Midterm Study Guide
Hemodynamic Monitoring & Support Devices
๐ Arterial Lines
Leveling: Transducer at 4th ICS, mid-axillary line (phlebostatic axis = right atrium = best place to get a reading and zero out device)
Radial is easiest access point for transducer
Purpose: Continuous BP monitoring, frequent ABGs
Complications: Infection, bleeding, thrombosis, neurovascular injury
๐ Square Wave Test
Assesses the accuracy and functionality of an arterial blood pressure monitoring system. Checks if fluid is properly transmitted to transducer.
Finding | What It Means | Action |
โ Normal: Square + 1โ2 oscillations + dicrotic notch | Accurate reading | No intervention |
โ Overdamped | Falsely low BP, no notch | Check for air, clots, kinks, loose connections |
โ Underdamped | Falsely high BP, extra oscillations | Shorten tubing, stabilize patient |
Steps: Rapid flush (creates square) โ observe waveform โ interpret
๐ง Central Venous Pressure (CVP)
Normal: 2โ8 mmHg
High (>13โ15): Hypervolemia / RV failure โ Diuretics (Lasix)
Low (<2): Hypovolemia โ Fluids
โค LVAD (Left Ventricular Assist Device)
Purpose: Pumps blood from LA โ aorta (supports LV)
โ Safety: If battery dies โ patient dies
Patient Teaching:
Carry spare batteries + charger at all times
Know how/when to change batteries (before alarm)
Avoid water immersion
Have emergency contact info ready
CAD, Angina, ACS, & MI
๐จ Not All Chest Pain = MI
May see cold, clammy, ashen/gray skin from vasoconstriction
Other causes: angina, anxiety, GI, pulmonary
Always: ECG + troponins + full assessment
๐ข Angina Quick Table
Type | Description | Relief | Troponins |
Stable | Predictable, exertional | Rest + sublingual nitro | Normal |
Unstable | New/worse, >10 min, unrelieved by rest | Medical emergency (can lead to MI) | Normal |
Silent | Ischemia w/o symptoms (common in DM) | N/A | Normal |
Key: Angina = no cardiac cell death โ no troponin rise
๐ STEMI vs NSTEMI
Feature | STEMI | NSTEMI |
ECG | ST elevation | No ST elevation |
Troponins | โ | โ |
Occlusion | Complete blockage | Partial blockage |
Urgency | Emergent: PCI <90 min / thrombolytic <30 min | Urgent cath 12โ72 hr |
Thrombolytics | โ | โ |
๐งช Troponins
Draw ASAP in suspected MI
โ = STEMI or NSTEMI
Normal = angina
๐ Nitrates
Action:
Vasodilate โ โ coronary blood flow, โ afterload, โ Oโ demand
Side Effects (too much): Hypotension, headache
Education:
Sit/lie down before taking
1 dose โ wait 5 min
No relief after 3 doses (15 min) โ call 911
Keep in dark glass container/same container, replace q6mo
No PDE-5 inhibitors (Viagra, Cialis)
๐ฉบ Modifiable CAD Risks
Hypertension
Smoking
Hyperlipidemia
Obesity
Inactivity
Diabetes control
Congestive Heart Failure (CHF)
๐ฉบ S/S of CHF
Chest pain
SOB / orthopnea
Dizziness / lightheadedness
Swelling (edema)
Sudden weight gain
Fatigue / weakness
Assessment Findings:
Crackles in lungs
S3 heart sound
Tachycardia
Hypotension (late, when heart canโt compensate)
Hypervolemia
Treatment:
Diuretics (first check potassium)
โ Common Reasons for Readmission
Noncompliance (not taking meds as prescribed)
Wrong meds or incomplete discharge orders
Cannot afford meds
๐ Patient Education
Daily weights โ report >2 lb in 1 day or >5 lb in 1 week
Low sodium diet โ โค2 g/day
Fluid restriction โ โค2 L/day
๐ Compensation Mechanisms in CHF
SNS activation โ โ HR, โ contractility, vasoconstriction
RAAS activation โ โ sodium & water retention โ โ preload
Ventricular remodeling โ dilation & hypertrophy (long-term damage)
๐ Medications for HF Patients
Med | Class/Action | Effect | Side Effects |
