Midterm Study Guide

Hemodynamic Monitoring & Support Devices

๐Ÿ“ Arterial Lines

  • Leveling: Transducer at 4th ICS, mid-axillary line (phlebostatic axis = right atrium = best place to get a reading and zero out device)

    • Radial is easiest access point for transducer

  • Purpose: Continuous BP monitoring, frequent ABGs

  • Complications: Infection, bleeding, thrombosis, neurovascular injury

๐Ÿ“Š Square Wave Test

Assesses the accuracy and functionality of an arterial blood pressure monitoring system. Checks if fluid is properly transmitted to transducer.

Finding

What It Means

Action

โœ… Normal: Square + 1โ€“2 oscillations + dicrotic notch

Accurate reading

No intervention

โš  Overdamped

Falsely low BP, no notch

Check for air, clots, kinks, loose connections

โš  Underdamped

Falsely high BP, extra oscillations

Shorten tubing, stabilize patient

Steps: Rapid flush (creates square) โ†’ observe waveform โ†’ interpret

๐Ÿ’ง Central Venous Pressure (CVP)

  • Normal: 2โ€“8 mmHg

  • High (>13โ€“15): Hypervolemia / RV failure โ†’ Diuretics (Lasix)

  • Low (<2): Hypovolemia โ†’ Fluids

โค LVAD (Left Ventricular Assist Device)

  • Purpose: Pumps blood from LA โ†’ aorta (supports LV)

  • โš  Safety: If battery dies โ†’ patient dies

  • Patient Teaching:

    • Carry spare batteries + charger at all times

    • Know how/when to change batteries (before alarm)

    • Avoid water immersion

    • Have emergency contact info ready

CAD, Angina, ACS, & MI

๐Ÿšจ Not All Chest Pain = MI

  • May see cold, clammy, ashen/gray skin from vasoconstriction

  • Other causes: angina, anxiety, GI, pulmonary

  • Always: ECG + troponins + full assessment

๐Ÿ’ข Angina Quick Table

Type

Description

Relief

Troponins

Stable

Predictable, exertional

Rest + sublingual nitro

Normal

Unstable

New/worse, >10 min, unrelieved by rest

Medical emergency (can lead to MI)

Normal

Silent

Ischemia w/o symptoms (common in DM)

N/A

Normal

Key: Angina = no cardiac cell death โ†’ no troponin rise

๐Ÿ“Š STEMI vs NSTEMI

Feature

STEMI

NSTEMI

ECG

ST elevation

No ST elevation

Troponins

โ†‘

โ†‘

Occlusion

Complete blockage

Partial blockage

Urgency

Emergent: PCI <90 min / thrombolytic <30 min

Urgent cath 12โ€“72 hr

Thrombolytics

โœ…

โŒ

๐Ÿงช Troponins

  • Draw ASAP in suspected MI

  • โ†‘ = STEMI or NSTEMI

  • Normal = angina

๐Ÿ’Š Nitrates

Action:

  • Vasodilate โ†’ โ†‘ coronary blood flow, โ†“ afterload, โ†“ Oโ‚‚ demand

Side Effects (too much): Hypotension, headache

Education:

  1. Sit/lie down before taking

  2. 1 dose โ†’ wait 5 min

  3. No relief after 3 doses (15 min) โ†’ call 911

  4. Keep in dark glass container/same container, replace q6mo

  5. No PDE-5 inhibitors (Viagra, Cialis)

๐Ÿฉบ Modifiable CAD Risks

  • Hypertension

  • Smoking

  • Hyperlipidemia

  • Obesity

  • Inactivity

  • Diabetes control

Congestive Heart Failure (CHF)

๐Ÿฉบ S/S of CHF

  • Chest pain

  • SOB / orthopnea

  • Dizziness / lightheadedness

  • Swelling (edema)

  • Sudden weight gain

  • Fatigue / weakness

  • Assessment Findings:

    • Crackles in lungs

    • S3 heart sound

    • Tachycardia

    • Hypotension (late, when heart canโ€™t compensate)

    • Hypervolemia

  • Treatment:

