OB complications
Uterine rupture
manifestations
symptomatic disruption and separation of layers of uterus/previous scar
mostly occur because of scarred uterus from previous c-section
sudden sharp abdominal pain/ripping or tearing sensation
bright red vaginal bleeding/signs of hypovolemic shock
visible palpation of fetal parts
risk factors
c-section
prior uterine rupture
trauma
abortion
multiparity
uterine overdistention
malpresentation
interventions
start IV fluids
transfuse blood products
administer oxygen
prepare for immediate surgery
support family and provide info about tx during emergency
maternal effects
severe hemorrhage
shock
hysterectomy
infection risk
fetal effects
abnormal FHR tracing
abrupt decrease in FHR
late/variable decelerations
absent baseline variability
tachy/bradycardia
loss of fetal station/no fetal descent
hypoxia
acidosis
post complication care
monitor for infection
assess for PPH
Shoulder dystocia
manifestions
head is born but anterior shoulder cannot pass under pubic arch
r/t excessive fetal size or maternal pelvic abnormality
cannot be predicted
risk factors
hx shoulder dystocia
maternal diabetes/obesity
prolonged second stage of labor
macrosomia >4000 g
interventions
assume McRoberts maneuver (pull mother’s knees to ears)
suprapubic pressure
clavicle fracture
symphsiotomy- incision in cartilage between pubic bones to enlarge pelvic opening
maternal effects
perineal trauma
uterine rupture
PPH
fetal effects
hypoxia
fractured clavicle
erb’s palsy
post complication care
perform neonatal injury assessment
maternal perineal care
Prolapsed cord
manifestations
cord lies below presenting part of fetus
visible/palpable after ROM
sudden onset variable/prolonged decels or bradycardia
report of feeling cord in vagina
risk factors
long cord
malpresentation (breech or transverse lie)
preterm labor
polyhydramnios
external cephalic version procedure
induction using balloon catheter
interventions
recognize ASAP as hypoxia from cord compression can cause CNS damage/death
relieve pressure off cord
hold presenting part off umbilical cord
assist into lateral position, trendelenburg, knee-chest
maternal effects
fetal effects
severe hypoxia
distress
bradycardia
variable decelerations
post complication care
neonatal resuscitation
monitor for hypoxic encephalopathy
Preterm labor
manifestations
onset of labor before 37 weeks
risk factors
spontaneous PTB
infection
decreased progesterone
maternal/fetal stress
indicated PTB
preeclampsia
multiple gestation
placental cause
maternal code blue
interventions
tocolytics to suppress uterine contractions (nifedipie, terbutaline, mg sulfate)
betamethasone for fetal lung maturity
maternal effects
fetal effects
respiratory distress syndrome
necrotizing enterocolitis
post complication care
admit to NICU
Meconium-stained fluid
manifestations
fetal stool in amniotic fluid
green, can be thin or thick
risk factors
hypoxia-induced peristalsis
sphincter relaxation
cord compression-induced vagal stimulation
interventions
suction fetal airway
intubation if thick meconium
maternal effects
fetal effects
meconium aspiration syndrome in newborn
cause severe aspiration pneumonia
post complication care
before birth
assess amniotic fluid for presence of meconium after ROM
gather equipment and supplies for neonatal resuscitation
immediately after birth
assess newborn respiratory effort, HR, muscle tone
suction only mouth and nose if WDL vital signs
Premature rupture of membranes
manifestations
SROM/leakage of fluid prior to onset of labor at any gestational age
risk factors
often preceded by infection (chorioamnionitis)
bacterial infection of amniotic cavity
maternal fever, tachycardia, uterine tenderness, foul odor
cervical insufficiency
