Leveling: Transducer at 4th ICS, mid-axillary line (phlebostatic axis = right atrium = best place to get a reading and zero out device)
Radial is easiest access point for transducer
Purpose: Continuous BP monitoring, frequent ABGs
Complications: Infection, bleeding, thrombosis, neurovascular injury
Assesses the accuracy and functionality of an arterial blood pressure monitoring system. Checks if fluid is properly transmitted to transducer.
Finding | What It Means | Action |
✅ Normal: Square + 1–2 oscillations + dicrotic notch | Accurate reading | No intervention |
⚠ Overdamped | Falsely low BP, no notch | Check for air, clots, kinks, loose connections |
⚠ Underdamped | Falsely high BP, extra oscillations | Shorten tubing, stabilize patient |
Steps: Rapid flush (creates square) → observe waveform → interpret
Normal: 2–8 mmHg
High (>13–15): Hypervolemia / RV failure → Diuretics (Lasix)
Low (<2): Hypovolemia → Fluids
Purpose: Pumps blood from LA → aorta (supports LV)
⚠ Safety: If battery dies → patient dies
Patient Teaching:
Carry spare batteries + charger at all times
Know how/when to change batteries (before alarm)
Avoid water immersion
Have emergency contact info ready
May see cold, clammy, ashen/gray skin from vasoconstriction
Other causes: angina, anxiety, GI, pulmonary
Always: ECG + troponins + full assessment
Type | Description | Relief | Troponins |
Stable | Predictable, exertional | Rest + sublingual nitro | Normal |
Unstable | New/worse, >10 min, unrelieved by rest | Medical emergency (can lead to MI) | Normal |
Silent | Ischemia w/o symptoms (common in DM) | N/A | Normal |
Key: Angina = no cardiac cell death → no troponin rise
Feature | STEMI | NSTEMI |
ECG | ST elevation | No ST elevation |
Troponins | ↑ | ↑ |
Occlusion | Complete blockage | Partial blockage |
Urgency | Emergent: PCI <90 min / thrombolytic <30 min | Urgent cath 12–72 hr |
Thrombolytics | ✅ | ❌ |
Draw ASAP in suspected MI
↑ = STEMI or NSTEMI
Normal = angina
Action:
Vasodilate → ↑ coronary blood flow, ↓ afterload, ↓ O₂ demand
Side Effects (too much): Hypotension, headache
Education:
Sit/lie down before taking
1 dose → wait 5 min
No relief after 3 doses (15 min) → call 911
Keep in dark glass container/same container, replace q6mo
No PDE-5 inhibitors (Viagra, Cialis)
Hypertension
Smoking
Hyperlipidemia
Obesity
Inactivity
Diabetes control
Chest pain
SOB / orthopnea
Dizziness / lightheadedness
Swelling (edema)
Sudden weight gain
Fatigue / weakness
Assessment Findings:
Crackles in lungs
S3 heart sound
Tachycardia
Hypotension (late, when heart can’t compensate)
Hypervolemia
Treatment:
Diuretics (first check potassium)
Noncompliance (not taking meds as prescribed)
Wrong meds or incomplete discharge orders
Cannot afford meds
Daily weights — report >2 lb in 1 day or >5 lb in 1 week
Low sodium diet — ≤2 g/day
Fluid restriction — ≤2 L/day
SNS activation → ↑ HR, ↑ contractility, vasoconstriction
RAAS activation → ↑ sodium & water retention → ↑ preload
Ventricular remodeling → dilation & hypertrophy (long-term damage)
Med | Class/Action | Effect | Side Effects |
Inotropes (dobutamine, milrinone, isoproterenol) | ↑ contractility | ↑ CO | Dobutamine → HTN; milrinone → hypotension |
Nitroprusside | Vasodilator | ↓ afterload | Cyanide toxicity |
Carvedilol | Beta-blocker | ↓ SNS, ↓ afterload | Can mask hypoglycemia, bradycardia |
Torsemide | Loop diuretic | ↓ preload (fluid offload) | Hypokalemia |
Eplerenone | Aldosterone antagonist | ↓ preload | Hyperkalemia |
Rhythm | Key Features | Shockable? | First Steps & Treatment |
Ventricular Tachycardia (VT) | Wide QRS, no P wave, fast rate | ✅ | Check pulse first → With pulse & stable = IV amiodarone. With pulse & unstable = IV amiodarone + synchronized cardioversion; Without pulse = CPR + defibrillation |
Ventricular Fibrillation (VF) | Chaotic, irregular QRS, no CO | ✅ | Defib VFib → CPR immediately |
Asystole | Flat-line, no ventricular activity | ❌ | CPR + ACLS (epi), Do NOT shock |
2nd Degree AV Block Type II | PR normal & consistent, randomly drops QRS | ❌ (pacing, not shock) | Can deteriorate to asystole → pacemaker |
3rd Degree AV Block | P waves & QRS independent, regular P-P & R-R | ❌ (pacing, not shock) | Pacemaker; dopamine/epi as bridge |
Rhythm | Key Features | Treatment |
SVT | Narrow complex tachycardia | Vagal maneuver → adenosine → cardioversion if SBP >90 |
Atrial Fibrillation | Irregularly irregular, no P waves | Rate control (β-blocker, CCB), amiodarone, anticoagulation |
Unstable Tachycardia | Any rapid rhythm causing instability | Immediate cardioversion |
1st Degree AV Block | PR >0.20 sec, consistent | Monitor, not lethal |
2nd Degree AV Block Type I (Wenckebach) | PR gets longer, drops QRS | Usually benign, monitor |
Method | Use For | |
Synchronized Cardioversion | Unstable VT (with pulse), SVT, A-Fib | |
Defibrillation | VFib, pulseless VT |
If PT has pauses in the heart on monitor -> anticipate pacemaker.
