AP Psychology
Theories
1. Research & Thinking Theories
Hindsight Bias
“I knew it all along” effect
After an outcome, people believe they predicted it all along
Makes us overconfident
Messes with accurate memory
Confirmation Bias
Tendency to search for, interpret and remember info in a way that confirms one’s existing beliefs
Ignores or downplays opposing evidence
Leads to biased thinking
Strengthens stereotypes
Poor judgements, polarized opinions
Overconfidence Effect
Tendency to overestimate the accuracy of one’s knowledge, judgments, or abilities
Riskier divisions
Reduces careful thinking
Causes mistakes
Theory of Mind
Ability to recognize that others have mental states that may be different from one’s own
“Other minds exist”
Empathy, social skills, communication
Operational Definition Principle
Variable must be defined in terms of specific procedures used to measure of manipulate it
Makes research clear
Prevents vague terms from meaning different things
EXAM
“How is the variable measured?” “Define in measurable terms?” = operational definition
2. Learning Theories (Major + Minor)
Classical Conditioning
Type of learning which an organism learns to associate two stimuli so that one stimulus predicts another
E.x. feeling anxious when you hear a dentist drill because of past pain
Pavlov’s Classical Conditioning
Learning process where a neural stimulus, after being repeatedly paired with an unconditioned stimulus becomes a conditioned stimulus that elicits a conditioned response
Learning by associating a neutral stimulus with a natural response
Before conditioning
Food (US) - salivation (UR)
Bell (NS) - no salivation
During conditioning
Bell (NS) + Food (US) - salivation (UR)
After conditioning
Bell (CS) - salivation (CR)
Terms
Unconditioned stimulus (US): food
Unconditioned response (UR): salivation
Conditioned stimulus (CS): bell
Conditioned response (CR): salivation to bell
Neutral stimulus (NS): bell during conditioning
Stimulus Generalization
Tendency to respond to stimuli that are similar to the conditioned stimulus in the same way
E.x. dog conditioned to salivate to a bell, dog also salivates to similar-sounding bell or tone
EXAM
Generalization: similar stimuli = same response
Discrimination: only the specific stimulus causes the response
Stimulus Discrimination
Learned ability to distinguish between a conditioned stimulus and other similar stimuli
E.x. god salivates only to the specific bell tone
EXAM
Generalization: similar stimuli = same response
Discrimination: only the exact stimulus = response
Extinction
Conditioned response decreases and eventually disappears when the conditioned stimulus is no longer paired with unconditioned stimulus
Weakening of a learned response
E.x. dog learned: bell = food = salivation, bell rings without food over and over, salivation to bell fades away = extinction
CR weakens and disappears
EXAM
Extinction does not erase learning, it suppresses it
“Response weakens” “CS no longer followed by US” = extinction
Spontaneous Recovery
Reappearance of a previously extinguished conditioned response after a rest period, without new conditioning
Opposite of extinction, CR reappears after a pause
Operant Conditioning (B.F. Skinner)
Learning in which behavior is strengthened or weakened based on the consequences that follow it
Learning by consequences
Behavior = consequence = future likelihood of behavior
EXAM
Consequence matters, not association
Operant = voluntary behavior (Contrast: classical = automatic response)
Used rats and pigeons in controlled experiments
Law of Effect (Thorndike)
Behaviors followed by satisfying consequences are more likely to be repeated, behaviors followed by unpleasant consequences are less likely to be repeated
Foundation of operant conditioning
Explains learning through trial and error
EXAM
Key difference: Thorndike studied cats, Skinner used rats and pigeons in controlled experiments
Positive Reinforcement
Desirable stimulus is added after a behavior, making if more likely the behavior will occur again
EXAM
Positive = adding something
Negative Reinforcement
Unpleasant stimulus is removed after a behavior making it more likely the behavior will occur again
EXAM
Negative = taking something away
Positive Punishment
Aversive stimulus is added after a behavior, making it less likely to occur again
Adding something unpleasant to decrease behavior
EXAM
Positive = adding something
Negative Punishment
Desirable stimulus is removed after a behavior making it less likely it will occur again
Taking away something pleasant
EXAM
Negative = taking something away
Primary Reinforcers
Stimuli that are innately rewarding because they satisfy biological needs
E.x. food, water, shelter, physical comfort
Unlearned = naturally motivating
Often related to survival
Secondary Reinforcers
Reinforcers are stimuli that become rewarding through association with primary reinforcers
Learned rewards
E.x. money (can buy food), grades (praise and future opportunities)
Learned = not naturally satisfying but becomes reinforcing through association
Signals access to primary reinforcer
Continuous Reinforcement
Schedule in which a behavior is reinforced every single time it occurs
Reward every time the behavior occurs
Fastest way to teach a behavior
But extinction happens quickly if reinforcement stops
EXAM
Contrasted with partial reinforcement which is slower to learn but more resistant to extinction
Partial (intermittent) Reinforcement
Schedule in which a behavior is reinforced only part of the time, not every time it occurs
Fixed-ratio (FR)
Reward after a set number of responses
High response, brief pause after reward
Variable-ratio (VR)
Reward after a random number of responses
Most resistant to extinction
Fixed-interval (FI)
Reward after set amount of time
Could cram
Variable-interval (VI)
Reward after a random amount of time
Slow, steady responding
Shaping
Operant conditioning technique in which successive approximations of a target behavior are reinforced until the desired behavior is achieved
Teaching a behavior step by step
Reward small steps toward the final behavior, makes learning complex behaviors easier
Often used in operant conditioning examples
Chaining
Individual behaviors are linked together in a sequence
Each step becomes the cue for the next step until a full behavior is learned
Teach small behaviors, connect them, they become one complete action
E.x. teaching a child to brush teeth (steps)
Chaining connects several behaviors into one sequence while shaping rewards small steps toward one behavior
EXAM
“Learning a sequence of steps”, “linking behaviors together”
Observational Learning
Learning that occurs by watching the behaviors of others and the consequences of those behaviors
Learning by watching others
EXAM
Modeling = copying behavior
Vicarious reinforcement/punishment = learning from consequences others receive
Social Learning Theory (Bandura)
People can learn behaviors by observing others and considering the rewards or punishments that follow those behaviors
Learning by observing and imitating others, considering consequences
E.x. bobo doll experiment: children watched adults acting aggressively = children imitated aggressive behavior
Children more likely to imitate when adults were rewarded or not punished
EXAM
Contrasts with classical condition (automatic responses) and operant conditioning (learning from your own behaviors consequences
3. Cognitive & Memory Theories
Information Processing Model
Describes memory as a series of steps: info is encoded, stored, and later retrieved
Encoding
Converting info into a form the brain can use
E.x. reading a textbook - understanding and noting key point
Storage
Keeping info in memory over time
E.x. remembering facts for a test
Retrieval
Getting info back when needed
E.x. recalling facts during an exam
Memory is active processing, not just passive storage
EXAM
Related to sensory memory, short-term memory, long-term memory
Parallel Processing
Ability to process multiple types of information simultaneously instead of one at a time
Handling many pieces of information at once
EXAM
Contrasted with serial processing (processing one thing at a time)
Key for vision, perception, and multitasking questions
Three-Stage Memory Model (Atkinson–Shiffrin)
Memory occurs in three stages:
Sensory memory
Duration: milliseconds-seconds
Function: briefly holds incoming sensory info
Example: Seeing a flash of lightning, hearing a loud bang
Capacity: very large but info faces quickly
Short-term memory/Working memory
Duration: 20 s without rehearsal
Capacity: 7 ± 2 items (miller’s magic number)
Function: temporarily holds and manipulates information
Example: remembering a phone number long enough to dial
Levels of Processing Theory
Memory depends on how deeply info in processed, not how long it is studied
Structural processing
focuses on appearance
Weak memory
Phonemic processing
Focuses on sound
Moderate memory
Semantic processing
Focuses on meaning
Strongest memory retention
EXAM
Making info meaningful improves memory
Encoding Specificity Principle
Memory is best when retrieval conditions match the conditions during encoding
When encoding a memory, you also encode environment, mood, surrounding cues
These cues help retrieve the memory later
Serial Position Effect
Tendency to recall the first and last items in a list better than the middle ones
Primacy effect
Better memory for first items
Stored in long-term memory
Recency Effect
Better memory for last items
Stored in short-term memory
Recency disappears after delay, primacy stays (long-term memory)
Spacing Effect
Tendency for distributed study or practice to lead to better long-term retention than cramming
Reduces fatigue, increases long-term retention, allows memory consolidation
Cramming helps with short-term recall
Testing Effect
Finding that actively retrieving info from memory leads to better long-term retention than passive review
Retrieval strengthens memory
EXAM
Works well with spacing effect
Flashbulb Memory
Vivid, emotion-laden memory for a surprising or significant event, held with high confidence but not necessarily accurate