passive/painful dilation of cervix leading to recurrent PTB during second trimester
tx with cerclage placement
multiple gestation
interventions
administer magnesium sulfate for 24 hrs for fetal neuroprotection
give tocolytic meds or nifedipine to suppress uterine activity
give antenatal glucocorticoids (betamethasone) to reduce respiratory distress syndrome, hemorrhage, necrotizing enterocolitis, death
maternal effects
increased risk of infection
fetal effects
PTB
risk for sepsis
post complication care
monitor infection
Hypertensive Disorders
CONTROLLING BLOOD PRESSURE IS THE OPTIMAL INTERVENTION TO PREVENT DEATH FROM STROKE/PREECLAMPSIA
gestational hypertension | development of HTN after week 20 of pregnancy in a woman with previously normal BP |
preeclampsia | HTN and proteinuria after 20 weeks of gestation who previously had neither condition in absence of proteinuria: new-onset HTN with new onset of ANY OF THESE—thrombocytopenia, renal sufficiency, impaired liver function, pulmonary edema, cerebral/visual symptoms |
chronic hypertension | present HTN before pregnancy/diagnosed before 20 weeks gestation |
superimposed preeclampsia | chronic HTN associated with preeclampsia |
Gestational HTN management
defined as systolic BP of >140 or diastolic of >90
frequent BP and weight measurement
report promptly if:
increase in BP
persistent headache
visual change
rapid weight gain
decreased fetal movement
uterine contractions
Preeclampsia
HTN and proteinuria after 20 weeks of gestation who previously had neither condition
can also develop in postpartum period
in absence of proteinuria, preeclampsia may be defined as HTN along with:
thrombocytopenia
renal sufficiency
impaired liver function
pulmonary edema
cerebral/visual symptoms (blinking stars/dots)
risk factors include:
preeclampsia hx
multifetal gestation
chronic HTN
pregestational/gestational diabetes
SLE
obstructive sleep apnea
nulliparity
BMI >30
AMA
thrombophilia
assisted reproductive technology
pathophysiology
placenta is root cause
begins to resolve after placenta expelled
spinal arteries fail to get larger and thicker
decreased placental perfusion and endothelial dysfunction= preeclampsia
placental ischemia=endothelial cell dysfunction
generalized vasospasm=poor tissue perfusion in organ system
preeclampsia with severe features
thrombocytopenia <100
renal insufficiency with elevated serum creatinine >1.1 mg/dl/doubling
pulmonary edema
headache unresponsive to medication
visual disturbances
systolic BP ≥160 or diastolic BP ≥110 at least twice 4 hrs apart
gestational age
onset prior to 34 weeks is most often severe
manage at facility with resources for management of serious maternal/neonatal complications
induction at 37 weeks indicated for preeclampsia without severe features
management
assess BP and edema
assess deep tendon reflex/hyperactive reflex (clonus)
assess PCR
evaluate for these s/s:
severe frontal headache
epigastric pain (heartburn)
right upper quadrant tenderness
visual disturbance
Eclampsia
onset of seizure activity/coma in preeclamptic patient
no hx of preexisting pathology
higher in multifetal gestation and women who did not receive prenatal care
caused by cerebral edema
watch for headache or visual disturbance
Chronic hypertension w/ superimposed preeclampsia
dx based on
sudden increase in BP that was previously well controlled
new-onset/sudden and sustained increase in proteinuria in woman known to have proteinuria before conception/early pregnancy
Signs and symptoms
persistent BP >140/90
proteinuria if preeclamptic
edema
severe headache
vision change
Treatment
Medications
magnesium sulfate
mag sulfate for seizure prophylaxis is indicated for:
preeclampsia with severe features
severe gestational HTN
