Respiratory System II Pathology Flashcards
WHO Classification of Tumours 5th Edition
- Thoracic Tumours
- International Histological Classification of Tumours No. 21
ICD-O Coding of Lung Tumours
- Lists various epithelial tumours, adenomas, adenocarcinomas, squamous cell carcinomas, and other tumour types with their corresponding ICD-O codes.
- Includes precursor lesions, salivary gland-type tumours and lung neuroendocrine neoplasms
- Covers tumours of ectopic tissues, mesenchymal tumours specific to the lung and haematolymphoid tumours.
Benign Epithelial Tumours
- Papillomas:
- Squamous cell papilloma, NOS (8052/0)
- Squamous cell papilloma, inverted (8053/0)
- Glandular papilloma (8260/0)
- Mixed squamous cell and glandular papilloma (8560/0)
- Adenomas:
- Sclerosing pneumocytoma (8832/0)
- Alveolar adenoma (8251/0)
- Papillary adenoma (8260/0)
- Bronchiolar adenoma / ciliated muconodular papillary tumour (8140/0)
- Mucinous cystadenoma (8470/0)
- Mucous gland adenoma (8480/0)
Squamous Cell Papilloma
- Benign tumor arising from epithelial surface, grows outward.
- Occurs in large bronchi, often with tracheal or laryngeal lesions.
- Presentation: endobronchial exophytic growth.
- Typically found in middle-aged male smokers.
- Associated with HPV 6 and HPV 11; high-risk HPV may be seen in cases associated with carcinoma.
- Clinical presentation: hemoptysis, recurrent pneumonia, asthma-like symptoms, dry cough.
Morphology Gross description
- Tan-white, friable, pedunculated / polypoid, smooth to verrucoid, glistening.
- Wart-like, cauliflower-like.
- Less than a few centimeters in size.
Microscopic Description
- Exophytic, papillary lesion with arborizing fibrovascular cores lined by keratinizing or nonkeratinizing mature squamous epithelium.
- Rarely inverted pattern.
- Lesion may grow into adjacent alveolar spaces.
- May contain areas lined by ciliated or non-ciliated columnar cells with cuboidal cells or mucin-filled cells (mixed squamous and glandular papilloma).
- May exhibit viral cytopathic effect: enlarged hyperchromatic nuclei, nuclear wrinkling, polychromasia, binucleate forms, perinuclear halos.
- Mild to moderate stromal inflammation which may be related to airway obstruction.
- No mitoses, no necrosis
Lung Cancer - Key Facts
- Leading cause of cancer-related deaths worldwide.
- Smoking is the leading cause (approximately 85% of cases).
- Often diagnosed at advanced stages.
- Screening high-risk individuals can improve survival rates.
- Primary prevention can reduce incidence.
Types of Lung Cancer
- Small Cell Lung Cancer (SCLC)
- Small Cell Carcinoma (SCC)
- Combined Small Cell Carcinoma (CSCC)
- Non-Small Cell Lung Cancer (NSCLC)
- Adenocarcinoma
- Squamous Cell Carcinoma (SCC)
- Large Cell Carcinoma (LCC)
Squamous Cell Carcinoma
- Malignant tumor from epithelial cells with squamous differentiation.
- Presence of keratinization or intercellular bridges.
- Strong association with tobacco smoking (80% of men and 90% of women).
- Primarily in patients over 50 years of age.
- 85% of primary lung cancers are non-small cell carcinomas, and squamous cell makes up 30% of these cases.
Sites
- Can present in any site of the lungs or bronchus but is more commonly central
- Mediastinal lymph nodes are the primary site of metastasis
- Hematogenous spread to distant organs:
- Bone
- Liver
Pathophysiology
- Squamous cells lining the respiratory tract transformed from carcinogen exposure, most frequently smoking.
- Occupational heavy metal exposure
Clinical Features
- Wide range of nonspecific pulmonary symptoms:
- Cough
- Chest pain
- Shortness of breath
- Hemoptysis
- Wheezing
- Weight loss
- Recurrent infections
- Loss of appetite and fatigue
Gross Description
- Cavitary mass with white, smooth cut surfaces that frequently contain hemorrhage and necrosis.
- Mass is more often central than peripheral and may be within the bronchus.
Microscopic Description
- Range from well differentiated squamous cell neoplasms showing keratin pearls and intercellular bridges to poorly differentiated neoplasms exhibiting only minimal squamous cell features.
- Poorly differentiated squamous cell carcinoma exhibits severe cellular and nuclear atypia, abundant mitosis and often requires IHC studies to differentiate from other poorly differentiated lesions.
Development of SCC
- Normal epithelium → Hyperplasia → Squamous metaplasia → Dysplasia → Carcinoma in situ → Invasive carcinoma
Characteristics
- Keratinization
- Intercellular bridges
- Abundant eosinophilic cytoplasm
Alveolar Adenoma
- Well circumscribed tumor consisting of cystic spaces lined by a single layer of type II pneumocytes overlying a spindle cell stroma.
- Solitary and peripheral tumors.
- Incidental diagnosis.
- Rare tumors
- Slightly female preponderance
- 39-74 y
Gross Description
- Size: 7-60 mm
- Well demarcated
- Smooth, lobulated multicystic pale yellow to tan surface cut.
Microscopic Description
- Cystic spaces filled with eosinophilic granular material.
- Lined by cytologically bland flattened to cuboidal epithelial cells (pneumocytes type II)
- Myxoid or collagenous stroma.
Lung Adenocarcinoma
- Most prevalent non-small cell lung carcinoma.
- Female > Male
- Most common type of lung cancer in male nonsmokers
- African Americans > Caucasians
- Age 60 - 70
Sites
- Upper lobe > lower lobe
- Peripheral > central
- Metastasis: brain (often only site) > bone > liver > adrenal
- Risk for brain metastasis increases with tumor size and lymph node stage
Pathophysiology
- Toxic cellular exposures → genetic mutations → proliferation of endobronchial cells
- Genetic events
Etiology
- Smoking is the greatest risk factor, including secondhand smoke
- Radon from soil, usually in residential areas
- Asbestos exposure
Clinical Features
- Cough (productive if mucinous adenocarcinoma), hemoptysis, dyspnea, weight loss, chest pain.
- Paraneoplastic / endocrine syndromes.
- Paraneoplastic syndromes are disorders that are triggered by an altered immune system response to a neoplasm. / Clinical syndromes involving nonmetastatic systemic effects that accompany malignant disease.
Gross Description
- Tan-white cut surface
- May have central area of scar or necrosis
- Usually well-defined but nonencapsulated
Microscopic Description
- 5 main histologic patterns:
- Lepidic: type II pneumocytes and club cells proliferate to line alveolar walls; lacks architectural complexity; no lymphovascular or perineural invasion
- Acinar: gland forming; round / oval glands invading the stroma (usually fibrous)
- Papillary: malignant cuboidal / columnar cells replace alveolar lining; contains fibrovascular cores.
- Micropapillary: ill-defined projection / tufting that lacks fibrovascular cores
- Solid: sheets of neoplastic cells