    • Diuretics (first check potassium)

โš  Common Reasons for Readmission

  • Noncompliance (not taking meds as prescribed)

  • Wrong meds or incomplete discharge orders

  • Cannot afford meds

๐Ÿ“ Patient Education

  • Daily weights โ€” report >2 lb in 1 day or >5 lb in 1 week

  • Low sodium diet โ€” โ‰ค2 g/day

  • Fluid restriction โ€” โ‰ค2 L/day

๐Ÿ”„ Compensation Mechanisms in CHF

  • SNS activation โ†’ โ†‘ HR, โ†‘ contractility, vasoconstriction

  • RAAS activation โ†’ โ†‘ sodium & water retention โ†’ โ†‘ preload

  • Ventricular remodeling โ†’ dilation & hypertrophy (long-term damage)

๐Ÿ’Š Medications for HF Patients

Med

Class/Action

Effect

Side Effects

Inotropes (dobutamine, milrinone, isoproterenol)

โ†‘ contractility

โ†‘ CO

Dobutamine โ†’ HTN; milrinone โ†’ hypotension

Nitroprusside

Vasodilator

โ†“ afterload

Cyanide toxicity

Carvedilol

Beta-blocker

โ†“ SNS, โ†“ afterload

Can mask hypoglycemia, bradycardia

Torsemide

Loop diuretic

โ†“ preload (fluid offload)

Hypokalemia

Eplerenone

Aldosterone antagonist

โ†“ preload

Hyperkalemia

Dysrhythmias & Lethal Rhythms

๐Ÿ’€ Lethal Rhythms

Rhythm

Key Features

Shockable?

First Steps & Treatment

Ventricular Tachycardia (VT)

Wide QRS, no P wave, fast rate

โœ…

Check pulse first โ†’

With pulse & stable = IV amiodarone.

With pulse & unstable = IV amiodarone + synchronized cardioversion;

Without pulse = CPR + defibrillation

Ventricular Fibrillation (VF)

Chaotic, irregular QRS, no CO

โœ…

Defib VFib โ†’ CPR immediately

Asystole

Flat-line, no ventricular activity

โŒ

CPR + ACLS (epi), Do NOT shock

2nd Degree AV Block Type II

PR normal & consistent, randomly drops QRS

โŒ (pacing, not shock)

Can deteriorate to asystole โ†’ pacemaker

3rd Degree AV Block

P waves & QRS independent, regular P-P & R-R

โŒ (pacing, not shock)

Pacemaker; dopamine/epi as bridge

๐Ÿ“Œ Other Key Dysrhythmias

Rhythm

Key Features

Treatment

SVT

Narrow complex tachycardia

Vagal maneuver โ†’ adenosine โ†’ cardioversion if SBP >90

Atrial Fibrillation

Irregularly irregular, no P waves

Rate control (ฮฒ-blocker, CCB), amiodarone, anticoagulation

Unstable Tachycardia

Any rapid rhythm causing instability

Immediate cardioversion

1st Degree AV Block

PR >0.20 sec, consistent

Monitor, not lethal

2nd Degree AV Block Type I (Wenckebach)

PR gets longer, drops QRS

Usually benign, monitor

Synchronized Cardioversion vs. Defibrillation

Method

Use For

Synchronized Cardioversion

Unstable VT (with pulse), SVT,ย  A-Fib

Defibrillation

VFib, pulseless VT

If PT has pauses in the heart on monitor -> anticipate pacemaker.ย 

AAA, TAA, & Dissection

๐Ÿ” Key Definitions

  • AAA (Abdominal Aortic Aneurysm) โ€“ Bulge in abdominal aorta

  • TAA (Thoracic Aortic Aneurysm) โ€“ Bulge in thoracic aorta

  • Dissection โ€“ Tear in aortic intima โ†’ blood enters media โ†’ separation of layers

๐Ÿ•’ Assessment

  • Any aneurysm: Assess q6โ€“12 hrs for changes

  • Goal: Detect expansion or rupture early

โš  Predisposing Factors

  • Older age

  • Hypertension

  • Atherosclerosis

  • Genetic connective tissue disorders (Marfan, Ehlers-Danlos)

๐Ÿ“ AAA Specifics

  • Clue: No circulation below diaphragm โ†’ Blue Toe Syndrome (poor perfusion to legs)

  • Possible abdominal bruit

  • Pulsatile abdominal mass (donโ€™t palpate vigorously!)