Emotion = stronger memory feeling, not better memory accuracy
Reconsolidation Theory
When a memory is retrieved, it becomes unstable and can be altered before being stored again
A memory is retrieved
It becomes unstable/flexible
New info, emotions, or context can alter it
Memory is re-stored, possibly changed
Retrieval failure
Information is stored in memory, but you can’t access it when you need it
E.x. you studied for a test but during the test your mind goes blank and later you remember the answer
Why: missing cues (environment, mood, context), stress or pressure, not enough strong connections to the memory
EXAM
“Tip of the tongue”, “memory is there but inaccessible”
Encoding failure
Info never gets properly stored in memory in the first place
Brain never really saved it, there’s nothing to retrieve
E.x. Someone introduces themselves, you immediately forget their name
Why: lack of attention, distractions, shallow processing (not thinking about meaning)
Encoding failure = never stored, retrieval failure = stored but can’t access
Proactive interference
Old info interferes with learning or remembering new info
Old blocks new
E.x. you learn your old password, then change it, you keep trying the old one by mistake
Previously learned info makes it harder to remember newer info
Retroactive interference
New info interferes with remembering old info
New blocks old
Learn a new phone number, now you can’t remember old one
Recently learned info makes it harder to recall earlier info
Retroactive interference = new-blocks old, proactive interference = old-blocks new
4.Developmental Theories
Piaget’s Cognitive Development
Children actively construct knowledge as they interact with the world
Thinking develops in 4 universal stages
Assimilation: fitting new info into existing schemas
Schema: mental framework for organizing info
Accommodation: changing schemas to fit new info
Equilibration: balancing assimilation and accommodation to create understanding
Sensorimotor stage (birth-2yrs)
Thinking through senses and actions
Object permanence develops
Knowing objects still exist when out of sight
E.x baby looks for a you hidden under a blanket
Preoperational stage (2-7yrs)
Thinking is symbolic but not logical
Egocentrism
Can’t see things from other’s perspectives
Centration
Focuses on one feature at a time
E.x thinking a taller glass has more juice even if the amount is the same
Concrete Operational stage (7-11yrs)
Logical thinking about concrete events
Understands conservation, less egocentric
Struggles with abstract ideas
E.x Knowing two equal balls of clay are still equal when one is flattened
Formal Operational stage (12+yrs)
Abstract and hypothetical thinking
Uses deductive reasoning
E.x solving algebra problems or debating ethical issues
Happens because children actively explore, adapt their schemas, build more complex ways of thinking over time
EXAM
Object permanence = sensorimotor
Egocentrism/conservation failure = preoperational
Logical thinking with real objects = concrete operational
Abstract or hypothetical reasoning = formal operational
Stranger Anxiety Theory
Fear or distress infants feel when they see an unfamiliar person
Happens around 6-8 months, decreases after 1 yr
Happens because infants form attachments to familiar caregivers
Can now distinguish familiar vs unfamiliar faces
EXAM
Infants around 8-12 months, crying around unfamiliar people, attachment to caregivers
Erikson’s Psychosocial Theory
Personality develops across the entire lifespan through 8 stages, each with a social conflict
Trust v.s Mistrust (0-1)
Care consistent = trust
Care unreliable = mistrust
E.x baby feels safe when needs are met
Autonomy v.s Shame & Doubt (1-3)
Encouraged independence - autonomy
Overly controlled = shame and doubt
E.x letting a toddler dress themselves
Initiative v.s Guilt (3-6)
Supported = initiative
Discouraged = guilt
E.x child starts games and activities
Industry v.s Inferiority (6-12)
Success in school = industry
Constant failure = inferiority
E.x feeling proud of school work
Identity v.s Role Confusion (12-18)
Exploring roles = identity
No exploration = role confusion
E.x teen exploring values, careers, beliefs
Intimacy v.s Isolation (Young adult)
Strong bonds = intimacy
Avoidance - isolation
Generativity v.s Stagnation (Middle adulthood)
Am I contributing to society?
Helping next generation = generativity
Self-focused = stagnation
Integrity v.s Despair (late adulthood)
Was my life meaningful?
Satisfaction = integrity
Regret = despair
Kohlberg’s Moral Development
Moral reasoning develops in stages, based on how people think about right and wrong, not what choice they make
Level 1: Preconventional morality
Moral thinking based on self-interest
Stage 1: Obedience & Punishment
Right = avoiding punishment
Stage 2: Individualism & Exchange
Right = what benefits me
Level 2: Conventional Morality
Moral thinking based on social rules
Stage 3: Good boy/girl
Right = pleasing others
Stage 4: Law & Order
Right = obeying laws and authority
Level 3: Postconventional Morality
Moral thinking based on principles
Stage 5: Social Contract
Laws are important but can be changed
Focus on rights and values
Stage 6: Universal Ethical Principles
Morality based on internal values
Justice, equality, human rights
EXAM
Fear of punishment = stage 1, Wanting approval - stage 3, Respect for laws = stage 4, Human rights over laws = stage 5/6
Vygotsky’s Sociocultural Theory
Cognitive development is shaped by social interaction and culture
Scaffolding = when helper gives support at first, then slowly removes it as the learner improves
EXAM
Learning happens with help from others
Zone of Proximal Development
Range between what a child can do alone and with help
Attachment Theory (Bowlby & Ainsworth)
Early relationships between babies and caregivers affect emotional development and future relationships
Attachment = emotional bond
Strong emotional connection between a baby and caregiver
Usually forms in the first years of life
Bowlby’s ideas
Secure attachment = children feels safe and protected
Affects confidence, relationships, emotional health
Ainsworth’s experiment
Observed babies when parent leaves, stranger enters, parent returns
Secure attachment
Upset when parent leaves, happy when parents returns, trusts caregiver
Insecure-Avoidant
Not very upset when parent leaves
Avoids parent when they return
Insecure-Anxious
Very upset when parent leaves
Hard to comfort when parent returns
Disorganized attachment
Confused or unusual behavior
May approach and avoid parent
early emotional bonds with caregivers influence development and relationships later in life
5. Motivation Theories
Instinct Theory
Behavior is motivated by innate biological instincts that prompt survival
Behavior = natural instincts we are born with
Survival, hunger, protection instinct
Drive-Reduction Theory
Motivation comes from desire to reduce physical discomfort and maintain balance in the body
We do things to reduce discomfort and return to balance
Homeostasis = keeping the body stable
Drive = need
Reduction = fix it
Incentive Theory
Behavior is motivated by external rewards and incentives
Behavior = motivated by rewards or incentives
EXAM
Opposite of drive-reduction theory
Needs
E.x eat because you’re hungry
Incentive theory
Motivated by rewards
E.x eat dessert because it tastes good
Arousal Theory
Motivated to maintain an optional level of excitement or alertness
Low arousal (bored)
High arousal (stressed)
Maslow’s Hierarchy of Needs
Motivation based on fulfilling needs from basic survival to personal growth
Physiological needs
Basic survival needs (food, water, sleep, etc)
Safety needs
Feeling safe and secure (shelter, protection, stability, etc)
Love and belonging
Relationships (friends, family, feeling accepted, etc)
Esteem needs
Feeling respected (confidence, achievement, etc)
Self - Actualization
Reaching full potential
Becoming best version of yourself
Lower needs must be met before higher needs
Bottom = survive, top = thrive
Self-Determination Theory
Motivated by needs for autonomy, competence, and connection with others
Autonomy
Feeling in control of your own choices
Competence
Feeling capable and skilled
Relatedness
Feeling connected to others
Intrinsic motivation = comes from inside
Extrinsic motivation = comes from rewards
6. Emotion Theories
James–Lange Theory
Emotions are caused by physical body reactions
EXAM
Physical changes come before emotions
E.x see a snake, heart beats fast, start shaking, then feel fear
Body first
Cannon–Bard Theory
Emotions and physical reactions occur at the same time
Stimulus = body reaction + emotion simultaneously
E.x see a snake, feel fear & heart races at the same time
Schachter–Singer (Two-Factor)
Emotions come from physical arousal plus the brain’s interpretation of that arousal
Emotion = arousal + cognitive label
E.x
Physical arousal (heart racing, sweating, trembling)
Cognitive label (“i’m scared” or “i’m excited” based on situation)
Emotion occurs (arousal + label = specific emotion)
Lazarus Cognitive-Mediational Theory
Emotions happen after we interpret a situation cognitively
Thought, emotion, physical reaction
Think about situation first, then feel emotion, then body reacts
Facial Feedback Hypothesis
Facial expressions can influence emotional experiences
Face muscles can send signals to your brain that affect how you feel
E.x even if you’re not happy, smiling can make you feel happier
EXAM
Body and mind influence each other
Emotions are not only reactions, they can be shaped by your body
7. Personality Theories
Freud’s Psychoanalytic Theory
Personality and behavior are shaped by unconscious motives, childhood experiences, and conflicts between Id, Ego, and Superego
Id (I want it now)
Operates on the pleasure principle
Wants immediate gratification
E.x eat a cake now because it looks tasty
Ego (Wait, plan)
Operates in the reality principle
Balances Id and superego
E.x wait to eat a cake after dinner
Superego (should/shouldn’t)
Operates on morality and ideals
Tells right from wrong
E.x “I shouldn't eat cake before dinner, it’s not proper.”
