Micro Exam 3
Chapter 13
13.1 -
Holobiont- a human plus all of its resident microbiota
Human Microbiome project - a project of the National Institutes of Health to identify microbial inhabitants of the human body and their role in health and disease
*we only knew about bacteria and fungi because we could grow it in a lab, with the project we discovered hundreds of microbes that cannot be cultured
Microbial antagonism- relationship in which microorganisms compete for survival in a common environment by taking actions that inhibit or destroy another organism
Endogenous- originating or produced within an organism or one of its parts
*an endogenous infection is a infection caused by biota already present in the body
Opportunistic- ordinarily non pathogenic or weakly pathogenic microbes that cause disease primarily in an immunologically compromised host
Virulence- in infection, the relative capacity of a pathogen to invade and harm host cells
*is the accurate term for describe the degree of pathogenicity
Virulence factor- a microbes structures or capabilities that allow it to establish itself in a host and cause damage
Infectious dose- infections will only occur is a minimum number is present
*numbers below the ID will not cause infection, numbers above the ID will
Portal of entry- route of entry for an infectious agent; typically a cutaneous or membranous route
Polymicrobial: Infections from more than one microbe
Portals of Entry; Becoming establish
Step 1: Finding a portal of entry
Exogenous: originating outside the body
*opposite of endogenous
Types of portals of entry:
Skin - through abrasions, broke skin, or bites
Gastrointestinal tract - by contaminated food, drink, or ingested substances
Respiratory tract - oral and nasal cavities
Urogenital - contracted by sexual means
Pathogens that infect during birth
T -toxoplasmosis
O -other diseases
R -rubella
C -cytomegalovirus
H - herpes
Step 2: Attaching to the host and interacting with the microbiome
Adhesion- process where microbes gain a more stable foothold on host tissues
-Bacteria, fungi, and protozoal pathogens attach by fimbriae, surface proteins, and adhesive slimes or capsules
-Viruses attach by specialized spikes and glycoproteins
-Helminths attach by suckers, hooks, and barbs
Step 3: Surviving Host Defenses
Phagocytes- a class of white blood cells capable of engulfing other cells and particles
*engulf and destroy pathogens by means of enzymes and antimicrobial chemicals
Leukocidin- a heat-labile substance formed by some pyogenic cocci that impairs and sometimes lyses leukocytes
Epigenetic- referring to changes in the way DNA is transcribed, not actual changes in the DNA sequence
Step 4: Causing Disease
The three main ways microbes can cause disease to the host are
By secreting proteins (enzymes or toxins) that directly damage host cells
Causing an overreaction by the body's defenses and those defenses cause host damage
Altering the host cell genome or transcription process through epigenetic changes
Direct damage via enzyme and toxins
Exoenzymes- break down and inflict damage on tissues, extracellular enzyme
Examples of exoenzymes are:
-Mucinas: digests the protective coating on mucous membranes
-Keratinase: digests keratin
-Hyaluronidase: digests hyaluronic acid
Toxigenicity- the power to produce toxins
Toxinoses- disease whose adverse effects are primarily due to the production and release of toxins
Toxemia- condition in which a toxin is spread throughout the bloodstream
Intoxications- poisoning that results from the introduction of a toxin into body tissues through ingestion or injection
Types of toxins
-Neurotoxins: act on the nervous system
-Enterotoxins: act on the intestines
-Hemotoxins: act on red blood cells
-Nephrotoxins: damage the kidneys
Exotoxin- a toxin that is secreted by a living bacterial cell into infected tissues
*causes more harm than endotoxin
Endotoxin- a toxin that is not actively secreted but is shed from the outer membrane
*found on all gram-negative bacteria
*lipopolysaccharide
Hemolysins- class of bacterial exotoxin that disrupts the cell membrane of red blood cells
Inducing an excessive host response
N/A on info
Epigenetic changes in host cells
N/A on info
Patterns of Infection
Localized infection- the microbe enters the body and remains confined to a specific tissue
-boils, fungal infections, and warts
Systematic infection- infection spreads to several sites and tissue fluids, usually in the bloodstream
Focal infection- infectious agent breaks loose from a local infection and is carried into other tissues
Leukocytosis- increase in white blood cells
Leukopenia- decrease in white blood cells
Step 5: Vacating the Host- Portals of Exit
Types of portals of exit
Respiratory and salivary portals - mucus, sputum and nasal drainage
Skin scales - shed skin and scalp
Fecal exit - poop
Urogenital tract - vaginal discharge or semen and urine
Removal of blood or bleeding- blood removed or released from a puncture
Latency- The state of being inactive
