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Fluid Excess – Edema
Edema: Excessive fluid in the interstitial compartment, causing tissue swelling or enlargement.
Characteristics:
Localized or generalized throughout the body.
May impair tissue perfusion and trap drugs in interstitial fluid.
Causes of Edema
Increased Capillary Hydrostatic Pressure:
Caused by higher blood pressure/increased blood volume.
Forces more fluid out of capillaries into tissues (e.g., pulmonary edema).
Loss of Plasma Proteins (e.g., Albumin):
Results in decreased plasma osmotic pressure.
Obstruction of Lymphatic Circulation:
Causes localized edema due to excessive fluid and protein not returning to the general circulation.
Increased Capillary Permeability:
Usually localized, may result from inflammatory responses or infections.
Histamines and other mediators increase capillary permeability; bacterial toxins/burns can cause widespread edema.
Effects of Edema
Swelling:
May appear pale or red.
Pitting Edema: Molds to pressure, leaving a pit once released.
Physiological Impacts:
Increase in body weight.
Functional impairment (joint movement).
Reduced vital capacity.
Pain due to pressure on nerves.
Ischemia can lead to tissue breakdown.
Lymphatic Filariasis (Elephantiasis)
More common in tropics; caused by nematodes from the family Filariodidea (e.g., Wuchereria bancrofti).
Majority of infections asymptomatic, damaging lymphatic system.
Acute inflammation results from infections where defenses are lost due to lymphatic damage.
Chronic conditions lead to complications: lymphoedema, elephantiasis, hydrocele.
Socioeconomic Impacts
Body deformities lead to social stigma and financial hardship due to loss of income and increased expenses.
Pathophysiology
Infection occurs through mosquito transmission of larvae to lymphatic vessels.
Adult worms block lymph drainage.
Usually acquired in childhood, visible symptoms develop later.
Diagnosis and Treatment
Diagnosed through blood sample examination.
Treatment includes anti-worm medications, compression clothing, and surgery.
Fluid Deficit – Dehydration
Causes:
Insufficient intake or excessive loss, often measured by weight changes.
More serious in infants and older adults; can include water and electrolyte loss during diarrhea.
Manifestations
Symptoms include decreased skin turgor, dry membranes, sunken eyes, and low blood pressure.
Urine analysis shows low volume but high specific gravity.
Compensatory Responses
Increasing thirst, heart rate, and constriction of cutaneous blood vessels to concentrate urine.
Fluid Excess
Two forms:
Volume Excess: Retain both sodium and water, often from renal failure.
Hypotonic hydration: More water than sodium is retained, leading to cellular swelling.
Sodium Imbalance
Hyponatremia:
Causes: excessive loss from sweating, vomiting, diuretics, hormonal imbalances.
Effects: fatigue, muscle cramps, and confusion.
Hypernatremia:
Causes: insufficient ADH, water loss, and excessive sodium.
Effects: weakness, dry membranes, agitation.
Potassium Imbalance
Hypokalemia:
Caused by losses through diarrhea or diuretics, affects cardiac rhythm and neuromuscular function.
Hyperkalemia:
Causes include renal failure, and can lead to muscle weakness and cardiac arrest.
Calcium Imbalance
Hypocalcemia:
Causes: hypoparathyroidism, malabsorption, renal issues.
Effects: muscle twitching, dysrhythmias.
Hypercalcemia:
Causes: bone malignancies, hyperparathyroidism.
Effects: muscle weakness and cardiac changes.
pH Imbalance
Acidosis: Increased hydrogen ions decrease pH; caused by respiratory/metabolic issues.
Alkalosis: Deficit of hydrogen ions; increases pH; can result from hyperventilation or excessive bicarbonate.
Effects: Both conditions can lead to nervous issues and muscle twitching.
Compensation and Decompensation
Compensation mechanisms are often short-lived and cannot address root causes; decompensation occurs when problems worsen or additional issues arise.