Inotropes (dobutamine, milrinone, isoproterenol) | โ contractility | โ CO | Dobutamine โ HTN; milrinone โ hypotension |
Nitroprusside | Vasodilator | โ afterload | Cyanide toxicity |
Carvedilol | Beta-blocker | โ SNS, โ afterload | Can mask hypoglycemia, bradycardia |
Torsemide | Loop diuretic | โ preload (fluid offload) | Hypokalemia |
Eplerenone | Aldosterone antagonist | โ preload | Hyperkalemia |
Dysrhythmias & Lethal Rhythms
๐ Lethal Rhythms
Rhythm | Key Features | Shockable? | First Steps & Treatment |
Ventricular Tachycardia (VT) | Wide QRS, no P wave, fast rate | โ | Check pulse first โ With pulse & stable = IV amiodarone. With pulse & unstable = IV amiodarone + synchronized cardioversion; Without pulse = CPR + defibrillation |
Ventricular Fibrillation (VF) | Chaotic, irregular QRS, no CO | โ | Defib VFib โ CPR immediately |
Asystole | Flat-line, no ventricular activity | โ | CPR + ACLS (epi), Do NOT shock |
2nd Degree AV Block Type II | PR normal & consistent, randomly drops QRS | โ (pacing, not shock) | Can deteriorate to asystole โ pacemaker |
3rd Degree AV Block | P waves & QRS independent, regular P-P & R-R | โ (pacing, not shock) | Pacemaker; dopamine/epi as bridge |
๐ Other Key Dysrhythmias
Rhythm | Key Features | Treatment |
SVT | Narrow complex tachycardia | Vagal maneuver โ adenosine โ cardioversion if SBP >90 |
Atrial Fibrillation | Irregularly irregular, no P waves | Rate control (ฮฒ-blocker, CCB), amiodarone, anticoagulation |
Unstable Tachycardia | Any rapid rhythm causing instability | Immediate cardioversion |
1st Degree AV Block | PR >0.20 sec, consistent | Monitor, not lethal |
2nd Degree AV Block Type I (Wenckebach) | PR gets longer, drops QRS | Usually benign, monitor |
Synchronized Cardioversion vs. Defibrillation
Method | Use For | |
Synchronized Cardioversion | Unstable VT (with pulse), SVT,ย A-Fib | |
Defibrillation | VFib, pulseless VT |
If PT has pauses in the heart on monitor -> anticipate pacemaker.ย
AAA, TAA, & Dissection
๐ Key Definitions
AAA (Abdominal Aortic Aneurysm) โ Bulge in abdominal aorta
TAA (Thoracic Aortic Aneurysm) โ Bulge in thoracic aorta
Dissection โ Tear in aortic intima โ blood enters media โ separation of layers
๐ Assessment
Any aneurysm: Assess q6โ12 hrs for changes
Goal: Detect expansion or rupture early
โ Predisposing Factors
Older age
Hypertension
Atherosclerosis
Genetic connective tissue disorders (Marfan, Ehlers-Danlos)
๐ AAA Specifics
Clue: No circulation below diaphragm โ Blue Toe Syndrome (poor perfusion to legs)
Possible abdominal bruit
Pulsatile abdominal mass (donโt palpate vigorously!)
๐ TAA Specifics
Compresses airway/lungs โ cough, dyspnea
May cause hoarseness, dysphagia if compressing recurrent laryngeal nerve/esophagus
๐ฅ AAA Rupture
Grey Turnerโs Sign โ flank bruising from retroperitoneal bleed
๐ข Dissection
Symptoms:
Sudden, severe tearing/ripping back pain
Hypotension/weakness
Muffled heart sounds (possible pericardial tamponade)
Intervention: Emergency surgery ASAP
๐ซ Treatment for Any Aneurysm
Control BP & HR to reduce aortic wall stress
ฮฒ-blockers, vasodilators as ordered
Surgery if:
Symptomatic
Rapidly expanding
ABGs, ARDS, ARF, Respiratory Failure
๐ ABG Basics
Value | Normal Range |
pH | 7.35 โ 7.45 |
PaCOโ | 35 โ 45 mmHg |
HCOโโป | 22 โ 26 mEq/L |
PaOโ | 80 โ 100 mmHg |
๐งฎ Interpreting ABGs
Look at pH โ Acidotic (<7.35) or alkalotic (>7.45)?
Look at PaCOโ โ If abnormal in opposite direction of pH โ respiratory cause.
Look at HCOโโป โ If abnormal in same direction as pH โ metabolic cause.