*There are four phases of infection and disease
Incubation period -from time of contact of disease to first symptoms
Prodrome stage - start of mild symptoms indicating the onset of disease
Acute period - the disease becomes well established and multiples
Convalescent period - immune system responds and you start to get better
*There can be a fifth stage that some diseases have which is the continuation phase in which the organism lingers or the symptoms linger
Passive carrier- person who mechanically transfers a pathogen without ever being infected by it; like a healthcare worker not washing their hands in between patients
Horizontal pattern of transmission: disease is spread through a population from one infected individual to another
Vertical pattern of transmission: transmission from parent to offspring via ovum, sperm, placenta, or milk
Vehicle: an inanimate material that serves as a transmission agent for pathogens
Fomite: inanimate object that harbors or transmits pathogens and is not a continuous source of infection
Kochs Postulates to Determine Etiology
The agent must be found
Inoculations of a pure culture must reproduce the same disease in animals
The agent must again be present in experimental animal
A pure culture must again be obtained
*Epidemiologists focus their work on the 5 W’s… Who, what, where, when, and why, and focuses on population more than individuals
Florence Nightingale: She is the first to collect and use health, disease, and death statistics for the purpose of improving public health
John Snow: Was the first to use a map to see the cause and events of a disease
Point-source: infectious agent came from one source and everyone that was exposed, was exposed of the same source at the same time.
Common-source: outbreak resulting from common exposure to a single source of infection that can occur over a period of time
Propagated epidemic: results from an infectious agent that is communicable from person to person and is therefore sustained over time in a population
Healthcare associated infection: any infection acquired as a direct result of a patient's presence in a hospital or health care setting
Herd immunity: the status of a collective acquired immunity in a population that reduces the likelihood that non immune individuals will contract and spread infection.
The lines of defense
-The first line of defense includes any barriers that block invasion at the portal of entry, and limits access to the internal tissues of the body
*the skin, hair follicles, and skin glands
-The second line of defense is an internalized system of protective cells and fluids and includes inflammation, phagocytosis, fever, and antimicrobial products.
The three main types of phagocytes are neutrophils, monocytes, and macrophages.
Neutrophils- react early in inflammatory response to bacteria and foreign materials
Eosinophils- are attached to sites of parasitic infections, play a minor phagocytic role
Monocytes-transformed into macrophages, the biggest leukocyte
-The third line of defense is acquired on an individual basis as each foreign substance is encountered by white blood cells called lymphocytes
Stages of the network of immunology
Lymphocyte development and differentiation
The presentation of markers (antigens)
Antigen challenge of B and T cells
B cell response and T cell response
Major Histocompatability complex (MHC)- a set of genes in mammals that produces molecules on surfaces of cells that differentiate among different individuals in the species. Has three classes
Class 1- gene codes for markers that appear on all nucleated cells
Class 2- gene code for immune regulatory markers
Class 3- genes encode proteins involved with the complement system
Thee immune system is responsible for…
Surveillance of the body
Recognition of foreign material
Destruction of anything foreign
Antigen- any cell that induces a specific immune response by B cells or T cells and can stimulate resistance to an infection or toxin.
Pathogen-associated molecular patterns (PAMP)- molecules on the surface of many types of microbes that are not present on host cells that mark the microbes as foreign.
Pattern recognition receptors (PRR)- molecules on the surface of host defense cells that recognize pathogen associated molecular patterns on microbes
Mononuclear phagocyte system- a collection of monocytes and macrophages scattered throughout the extracellular spaces that function to engulf and degrade foreign molecules
Lymph- a plasma like fluid carried by the lymphatic circulation
Skin-associated lymphoid tissues (SALT)- discrete bundle of lymphatic tissue located just under the surface of the skin
Mucosa-associated lymphoid tissue (MALT)- small patches of lymphoid tissue situated in and on mucosal surfaces, containing T cells, B cells, phagocytes, and other immune cells
Gut associated lymphoid tissue (GALT) - A collection of lymphoid tissue in the gastrointestinal tract that includes the appendix, the lacteals, and Peyer’s patches.