Ego uses defence mechanisms to reduce anxiety from conflict between Id and Super ego
Repression: forgetting bad memories
Denial: refusing to accept reality
Projection: blaming others for your feelings
Psychosexual Stages
Childhood stages of personality development, each focused on a different erogenous zone, where unresolved conflicts can affect adult personality
Oral (0-1)
Pleasure from sucking, chewing, biting
Fixation - smoking, nail-biting, overeating
Anal (1-3)
Pleasure from controlling bladder/bowels
Fixation - overly neat (anal-retentive) or messy (anal-expulsive)
Phallic (3-6)
Oedipus/Electra complex: desire for opposite-sex parent
Fixation - problems with authority or relationships
Latency (6-puberty)
Sexual feelings are dormant; focus on school friends
Fixation - minimal direct effect, social skills develop
Genital (puberty-adult)
Mature sexual interest develop
Fixation - difficulties in adult relationships if earlier stages unresolved
Fixation: being stuck at a stage, affects adult personality
Oedipus complex: boys desire for mother, rivalry with father
Electra complex: girls desire for father, rivalry with mother
Defense Mechanisms Theory
Unconscious strategies used by the ego to reduce anxiety caused by conflict between id, ego, and superego
Ego protects you from stress and anxiety by distorting reality
Repression: pushes unwanted thoughts into the unconscious
E.x forgetting a traumatic event
Denial: refusing to accept reality
E.x refusing to admit you failed a test
Projection: attributing your feelings to someone else
E.x blaming someone else for your anger
Displacement: redirecting emotions to a safer target
E.x yelling at a sibling instead of your teacher
Regression: returning to an earlier stage of development
E.x throwing a tantrum when stressed
Rationalization: justifying behavior with logic
E.x “I failed because the test was unfair”
Sublimation: turning negative impulses into positive actions
E.x canneling anger into sports
Reaction formation: acting opposite to your feelings
E.x being overly nice to someone you dislike
EXAM
Unconscious= you’re not aware you’re using them
Protects ego from anxiety
Can be healthy (sublimation) or unhealthy (denial, projection) depending on usage
Trait Theory
Theory of personality that focuses on stable, measurable characteristics that define a person
Personality = a collection of traits that are stable over time
Cardinal traits: dominant traits that define a person
Central traits: general characteristics found in everyone
Secondary traits: situational traits (less obvious)
Big Five Personality Theory (OCEAN)
Model that describes personality using five broad traits: openness, conscientiousness, extraversion, agreeableness, neuroticism
Openness
Curious, imaginative, creative, open to new experiences
Conscientiousness
Organized, responsible, dependable
Extraversion
Outgoing, energetic, sociable
Agreeableness
Friendly, compassionate, cooperative
Neuroticism
Tendency to experience negative emotions, emotionally
EXAM
Each trait exists on a spectrum (low-high)
Traits predict behavior across situations
Often measured with personality tests
Humanistic Personality Theory
Personality develops through personal growth and the desire to reach one’s full potential
Maslow: believed people are motivated to reach self-actualization (best possible self)
Rogers: believed personality develops through self-concept and acceptance from others
Free will
People make their own choices, not controlled only by instincts or environment
Self-concept
How you see yourself (real self: who you are v.s ideal self: who you want to be)
E.x student works hard in school because they want to be the best version of themselves, not because of rewards or punishment
Rogers’ Self Theory
Personality develops from self-concept and acceptance from others
Congruence (real self ≈ ideal self) v.s Incongruence (real self ≠ ideal self)
E.x if student feels accepted by family/friends they are more likely to develop a positive self-concept
Maslow’s Self-Actualization Theory
People are motivated to reach their full potential after basic needs are satisfied
Cannot focus on self-actualization until lower needs are met
EXAM
Part of humanistic psychology
Personal growth, free will, positive human nature
Social-Cognitive Theory
Personality is shaped by the interaction of thoughts, behavior and environment
People learn by watching others and thinking about what they see
Observational learning
Personality shaped by thoughts (cognition), behavior, environment
All three influence each others
Belief in your ability to succeed
High self efficacy: I can do this
Low self efficacy: I can’t do this
E.x student studies harder because they believe they can succeed (self-efficacy), they saw others succeed (observational learning)
Reciprocal Determinism
Behavior, personal factors, and environment influence each other
Part of Social-cognitive theory
E.x student think “i am good at math”, behavior: studies more, environment: gets praise from teacher
EXAM
Reciprocal = back and forth (each part influences each other)
Self-Efficacy Theory
Belief in one’s ability to succeed influences behavior and performance
Self efficacy: how much you believe you can do something successfully
E.x student A believes they can do well on the test = studies harder and tries, student B thinks they will fail = gives up sooner
Student A has higher self-efficacy
Affects motivation, effort, confidence, success
Projection v.s Objective tests
Respond to ambiguous stimuli, and your answers reveal unconscious thoughts or feelings
No clear right or wrong answer, you “project your inner thoughts onto the stimulus
E.x. inkblot test
What you see reflects you
Pros: can reveal hidden emotions/unconscious thoughts, Cons: less reliable
Projective = interpreted responses, Objective = scored responses
8. Intelligence Theories
Spearman’s g Factor
Intelligence includes one general mental ability that influences performance on many tasks
Believes that people who do well in one mental task usually does well in other mental tasks
g (general intelligence): overall mental ability
Affects problem solving, learning, reasoning, memory
E.x student is good at math, reading, science = high g
Believed intelligence is mostly one general ability not many separate abilities
Thurstone’s Primary Mental Abilities
Intelligence is made up of several different mental abilities instead of one general intelligence
Disagrees with Spearman
Seven primary mental abilities
Verbal comprehension: understanding words and language
Word fluency: producing words quickly
Number ability: working with numbers
Spatial ability: understanding shapes and space
Associative memory: remembering information
Perceptual speed: quickly noticing details
Reasoning: solving problems logically
E.x student is good at math but not reading
Gardner’s Multiple Intelligences
Intelligence consists of multiple independent intelligences
8 intelligences
Linguistic (words, reading, writing), Logical/Mathematical (reasoning, man, problem solving), Spatial (visualizing, maps, design), Musical (rhythm, pitch, instruments), Bodily/Kinesthetic (physical coordination, movement), Interpersonal (understanding others), Intrapersonal (understanding yourself), Naturalistic (recognizing patterns in nature
Sternberg’s Triarchic Theory
Intelligence has three distinct but related components
Analytical
Problem-solving, logical reasoning, academic skills, measured by IQ test
Creative
Ability to adapt to new situations, thinking outside the box, generating new ideas
Practical
Applying knowledge to real-word situations, adapting to everyday life knowing how to handle real-life problems
Emotional Intelligence Theory
Ability to perceive, understand, manage, and use emotions effectively
EXAM
Reading people well, controlling emotional reactions
Handling conflict effectively, being socially skilled
Nature v.s Nurture
What shapes human behavior and development more: biology or environment
Nature: genetics/biology, Nurture: environment and experience
Most psychologists agree it’s both working together, not one or the other
9. Social Psychology Theories
Attribution Theory
How people interpret and explain the causes of behavior, both their own and others
Internal (dispositional) attribution
Caused by personality, traits, effort, attitude
Ex. “she failed because she’s lazy”
External (situational) attribution
Caused by environment or circumstances
E.x “She failed because the test was unfair”
EXAM
Blaming personality instead of situation
Explaining behavior as traits v.s environment
Protecting self-esteem by blaming outside factors
Fundamental Attribution Error
Tendency to overestimate personality (internal) causes and underestimate situational (external) causes when explaining other people’s behavior
When someone does something, we assume “that’s just who they are” instead of “maybe something happened to them”
We see the person, not situation
We assume behavior reflects character
EXAM
blames personality instead of situation
Underestimates external factors
overestimates dispositional causes
Self-Serving Bias