AAA (Abdominal Aortic Aneurysm) – Bulge in abdominal aorta
TAA (Thoracic Aortic Aneurysm) – Bulge in thoracic aorta
Dissection – Tear in aortic intima → blood enters media → separation of layers
Any aneurysm: Assess q6–12 hrs for changes
Goal: Detect expansion or rupture early
Older age
Hypertension
Atherosclerosis
Genetic connective tissue disorders (Marfan, Ehlers-Danlos)
Clue: No circulation below diaphragm → Blue Toe Syndrome (poor perfusion to legs)
Possible abdominal bruit
Pulsatile abdominal mass (don’t palpate vigorously!)
Compresses airway/lungs → cough, dyspnea
May cause hoarseness, dysphagia if compressing recurrent laryngeal nerve/esophagus
Grey Turner’s Sign → flank bruising from retroperitoneal bleed
Symptoms:
Sudden, severe tearing/ripping back pain
Hypotension/weakness
Muffled heart sounds (possible pericardial tamponade)
Intervention: Emergency surgery ASAP
Control BP & HR to reduce aortic wall stress
β-blockers, vasodilators as ordered
Surgery if:
Symptomatic
Rapidly expanding
Value | Normal Range |
pH | 7.35 – 7.45 |
PaCO₂ | 35 – 45 mmHg |
HCO₃⁻ | 22 – 26 mEq/L |
PaO₂ | 80 – 100 mmHg |
Look at pH – Acidotic (<7.35) or alkalotic (>7.45)?
Look at PaCO₂ – If abnormal in opposite direction of pH → respiratory cause.
Look at HCO₃⁻ – If abnormal in same direction as pH → metabolic cause.
Compensation:
Uncompensated – opposite value normal
Partially compensated – opposite value abnormal but pH still abnormal
Fully compensated – opposite value abnormal but pH normal
Condition | pH | PaCO₂ | HCO₃⁻ |
Respiratory Acidosis | ↓ | ↑ | Normal or ↑ (comp) |
Respiratory Alkalosis | ↑ | ↓ | Normal or ↓ (comp) |
Metabolic Acidosis | ↓ | Normal or ↓ (comp) | ↓ |
Metabolic Alkalosis | ↑ | Normal or ↑ (comp) | ↑ |
PaO₂ <60 mmHg (hypoxemia) and/or PaCO₂ >45 mmHg (hypercapnia)
Types:
Hypoxemic → Low O₂, normal/low CO₂ (O₂ problem)
Hypercapnic → High CO₂ (ventilation problem)
Cause: Hypoventilation, airway obstruction, neuromuscular weakness
On vent: ↑ Peak Inspiratory Pressure (PIP) = bad (obstruction, bronchospasm, kinked tube, mucus plug)
Fix: Suction, check tubing, bronchodilators
Cause: Inflammatory injury to alveoli → ↑ permeability → pulmonary edema
Criteria:
Acute onset
Refractory hypoxemia
PaO₂/FiO₂ ratio ≤300
Treatment:
Low tidal volume ventilation
↑ PEEP
Prone positioning
Treat cause
HOB ≥30° (prevent aspiration)
Oral hygiene (chlorhexidine rinse per protocol)
Suction as needed (keep airway clear, prevent micro-aspiration)
Daily sedation vacation & readiness-to-wean assessment (if protocol)
If necessary → Paralytic (neuromuscular blocker) per order to improve synchrony with ventilator
Always pair paralytics with sedation & analgesia
Steps:
Remove from ventilator
Manually ventilate with Ambu bag + 100% O₂
Call for help
Bad Respiratory Pattern:
Apneustic (spaced mountains) -> caused by brain stem injury/lesion or CVA
CPP (Cerebral Perfusion Pressure) = MAP − ICP
Normal CPP: 60–100 mmHg
<50 mmHg: Ischemia risk
<30 mmHg: Incompatible with life
MAP (Mean Arterial Pressure) = (SBP + 2 × DBP) ÷ 3
Halo Sign: Clear/yellow CSF ring around blood on dressing
Battle Sign: Bruising over mastoid (possible basilar skull fracture).