๐Ÿ“ TAA Specifics

  • Compresses airway/lungs โ†’ cough, dyspnea

  • May cause hoarseness, dysphagia if compressing recurrent laryngeal nerve/esophagus

๐Ÿ’ฅ AAA Rupture

  • Grey Turnerโ€™s Sign โ†’ flank bruising from retroperitoneal bleed

๐Ÿ’ข Dissection

  • Symptoms:

    • Sudden, severe tearing/ripping back pain

    • Hypotension/weakness

    • Muffled heart sounds (possible pericardial tamponade)

  • Intervention: Emergency surgery ASAP

๐Ÿซ€ Treatment for Any Aneurysm

  • Control BP & HR to reduce aortic wall stress

    • ฮฒ-blockers, vasodilators as ordered

  • Surgery if:

    • Symptomatic

    • Rapidly expanding

ABGs, ARDS, ARF, Respiratory Failure

๐Ÿ“Š ABG Basics

Value

Normal Range

pH

7.35 โ€“ 7.45

PaCOโ‚‚

35 โ€“ 45 mmHg

HCOโ‚ƒโป

22 โ€“ 26 mEq/L

PaOโ‚‚

80 โ€“ 100 mmHg

๐Ÿงฎ Interpreting ABGs

  1. Look at pH โ€“ Acidotic (<7.35) or alkalotic (>7.45)?

  2. Look at PaCOโ‚‚ โ€“ If abnormal in opposite direction of pH โ†’ respiratory cause.

  3. Look at HCOโ‚ƒโป โ€“ If abnormal in same direction as pH โ†’ metabolic cause.

  4. Compensation:

    • Uncompensated โ€“ opposite value normal

    • Partially compensated โ€“ opposite value abnormal but pH still abnormal

    • Fully compensated โ€“ opposite value abnormal but pH normal

๐Ÿ“ Acid-Base Types

Condition

pH

PaCOโ‚‚

HCOโ‚ƒโป

Respiratory Acidosis

โ†“

โ†‘

Normal or โ†‘ (comp)

Respiratory Alkalosis

โ†‘

โ†“

Normal or โ†“ (comp)

Metabolic Acidosis

โ†“

Normal or โ†“ (comp)

โ†“

Metabolic Alkalosis

โ†‘

Normal or โ†‘ (comp)

โ†‘

๐Ÿซ Respiratory Failure

  • PaOโ‚‚ <60 mmHg (hypoxemia) and/or PaCOโ‚‚ >45 mmHg (hypercapnia)

  • Types:

    • Hypoxemic โ†’ Low Oโ‚‚, normal/low COโ‚‚ (Oโ‚‚ problem)

    • Hypercapnic โ†’ High COโ‚‚ (ventilation problem)

๐Ÿ”ด Hypercapnic Respiratory Failure

  • Cause: Hypoventilation, airway obstruction, neuromuscular weakness

  • On vent: โ†‘ Peak Inspiratory Pressure (PIP) = bad (obstruction, bronchospasm, kinked tube, mucus plug)

  • Fix: Suction, check tubing, bronchodilators

๐Ÿ’ฅ ARDS (Acute Respiratory Distress Syndrome)

  • Cause: Inflammatory injury to alveoli โ†’ โ†‘ permeability โ†’ pulmonary edema

  • Criteria:

    • Acute onset

    • Refractory hypoxemia

    • PaOโ‚‚/FiOโ‚‚ ratio โ‰ค300

  • Treatment:

    • Low tidal volume ventilation

    • โ†‘ PEEP

    • Prone positioning

    • Treat cause

Airway & Ventilator Management

๐Ÿ›ก VAP (Ventilator-Associated Pneumonia) Bundle

  • HOB โ‰ฅ30ยฐ (prevent aspiration)