Attribute successes to internal factors (personality, effort, ability, etc), attribute failures to external factors (luck, other people, situation, etc)
E.x “I succeeded because I'm awesome.” “I failed because the situation was unfair”
Self-esteem, confidence, mental health
EXAM
About yourself
Fundamental attribution error about others
Cognitive Dissonance Theory
We feel psychological discomfort when we hold two conflicting thoughts or when our behavior contradicts our beliefs
Thoughts and actions don’t match, feel uncomfortable, try to fix it
E.x “smoking is bad for my health" but you smoke
Creates dissonance (mental discomfort)
Might quite smoking
Ways to reduce dissonance: change behavior, change belief, add a new justification
EXAM
Feeling uncomfortable after acting against beliefs
Justifying behavior
Changing attitudes to match behavior
Social Identity Theory
Explains how our self-concept is shaped by the groups we belong to
Part of who you are comes from your group memberships
Ingroup bias
View our own group more positively, give our group credit for success, blame other groups more easily
Boosts self-esteem, provides identity, makes us feel connected
EXAM
Ingroup v.s outgroup
Group-based self-esteem
Favoring your group
Prejudice from group identity
Mere Exposure Effect
Tendency to develop a preference for things simply because we are repeatedly exposed to them
E.x song you didn’t like start sounding good after hearing it many times
Makes things feel familiar, familiar things feel safer, safety increases positive feelings
EXAM
Repeated exposure increasing liking
Advertising repetition
Growing preference just from familiarity
Foot-in-the-Door Phenomenon
Getting someone to agree to a small request first makes them more likely to agree to a larger request later
After agreeing to the small request people think “i must care about this issue” “i’m the type of person who supports this”
EXAM
Small request first, larger request later
Gradual commitment, consistency
Door-in-the-Face Technique
Large request first (likely be refused), then smaller more reasonable request
Second request feels like a compromise
Reciprocity: when someone “backs down” from a big request, we feel pressure to meet them halfway
Normative Social Influence
Someone conforms to a group to be liked, accepted, or avoid rejection
Change behavior because you care about social approval
E.x dressing a certain way to match your friend group
Want to be liked, public compliance, “I don’t want to stand out”
Informational Social Influence
Someone conforms because they believe others are correct and have better information
You change your behavior because you think the group knows better than you
E.x trying a restaurant because it’s crowded and you assume it must be good
Want to be correct, private acceptance, happens in ambiguous situations
Groupthink
When a group makes a bad or irrational decision because members value harmony and agreement over critical thinking
Desire for consensus overrides realistic evaluation of alternatives
E.x a group of students agrees on a weak project idea because no one wants to challenge the “popular” person’s suggestion
Deindividuation
Loss of self-awareness and self-restraint that can occur in group situations, leading people to act in ways they normally wouldn't
People feel anonymous in a group
More likely to lose personal responsibility, follow group behavior, and act impulsively or aggressively
EXAM
loss of self awareness, impulsive behavior, happens in crowds
Bystander Effect
People are less likely to help someone in an emergency where other people are present
More people = less likely someone is to help
Diffusion of responsibility, pluralistic ignorance, evaluation apprehension
Social Facilitation
People perform better on simple or well-learned tasks when others are watching, but worse on difficult or new tasks
Presence of others increases arousal which improved performance on easy tasks but hurts performance on hard tasks
Social Loafing
Individuals put in less effort when working in a group than when working alone
When responsibility is shared, individual effort decreases
Diffusion of responsibility, lack of individual accountability, belief that individual effort won’t be noticed
Conformity
When a person changes their behavior, beliefs or attitudes to match those of a group
Change to “fit in” with others
Why: want to be liked (normative social influence), want to be correct (informational social influence)
Obedience
Person follows direct orders form someone in a position of authority
Why: Authority figures carry power or status, people feel pressure to comply or avoid consequences
Obedience = following authority, conformity = following group
Attitude behavior gap
People's actions do not always match their attitudes or beliefs
What you say ≠ what you actually do
Why: habits are stronger than intentions, social pressure/convenience, lack of self-control/motivation, situational factors (time, stress, environment)
Often linked with cognitive dissonance
Disorders
Unit 7: Clinical Psychology
7.4 Anxiety Disorders
Generalized Anxiety Disorder (GAD)
Excessive, persistent, and uncontrollable worry about everyday life events
Worry is chronic, anxiety is not tied to one specific thing (unlike phobias)
Person often feels on edge most of the time, difficulty controlling the worry
Symptoms: restlessness, fatigue, difficulty concentrating, irritability, muscle tension, sleep problems
GAD = vague, constant worry about many things, Phobia = intense fear of a specific object or situation
GAD = ongoing anxiety, Panic disorder = sudden, intense panic attacks
E.x someone constantly worries about school, health, family, future even when there is no immediate reason, and they can’t “turn it off”
Causes:
Biological: genetic predisposition, overactive amygdala
Cognitive: tendency to expect the worst
Environmental: stress, trauma
Treatments:
Cognitive therapy: helps patients identify and challenge irrational “worst case scenario” thinking
Medications: SSRIs (selective serotonin reuptake inhibitors) (like fluoxetine) used long-term
Relaxation techniques, lifestyle changes
Panic Disorder
Recurrent, unexpected panic attacks and ongoing fear of having more attacks
Panic attack: sudden surge of intense fear or discomfort that peaks within minutes
Unexpected panic attacks (not tied to specific trigger), persistent worry about future attacks, behavioral changes (avoiding places/situations)
Symptoms: rapid heart rate, shortness of breath, chest pain, dizziness, sweating, trembling, feeling like your choking or losing control, fear of dying
Panic disorder = attacks are unpredictable, Phobia = fear tied to a specific trigger
E.x. A person suddenly feels intense fear while sitting in class: heart racing, can’t breath, thinks they are dying. They start avoiding school because they fear another attack
Causes:
Biological: sensitive “fight or flight” system
Cognitive: misinterpreting physical sensations (“i’m dying”)
Learning: conditioning from past panic experiences
Treatments:
Cognitive behavioral therapy (CBT): teaches patients to reinterpret panic symptoms
Exposure therapy: gradually exposes person to panic sensations to reduce fear
Medications: SSRIs (short term)
Breathing retraining
Separation Anxiety Disorder
Excessive fear or anxiety about being separated from attachment figures
Fear is developmentally inappropriate (too intense for age)
Strong distress when anticipating or experiencing separation
Persistent (typically at least 4 weeks in children, longer in adults)
Symptoms: extreme distress when leaving home/caregivers, constant worry about losing attachment figures, refusal to go to school or be alone, clinging behavior, nightmares about separation, physical complaints when separation is expected (headaches, etc)
Normal = young children may briefly fear separation, Disorder = intense, persistent, interferes with daily life
Separation anxiety = fear focused on attachment figures, GAD = broad, generalized worry about many things
E.x A child refuses to go to school because they are terrified something bad will happen to their parents while they’re away, and experiences stomachaches every morning before leaving
Causes:
Biological: temperament (high anxiety sensitivity
Environmental: overprotective parenting, stressful events
Learning: past experiences of separation or loss
Treatments:
Behavioral therapy: gradual separation exposure
Cognitive behavioral therapy: helps reduce catastrophic thinking
Family therapy: helps caregivers avoid reinforcing anxiety
Reassurance + routines
Specific Phobia
Intense, irrational fear of a specific object or situation that leads to avoidance
Symptoms: immediate fear or anxiety when exposed to trigger, fear is excessive and unreasonable compared to actual threat. Person often avoids the object/situation, causes distress or interferes with daily life
Common triggers: animal, natural environment, situational, blood injection injury
E.x. someone refuses to go hiking because they have an extreme fear of snakes, even in areas where snakes are rare
Specific phobia = fear tied to one specific trigger, GAD = broad, generalized worry about many things