Cushing’s Triad:
Widened pulse pressure
Bradycardia
Irregular respirations
Mild: 13–15
Moderate: 9–12
Severe: <8 → consider intubation
Surgical: Craniotomy if indicated
Medical: Mannitol (osmotic diuretic) to pull fluid from brain
Positioning: HOB 30°, neck neutral/midline
Environment: Calm, minimal stimulation
Avoid: Coughing, straining, Valsalva
Temp control: Prevent fever (↑ metabolic demand)
Ventilation: Keep CO₂ in normal range (hypercapnia → vasodilation → ↑ ICP)
Violence (e.g., gunshots, stab wounds)
Sports injuries (contact sports, tackles)
Diving accidents (hitting head on shallow water)
Falls (especially in elderly or high falls)
Primary Injury:
Direct trauma to spinal cord
Penetrating (stab, bullet) or blunt trauma (impact, compression)
Immediate damage
Secondary Injury:
Progressive damage after primary injury
Inflammation, ischemia, edema cause further permanent damage
Monitor carefully for worsening neuro status
Flexion-Rotation injury = Most unstable spinal injury
Cervical injuries (C1–C8) especially risky for breathing
Higher cervical lesions → respiratory muscle paralysis (diaphragm, intercostals)
Occurs in SCIs above T6 and can lead to a life-threatening hypertensive crisis if not managed properly.
Avoid: Full bladder, bowel impaction, tight clothing, SCDs, pain stimuli
Symptoms:
Sudden hypertension (↑BP)
Flushing above injury level (facial)
Bradycardia (slow HR)
Headache, sweating
Emergency!
Sit patient up
Remove triggering factors immediately
Catheterize bladder if needed
Notify provider promptly
Injury below L1–L2 (nerve roots, not spinal cord itself)
Symptoms:
Complete sensory loss in saddle area (between legs, buttocks, inner thighs, back of legs)
Asymmetric lower extremity weakness & pain
Bowel/bladder dysfunction (urgent!)
Needs urgent surgical decompression
Autoimmune ascending paralysis (starts in legs → moves up)
Severe respiratory muscle weakness → monitor closely
Treatment: IV Immunoglobulins (IVIG) or plasmapheresis
CN # & Name | Type | Main Function |
---|---|---|
I – Olfactory | Sensory | Smell |
II – Optic | Sensory | Vision |
III – Oculomotor | Motor | Eye movement (up, down, medial), eyelid opening, pupil constriction |
IV – Trochlear | Motor | Eye movement (down & inward) |
V – Trigeminal | Both | Facial sensation, corneal reflex, chewing |
VI – Abducens | Motor | Lateral eye movement |
VII – Facial | Both | Facial expressions, taste (anterior 2/3 tongue), eyelid closing, saliva/tear production |
VIII – Vestibulocochlear | Sensory | Hearing & balance |
IX – Glossopharyngeal | Both | Taste (posterior 1/3 tongue), gag reflex, swallowing |
X – Vagus | Both | Parasympathetic control of heart, lungs, digestion; gag reflex |
XI – Accessory | Motor | Shoulder shrug & head rotation |
XII – Hypoglossal | Motor | Tongue movement |
Assessments:
CN I Olfactory (Smell)
Assessment: Close 1 nostril and identify familiar scents.
CN II Optic (Vision)
Assessment:
Visual Field: Ask pt to look at bridge of your nose and indicate when object from periphery is seen (peripheral vision).
Visual Acuity: Ask pt to read Snellen chart.
CN III (Oculomotor), CN IV (Trochlear), CN VI (Abducens)
Assessment: Ask pt to hold head steady and assess extraocular muscles by moving finger in 6 directions and PERRLA (Pupils Equal, Round, Reactive to Light & Accommodation)
CN V Trigeminal (facial sensation/corneal reflex/chewing)
Assessment: Ask pt to close eyes and identify light touch & pinpricks on face, clench teeth and assess strength
CN VII Facial (Facial expression/taste anterior 2/3 tongue)
Assessment: Ask pt to raise eyebrows, close eyes tightly, purse lips, smile & frown. Taste test (sweet/salty).
CN VIII Vestibulocochlear (hearing/balance) → Sensory
Assessment:
Hearing: Ask pt to close eyes and indicate when he/she hears rustling of fingertips.
CN IX Glossopharyngeal (swallowing/gag reflex/taste posterior ½ tongue) and CN X Vagus (Parasympathetic to heart, lungs, digestive system, gag reflex)
Assessment: Gag reflex
CN XI Accessory (Sternocleidomastoid/neck and trapezius muscle movement) → Motor
Assessment: Shoulder shrug and turns head side-to-side against resistance.
CN XII Hypoglossal (Tongue movement) → Motor
Assessment: “Stick out your tongue” and push tongue side-to-side against resistance from a tongue blade.
Topic | Key Points |
Ischemic Stroke | Clot → ischemia → thrombectomy + thrombolytics if within window |
Hemorrhagic Stroke | Bleeding → avoid worsening bleed → stop bleed (causes: trauma) |
Right-sided stroke signs | Impulsive, left neglect, memory, safety risks |
*Left-sided stroke signs | Slow, cautious, speech difficulties (expressive aphasia) |
*Aneurysm rupture | Worst headache + N/V → emergency |
*Aphasia | Language impairment → use simple, supportive communication (gestures, yes/no, picture boards) Expressive aphasia: Difficulty producing speech Receptive aphasia: Difficulty understanding speech |