternal thyroid hormone:
secretion increases
causes BMR (the amount of oxygen consumed by the body) to progressively increase
transferred to the fetus beginning soon after conception
critical for fetal brain development, neurogenesis, and organizational processes
especially important prior to 20 weeks as fetal production becomes more functional at 20 weeks
after 20 weeks, much of the thyroxin (T4) needed for development continues to be provided by mom
Low maternal thyroid levels places the fetus at higher risk of fetal hypothyroidism
Pituitary Gland:
enlarges and grows in size due to an increase in blood supply
when hormones from target glands rise, the anterior pituitary ceases its own hormones
FSH and LH:
secreted by anterior pituitary
secretion is inhibited as a result of hCG produced by the placenta and corpus luteum, plus increased secretion of prolactin by the anterior pituitary gland
hormone levels remain decreased until after delivery
TSH levels: reduced during the 1st trimester but return to normal for the remainder of the pregnancy
secreted by anterior pituitary
GH levels:
secreted by anterior pituitary
decreased
the action of human chorionic somatomammotropin (hCS), aka human placental lactogen (hPL), likely decreases the need and use of GH
Prolactin:
secreted by anterior pituitary
secreted in pulses and increases 10-fold to promote breast development and the lactation process
high progesterone levels secreted by the placenta → inhibition of prolactin on the breast → suppresses lactation
placenta is expelled at birth, progesterone levels drop → lactogenesis begins
Melanocyte-stimulating hormone (MSH):
secreted by anterior pituitary
levels increase
causes skin changes along with estrogen and possibly progesterone
Oxytocin:
made in the hypothalamus, secreted by posterior pituitary
gradually increases as fetus matures
responsible for uterine contractions, both before and after delivery
muscle layers of the uterus (myometrium) become more sensitive to oxytocin near term
at the end of a term pregnancy, levels of progesterone levels decline → contractions occur more frequently and intensely (previously suppressed by progesterone)
believed to be one of the initiators of labor
After delivery, oxytocin secretion continues → myometrium contracts → constriction of uterine blood vessels → decreases amount of vaginal bleeding after delivery
responsible for milk ejection during breastfeeding
Stimulation of the breasts through sucking or touching stimulates the secretion of oxytocin
can increase the severity of after pains since oxytocin stimulates uterine contractions
ADH, aka vasopressin:
made in the hypothalamus, secreted by posterior pituitary
its release is unchanged in pregnancy
helps the body conserve water by reducing urine formation and causes vasoconstriction, which raises BP and hypervolemia
Pancreas:
increased demand to meet needs of pregnancy → increased secretion of insulin
Maternal insulin does not cross the placenta, so the fetus must produce its own supply to maintain glucose control
In early pregnancy, maternal glucose goes to the fetus, so the mother's levels are low.
fetus is also drawing amino acids and lipids from mom, decreasing her ability to synthesize glucose.
Hormonal antagonists rise in late pregnancy, so more insulin is needed to overcome their effects.