Causes:
Biological: genetic predisposition, heightened fear response
Environmental: traumatic experience with the object/situation
Learning: classical conditioning (learned fear), Operant conditioning (avoidance reinforces fear)
Treatments:
Exposure therapy (most effective): gradual or direct exposure to feared object/situations
Systematic desensitization, flooding (immediate exposure in safe environment)
Modeling: watching others interact safely with feared object
Cognitive behavioral therapy: challenges irrational fear beliefs
Medication (rare): short term anti-anxiety meds
Social Anxiety Disorder
Excessive fear or anxiety of social situations where you might be judged, embarrassed, or rejected
Fear is out of proportion to the actual situation
Symptoms: avoids social situations, endures them with extreme anxiety, physical symptoms, overthinking social interactions
EXAM: fear is irrational or excessive compared to actual threat, causes significant distress or impairment, persistent
E.x. Someone is scared of public speaking
Causes:
Biological: overactive amygdala, genetic disposition, neurotransmitter imbalance (especially serotonin)
Environmental: negative social experiences, overly critical or controlling parenting, lack of social exposure/skills growing up
Cognitive: negative thinking patterns, high self-focus in social situations, overestimating how bad things will go
Behavioral: avoidance reinforces fear, conditioning
*Think biopsychosocial model
Treatments:
Cognitive behavioral therapy: changes negative self-beliefs
Exposure therapy: practicing social situations
Social skills training: learning eye contact, conversation skills
SSRIs: severe cases
Group therapy: safe environment to practice social interactions
Agoraphobia
Intense fear or anxiety about being in situations where escape might be difficult or help wouldn’t be available if panic occurs
Scared of being trapped, helpless, or unable to escape
Situations: crowds, malls, public, open spaces or enclosed spaces, being outside alone
Symptoms: avoidance of these situations, needs a companion to go out, panic-like symptoms (rapid heartbeat, etc)
EXAM: fear is excessive/unreasonable, causes significant impairment in daily life, persistent
Causes:
Biological: genetic predisposition to anxiety disorders, overactive amygdala
Psychological factors: fear conditioning, catastrophic thinking, heightened sensitivity to bodily sensations
Behavioral: avoidance of feared places, avoidance reduces anxiety short-term but reinforces the fear long-term
Social/Environmental factors: traumatic or embarrassing panic attack in public, stressful life events increasing overall anxiety levels, learned fear from observing others anxiety or reactions
Panic - avoidance - more fear - more avoidance
Treatments:
Exposure therapy (key treatment): gradual exposure to feared places
CBT: addresses fear of having panic attacks in public
Systematic desensitization: relaxation + gradual exposure hierarchy
SSRIs: often used for long-term symptom reduction
Support systems: sometimes a trusted person accompanies initial exposures
7.5 Obsessive-Compulsive and Related Disorders
Obsessive-Compulsive Disorder (OCD)
Unwanted, intrusive, thoughts, and repetitive behaviors or mental acts performed to reduce anxiety caused by those thoughts
E.x. “my hands are contaminated” - excessive handwashing
Symptoms: feeling anxious or distressed when obsessions occur, performing rituals according to strict rules or patterns, temporary relief after completing compulsions, followed by return of anxiety
Causes:
Biological: genetics, overactivity in basal ganglia, serotonin imbalance
Psychological: maladaptive thought patterns, inflated responsibility, overestimation of danger, difficulty tolerating uncertainty
Behavioral: negative reinforcement loop, compulsion reduces anxiety temporarily, reinforces compulsion (makes it stronger over time), learned associations between thoughts and danger
Environmental: Stressful or traumatic experiences can trigger or worsen symptoms, learned behaviors from environment
EXAM: obsessions = thoughts, compulsions = behaviors
The disorder is maintained because compulsions relieve anxiety short-term but strengthen the cycle long-term
Treatments:
Exposure and response prevention (ERP) (best behavioral treatment): patient is exposed to obsession trigger but prevented from doing compulsion
CBT: challenges irrational obsessive thoughts
SSRIs (antidepressants): help reduce obsessive thoughts
Brain stimulation: extreme cases
Body Dysmorphic Disorder
A person is excessively preoccupied with a perceived flaw in their appearance that is either minor or not observable to others, causing distress and repetitive behaviors
Symptoms: preoccupation with a perceived flaw in appearance, repeated comparing appearance to others, excessive covering up “flaws”, strong distress shame or embarrassment, difficulty focusing on anything else
Causes:
Biological: possible serotonin involvement, brian activity differences in areas linked to perception and self-evaluation
Psychological: distorted body image, cognitive distortions like magnifications, low self-esteem and high self-criticism
Behavioral: repetitive checking, seeking reassurance, behaviors temporarily reduce anxiety but maintain the disorder
Social/Environmental: media and social media pressure about appearance, bullying or teasing about looks, cultural emphasis on beauty standards
EXAM: BDD = focus on imagined or minor physical flaw, eating disorders = focus on weight, OCD = obsessions + compulsions not centered on looks
Treatments:
CBT: challenges distorted body image beliefs
SSRIs: reduce obsessive focus on appearance
Exposure therapy: reduces avoidance of mirrors/social situations
Avoidance of cosmetic reinforcement
Hoarding Disorder
Persistent difficulty discarding possessions, leading to excessive accumulation of items that cluster living spaces and impair daily functioning
Symptoms: difficulty throwing away items, strong emotional attachment to possessions, distress when attempting to discard items, avoidance of letting others touch or remove belongings
Causes:
Biological: possible abnormalities in prefrontal cortex, may overlap genetically with obsessive-related disorders
Psychological: beliefs that items have strong emotional or practical value, difficulty making decisions and categorizing importance, anxiety about losing important memories tied to objects
Behavioral: saving items that reduces anxiety - reinforces hoarding behavior
Cognitive: poor organization and categorization skills, overestimation of future need for items
Environment: Stressful life events, learned saving behaviors from family or environment
Treatments:
CBT (most effective): targets beliefs about saving items
Skills training: decision:making and organizational skills
Exposure therapy: practice discarding items gradually
SSRIs: sometimes used, but less effective than CBT
EXAM:
hoarding disorder = difficulty discarding + cluttered living space, OCD = obsessions + compulsions, not necessarily clutter
Hoarding is its own disorder in DSM-5 not just a subtype of OCD
Trichotillomania
Recurrent irresistible urges to pull out one’s own hair resulting in noticeable hair loss and distress or impairment
Symptoms: repeated pulling out of hair, feeling relief after pulling, difficulty stopping the behavior despite wanting to, sometimes occurs during stress, boredom, or anxiety
Causes:
Biological: possible genetic links with obsessive-related disorders, involvement of brain circuits related to impulse control and habit formation
Psychological: urges build tension - pulling reduces tension, anxiety or stress triggers the behavior
Behavioral: negative reinforcement (pulling hair reduces unpleasant feelings - behavior continues)
Environment: stressful situations or emotional distress, learned coping mechanism
EXAM: Part of obsessive-related disorder spectrum
Treatments:
Habit reverse training (HRT): key behavioral therapy: replaces hair-pulling with competing response (like clenching fists)
CBT: addresses triggers and urges
SSRIs: sometimes used
Stress management techniques
Excoriation Disorder
Recurrent compulsive picking at one’s skin resulting in lesion, distress, and difficulty stopping the behavior
Symptoms: repeated picking at skin, tension or urge before picking, temporary relief after picking, difficulty resisting the behavior despite attempts to stop
Causes:
Biological: possible genetic overlap with obsessive-related and impulse control disorders, brain differences in habit and reward systems
Psychological: anxiety or stress increases urge to pick, perfectionism or focus on perceived skin “imperfections”, tension builds - picking relieves it (reinforcement cycle
Behavioral: negative reinforcement (picking reduces anxiety or discomfort, so behavior continues
Environmental factors: stressful life events or boredom, triggers like noticing small skin irregularities
Treatments:
Habit reversal training (HRT): main treatment
CBT: reduces
Stimulus control: removing triggers (like mirrors, picking tools
SSRIs: sometimes helpful
