Evolutionary psych – study how natural selection influences behavior
NERVOUS SYSTEM
CENTRAL NS: Brain and spinal cord
PERIPHERAL NS: Rest of the NS – relays to Central NS
Somatic NS: Voluntary movement, has sensory and motor neurons
Autonomic NS: Involuntary organs (heart, lungs, etc) – contains the:
Sympathetic NS: fight/flight (generally activates – exception digestion)
Parasympathetic NS: rest/digest (generally inhibits - exception digestion)
NEURONS
Dendrites: Receive incoming NTs
Axon: AP travels down this
Myelin Sheath: speeds up AP down axon, protects axon
Synapse: gap w neurons, where APs happen
SENSORY neurons: receive sense signals from environ.–send signal to brain
MOTOR neurons: signals to move – send signals from brain
Interneurons: cells in spinal cord /brain responsible for reflex arc
Reflex arc: important stimuli skips the brain and routes through the spinal cord for immediate reactions (hand on a hot flame)
Glial cells: support cells – give nutrients and clean up around neurons
Neurons Fire w/ an Action Potential: ions move across membrane sends an electrical charge down the axon
Resting potential: neuron maintains a -70mv charge when not doing anything
Depolarization: (+) sodium ions go into cell causing it to be more positive, triggers action potential
Threshold of depolarization: stimulus strength must reach this point to start the AP
Refractory period: time that a neuron must reset before it can fire again
NEUROTRANSMITTERS
chemicals that are released in synaptic cap, received by neurons
Excitatory: increase action potential
Inhibitory: decrease action potential
GABA: inhibitory, slows down brain activity
Glutamate: excitatory, for normal brain functions
Dopamine: reward and fine movement—in hypothalamus, associated with addiction
Serotonin: moods, emotion, sleep—in amygdala, lack of it is associated w depression
Acetylcholine: Memory and movement—in hippocampus, associated w Alzheimer’s
Norepinephrine: alertness and arousal, sympathetic NS—lack of it is associated w depression
Endorphins: decrease pain
Substance P: pain signals
HORMONES (not in NS)
Oxytocin: love, bonding, childbirth, laxation
Adrenaline: fight or flight
Leptin: makes you full
Ghrelin: makes you hungry
Melatonin: sleepy
Reuptake: unused neurotransmitters taken back up into sending neuron
PSYCHOACTIVE DRUGS
Agonist: drug that mimics neurotransmitter
Antagonist: drug that blocks a neurotransmitter
Depressants: Decrease NS activity, relaxed (alcohol)
Stimulants: increase NS activity, alertness (caffeine, nicotine, amphetamines)
Hallucinogens: hallucinations and altered perceptions (marijuana)
Opioids: relieve pain, increases dopamine levels (endorphin agonists) (heroin)
Tolerance: needing more of a drug to achieve the same effects
Addiction: must have it to avoid withdrawal symptoms
Withdrawal: painful (psychological and physical) symptoms associated with sudden stoppage
BRAIN
Cerebellum: movement, balance, coordination, procedural memory (ability to perform learned skills without conscious effort)
Brainstem/Medulla: vital organs, automatic functions (HR, BP, breathing)
Reticular Activating System: alertness, arousal, sleep, eye movement
Cerebral Cortex: outer portion of brain, higher order thought processes, includes limbic system, lobes, corpus callosum
Limbic System (think emotions)
Amygdala: negative emotions
Hippocampus: episodic and semantic memory, creating new memories
Hypothalamus: reward center, eating behaviors—like to endocrine system, homeostasis
Thalamus: relay center for all senses except smell
Pituitary gland: talks w endocrine system and hypothalamus—releases hormones
Occipital Lobe: vision, in back of brain
Frontal Lobe: decision making, planning, judgement, movement, personality, language, executive functioning
Prefrontal cortex: front of frontal lobe—executive function, decision making, socializing
Motor cortex: back of frontal lobe, controls skeletal movements (voluntary body movements)
Parietal Lobe: sensations and touch
Somatosensory cortex: registers and provides touch sensations
Temporal lobe: auditory and face recognition, language
Aphasia: damaged speech, comes from damage to left hemisphere
Broca’s Area: producing speech
Wernicke’s Area: comprehending speech
Corpus Callosum: connects 2 hemispheres
Split-brain experiments: split the corpus callosum—image shown to right eye is processed in left hemisphere, patient can say what they saw—image shown to left eye processed in right hemisphere, can’t say what was seen
Plasticity: brain changed through damage and experiences
Endocrine System: sends hormones throughout body
Pituitary gland: controlled by hypothalamus, releases growth hormones
EEG: shows broad brain activity, not specific, electrical output, with electrodes
fMRI: shows brain activity in specific regions, measures oxygen
Lesion: intentional destruction of brain tissue to see effect
DISEASES AND DISORDERS