examples of hormonal antagonists: hCS, steroids (cortisol), placental GH, glucocorticoids, prolactin, estrogen, and progesterone
results in glucose being less likely to enter mom’s cells and is more likely to cross over the placenta to the fetus
mom becomes slightly insulin resistance to make sure the growing fetus gets enough glucose
if mom doesn’t have enough beta cells (of the islets of Langerhans) → mom cant make enough insulin → blood sugar levels rise
in early pregnancy, maternal insulin production and insulin level decreases
Adrenal Glands:
hormone production increases
increased cortisol levels helps in times of stress
regulates carb and protein metabolism
secretion doesnt increase, the rate of clearance decreases
Levels rise in the 2nd and 3rd trimesters due to higher estrogen and placental release of corticotropin-releasing hormone (CRH)
most cortisol is deactivated to cortisone by a placental enzyme to protect the fetus
aldosterone increases
normally regulates absorption of sodium from within the kidney (at the distal tubules)
During pregnancy, progesterone promotes salt loss in urine, while aldosterone helps regulate electrolytes, water balance, and blood pressure
Prostaglandin Secretion:
theorized: increased prostaglandins production → facilitates uterine contractions, promotes cervical ripening, and increases myometrial sensitivity to oxytocin needed for labor
Placental Secretion:
placenta can make enzymes, proteins, fats, and carbs for energy storage
placenta functions as an endocrine gland, manufacturing and secreting hormones (protein and steroid hormones)
the placenta begins to produce these hormones:
hCG (protein hormone): maintains the maternal corpus luteum, which secretes progesterone and estrogens
hPL/hCS (protein hormone):
prepares mammary glands for lactation
involved in the process of making glucose available for fetal growth by altering maternal carb, fat, and protein metabolism
Antagonist of insulin because it decreases tissue sensitivity or alters the ability to use insulin
relaxin (protein hormone):
secreted by the placenta and corpus luteum
act synergistically with progesterone
increases flexibility of the pubic symphysis, permitting the pelvis to expand during delivery
cervical dilation
suppresses the release of oxytocin by the hypothalamus, thus delaying the onset of labor contractions
progesterone (steroid hormone):
Produced by the corpus luteum during the first few weeks of pregnancy and then by the placenta until term
supports and maintains the uterine endometrium
inhibits uterine contractility, maternal immune response
assists in the development of the breasts for lactation (the milk-producing glands)
estrogen (steroid hormone):
Promotes enlargement of the genitals, uterus, and breasts and increases vascularity, causing vasodilatation
Relaxation of pelvic ligaments and joints
Associated with hyperpigmentation, vascular changes in the skin, increased activity of the salivary glands, and hyperemia of the gums and nasal mucous membranes
Aids in developing the ductal system of the breasts in preparation for lactation
*DRI = daily recommended intake*
Iron and folic acid: needed to make more blood cells (since maternal blood volume increases), and must be supplemented since diet alone usually can't meet the increased demand
Iron: essential for fetal growth & brain development and in the prevention of maternal anemia
DRI during pregnancy: 27 mg/day of ferrous iron
lactating = 9-10 mg/day
folic acid: should be increased before and early in pregnancy is key to preventing fetal neural tube defects
DRI during pregnancy: 400-800 mcg/day of folic acid, possibly higher if there is previous history of fetuses with neural tube defects
lactating = 500 mcg/day (within same range as during pregnancy)
food sources: dark green vegetables (broccoli, romaine lettuce, and spinach), baked beans, black-eyed peas, citrus fruits, peanuts, liver
Eat at least 8-12 oz of lower methylmercury fish weekly, with one of them being an oily fish
Protein DRI during pregnancy: 71 g/day during 2nd & 3rd trimesters
same if lactating
Calorie DRI during pregnancy: 2,200–2,400 (2nd trimester), 2,400–2,600 (3rd trimester) daily
lactating = 2,200–2,400 daily
Fish, Shellfish, and levels of Mercury:
Most fish and shellfish contain some mercury
high levels of mercury can harm a fetus’s CNS if eaten in large amounts
also associated with pregnancy complications
mercury exposure is likely to arise from predatory fish consumption
before becoming pregnant, when pregnant, and if breastfeeding:
avoid fish with moderate to high mercury levels for 6–12 months before conception and during pregnancy