all cases of severe HTN regardless of classification
high Mg levels can causes relaxation of smooth muscle
initiate when diagnosed with preeclampsia
continue until 24 hrs post delivery or 24 hrs after last seizure if eclamptic
dose: 4-6gm loading dose over 20-30 min, then 2gm/hr
side effects
flushing/warm
drowsiness
sweating
N/V
HoTN
dizziness
headaches
slurred speech
visual disturbance
muscle weakness/decreased DTR
toxicity
absence/change in DTR
decreased LOC
SOB
decreased respiratory rate
persistent HoTN
chest pain
bradycardia/cardiac arrest
Mg greater than 8 mg/dL
give 1 g IV calcium gluconate over 3 min for toxicity
Meds for chronic HTN during pregnancy | dose | |
labetalol | 200-2400 mg | first-line in pregnancy, does not reduce uterine blood flow |
nifedipine | 30-90 mg daily | use with caution with mg sulfate |
methyldopa | 500-2000 mg | not effective when taken less than three times a day |
hydrochlorothiazide | 12.5-25mg daily | may be continued if taken before conception but not started as new med in pregnancy risk for thrombocytopenia for newborn |
medications to avoid | |
angiotensin-converting enzyme inhibitors (captopril) angiotensin II receptor antagonist (losartan) | assoc. with birth defects and impaired fetal renal fx |
atenolol | assoc. with growth restriction avoid use in early pregnancy, caution in late pregnancy |
HELLP syndrome
characterized by:
hemolysis
elevated liver enzymes
low platelet count
symptoms
upper right abdominal pain
N/V
headache
blurry vision
can lead to:
liver hematoma/rupture
ARDS
sepsis
hypoxic encephalopathy
fetal/maternal death
preterm delivery
recurrent preeclampsia
DIC
treatment
induction regardless of gestational age
monitor CBC and liver enzymes Q6H
magnesium infusion
BP control
early epidural placement
Fetal changes
impaired uteroplacental blood flow can cause:
IUGR
oligohydramnios
placental abruption
nonreassuring fetal status
preterm labor
Delivery recommendations
induction if severe preeclampsia/eclampsia
Hemorrhagic Disorders
Hemorrhage
Antepartum hemorrhagic disorders
antepartum hemorrhage
bleeding in pregnancy jeopardizes maternal/fetal well-being
blood loss decreases oxygen carrying capacity, increases risk for:
hypovolemia
anemia
infection
preterm labor
impaired O2 to fetus
hypoxemia of fetus
hypoxia of fetus
spontaneous abortion (miscarriage)
occurs before 20 weeks
loss of fetus weighing ≤500g
management
assess pregnancy hx, vital signs, pain, bleeding, labs, emotional status
administer misoprostol (cytotec)
dilation and curettage
emphasize rest
ectopic pregnancy
fertilized ovum implants outside uterine cavity
manifestations
dull, lower quadrant pain on one side
delayed menses
abnormal vaginal bleeding (spotting)
management
assess b-hCG level and transvaginal UA exam
administer methotrexate
refrain from taking folate
surgery
ruptured ectopic pregnancy
abdominal discomfort to colicy pain when tube stretches to sharp, stabbing pain
referred shoulder pain
management
pain meds
assess for s/s shock (faintness, dizziness)
surgery
hydatidiform mole
placental trophoblast growth in which chorionic villi develop into edematous, cystic, avascular transparent vesicles that hang in grape-like cluster
complete
no embryonic/fetal parts
sperm fertilizes empty egg
partial
often have embryonic/fetal parts and amniotic sac
abnormal placenta forms, two sperm fertilize one egg
generally can’t survive
manifestations
anemia from blood loss
hyperemesis gravidarum
abdominal cramps
large fundal height
preeclampsia
management
transvaginal UA
monitor b-hCG levels weekly for 3 weeks then monthly for 6-12 months
suction curettage
instruct to not get pregnant for 12 months
can increase risk for cancer
placenta previa
placenta implanted in lower uterine segment near/over internal cervical os