  • Oral hygiene (chlorhexidine rinse per protocol)

  • Suction as needed (keep airway clear, prevent micro-aspiration)

  • Daily sedation vacation & readiness-to-wean assessment (if protocol)

๐Ÿ’ข If Pt is โ€œFighting the Ventโ€

  • If necessary โ†’ Paralytic (neuromuscular blocker) per order to improve synchrony with ventilator

  • Always pair paralytics with sedation & analgesia

๐Ÿšจ Sudden Respiratory Distress on Vent

Steps:

  1. Remove from ventilator

  2. Manually ventilate with Ambu bag + 100% Oโ‚‚

  3. Call for helpย 

Bad Respiratory Pattern:ย 

  • Apneustic (spaced mountains) -> caused by brain stem injury/lesion or CVA

Acute Intracranial Problems

๐Ÿงฎ Key Formulas

  • CPP (Cerebral Perfusion Pressure) = MAP โˆ’ ICP

    • Normal CPP: 60โ€“100 mmHg

    • <50 mmHg: Ischemia risk

    • <30 mmHg: Incompatible with life

  • MAP (Mean Arterial Pressure) = (SBP + 2 ร— DBP) รท 3

โš  Signs of Increased ICP

  • Halo Sign: Clear/yellow CSF ring around blood on dressing

  • Battle Sign: Bruising over mastoid (possible basilar skull fracture).

  • Cushingโ€™s Triad:

    • Widened pulse pressure

    • Bradycardia

    • Irregular respirations

๐Ÿ“Š Glasgow Coma Scale (GCS)

  • Mild: 13โ€“15

  • Moderate: 9โ€“12

  • Severe: <8 โ†’ consider intubation

๐Ÿ’ฅ Management of High ICP

  • Surgical: Craniotomy if indicated

  • Medical: Mannitol (osmotic diuretic) to pull fluid from brain

  • Positioning: HOB 30ยฐ, neck neutral/midline

  • Environment: Calm, minimal stimulation

  • Avoid: Coughing, straining, Valsalva

  • Temp control: Prevent fever (โ†‘ metabolic demand)

  • Ventilation: Keep COโ‚‚ in normal range (hypercapnia โ†’ vasodilation โ†’ โ†‘ ICP)

Spinal Cord Injuries (SCI)

1. Causes of Spinal Cord Injuries

  • Violence (e.g., gunshots, stab wounds)

  • Sports injuries (contact sports, tackles)

  • Diving accidents (hitting head on shallow water)

  • Falls (especially in elderly or high falls)

2. Types of SCI Injury

  • Primary Injury:

    • Direct trauma to spinal cord

      • Penetrating (stab, bullet) or blunt trauma (impact, compression)

    • Immediate damage

  • Secondary Injury:

    • Progressive damage after primary injury

      • Inflammation, ischemia, edema cause further permanent damage

    • Monitor carefully for worsening neuro status

3. Most Unstable Injury

  • Flexion-Rotation injury = Most unstable spinal injury

4. Respiratory Concerns in SCI

  • Cervical injuries (C1โ€“C8) especially risky for breathing

  • Higher cervical lesions โ†’ respiratory muscle paralysis (diaphragm, intercostals)

5. Autonomic Dysreflexia (AD)

  • Occurs in SCIs above T6 and can lead to a life-threatening hypertensive crisis if not managed properly.

  • Avoid: Full bladder, bowel impaction, tight clothing, SCDs, pain stimuli

  • Symptoms:

    • Sudden hypertension (โ†‘BP)

    • Flushing above injury level (facial)

    • Bradycardia (slow HR)

    • Headache, sweating

  • Emergency!

    • Sit patient up

    • Remove triggering factors immediately

    • Catheterize bladder if needed

    • Notify provider promptly

6. Cauda Equina Syndrome (CES)

  • Injury below L1โ€“L2 (nerve roots, not spinal cord itself)

  • Symptoms:

    • Complete sensory loss in saddle area (between legs, buttocks, inner thighs, back of legs)

    • Asymmetric lower extremity weakness & pain

    • Bowel/bladder dysfunction (urgent!)