7.6 Depressive Disorders
Major Depressive Disorder (MDD)
Persistent depressed mood and loss of interest, along with other symptoms that impair daily functioning for at least 2 weeks
Symptoms: depressed mood, loss of interest or pleasure, change in appetite or weight, sleep disturbances, fatigue or low energy, feeling worthlessness, difficulty concentrating, recurrent thoughts of death
Causes:
Biological: genetic predisposition, low levels of serotonin, norepinephrine, dopamine, brain differences in mood regulation
Psychological: negative thinking, learned helplessness, low self-esteem
Behavioral: withdrawal reduces positive reinforcement - deepens depression
Environment: stress, trauma, lack of support
2+weeks+sadness+loss of interest = MDD
Bipolar disorder - includes mania
Persistent depressive disorder - longer-lasting but less severe
Treatments:
CBT: changes negative thinking patterns - improves mood
SSRIs: antidepressant medications (increase serotonin levels)
Electroconvulsive therapy (ECT): used for severe cases when other treatments don’t work
Behavioral activation: encouraging engagement in positive/rewarding activities
Lifestyle factors: exercise, sleep, social support
Persistent Depressive Disorder (Dysthymia)
Chronic depressed mood lasting most of the day, more days than not, for at least 2 year (1 yr in children/teens), with less severe but longer-lasting symptoms than major depression
Symptoms: ongoing low mood, low energy or fatigue, low self-esteem, poor concentration or difficulty making decisions, changes in appetite, sleep problems, feelings of hopelessness
Causes:
Biological: genetic predisposition, neurotransmitter imbalances
Psychological: chronic negative thinking patterns, low self-esteem
Behavioral: reduced engagement in rewarding activities
Environment: long-term stress or difficult life conditions, lack of support
Treatments:
CBT: helps break long-term, negative thought patterns
SSRIs: antidepressant meds similar to MDD
Long-term therapy: focus on chronic patterns and coping strategies
Lifestyle changes: regular routines, exercise, social connection
Seasonal Affective Disorder (SAD)
Symptoms occur at specific times of the year, most commonly during fall and winter when daylight hours are shorter
Symptoms: depressed mood, low energy and fatigue, increased sleep, increased appetite, weight gain, loss of interest in activities, difficulty concentrating
Causes:
Biological: reduced sunlight - disrupts circadian rhythms, decreased serotonin levels, increased melatonin production
Environmental: shorter daylight hours in fall/winter, less exposure to natural sunlight
Treatments:
Light therapy: exposure to bright artificial light to mimic sunlight
SSRIs, CBT
Lifestyle changes: more daylight exposure, regular sleep, exercise
Disruptive Mood Dysregulation Disorder (less common)
Childhood disorder marked by severe, chronic irritability and frequent temper outbursts that are out of proportion to the situation
Symptoms: severe temper outbursts, occur 3 or more times a week, persistently irritable or angry mood
Symptoms present for at least 12 months, diagnosed between ages 6-18
EXAM: DMDD ≠ Bipolar Disorder
Causes:
Combination of genetic vulnerability and environmental stress, difficulty regulating emotions
Treatments:
CBT: emotion regulation, coping skills
Parent training: behavior management strategies
Medication: stimulants, antidepressants, depending on symptoms
*think “constant anger + explosive outburst in kids”
7.7 Bipolar and Related Disorders
Bipolar I Disorder
Presence of at least one full manic episode, often alternating with episodes of depression
Manic episode (KEY CONCEPT):
Inflated self-esteem or grandiosity
Decreased need for sleep
More talkative, racing thoughts
Increased goal-directed activity or agitation, distracted
Risky behaviors
Depressive episodes (common but not required for diagnosis)
Persistent sadness, loss of interest, fatigue, sleep/appetite changes, etc
Mania causes major impairment, may include psychosis (loss of touch with reality)
EXAM: Bipolar I = full mania required
Causes:
Strong genetic component
Brain chemistry (neurotransmitter imbalance)
Stress can trigger episodes
Treatments:
Mood stabilizers (E.x. lithium)
Antipsychotic medications
Psychotherapy (E.x. CBT, psychoeducation)
*think mania = high highs (can be extreme)”
Bipolar II Disorder
Involves at least one hypomanic episode and at least one major depressive episode, without ever having a full manic episode
Hypomanic Episode (KEY CONCEPT)
Elevated or irritable mood, but less severe than mania (lasts at least 4 consecutive days
Overlap symptoms with mania: increased energy, decreased need for sleep, talkative, racing thoughts, distracted, elevated confidence)
No psychosis and does not cause major impairment or require hospitalization
Major depressive episode (required)
Persistent sadness or loss of interest, changes in sleep, appetite, energy, feelings of worthlessness, or difficulty concentrating (must last at least 2 weeks)
EXAM:
Bipolar II ≠ Bipolar I Disorder
Bipolar II = hypomania + depression, Bipolar I = full mania (more severe)
Depression lasts longer and more frequent than hypomania
Hypomania may seem “productive” but is still abnormal
Causes:
Genetic factors
Neurotransmitter imbalance
Stress triggers episodes
Treatments:
Mood stabilizers: E.x. lithium
Antidepressants: used carefully
Psychotherapy: CBT, coping strategies
* think lower highs (hypomania + deep lows (depression)
Cyclothymic Disorder
Frequent mood swings between hypomanic symptoms and mild depressive symptoms, but not severe enough to meet criteria for Bipolar I or II
Symptoms: periods of hypomanic symptoms, periods of depressive symptoms, symptoms are less intense than full hypermania or major depression, mood swings are chronic and ongoing
At least a yr in children (2 yrs in adults)
Symptoms present at least half the time
No symptom-free period longer than 3 months
Cyclothymic = milder but long-lasting mood swings
Less severe than bipolar disorders, but can still impact functioning
May develop into bipolar over time
Causes:
Genetic predisposition
Brain chemistry (neurotransmitters)
Environmental stressors
Treatments:
Psychotherapy: especially CBT
Mood stabilizers: in some cases
Lifestyle regulation (sleep, routines)
* think “chronic, milder ups and downs”
7.8 Schizophrenia Spectrum and Psychotic Disorders
Schizophrenia
Distorted thinking, perceptions, emotions, and behavior, often involving a loss of contact with reality (psychosis)
Core symptoms (VERY TESTED):
Positive Symptoms (ADD something abnormal):
Hallucinations, delusions, disorganized speech, disorganized speech, disorganized or abnormal behavior
Negative symptoms (REMOVE normal behavior):
Reduced emotional expression, lack of motivation, social withdrawal, reduced speech
Symptoms persist for at least 6 months, includes at least 1 month of active symptoms
Causes:
Biological: genetics, brain structure differences
Neurotransmitters: especially excess dopamine activity
EnvironmentL prenatal issues, stress, trauma
Treatment:
Antipsychotic medications: reduces dopamine activity
Psychotherapy: support, coping skills, community support programs
Schizophrenia ≠ “multiple personalities”
About psychosis, not split identity
* think “lost touch with reality (hallucinations+delusions)”
Schizoaffective Disorder
Combines symptoms of schizophrenia with mood disorder symptoms (depression or mania)
Symptoms:
Psychotic symptoms: hallucinations, delusions
Mood episodes: major depressive episodes and/or manic episodes
There must be at least 2 weeks of psychotic symptoms without any mood symptoms
Separates it from mood disorders with psychotic features
Can significantly affect thinking, mood and daily functioning
Often involves cycles of mood symptoms+ongoing psychosis
Causes:
Genetic factors
Brain chemistry: dopamine imbalance
Environmental stressors
Treatments:
Antipsychotic medications: for psychosis
Mood stabilizers or antidepressants: for mood symptoms
Psychotherapy and support
Schizoaffective = psychosis + mood disorder together
Schizophrenia = psychosis only (no major mood episodes required)
Mood disorders with psychotic features = psychosis only during mood episodes
* think “schizo (psychosis) + affective (mood)”
Brief Psychotic Disorder
Sudden onset of psychotic symptoms that last for a short period and then fully resolve
SymptomsL delusions, hallucinations, disorganized speech, grossly disorganized or abnormal behavior
Lasts at least 1 day but less than 1 month, person returns to previous level of functioning (VERY TESTED)
Onset: sudden (often triggered by extreme stress or trauma)
Causes: severe stress, biological vulnerability (genetics, brain chemistry
Treatments:
Short-term antipsychotic medications
Psychological support/therapy
Often revolves completely with treatment
EXAM:
Brief psychotic disorder = short duration (<1 month)
Schizophrenia = long-term (6+ months)
Schizoaffective disorder = psychosis + mood disorder
* think “short, sudden break from reality”
7.9 Trauma- and Stressor-Related Disorders
Post-Traumatic Stress Disorder (PTSD)