shark, swordfish, king mackerel, marlin, orange roughy, bigeye tuna, tilefish
eat 8-12 oz (two average meals) weekly of low mercury fish
shrimp, white albacore tuna (limit to 6 oz/week), salmon, lobster, sole, tilapia, cod, haddock, pollock, catfish
check local advisories about the safety of fish caught in local lakes, rivers, and coastal areas
amount of weight gain is not as important as what they eat
extra weight can be lost after pregnancy, but poor nutrition during pregnancy cant be reversed
Dieting during pregnancy is never recommended, even for people with higher weight
Severe calorie restrictions are associated with low birth weight
If pt is gaining weight in a steady, gradual manner, then they are taking in enough calories
adequate calories doesn’t always mean sufficient nutrient intake
quantity and quality of foods must be evaluated
recommended weight gain in 1st trimester based on prepregnancy BMI:
underweight (<18.5) = 5 lb
healthy (18.5 to 24.9) = 3.5-5 lb
high BMI (25 or higher) = 2 lb
much of the weight gain here is due to uterine growth and blood volume expansion
recommended weight gain in 2nd & 3rd trimester based on prepregnancy BMI:
underweight (<18.5) = slightly more than 1 lb/week
healthy (18.5 to 24.9) = 1 lb/week
high BMI (25 or higher) = 0.5-0.6 lb/week
recommended total weight gain throughout pregnancy based on prepregnancy BMI:
underweight (<18.5) = 28-40 lb
healthy (18.5 to 24.9) = 25-35 lb
overweight (25 to 29.9) = 15-25 lb
obese (>30) =11-20 lb
Teaching to Promote Optimal Nutrition During Pregnancy:
Follow a healthy dietary pattern and select a variety of foods from each food group
Gain weight in a gradual and steady manner depending on prepregnancy BMI
refer to red text under previous heading
Avoid weight reduction diets during pregnancy
Do not skip meals
Do not restrict salt use unless instructed by your HCP
Engage in reasonable physical activity for 150 min/week
Lactose intolerance:
Additional or substitute sources of calcium and vitamin D may be necessary
Encourage the pt to drink and eat lactose-free dairy products or calcium-enriched orange juice or soy milk
Vegetarians:
choose not to eat meat, poultry, and fish; plant-based foods only
Lacto-ovo-vegetarians:
no: red meat, fish, and poultry
yes: eggs, milk, dairy products, plant-based foods
Lacto vegetarians:
no: eggs, meat, fish, and poultry
yes: milk, dairy products, plant-based foods
Vegans:
no: foods originating from animals (eggs, milk, milk products)
yes: plant-based foods ONLY
must pay special attention to their intake of protein, iron, calcium, vitamin D, and vitamin B12
Suggestions include:
For protein: Substitute soy foods, beans, lentils, nuts, grains, and seeds.
For iron: Eat a variety of meat alternatives along with vitamin C–rich foods.
For calcium: Substitute soy, calcium-fortified orange juice, kale, broccoli, and tofu.
For vitamin D: Consume fortified milk, fish, cereal, and take prenatal vitamins
For vitamin B12: Eat fortified soy foods and cereals, and take a B12 supplement.
Pica
intense craving for and eating of nonfood items over at least 1 month
Common cravings:
geophagia = dirt, clay, soil
amylophagia = raw starch (cornstarch, laundry starch, flour)
pagophagia = ice, freezer frost
Other possible cravings: paper, toilet paper, charcoal, baby powder, chalk, coffee grounds, paint chips, ashes
Nutritional and clinical implications include the following:
geophagia: replaces nutritive sources and can contain toxic substances
causes iron-deficiency anemia, low gestational weight gain, constipation, hypokalemia, parasitic infection
pagophagia: can cause iron-deficiency anemia, low gestational weight gain, tooth fractures, jaw pain, freezer burn injuries
amylophagia: replaces iron-rich foods → iron deficiencies, and also leads to abdominal discomfort, poor glucose control, and excessive weight gain
having conflicting feelings at the same time; a normal, universal feeling
the person may feel proud and excited, while simultaneously fearful and anxious
mom commonly experiences this during the 1st trimester
evolves into acceptance by 2nd trimester, when fetal movement is felt
reactions are influenced by several factors: the way they were raised, their current family situation, the quality of the relationship with their partner, their hopes for the future, their personality, and their ability to adapt to changing circumstances
focusing on oneself
common during the early part of pregnancy
heightening in the 1st and 3rd trimesters when their focus is on behaviors that will ensure a safe and health pregnancy outcome