  • Needs urgent surgical decompression

7. Guillain-Barrรฉ Syndrome (GBS)

  • Autoimmune ascending paralysis (starts in legs โ†’ moves up)

  • Severe respiratory muscle weakness โ†’ monitor closely

  • Treatment: IV Immunoglobulins (IVIG) or plasmapheresis

Cranial Nerves

CN # & Name

Type

Main Function

I โ€“ Olfactory

Sensory

Smell

II โ€“ Optic

Sensory

Vision

III โ€“ Oculomotor

Motor

Eye movement (up, down, medial), eyelid opening, pupil constriction

IV โ€“ Trochlear

Motor

Eye movement (down & inward)

V โ€“ Trigeminal

Both

Facial sensation, corneal reflex, chewing

VI โ€“ Abducens

Motor

Lateral eye movement

VII โ€“ Facial

Both

Facial expressions, taste (anterior 2/3 tongue), eyelid closing, saliva/tear production

VIII โ€“ Vestibulocochlear

Sensory

Hearing & balance

IX โ€“ Glossopharyngeal

Both

Taste (posterior 1/3 tongue), gag reflex, swallowing

X โ€“ Vagus

Both

Parasympathetic control of heart, lungs, digestion; gag reflex

XI โ€“ Accessory

Motor

Shoulder shrug & head rotation

XII โ€“ Hypoglossal

Motor

Tongue movement

Assessments:

  • CN I Olfactory (Smell)

    • Assessment: Close 1 nostril and identify familiar scents.

  • CN II Optic (Vision)

    • Assessment:

      • Visual Field: Ask pt to look at bridge of your nose and indicate when object from periphery is seen (peripheral vision).

      • Visual Acuity: Ask pt to read Snellen chart.

  • CN III (Oculomotor), CN IV (Trochlear), CN VI (Abducens)

    • Assessment: Ask pt to hold head steady and assess extraocular muscles by moving finger in 6 directions and PERRLA (Pupils Equal, Round, Reactive to Light & Accommodation)

  • CN V Trigeminal (facial sensation/corneal reflex/chewing)

    • Assessment: Ask pt to close eyes and identify light touch & pinpricks on face, clench teeth and assess strength

  • CN VII Facial (Facial expression/taste anterior 2/3 tongue)

    • Assessment: Ask pt to raise eyebrows, close eyes tightly, purse lips, smile & frown. Taste test (sweet/salty).

  • CN VIII Vestibulocochlear (hearing/balance) โ†’ Sensory

    • Assessment:

      • Hearing: Ask pt to close eyes and indicate when he/she hears rustling of fingertips.

  • CN IX Glossopharyngeal (swallowing/gag reflex/taste posterior ยฝ tongue) and CN X Vagus (Parasympathetic to heart, lungs, digestive system, gag reflex)

    • Assessment: Gag reflex

  • CN XI Accessory (Sternocleidomastoid/neck and trapezius muscle movement) โ†’ Motor

    • Assessment: Shoulder shrug and turns head side-to-side against resistance.

  • CN XII Hypoglossal (Tongue movement) โ†’ Motor

    • Assessment: โ€œStick out your tongueโ€ and push tongue side-to-side against resistance from a tongue blade.

Strokeย 

Topic

Key Points

Ischemic Stroke

Clot โ†’ ischemia โ†’ thrombectomy + thrombolytics if within window

Hemorrhagic Stroke

Bleeding โ†’ avoid worsening bleed โ†’ stop bleed (causes: trauma)

Right-sided stroke signs

Impulsive, left neglect, memory, safety risks

*Left-sided stroke signs

Slow, cautious, speech difficulties (expressive aphasia)

*Aneurysm rupture

Worst headache + N/V โ†’ emergency

*Aphasia

Language impairment โ†’ use simple, supportive communication (gestures, yes/no, picture boards)


Expressive aphasia: Difficulty producing speech

Receptive aphasia: Difficulty understanding speech