Proteins -II
Protein Folding
- Information for correct folding is in the primary sequence of proteins.
- Driven by thermodynamics; proteins fold to maximize weak non-covalent interactions, reducing free energy.
- Protein folding is cooperative.
- Some proteins require chaperones (e.g., Heat-shock proteins, Peptidyl-prolyl cis-trans isomerases, Protein-disulfide isomerases) to assist folding, prevent aggregation, and stabilize unfolded chains.
Denaturation of Proteins
- Loss of protein structure and function.
- Caused by: pH changes, temperature (T), chaotropic agents (urea, guanidine hydrochloride), detergents (SDS).
Post-Translational Modification (PTM) of Proteins
- Introducing chemical groups onto polypeptide chain after translation.
- Glycosylation:
- O-linked glycoproteins: Golgi, ether linkage to -OH of Ser and Thr.
- N-linked glycoproteins: ER, amide linkage to N of Asn within consensus sequence (Asn-X-Ser; Asn-X-Thr).
- Lipid modification (Lipoprotein):
- Acetylation: a- and ϵ-NH2 groups (e.g., histones).
- Myristoylation: N-terminal Gly (C14), amide linkage, involved in protein-protein/protein-lipid interactions, signal transduction.
- Palmitoylation: Cys residues near C-terminus (C16), thioester linkage, protein-protein interactions, membrane association.
- Polyisoprenylation: Cys residues near C-terminus (farnesyl PP [C:15], geranylgeranyl PP [C:20]), protein-protein interactions, signal transduction.
- GPI (glycosylphosphatidylinositol) anchor: At C-terminus of some membrane proteins.
- Phosphorylation:
- Dynamic, regulatory modification by protein kinases and phosphatases.
- Protein kinases: Transfer of γ-phosphate from ATP onto -OH group of Ser, Thr, or Tyr.
- Types: Ser/Thr kinases, Tyr protein kinases.
- Other modifications: Hydroxylation (e.g., HO-proline in collagen), Sulfation (Tyr), Ubiquitination (e.g., histones), Sumoylation (e.g., histones), Carboxylation.
Pathophysiology of Misfolding Proteins
- Misfolding leads to denaturation (loss of function) or partial function loss, causing diseases.
- Alzheimer's Disease (AD): Neurological degenerative disease marked by memory loss, confusion, and behavioral changes.
- Results from dense plaques of fibrillar β-amyloid proteins with well-ordered β-sheet secondary structure.
- A protein misfolding disease, possibly initiated by oxidative stress.
- Cystic Fibrosis (CF): Chronic disease affecting lungs and pancreas due to thick, sticky mucus.
- Caused by misfolding of the Cystic Fibrosis Transmembrane Conductance Regulator (CFTR) protein.
- Approximately 70% of cases involve a deletion of phenylalanine at position 508 (Phe508) in CFTR.
Protein Purification
- Separation of a protein of interest from a complex mixture.
- Procedure: Cell breakage/homogenization, protein extraction, purification (precipitation, chromatography).
- Requires an assay method for protein abundance (activity or antibody).
- Principles based on differences in:
- Molecular weight (gel filtration / size-exclusion chromatography).
- Charge properties (ion-exchange chromatography).
- Hydrophobicity (hydrophobic interaction chromatography).
- Affinity for substrates/ligands (affinity interaction chromatography).
- Gel filtration: Separates proteins by size; larger molecules elute first.
- Ion-exchange chromatography: Separates by net charge.
- Anion exchanger (positively charged stationary phase) binds negatively charged analytes.
- Cation exchanger (negatively charged stationary phase) binds positively charged analytes.
- Elution occurs via pH or salt gradient.
Protein Analysis
- Electrophoresis: Follows protein purification progress.
- SDS-PAGE: Migration is determined only by polypeptide size; SDS (anionic detergent) binds to proteins (1 SDS per 2 amino acid residues), dissociates them, and overwhelms native charge.
- Native PAGE: Migration is determined by both size and charge.
- 2D Gel Electrophoresis: Identifies proteins by two steps:
- 1. Isoelectric focusing (IEF): Separates proteins based on their isoelectric point (pI, charge).
- 2. SDS-PAGE: Separates proteins based on size.
- Proteins are often identified by mass spectrometry (MS) or Western blot after 2D gel.
- Western blot: SDS-PAGE followed by transfer of proteins onto a membrane and visualization with a specific antibody (Y).