normal psychological adaptation
person may withdraw and become increasingly preoccupied with themselves and their fetus
may participate less with the outside world, may appear passive to their family and friends
happens during the 2nd trimester
the physical changes of the growing fetus bring a sense of reality and validity to the pregnancy
many tangible signs that someone separate from themselves is present; they conceptualize the fetus as a person
enlarging abdomen, fetal movement, hearing the heartbeat, seeing the fetal image on an ultrasound, feeling distinct parts, recognizing independent sleep and wake patterns
may verbalize positive feelings about the pregnancy
may accept their new body image and talk about the new life within
Emotional lability: having rapid, intense mood swings, where emotions can change quickly and feel hard to control
mom may start to cry without any apparent cause
Some people feel as though they are riding an “emotional roller coaster”
common throughout pregnancy
normal but can be stressful
Some people feel beautiful, while others feel uncomfortable with their appearance
some people may view pregnancy as a from worrying about weight, while others worry about weight gain
maternal tasks must be accomplished to incorporate the maternal role into her personality
Accomplishing these tasks helps mom develop her self-concept as a parent and build a strong mutual bond with the baby
Primary focus: ensuring safe passage throughout pregnancy and birth by participating in positive self-care activities
1st trimester: person focuses on themselves, not on the fetus
2nd trimester: person develops a great attachment to the fetus
3rd trimester: person has concern for themselves and the fetus as one
Seeking acceptance of infant by others
1st trimester: acceptance of pregnancy by themselves and others
2nd trimester: family needs to relate to the fetus as member
3rd trimester: unconditional support by others
Seeking acceptance of self in maternal role to infant (“binding in”)
1st trimester: pregnant person accepts idea of pregnancy, but not of an infant
2nd trimester: sensation of fetal movement (quickening) causes mom to acknowledges fetus as a separate entity within them
3rd trimester: person longs to hold infant and becomes tired of being pregnant
Learning to give of oneself
1st trimester: identifies what must be given up to assume new role
2nd trimester: identifies with infant and learns how to delay own desires
3rd trimester: questions their ability to become a good parent
discomforts of pregnancy can produce stress on the sexual relationship between mom and her partner
mom’s sexual desire may change throughout the pregnancy
1st trimester: may be less interested in sex because of fatigue, nausea, and fear of disturbing the early embryonic development
2nd trimester: interest may increase because of the stability of the pregnancy
3rd trimester: enlarging physical size may produce discomfort during sexual activity
Abstinence recommended for people with pregnancy complications [vaginal bleeding, preterm cervical dilation, preterm labor risk, risk of antepartum (during labor, before birth) hemorrhage because of placenta previa]
2nd trimester: dad accepts role as caregiver and support person, accepts the reality of the fetus when movement is felt, and experiences confusion when dealing with their pregnant partner’s mood swings and introspection
3rd trimester: dad prepares for the reality of this new role and negotiates what the role will be during the labor and birthing process
dad’s response depends on the state of the relationship
a struggling relationship may push the partner away near the end of pregnancy when responsibilities grow
may manifest as working late, staying out late with friends, or beginning new or superficial relationships
in a stable relationship, the partner now finds concrete tasks to do (painting the nursery, assembling the car seat, attending childbirth classes)
Expectant partners share many of the same anxieties as their pregnant partners
sibling’s reaction to pregnancy is age dependent
young toddler’s may regress in toilet training or ask to drink from a bottle again
older school-aged child may ignore the new addition to the family and engage in outside activities to avoid the new member
Preparing siblings for a new baby should match their age and life experiences at home
child may fear changes in their bond with their parents or worry about being replaced by the new family member → beginning of sibling rivalry
reduce this by constant reinforcement of love and caring