risk factors
previous c-section
AMA
multiparity
hx suction curettage
maternal cocaine use
smoking
low-lying
placenta in lower segment of uterus
does not reach opening of cervix
marginal
placenta next to cervix but does not cover
partial
placenta covers part of opening
complete
placenta covers entire opening
manifestations
painless bright red vaginal bleeding during second/third trimester
soft, relaxed, nontender abdomen
outcomes
major complication is hemorrhage
morbidly adherent placenta
surgery-related trauma
preterm birth/IUGR
late decels, absent/variable variability
management
no vaginal exams/internal monitors EXCEPT transvaginal ultrasound determining placental location
c-section is only option
cut right through placenta
come to hospital if any bleeding occurs
placental abruption
detachment of all/part of placenta after 20 weeks of gestation
risk factors
maternal HTN
cocaine/methamphetamine use
penetrating/blunt external abdominal trauma
smoking
hx abruption
PPROM
manifestations
painful, tight belly
wave-like contractions
uterine hypertonicity
treatments
emergency c-section
IV fluids
blood transfusion
vasa previa
fetal vessels lie over cervical os
vessels implanted into fetal membranes rather than into placenta
velamentous insertion
cord vessels branch at membranes and onto placenta
succenturiate placenta
placenta divided into 2+ lobes
battledore insertion of cord increases risk of fetal hemorrhage
Intrapartum hemorrhagic disorders
disseminated intravascular coagulation (DIC)
acquired secondary complication resulting in formation of clots in microsystem
clotting occurs throughout entire circulation
clotting factors are consumed, unable to keep up with demand
fibrinolysis occurs
fibrin split product damage RBC, cause hemolysis
damage endothelial lining of vessels, capillaries in lungs, platelets
leads to ARDS, pulmonary edema
lab findings
low fibrinogen <100
PT prolonged before PTT
management
administer fresh frozen plasma (FFP)
Postpartum hemorrhagic disorders
postpartum hemorrhage
cumulative blood loss ≥1000 ml OR bleeding associated with s/s of hypovolemia within 24 hrs of birth
primary PPH (early/acute)
occurs within 24 hrs of birth
uterine atony/rupture/inversion
coagulopathy
genital laceration
retained/invasive placenta
secondary PPH (late)
occurs more than 24 hrs to 12 weeks after birth
infection
retained placenta
coagulopathy
subinvolution of uterus
prolonged lochial discharge
irregular/excessive bleeding
sometimes hemorrhage
hemorrhagic (hypovolemic) shock
results of hemorrhage, death may occur
assessment
respirations
pulse
BP
skin
UO
LOC
characteristics
cool, pale, clammy
rapid, shallow respirations
rapid, weak, irregular pulse
lethargy, anxiety
management
fluid/blood replacement therapy
O2 delivery/maintain CO
restore blood volume
Early pregnancy bleeding
diagnosis
vaginal bleeding before 20 weeks gestation
confirmed by ultrasound and hCG levels
treatment
Late pregnancy bleeding (refer to previous sections)
placenta previa
placental abruption
Placental disorders (refer to previous sections)
placenta previa
placental abruption
placenta accreta—slight penetration on myometrium
placenta increta—deep penetration of myometrium
placenta percreta—perforation of myometrium and uterine serosa
management
early delivery
hysterectomy in severe cases
Hemorrhage medications
Medication | Route | Dose | Contraindications/uses |
pitocin (oxytocin) | IM or IV | 10-30 MU | |
misoprostol (cytotec) | rectal/vaginal | 800-1000 mcg | |
methergine (methylergonvine) | PO or IM | 0.2 mg | hypertension |
hemabate (carboprost) | IM | 250 mcg | asthma |
tranexamic acid | IV | 1 gm | tx lacerations |
Which condition is a potential cause of an early postpartum hemorrhage?