Occurs after exposure to a traumatic event, leading to persistent psychological distress and impaired functioning
Symptoms: intrusion (flashbacks, nightmares, intrusive, distressing memories), avoidance (thoughts, feelings, conversations, places, people associated with trauma), negative changes in mood & cognitions, arousal and reactivity (hyperarousal)
Symptoms last more than 1 month
Causes significant distress or impairment
Occurs after a traumatic event
Biological:
Overactive amygdala
Reduced hippocampal function
Stress hormones dysregulations
Learning:
Classical conditioning: neutral cues become associated with trauma
Operant conditioning: avoidance is negatively reinforced
Cognitive:
Maladaptive beliefs (“ the world is unsafe”)
Memory processing problems
Treatments:
CBT: changes negative thought patterns
Exposure therapy: gradual confrontation of trauma-related cues
Eye movement desensitization and reprocessing
SSRIs: sertraline, fluoxetine
Acute Stress Disorder
Occurs shortly after exposure to a traumatic event, with symptoms similar to PTSD but lasting for a shorter duration
Symptoms: same as PTSD
Symptoms occur 3 days to 1 month after trauma
Must have 9+ symptoms across categories
Causes significant distress or impairment
If symptoms last longer than 1 month may become PTSD
Treatments:
Early intervention: trauma-focused CBT, stress management techniques
Medication (less common): short-term use of anti-anxiety meds or sleep aids
*dissociation
7.10 Dissociative Disorders
Dissociative Identity Disorder (DID)
Presence of two or more distinct personality states that recurrently take control of behavior, along with memory gaps that are too extensive to be explained by normal forgetting
Symptoms: multiple identities (each may have different names, traits, voices, or behavior), Amnesia (gaps in recall of everyday events, personal info, or trauma), disruption of identity (sense of self is fragmented), feels detached from oneself, feeling the world is unreal
2 or more distinct identities/personality states
Recurrent gaps in memory
Causes distress or impairment
Causes:
Often linked to severe childhood trauma or abuse
Dissociation acts as a coping mechanism to separate from trauma
Symptoms may be influenced by therapist expectations or media
Role-playing or reinforcement of identities
Differences in brain activity, but not fully understood
EXAM: dissociation - defense mechanism where a person disconnects from thoughts, memories, or identity to reduce psychological distress
Treatments:
Psychotherapy (long-term): integration of identities or improved coordination
CBT techniques: managing symptoms and coping
Dissociative Amnesia
Inability to recall important personal information usually related to trauma or stress, that is too extensive to be explained by ordinary forgetting
Symptoms: memory loss, sudden onset, memory reversible, intact general functioning
Types of dissociative Amnesia:
Localized
Inability to recall events during a specific time period (most common)
Selective
Can recall some, but not all, events from a period
Generalized (rare)
Loss of memory for one’s entire life/identity
Continuous
Ongoing inability to form new memories about current events
Causes:
Trauma-based (most accepted)
Retrieval failure of traumatic memories
No clear brain damage, differs from neurological amnesia
Treatments:
Psychotherapy: helps recover memories safely, addresses underlying trauma
CBT: coping strategies and emotional regulation
EXAM: “can’t remember personal trauma”
7.11 Somatic Symptom and Related Disorders (sometimes included)
Somatic Symptom Disorder
A person experiences significant physical symptoms along with excessive thoughts, feelings, or behaviors related to those symptomes
Symptoms are real, but psychological response is disproportionate
Symptoms: Physical: pain, headaches, stomach issues, fatigue, excessive concern about symptoms, high health-related anxiety, disruption of daily life
Persistent symptoms (6+ months)
Disproportionate thoughts/behaviors about the symptoms
Not fully explained by a medical condition or reaction is excessive relative to it
Causes:
Misinterpretation of normal bodily sensations, catastrophic thinking
Attention and reinforcement
Heightened sensitivity to bodily sensations
Treatments:
CBT: helps challenge catastrophic thinking, reduces symptom focus
Stress management
Sometimes SSRIs for anxiety/depression
EXAM: symptoms are not faked, focus is excessive concern
Illness Anxiety Disorder
Preoccupation with having or developing a serious illness, despite having little or no physical symptoms
mainly about fear of illness, not the symptoms themselves
Symptoms: minimal or no somatic symptoms, high health anxiety, misinterpretation of normal sensations, excessive health behaviors or avoidance
Preoccupation with illness for 6+ months
Disproportionate anxiety about health
Not better explained by another disorder
Causes:
Catastrophic thinking about health
Selective attention to bodily sensations
Reinforcement of worry
Possible predisposition to anxiety
Treatments:
CBT: challenge irrational health beliefs, reduce chacking/avoidance behaviors
SSRIs may help with anxiety
7.12 Feeding and Eating Disorders
Anorexia Nervosa
Eating disorder characterized by significantly low body weight, intense fear of gaining body weight and distorted body image
May involve excessive exercise, fasting, or purging behaviors
Symptoms: extreme dieting, dramatic weight loss, obsession with body image, denial of seriousness of low body weight, social withdrawal and irritability
Causes:
Biological: genetics, abnormal neurotransmitter activity
Psychological: perfectionism, anxiety, low self-esteem
Social/Cultural pressures: thin ideal, media influence
Treatments:
Nutritional rehabilitation: restoring healthy weight
CBT
Family therapy (especially for adolescents
EXAM: anorexia = severe weight loss + restriction of food intake
Bulimia Nervosa
Eating disorder characterized by binge eating followed by compensatory behaviors to prevent weight gain
Often feels a loss of control during binge episodes
Unlike anorexia nervosa, body weight is often normal or slightly above average
Symptoms: repeated episodes of eating unusually large amounts of food in a short time, feeling unable to stop, strong concern with body weight and shape, shame/guilt around eating, frequent bathroom use after meals
Causes:
Biological: genetics, neurotransmitter imbalance
Psychological: low self-esteem, depression, perfectionism
Social/cultural: pressure about appearance and thinness
Treatments:
CBT: most common
Nutritional counselling
Family therapy: in some cases
SSRIs: especially for related depression/anxiety and reducing binge-purge cycles
Binge-Eating Disorder
Recurrent episodes of binge eating without compensatory behaviors
Person eats unusually amounts of food in a short period and feels a loss of control
Unlike Bulimia nervosa, there is not regular purging
No vomiting, laxatives, or excessive exercise to compensate
Symptoms: eating quickly, until uncomfortably full, large amounts when not physically hungry
Causes:
Biological: genetics, brian chemistry
Psychological: stress, depression, low self-esteem
Emotional coping through food, social and environmental influences
EXAM:
Bulimia: bing eating + purging
Bing-eating disorder: binge eating without purging
7.13 Personality Disorders
Cluster A (odd/eccentric)
Paranoid
Persistent distrust and suspicion of others
Believes others may be trying to harm, deceive, or exploit them, even without strong evidence
Unwilling to trust people and may interpret harmless actions as threatening
Symptoms: Constant suspicion of others motives, belief that others are lying, cheating, or taking advantage, reluctant to confide in others, holding grudges, reading hidden threats or insults into normal comments
Causes:
Combination of genetics and early childhood experiences
Trauma, neglect, or harsh family environments may increase risk
Treatments:
Difficult to treat because the person often does not trust therapists
Psychotherapy (talk therapy): main treatment
Building trust is major focus
Medication may help related anxiety or anger, no specific cure
Schizoid
Detachment from social relationships and a limited range of emotional expression
Prefers to be alone and usually has little interest in close friendships or romantic relationships
Often appears emotionally cold, distant or indifferent to praise and criticism
Symptoms: limited emotional expression (“flat”), little desire for relationships including family, prefers solitary activities
Cause:
Exact cause is unclear
Likely combination of genetics and early environmental influences
May be linked to family history of personality disorders or schizophrenia-spectrum disorders
Treatments:
Person may not seek help because they often do not feel distressed by isolation
Psychotherapy: may help improve social functioning
Building trust and communication skills is often the focus
EXAM:
Schizoid: social detachment without hallucinations or delusions
Schizophrenia: psychosis involving hallucinations, delusions, and disorganized thinking
Schizotypal