Multiple Sclerosis: destruction of myelin sheath (pathway in neurons), disrupts action potentials, causes impaired mobility, paralysis, pain
Myasthenia gravis: acetylcholine blocked, disrupts action potentials, causes poor motor control and paralysis
Blindsight: caused by lesions to a primary visual cortex, individuals can still respond to visual stimuli without conscious awareness of seeing them, information from the eye is being processed unconsciously
Prosopagnosia: face blindness, caused by damage to occipital and/or temporal lobe
Broca’s aphasia: broken speech
Wernicke’s aphasia: jumbled speech
Phantom limb pain: pain from an amputated limb, caused by brain plasticity
Epilepsy: seizures, caused by too much/too little glutamate/GABA
Alzheimer’s: memory loss, caused by destruction of acetylcholine
SLEEP
Consciousness: awareness of cognitive processes
Circadian rhythm: biological clock of body
Sleep stages:
NREM 1: light sleep, hypnagogic sensations (ex. feeling of falling)
NREM 2: sleep spindles (random bursts of brain activity) and K complexes, light sleep
NREM 3: deep sleep, delta waves (slow and large)
REM: dreaming, muscles relaxed but other body systems active, body paralyzed
REM rebound: after sleep disruptions and/or lack of REM sleep REM becomes more intense
Dream theories:
Activation synthesis: the brain produces random bursts of energy stimulating lodged memories in limbic system and brain stem, brain’s attempt at making sense of random neural activity
Consolidation dream theory: brain is combining and processing memories for storage
Why do we need sleep
Consolidation: storage of memories
Restoration: helps regenerate the immune system and restore energy
Sleep disorders:
Insomnia: inability to fall/stay asleep
Somnambulism: sleep walking, happens during NREM3
Narcolepsy: fall into REM out of nowhere
Sleep apnea: stop breathing while asleep, usually due to obesity
REM behavior disorder: malfunction of mechanism that paralyzes you during REM, physically act out dreams
SENSATION
Transduction: convert stimulus to action potential
Absolute threshold: minimum amount of stimulus to detect sound, 50% of time detected
Just noticeable difference: minimum difference between 2 stimuli required for detection
Weber’s Law: 2 stimuli must differ by a constant minimum proportion, depends on intensity of original stimulus
Sensory Adaptation: diminished sensitivity as a result of constant exposure (ex. getting used to AC blowing), allow us to focus on more important sensations
VISUAL SYSTEM
Lens: focuses light on retina, behind pupil
Retina: light sensitive inner surface of eye, contains receptor rods, cones, neurons, processes visual information
Fovea: area of best vision, cones located there
Rods: black and white, dark adaptation, more rods than cones, along sides of retina
Cones: color, bright light, in fovea
Ganglion cells: creates optic nerve
Blind spot: occurs where optic nerve leaves eye, can’t see there, brain fills in blank
light → retina (rods/cones → bipolar cells → ganglion cells) → fovea → optic nerve → brain
other vocab:
Accomodation: lens changes curvature to focus images on retina
Nearsightedness: better vision near, light focused in front of retina
Farsightedness: better vision far, light focused behind retina
Color vision theories:
Trichromatic: three cones for receiving color, blue-short waves, green-medium waves, red-long waves (ex. when see purple, red and blue cones activated)
Opponent Process: complementary colors are processed in ganglion cells, explains why we see an afterimage
Color deficiency: damage to/missing cones or ganglion cells
dichromatism: missing 1 cone, trouble distinguishing between two colors
monochromatism: only have rods, see black and white
AUDITORY SYSTEM
Wavelength: long waves=low pitch, short waves=high pitch
Amplitude: short waves=quiet, tall waves=loud
Theories of hearing (in cochlea):
Place theory: location where hair cells in cochlea bends allow us to perceive pitch
Frequency theory: the rate at which action potentials are sent determines sound
Volley theory: groups of neurons fire action potentials out of sync
Sound localization: which ear gets the waves first tells the location of sound
Conduction deafness: damage to middle ear (bones of ear and eardrums) can cause hearing loss
Sensorineural: damage to cochlea, hairs in cochlea, or nerve—usually due to old age and loud noise
OTHER SENSES
Vestibular: sense of balance, hair cells in ear
Kinesthetic: sense of body position and movement without looking
Pain: gate-control theory—gate that controls how much pain is felt, physical and psychological
Hot: activation of warm and cold receptors
Taste: 6 taste receptors: sweet, salty, bitter, umami, sour, and oleogustus (oily)
some people have more (supertasters) or less (nontasters)
connected with smell
Smell (olfaction): only sense that does not route through the thalamus, processed by olfactory bulb in limbic system, pheromones (odor molecules)