Biology 173 - Intro Bio Lab Unit 1; Enzyme Action, Gut Microbiome, & Basic PCR
Lecture 1: Intro
Scientific Method:
Explore/Observe → Ask a Question → Conduct Research → For a Hypothesis → Experiment → Analyze data and Form Conclusions
→ Do results support hypothesis?
Yes → Report Results
No → Go back to hypothesis, retesting
Observations
Qualitative: recording descriptions, describing, thinking of things
Quantitative: numerical measurement, organized into tables and graphs
Hypothesis: possible explanation that leads to a testable prediction and motivates experiments
More broad
Based on observations and assumptions
NOT an “educated guess:, “if-then” statement, or “if I do X, Y will occur”
Must be falsifiable; must make specific predictions that can be critically tested by experiments or observational studies
Prediction: a description of an expected outcome in the test (experimental) group and in each control group
Confirming a prediction supports (but does not “prove”) that hypothesis
Negating a prediction suggests that it may be wrong
Ex.
Observation: When you turn it on, your desk lamp doesn’t work
Question: Why doesn’t your desk lamp work?
Hypothesis 1: Bulb is burnt out
Hypothesis 2: Bulb is not plugged in; it’s loose
Some hypotheses are untestable; supernatural and religious explanations are outside the bounds of this course
Ex. If a plant receives fertilizer, then it will grow to be bigger than a plant that doesn’t receive fertilizer.
This is NOT a hypothesis; hypothesis might be “Plant growth is affected by fertilizer.”
Forming and Testing Hypotheses
Controlled Experiment
A single variable is changed in the experimental group and compared to the control group
Independent variable: manipulated/changed by researchers
Dependent variable: affected in response to the independent variable
Controls: makes sure other factors aren’t influencing the experiment → is the independent variable really the thing influencing the results?
Provides a baseline value for comparison
Makes sure everything is working properly
Negative Control: variable is not changed; expect no effect
Ex. Placebo, Saline
Positive Control: variable with a known effect; expect a specific effect
Another cancer drug that you know works
Dilutions:
Ex. Making a solution that is ⅓ its original concentration; how much solution + how much water?
⅓ concentration → 1 out of 3 parts
1 part substance + 2 parts water = 3 total parts
100µ substance + 200µ water = 300µ
Ex. 60% = 60/100 = 60 parts out of 100 parts
60µ substance + 40µ water = 100µ total
Stepwise/Serial Dilutions:
Step-wise series of dilutions, where the dilution factor states the same for each step
Each new dilution sues the PREVIOUS solution as the source; add H2O to dilute
Spectrophotometer:
Instrument that measure light absorbance of samples
Absorbance: light absorbed by a sample
Transmittance: amount of light passing through a sample
Product is proportional to the amount of light that passes through
Ecology I: Species Interactions
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Lecture 2: Enzyme Action, Part I
Bioluminescence = a spark of energy
Enzymes & Metabolism:
Metabolism = the totality of an organism’s chemical reactions
A metabolic pathway begins with a specific molecule and ends with a product
Catalyst = chemical agent that speeds up a reaction without being consumed in the reaction
Enzyme = catalytic protein
In catalysis, enzymes or other catalysts speed up specific reactions by lowering the Ea (activation energy) barrier
We’re talking about Gibbs free energy, we want to be at a lower energy level
Enzymes hasten spontaneous reactions that would occur eventually
The Activation Energy Barrier
It takes energy to break bonds, enzyme gives you the push to break the bonds and form new ones to get to a lower energy state
Reaction is exergonic, and occurs spontaneously, but the rate is slow due to Ea
Energy is released from the system
You end up at a lower energy level
Exergonic reaction = spontaneous because it’s easier to go down than go up
The Catalytic Cycle
Usually can catalyze the forward or reverse reactions
How does the active site lower the activation energy barrier?
The enzyme:
Orients the substrates correctly
Strains the bonds
Provides a favorable environment
Ex. Lucy & Chocolate Factory:
Substrate: chocolate + wrapper
Enzyme: lucy & ethel
Active site: their hands
Product: wrapped chocolate
How to measure the rate of the reaction?