Whole blood- A liquid connective tissue consisting of blood cells suspended in plasma.
Pluripotential stem cells- is a primary precursor of new blood cells, is a pool of undifferentiated cells
Types of white blood cells
Granulocytes- a mature leukocyte that contains noticeable granules in a wright stain
*Neutrophils, eosinophils, and basophils
Agranulocytes- one form of a leukocyte having globular, non lobed nuclei and lacking prominent cytoplasmic granules.
Cytokines- a chemical substance by white blood cells and tissue cells that regulates development, inflammation, and immunity
B cells mature in red bone marrow, and T cells mature in the thymus
Lysozyme- an enzyme found in sweat, tears, and saliva that breaks down bacteria peptidoglycan
Rubor- redness
Calor- warmth
Tumor-swelling
Dolor-pain
Diapedesis- the migration of intact blood cells between endothelial cells of a blood vessel such as a venule
Interferon- is a small protein produced naturally by certain white blood and tissue cells, is a type of cytokine
*three types, alpha, beta(stimulate phagocytes) and gamma (regulator of macrophages and T and B cells)
Natural killer cells- type of lymphocyte related to T cells that lack specificity for antigens.
Dendritic cells- a type of phagocyte and immune cell that is found in tissues and boost immune responses by showing antigens on its surface
Antimicrobial peptides- short protein molecules found in epithelial cells, have the ability to kill bacteria
Immunogens- any substance that induces a state of sensitivity or resistance after processing by the immune system of the body
B cells- a type of lymphocyte or white blood cell, produce antibodies and neutralize pathogens
T cells- a type of lymphocyte or white blood cell, destroy infected or cancerous cells.
Major Histocompatibility complex (MHC)- a set of genes in mammals that produces molecules on surfaces of cells that differentiate among different individuals in the species
-gives rise to glycoproteins called MHC molecules and is found on all cells but red blood cells
Three classes of MHC genes have been identified:
Class 1 genes- markers that appear on all nucleated cells
Class 2 genes- genes code for immune regulatory markers
Class 3 genes- genes encode proteins involved with the complement system
Clone- a group of genetically identical cells
How T cells respond to antigens
Helper t cells help B cells to activate cytotoxic T cells (CD4 cells). The most prevalent type of T cell.
Regulatory T cells guard against excessive or inappropriate inflammation. Also carry CD4 markers.
Cytotoxic T cells lead to the destruction of infected host cells. Can kill virally infected cells, cancer cells, and cells from other animals and humans
How B cells respond to antigens
A B cell divides giving rise to plasma that releases the five classes of antibodies.
Regulatory B cells regulate the degree of response from T cells
Characteristics of good antigens
-their chemical composition
-their context
-their size
Epitope- primary signal that the molecule is foreign
Haptens- a partial antigen, constitutes the determinative group and cannot stimulate a full immune response
Alloantigens- cell surface markers that occur in some species but not others
Superantigens- bacterial toxins that are potent stimuli for T cells
Cytokine storm- an overwhelming release of immune-modulating chemicals known as cytokines
3 different cells that can serve as Antigen-presenting cells: APC
Macrophages
B cells
Dendritic cells
Cytotoxicity- is the capacity of certain T cells to kill a target cell
Apoptosis- genetically programmed death of cells that is both a natural process of development and the body’s means of destroying abnormal cells.
Classes of Antibodies:
IgG- a monomer of immunoglobulin, it is the most prevalent antibody, and neutralizes toxins. The only antibody capable of crossing the placenta
IgA- coats the surface of membranes and is found in saliva, tears, colostrum, and mucus
IgM- composed of five monomers, binds antigens, circulates mainly in the blood
IgD- found in miniscule amounts, main function is that it is the receptor for antigen on B cells
IgE- its role is to stimulate an inflammatory response, can lead to asthma and certain other allergies
Active Immunity- occurs when an individual received an immune stimulus that activates the B and T cells, causing the body produce antibodies
Passive Immunity- occurs when an individual receives immune substances that were produced actively in the body of another human.
Natural Immunity- ecompasses any immunity that is acquired during the normal biological experiences of an individual
Artificial Immunity- protection from infection obtained through medical procedures