a. Subinvolution of the uterus
b. Incomplete placental separation
c. Infection
d. Coagulopathy
Nursing interventions
Fetal Positioning
Lie
longitudinal (normal)
transverse
sideways, requires c-section
Attitude
flexion
normal, chin tucked to chest
extension
head tilted backwards
Presentation
cephalic
normal, head first
breech
feet/bottom first
may require c-section
shoulder
transverse lie with shoulder leading into birth canal
requires c-section
Position
occiput anterior
normal
occiput posterior
face facing belly button
cause painful back labor
use hand and knees position, counterpressure
Fetal Heart Rate Tracing
FHR Variability
refers to fluctuations in the fetal heart rate around the baseline
absent variability
fluctuations are undetectable
caused by fetal hypoxemia, metabolic acidemia, congenital anomaly
minimal variability
change is greater than undetectable but less than or equal to 5 bpm
caused by fetal hypoxemia, metabolic acidemia, congenital anomaly, neuro injury, CNS depressants, fetus in a sleep state
moderate variability
normal change of 6-25 bpm, shows healthy nervous system
marked variability
change is greater than 25 bpm
Accelerations
temporary increases in the FHR, usually a sign of a healthy baby
defined as 10 bpm for 10 seconds if less than 32 weeks, 15 bpm for 15 seconds if greater than or equal to 32 weeks
Decelerations
temporary decrease in FHR and are categorized by their shape, timing, and relationship to contractions
variable decelerations
abrupt decrease in FHR that can occur at any time
caused by cord compression
visually abrupt (onset to lowest point less than 30 seconds) decrease in FHR by 15 bpm+ and lasts at least 15 seconds
returns to baseline in less than 2 minutes
early decelerations
gradual decrease in FHR that mirror contractions
caused by fetal head compression
begin and end with the contraction, and onset to the lowest point is greater than or equal to 30 seconds.
late decelerations
gradual decreases in FHR that begin after contraction starts and don't return to baseline until after contraction ends
caused by placental insufficiency.
prolonged decelerations
decrease in FHR of at least 15 bpm below the baseline, lasting more than 2 minutes but less than 10 minutes
Category
Category 1 (Normal)
baseline rate of 110-160 bpm
moderate baseline variability
no late or variable decelerations
early decelerations and accelerations may be present or absent
indicates a well-oxygenated fetus
Category 3 (Abnormal):
absent baseline FHR variability AND any of the following
recurrent late decelerations
recurrent variable decelerations
bradycardia,
sinusoidal pattern.
Indicates a poorly oxygenated fetus.
Category 2 (Indeterminate):
Any FHR pattern that does not fit into Category 1 or 3:
moderate variability with recurrent late or variable decelerations
minimal variability with recurrent variable decelerations
absent variability without recurrent decelerations
bradycardia with moderate variability
prolonged decelerations
tachycardia
indicates fetus that is showing compensatory responses to lack of oxygen
periodic change=associated with ctx
episodic change=not associated with ctx
Obstetrical Procedures
Induction of labor
equipment
IV oxytocin
amniotomy
indications
continuing pregnancy poses risk to mother/fetus
maternal DM
postterm pregnancy
HTN complications
IUGR
chorioamnionitis
PROM
before induction begins:
assess cervix with bishop score, uterine activity, fetal size/presentation
determine gestational age
evaluate maternal/fetal wellbeing
contraindications
Episiotomy
Vacuum extraction
Forceps delivery
Cesarean section
Contraindications
active maternal infection (herpes for vaginal birth)
fetal distress
risk of uterine rupture
Intimate Parter Violence
physical, emotional, sexual, and financial abuse during pregnancy
signs
unexplained injuries
interventions
support services
effects on pregnancy
PTB
LBW
placental abruption
Transition to Parenthood
Common issues that couples face as they become parents include:
Changes in their relationship with one another
Sexual intimacy
Division of household and infant care responsibilities
Financial concerns
Balancing work and parental responsibilities
Social activities
Nursing interventions
encourage partners during pregnancy and in the postpartum period to share personal expectations and assess their relationship
prioritize one-on-one conversations, schedule time apart from the infant, and express appreciation for each other and the child
identify and utilize support from close relationships, healthcare team
explore new approaches to lifestyle and habits that may ease the transition to parenthood
Bonding and attachment
encourage skin to skin contact
breastfeeding
assign meaning to infant’s actions
talk, coo, sing to infant
play peek-a-boo