Experiment 1: Measuring product formation over time
Count total number of wrapped chocolates each minute for ten minutes
[ ] = reate
[Enzyme] is constant (2 units)
[Substrate] is the speed of the conveyor belt delivering chocolates
Reaction Rate: Take the slope of the graph of product formed over time
slope = # of wrapped chocolates produced per minute
Enzyme Kinetics:
Enzyme kinetics = study of enzyme mechanisms through reaction rates under various conditions
When an enzyme is saturated, it can’t handle any more substrate
Think of enzyme catalysis in two parts
1. find time - time to bind next sub
2. processing time - time to convert substrate into product
High [substrate] decreases find time but does not affect process time
How to Measure Enzyme Function?
Determine the rate of enzyme reaction
Amount of substrate converted to product per unit time
Follow the increase in [product] or decrease in [substrate]
Spectrophotometry – measure light absorbance
Absorbance is proportional to [compound]
What affects reaction rate?
[substrate] & [enzyme]
Substrate concentration increase would increase reaction speed but only up to a certain point
Temperature, pH
Reaction order
Efficiency of enzyme
Factors that affect enzyme shape
Polyphenoloxidase (= our “model system”)
Oxidizes polyphenolics to quinones
Orthoquinone breaks down into other compounds
Some of those are colored (absorb strongly at 525nm)
Increase in absorbance over time tells us rate (= slope)
Polyphenyloxidase = responsible for the reaction that turns food brown
Non-essential – the gene can be deleted from the plant and it looks normal/grows normally
Found in a lot of plants (i.e., avocado, mango, apple, potato, walnuts, and many more)
What Does Polyphenoloxidase Do?
Presumably, it is beneficial to the plant in some way
Causes browning/bruising in damaged part of plant
Is it a defense mechanism?
May deter herbivory (of at least some organisms). Deters the whole plant from being consumed
If so, how would you test that?
Enzyme Action Lab
Substrate: Catechol
Product: Orthoquinone (brown)
How is it measured?
Absorbance vs time
How do we find the reaction rate?
Slope of the absorption vs time graph
Factors influencing enzymatic activity
Enzyme concentration (Enzyme 1)
Substrate concentration (Enzyme 2)
Temperature
Inhibitors, cofactors
Salt concentration
Lab 2:
Part I- varying amounts of enzyme
Organize group, see effect of VOLUME of enzyme
Part II- varying [enzyme]
Improve on design of Part I
Dilute enzyme to see impact on rate
Difference between Volume & Concentration
How much of something/how much space it takes up
Concentration: amount/weight PER volume
Lecture 3: Enzyme Action, Part II
Enzyme Kinetics
Vmax = maximum rate of reaction
All active sites are full (active saturation)
KM = affinity of the enzyme for the substrate
The lower KM = greater affinity
[substrate concentration] at which the reaction rate is half-maximal
Higher Vmax is better, lower KM = “better”
We want enzymes that are efficient; don’t have to use a lot of substrate to get the reaction to go
Vmax & KM = quantitative measures of enzyme efficiency
How to Measure Initial Reaction Rate?
Abs vs. time graph
Initial rate = slope over first 0-2 minutes of reaction
Rate vs. [substrate] graph
Use to determine Vmax & KM
Dependent on [enzyme], temp, pH, inhibitors, etc.
Optimal Conditions for Enzyme Activity
Each enzyme has an optimal environment in which it can function; favors the most active shape
Competitive inhibitors: binds to the active site
Competes with substrate
Noncompetitive inhibitors: binds to another part of the enzyme and changes its shape
Doesn’t compete based on same site, but prevents other things from binding
Shuts down the enzyme, not affected by amount of substrate
Non-specific inhibitors: affects ALL enzymes in the system
Something like a poison, completely denatures all enzymes
Percent Inhibition = (rate without inhibitor) - (rate with inhibitor)(rate without inhibitor) x 100
Competitive Inhibitors - % inhibition varies with different substrate concentrations
Increasing substrate reduces % inhibition
Vmax stays the same
Noncompetitive inhibitors: % inhibition is constant throughout the substrate concentrations
Noncompetitive inhibitors lower the rate of vmax
Cofactors
Non-protein enzyme helpers
Might be inorganic (metal in ionic form)
Chelating agents can pull metal ions from solution, and inhibit enzyme function
Lecture 4: Enzyme Action, Part 3
Plant Defense
Organisms can control their reactions by regulating enzyme activity
Physical Defenses:
Thorns, spines, hairs
Thick bark, rind or cuticle, hard shell
Mineral crystals, silica, calcium oxalate
Chemical Defenses:
Toxins, irritants, foul taste or smell
Think of caffeine, capsaicin, poison ivy oils
Ironically, some may be consumed because of these compounds
Animal Partners:
Ants given nectar to live on plant and attack predators
All of these defenses have associated costs to the plant
Plant Hierarchical Organization
Plants evolved three basic organs: roots, stems, and leaves
Organized into a root system and a shoot system
Plant response to environment depends on those two systems
Last week:
The rate doesn’t double until about 12
The graph levels off because the enzymes are saturated
Microbiome 0 Activity:
The rate doesn’t double until about 12
The graph levels off because the enzymes are saturated
Competitive vs Noncompetitive Inhibitors:
Competitive:
Competes with substrate for reaction site
Lowers rate of true reaction
Less effective at high substrate
Fraction of active sites are occupied by inhibitor
% inhibition changes with increasing substrate concentration
V max will ultimately be just as high as without inhibitor
K M will be ___________, because it takes a higher [substrate] to get the same rate of product formation (or same proportion of active sites filled by substrate)
Noncompetitive Inhibitors
Changes shape of active site, crippling enzyme
% inhibition constant with increasing [substrate]
Increasing [substrate] speeds up reaction somewhat, but V max is ultimately lower, active site no longer efficient
_______________________K M , depending on whether substrate binding/processing is affected
Km: to find Km:
look at where vmax is:
Look for half of vmax
Look at where the substrate concentration is for this half value
Lecture 5: Guest Lecture
Plant Defense
Lecture 6: Enzyme Recap
Part IV:
Results of running the reaction at 0C, 20C (room temp), 45C, and 65C
You have to prewarm and precool the enzyme at substrate: pretreatments
Water has a high specific heat, so it doesn’t change temperature quickly
Results:
45, 20, 0, 65 → fastest to slowest temperatures
Warmth helps the reaction run a little faster; high heats denature the enzyme
Part V:
Results of freezing and thawing the enzyme vs boiling it and letting it cool
Control = never-frozen, never-boiled
Results:
Control ≈ frozen, boiled
The boiled enzyme had a cloudiness meant that the protein unfurled, which made the vial cloudy and increased absorbance
This is why absorbance doesn’t matter → look at reaction rates
Boiled enzyme was denatured
Part VI:
Results of using potassium arsenite
Use 100µl of arsenite with varying substrate concentration
Percent inhibition should be close at each concentration
Arsenite inhibits very little or not at all; this is unexpected but the true result
This might be confused for non-competitive, but it is NOT
There is no inhibition happening; arsenite is not an inhibitor
Learned that there are no - SH groups in PPO (enzyme)
Part VII:
Results of checking whether para-hydroxybenzoic acid is in inhibitor
PHBA has a chemical structure similar to catechol
PHBA show partial inhibition
% inhibition at high substrate concentration is less than % inhibition at low substrate concentration
PHBA is a competitive inhibitor
Part VIII:
Results of using polyphenoloxidase have a metal ion cofactor (copper or iron)
Cofactor required for reaction to occur
Incube enzyme with different chelating agents (compound which tightly binds metal ions)
Chelators will strip the metal ions from the active site of the enzyme
PTU reacts with copper
Potassium cyanide reacts with both copper and iron
The enzyme has a copper cofactor for sure
You don’t know if the enzyme for sure has an iron cofactor
Need to test a chelator that only draws out iron to improve this experiment
Part IX:
The effect of increasing salt
% inhibition is not truly appropriate, as it isn’t actually an inhibitor
Inhibitor has to bind to the enzyme
Salt denatures the enzyme
Should have pretreated your enzyme to allow for denaturation
As the concentration of salt increases, reaction rate decreases (to a certain point)
How is this biologically relevant?
We need salt to live; major ecosystems on the planet that require saltwater
The saltiness of the water decreases because the glaciers are melting
Gut Microbiome:
Most of the human GI tract microbes are in the colon
Four rates dictate composition of colonic microbiota
Gut microbiome is ALL microbes in the gut environment
Large intestine/colon
Food for the colonic microbiota = Fiber
What happens to fiber?
Fermentation
1.) Hydrogen and Carbon Dioxide are common byproducts
2.) methane producing microbes are present in ~20% of people
1º Degraders take fiber, and turn it into intermediate products like acetate
Butyrate producers take that and turn into butyrate
Gasses produced by microbes in the large intestine (hydrogen (H2 ) and methane (CH4)) can be detected in breath
Gasses produced during microbial fermentation of fiber in colon (gold color)
Diffuse into blood stream
Released from blood into lungs
Exhaled in breath
We take a big breath for breath samples because we want what’s at the bottom of the breath