Mandela 2.5
Test Questions
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Phases of SA Node Depolarization
The SA node depolarization consists of three distinct phases:
Phase 4 (Slow Depolarization Phase)
Phase 0
Phase 3
Phase 4: Slow Depolarization Phase
Membrane Potential at Phase 4 Start:
Begins at -60 mV
Permeability Changes:
PNA (Permeability of Sodium) is increasing.
PK (Permeability of Potassium) is decreasing.
Current Generation:
The current generated due to sodium influx is referred to as Funny Current (IF).
The channel responsible for the influx of sodium ions is the HCN channel.
Reason for Naming (Funny Current):
Hyperpolarization triggers the opening of the HCN channel, contrary to the norm, which is unexpected (hence the term funny).
Requires:
Hyperpolarization
Cyclic AMP (cAMP)
Cyclic Nucleotide Binding Domain (CNBD):
Domain where cAMP binds to enable channel opening.
Ion Influx During Phase 4:
Sodium ions (Na⁺) enter the cell leading to the generation of funny current (IF).
Phase 0: Rapid Depolarization
Threshold Potential:
Reached at -40 mV, necessary to initiate an action potential in the SA node.
Permeability Changes:
At threshold:
Permeability for L-type Calcium channels increases.
Permeability for T-type Calcium channels decreases.
Calcium's Role:
The driving ion for Phase 0 is calcium ions (Ca²⁺).
This contrasts with Phase 0 in ventricular myocytes, which is driven by sodium ions.
Action Potential Shape Characteristics:
The upward trend of the action potential is smooth; there is no flat line in Phase 4.
Phase 3: Repolarization
Key Events in Phase 3:
Potassium channels open (K⁺ influx).
L-type calcium channels close.
Reiteration of Cycle:
After Phase 3, the cycle returns to Phase 4.
Summary of Driving Forces and Ion Channels
Phase 4 Start:
Driving force: Sodium (Na⁺)
Channels: HCN channels
Phase 0 Initiation:
Driving force: Calcium (Ca²⁺)
Phase 3 Dynamics:
Driving force: Potassium (K⁺)
Differences between SA Node and Ventricular Myocytes
True Resting Potential:
Ventricular myocytes have a true resting potential of -90 mV, while SA node does not.
Ion Channel Activity in Phase 4:
Only leaky potassium channels are open during Phase 4 in ventricular myocytes; sodium and calcium channels are closed.
Phase 0 Dynamics in Ventricular Myocytes:
Rapid depolarization due to sodium influx, in contrast to calcium primarily driving phase in SA node.
Phase 2: Plateau Phase (Ventricular Myocytes)
Importance of Phase 2:
This phase is crucial for cardiac muscle contraction.
Channel Activity:
Opening of L-type Calcium channels (Ca²⁺ influx).
Potassium ion (K⁺) efflux.
Electrical Neutrality in Phase 2:
Influx of calcium and efflux of potassium create a flat plateau as the charges neutralize each other.
Phase 3 (Ventricular Myocytes) Recollection
Ion Channels Characteristics:
All channels closed except delayed rectifier potassium channels (K ext{+}).
Involvement of Sodium-Potassium ATPase pump plays a significant role in restoring membrane potential.
On Drugs and Arrhythmia Understanding
Knowledge of the differences between SA node and ventricular myocytes assists in understanding the effects of drugs and depolarization processes in arrhythmias.