Odd thinking, unusual beliefs, eccentric behavior, and difficulty forming close relationships
May seem socially anxious, suspicious, and uncomfortable in relationships
May have unusual perceptions or “magical thinking”, but don’t fully lose touch with reality like in psychotic disorders
Symptoms: strange or eccentric behavior or appearance, odd beliefs, unusual speech patterns or thoughts, social anxiety and discomfort with close relationships, suspiciousness or mild paranoia, inappropriate or limited emotional expression
Causes:
Likely influenced by genetics and brian differences
Often linked to family history of schizophrenia
Environmental stress may contribute
Treatments:
Psychotherapy: improve social skills and coping
Sometimes low=dose medication for anxiety, depression, or unusual thinking
Support for social functioning is important
EXAM
Compare “schizo-” personality disorders
Paranoid: distrust and suspicion
Schizoid: detached, prefers isolation
Schizotypal: odd beliefs+eccentric behavior+social discomfort
Cluster B (dramatic/emotional)
Antisocial
Disregard for the rights of others, lack of empathy, and violation of social rules or laws
Person may be manipulative, deceitful, impulsive, and show little guilt or remorse for harmful actions
Sometimes associated with the term “sociopathy” (not the official diagnosis)
Symptoms: repeated lying or manipulation, aggressive or reckless behavior, breaking rules/laws repeatedly, lack of guilt or remorse, impulsive, irresponsible, difficulty maintaining work or relationships
Shows low empathy for others
Causes;
Combination of genetics and environment
Childhood abuse, neglect, harsh parenting, or early conduct problems may increase risk
Often linked to a history of conduct disorder before age 15
Treatments:
Difficult to treat because the person may not believe they need help
Psychotherapy: focus on behavior control and responsibility
No specific medication cures it, but some may help with aggression or impulsivity
EXAM:
Antisocial: violates others rights, little remorse
Borderline: unstable emotions, fear of abandonment, unstable relationships
Borderline
Unstable emotions, relationships, self-image, and behavior
Person has intense fear of abandonment and may experience rapid mood swings
Relationships are often intense and unstable( ”love-hate” pattern)
Symptoms: fear of abandonment, unstable or intense relationships, rapid mood swings, impulsive or risky behaviors, unstable self-image or identity, strong anger or difficulty controlling anger, feelings of emptiness, emotional instability
Causes:
Combination of genetics, brain differences, and environment
Childhood trauma, neglect, or unstable family relationships may increase risk
Treatment:
Dialectical behavior therapy (DBT): type of CBT designed to help people manage intense emotions, reduce harmful behaviors, and improve relationships
Other psychotherapy approaches
Medication: helps symptoms like anxiety or depression but no single cure
Histrionic
Personality disorder characterized by excessive emotionality and attention-seeking behavior
Person often feels uncomfortable when not the center of attention and may use dramatic, flirtatious or theatrical behavior to gain attention
Symptoms: strong need to be the centre of attention, inappropriately sexually seductive, rapidly shifting and shallow emotions, easily influenced by others, considers relationships more intimate than they actually are
Causes
Combination of genetics and environment
Childhood reinforcement of attention-seeking patterns
Temperament and learned behavior patterns
Treatments:
Psychotherapy: main treatment
Focus on developing more stable emotions and healthier relationship patterns
Medication may be used for anxiety or depression, but does not treat the disorder itself
EXAM:
Narcissistic personality disorder: focuses on superiority, admiration, and lack of empathy
Histrionic: focuses on attention-seeking and dramatic emotional expression
Narcissistic
Grandiosity, need for admiration, and lack of empathy
Person has an inflated sense of self-importance and often believes they are superior to others
Symptoms: exaggerated sense of self-importance, preoccupation with fantasies of success/power, need for admiration and attention, sense of entitlement, lack of empathy, arrogant or dismissive attitudes
Causes:
Combination of genetics and environment
Overvaluation or excessive praise in childhood
Inconsistent parenting (overindulgence or extreme criticism)
Treatments:
Psychotherapy: main approach
Focus on building empathy and realistic self-image
Often difficult because individuals may not see their behavior as a problem
Cluster C (anxious/fearful)
Avoidant
Extreme social inhibition, feelings of inadequacy, and hypersensitivity to criticism
Person wants social connection but avoids relationships due to fear of rejection or embarrassment
Symptoms: avoids social situations due to fear of rejection, stong feelings of inadequacy or inferiority, extremely sensitive, reluctance to take personal risks, socially inhibited even though they desire relationships, low self-esteem
Causes:
Combination of genetics and temperament (inborn shyness or anxiety)
Early experiences of rejection, criticism, or social anxiety
Learned fear of social judgment
Treatments:
CBT: challenge negative beliefs about self and others
Social skills training
Gradual exposure to social situations
Medication: anxiety or depression
EXAM;
Schizoid: does not want social relationships
Avoidant: wants relationships but fears rejection and avoids them
Dependent
Excessive need to be taken care of, leading to submissive and clingy behavior
Has difficulty making decisions without reassurance from others and fears being alone or abandoned
Symptoms: difficulty disagreeing with others, people-pleaser, feels helpless or uncomfortable when alone, quickly seeks a new relationship when one ends
Causes:
Overprotective or controlling parenting
Reinforced dependence in childhood
Temperament: anxious or insecure attachment stupe
Learned helplessness over time
Treatments:
CBT: build independence and confidence
Gradual encouragement of decision-making and autonomy
Addressing anxiety about separation
Medication: anxiety or depression
EXAM:
Avoidant: Fears rejection/criticism and avoids relationships
Dependent: fears separation and relies heavily on others for decisions and support
Obsessive-Compulsive Personality Disorder
Preoccupation with orderliness, perfectionism, and control
Overly focused on rules, structure, and doing things “the right way”
*different than obsessive-compulsive disorder, which involves unwanted intrusive thoughts and compulsions
Symptoms: extreme perfectionism that interferes with task completion, devotion to work and productivity, inflexible about morals, ethics, or “correct” behavior, overly controlling in personal and social situations, discomfort when things are messy or out of place
Causes:
Genetic predisposition toward anxiety/perfectionism
Strict or controlling upbringing
Learned need for order and control to reduce anxiety
Treatment:
CBT: reduce rigid thinking patters
Learning flexibility and stress management skills
Medication: helps with anxiety or obsessive thinking is some cases
7.14 Neurodevelopmental Disorders (teacher-dependent but often included)
Attention-Deficit/Hyperactivity Disorder (ADHD)
Persistent patterns of inattention and/or hyperactivity-impulsivity that interfere with functioning or development
Symptoms usually begin in childhood and can continue into adolescence and adulthood
Symptoms: inattention, hyperactivity, impulsivity
Causes:
Strong genetic component
Differences in brain activity: especially attention and impulse-control systems
Neurotransmitter involvement: especially dopamine and norepinephrine
Environmental factors: not the main cause
Treatments:
Stimulant medication: E.x. increase dopamine activity
Behavioral therapy: especially for organization and impulse control
Classroom supports: structure, routines, reduced distractions
Parent training and school interventions
EXAM: ADHD is a neurodevelopmental disorder, not an anxiety or personality disorder
Autism Spectrum Disorder (ASD)
Persistent differences in social communication and restricted or repetitive behaviors/interests
Called a “spectrum” because symptoms range from mild to severe and vary wildly between individuals
Diagnostic areas:
Social communication differences
Difficulty with social interaction and back-and-forth conversation
Challenges understanding social cues
Difficulty forming or maintaining relationships
Restricted/repetitive behaviors
Repetitive movements or speech
Strong need for routines and sameness
Highly focused on intense interests
Sensory sensitivities (sounds, textures, lights)
Treatment/support
No cure, but early intervention improves functioning
Behavioral therapies: applied behavior analysis, etc
Speech and occupational therapy
Social skills training
School accommodations and structured routines
EXAM
ASD = social communication differences+repetitive behaviors
Not explained by fear (like anxiety disorders) or personality traits alone
Parts of the brain