Compensation:
Uncompensated โ opposite value normal
Partially compensated โ opposite value abnormal but pH still abnormal
Fully compensated โ opposite value abnormal but pH normal
๐ Acid-Base Types
Condition | pH | PaCOโ | HCOโโป |
Respiratory Acidosis | โ | โ | Normal or โ (comp) |
Respiratory Alkalosis | โ | โ | Normal or โ (comp) |
Metabolic Acidosis | โ | Normal or โ (comp) | โ |
Metabolic Alkalosis | โ | Normal or โ (comp) | โ |
๐ซ Respiratory Failure
PaOโ <60 mmHg (hypoxemia) and/or PaCOโ >45 mmHg (hypercapnia)
Types:
Hypoxemic โ Low Oโ, normal/low COโ (Oโ problem)
Hypercapnic โ High COโ (ventilation problem)
๐ด Hypercapnic Respiratory Failure
Cause: Hypoventilation, airway obstruction, neuromuscular weakness
On vent: โ Peak Inspiratory Pressure (PIP) = bad (obstruction, bronchospasm, kinked tube, mucus plug)
Fix: Suction, check tubing, bronchodilators
๐ฅ ARDS (Acute Respiratory Distress Syndrome)
Cause: Inflammatory injury to alveoli โ โ permeability โ pulmonary edema
Criteria:
Acute onset
Refractory hypoxemia
PaOโ/FiOโ ratio โค300
Treatment:
Low tidal volume ventilation
โ PEEP
Prone positioning
Treat cause
Airway & Ventilator Management
๐ก VAP (Ventilator-Associated Pneumonia) Bundle
HOB โฅ30ยฐ (prevent aspiration)
Oral hygiene (chlorhexidine rinse per protocol)
Suction as needed (keep airway clear, prevent micro-aspiration)
Daily sedation vacation & readiness-to-wean assessment (if protocol)
๐ข If Pt is โFighting the Ventโ
If necessary โ Paralytic (neuromuscular blocker) per order to improve synchrony with ventilator
Always pair paralytics with sedation & analgesia
๐จ Sudden Respiratory Distress on Vent
Steps:
Remove from ventilator
Manually ventilate with Ambu bag + 100% Oโ
Call for helpย
Bad Respiratory Pattern:ย
Apneustic (spaced mountains) -> caused by brain stem injury/lesion or CVA
Acute Intracranial Problems
๐งฎ Key Formulas
CPP (Cerebral Perfusion Pressure) = MAP โ ICP
Normal CPP: 60โ100 mmHg
<50 mmHg: Ischemia risk
<30 mmHg: Incompatible with life
MAP (Mean Arterial Pressure) = (SBP + 2 ร DBP) รท 3
โ Signs of Increased ICP
Halo Sign: Clear/yellow CSF ring around blood on dressing
Battle Sign: Bruising over mastoid (possible basilar skull fracture).
Cushingโs Triad:
Widened pulse pressure
Bradycardia
Irregular respirations
๐ Glasgow Coma Scale (GCS)
Mild: 13โ15
Moderate: 9โ12
Severe: <8 โ consider intubation
๐ฅ Management of High ICP
Surgical: Craniotomy if indicated
Medical: Mannitol (osmotic diuretic) to pull fluid from brain
Positioning: HOB 30ยฐ, neck neutral/midline
Environment: Calm, minimal stimulation
Avoid: Coughing, straining, Valsalva
Temp control: Prevent fever (โ metabolic demand)
Ventilation: Keep COโ in normal range (hypercapnia โ vasodilation โ โ ICP)
Spinal Cord Injuries (SCI)
1. Causes of Spinal Cord Injuries
Violence (e.g., gunshots, stab wounds)
Sports injuries (contact sports, tackles)
Diving accidents (hitting head on shallow water)
Falls (especially in elderly or high falls)
2. Types of SCI Injury
Primary Injury:
Direct trauma to spinal cord
Penetrating (stab, bullet) or blunt trauma (impact, compression)
Immediate damage
Secondary Injury:
Progressive damage after primary injury
Inflammation, ischemia, edema cause further permanent damage
Monitor carefully for worsening neuro status
3. Most Unstable Injury
Flexion-Rotation injury = Most unstable spinal injury
4. Respiratory Concerns in SCI
Cervical injuries (C1โC8) especially risky for breathing
Higher cervical lesions โ respiratory muscle paralysis (diaphragm, intercostals)
5. Autonomic Dysreflexia (AD)
Occurs in SCIs above T6 and can lead to a life-threatening hypertensive crisis if not managed properly.
Avoid: Full bladder, bowel impaction, tight clothing, SCDs, pain stimuli
Symptoms:
Sudden hypertension (โBP)
Flushing above injury level (facial)
Bradycardia (slow HR)
Headache, sweating
Emergency!
Sit patient up
Remove triggering factors immediately
Catheterize bladder if needed
Notify provider promptly
6. Cauda Equina Syndrome (CES)
Injury below L1โL2 (nerve roots, not spinal cord itself)
Symptoms:
Complete sensory loss in saddle area (between legs, buttocks, inner thighs, back of legs)
Asymmetric lower extremity weakness & pain
Bowel/bladder dysfunction (urgent!)
Needs urgent surgical decompression
7. Guillain-Barrรฉ Syndrome (GBS)
Autoimmune ascending paralysis (starts in legs โ moves up)
Severe respiratory muscle weakness โ monitor closely
Treatment: IV Immunoglobulins (IVIG) or plasmapheresis
Cranial Nerves
CN # & Name | Type | Main Function |
|---|---|---|
I โ Olfactory | Sensory | Smell |
II โ Optic | Sensory | Vision |
III โ Oculomotor | Motor | Eye movement (up, down, medial), eyelid opening, pupil constriction |
IV โ Trochlear | Motor | Eye movement (down & inward) |
V โ Trigeminal | Both | Facial sensation, corneal reflex, chewing |
VI โ Abducens | Motor | Lateral eye movement |
VII โ Facial | Both | Facial expressions, taste (anterior 2/3 tongue), eyelid closing, saliva/tear production |
VIII โ Vestibulocochlear | Sensory | Hearing & balance |
IX โ Glossopharyngeal | Both | Taste (posterior 1/3 tongue), gag reflex, swallowing |
X โ Vagus | Both | Parasympathetic control of heart, lungs, digestion; gag reflex |
XI โ Accessory | Motor | Shoulder shrug & head rotation |
XII โ Hypoglossal | Motor | Tongue movement |
Assessments:
CN I Olfactory (Smell)
Assessment: Close 1 nostril and identify familiar scents.
CN II Optic (Vision)
Assessment:
Visual Field: Ask pt to look at bridge of your nose and indicate when object from periphery is seen (peripheral vision).
Visual Acuity: Ask pt to read Snellen chart.
CN III (Oculomotor), CN IV (Trochlear), CN VI (Abducens)
Assessment: Ask pt to hold head steady and assess extraocular muscles by moving finger in 6 directions and PERRLA (Pupils Equal, Round, Reactive to Light & Accommodation)
CN V Trigeminal (facial sensation/corneal reflex/chewing)
Assessment: Ask pt to close eyes and identify light touch & pinpricks on face, clench teeth and assess strength
CN VII Facial (Facial expression/taste anterior 2/3 tongue)
Assessment: Ask pt to raise eyebrows, close eyes tightly, purse lips, smile & frown. Taste test (sweet/salty).
CN VIII Vestibulocochlear (hearing/balance) โ Sensory
Assessment:
Hearing: Ask pt to close eyes and indicate when he/she hears rustling of fingertips.
CN IX Glossopharyngeal (swallowing/gag reflex/taste posterior ยฝ tongue) and CN X Vagus (Parasympathetic to heart, lungs, digestive system, gag reflex)
Assessment: Gag reflex
CN XI Accessory (Sternocleidomastoid/neck and trapezius muscle movement) โ Motor
Assessment: Shoulder shrug and turns head side-to-side against resistance.
CN XII Hypoglossal (Tongue movement) โ Motor
Assessment: โStick out your tongueโ and push tongue side-to-side against resistance from a tongue blade.
Strokeย
Topic | Key Points |
Ischemic Stroke | Clot โ ischemia โ thrombectomy + thrombolytics if within window |
Hemorrhagic Stroke | Bleeding โ avoid worsening bleed โ stop bleed (causes: trauma) |
Right-sided stroke signs | Impulsive, left neglect, memory, safety risks |
*Left-sided stroke signs | Slow, cautious, speech difficulties (expressive aphasia) |
*Aneurysm rupture | Worst headache + N/V โ emergency |
*Aphasia | Language impairment โ use simple, supportive communication (gestures, yes/no, picture boards) Expressive aphasia: Difficulty producing speech Receptive aphasia: Difficulty understanding speech |