addictions vol 1
Foundations of Addictions Counseling
Chapter 1: History and Etiological Models of Addiction
Substance Use in the United States
The Temperance Movement and Prohibition
Early views framed alcohol as healthy.
1784: Dr. Benjamin Rush identified alcoholism as a disease.
1800s: Clergy warned that alcohol corrupted body and mind.
Temperance movement initially promoted moderation, later shifting to abstinence.
1840: The Washingtonian Total Abstinence Society emerged, a precursor to AA.
Women played a major role through groups such as the Women’s Christian Temperance Movement, Daughters of Rechab, and Sisters of Sumaria.
1919: The Volstead Act prohibited manufacturing and sale of alcohol.
Prohibition reduced consumption but increased crime and social turmoil; repealed in 1933.
Post-Prohibition: States regulated alcohol sales; problems were attributed to people, not substances.
Early 20th century: Temperance movement spread internationally.
Early Drug Use and Regulation
Cocaine, opium, morphine widely used in medicines.
These substances were legal through the 19th century; marijuana legal until 1930s; LSD legal in 1950s.
1906: Pure Food and Drug Act passed.
1914: Harrison Act taxed opium and coca products.
1971: NIAAA established; President Nixon launched the War on Drugs.
1974: NIDA created.
1986: Anti-Drug Abuse Prevention Act created the U.S. Office for Substance Abuse Prevention.
1990s: Focus on controlling prescription drug misuse and drug importation.
Recent decades: Emphasis on incarceration over prevention and treatment.
2023: President Biden’s Unity Agenda prioritized addressing the opioid and overdose epidemic.
Current Policies Influencing Prevention
Policies emphasize punitive approaches, contributing to mass incarceration.
All states set minimum drinking ages and penalize sales to minors.
Some states maintain dry counties.
Alcohol pricing influenced through taxation and state-owned liquor stores.
Military settings may ban alcohol and tobacco.
DUI is a criminal offense; convictions raise insurance rates.
Establishments serving alcohol may be held civilly liable for harms caused by intoxicated patrons.
Illicit drug policies remain less specific than alcohol regulations.
2000: SAMHSA reauthorized, funding integrated treatment for co-occurring disorders.
2020: Oregon decriminalized drug use, shifting toward ticketing rather than incarceration.
Models Explaining the Etiology of Addiction
Moral Model
Addiction viewed as moral failing; no biological basis.
Influential in religious and legal systems.
Psychological Models
Cognitive-Behavioral Models: Addiction driven by cognitive and behavioral reinforcers.
Learning Models: Social learning shapes substance use behaviors.
Psychodynamic Models: Addiction linked to ego deficits, attachment issues, hostility, or early experiences.
Personality Models: Certain traits predispose individuals to addiction.
Family Models
Behavioral Model: Family members reinforce substance-using behavior.
Family Systems Model: Addiction maintains family roles; change disrupts system balance.
Family Disease Model: Addiction transmitted genetically and maintained environmentally.
Disease Model
Addiction is a primary, chronic, incurable disease.
Individuals identify as "recovering" rather than "recovered."
Public Health Model
Addiction results from societal influences; emphasizes promoting healthy behaviors.
Developmental Model
Risk and vulnerability change across the lifespan.
Developmental factors influence likelihood of substance misuse.
Biological Models
Genetic Models: Inherited metabolic defects interact with environment.
Neurobiological Models: Neurotransmitter systems shape addiction; includes Opponent Process and Incentive-Sensitization theories.
Supracultural Models: Cultural norms around guilt, aggression, and alcohol use influence addiction rates.
Culture-Specific and Subcultural Models
Cultural customs and attitudes shape substance use patterns.
Subgroups defined by age, gender, ethnicity, SES, religion, and family background show distinct patterns.
Multicausal Models
No single model explains addiction.
Etiology involves multiple interacting variables.
Includes Syndrome and Integral Models.
Chapter 2: Substances of Addiction
The age group most affected by substance use disorders is 18 to 25 years old.
Within the first six months of the COVID-19 pandemic, 13% of Americans reported increased substance use as a means of coping with stress and negative emotions related to the pandemic.
The ingestion of chemicals results in a substance use disorder for only a small subset of people.
Depressants
Depressants are a class of substances that cause depression of the central nervous system (CNS), generally resulting in drowsy or calming behavioral effects.
This depressant effect results from slowing messages between the brain and body.
A key neurotransmitter involved in this effect is γ-aminobutyric acid (GABA).
Alcohol
Ethanol, or ethyl alcohol, is the most abused mood-altering substance today.
Approximately 50% of people in the United States over age 12 reported alcohol use within the past month.
Guidelines for daily alcohol use: 2 drinks for men, 1 drink for women.
A common misconception is that some alcoholic beverages are safer or less addicting than others.
The effects of alcohol are experienced biologically for as long as the ethanol remains in the body, and the liver processes ethanol at a relatively the same rate for most people.
Respiratory arrest or aspiration of vomit are leading causes of death due to alcohol use.
Sedatives/Hypnotics
The most common drugs in this group are benzodiazepines, barbiturates, and nonbarbiturates.
Benzodiazepines, referred to as tranquilizers, are frequently prescribed for symptoms like sleeplessness, anxiety, muscle strains, and seizures.
Researchers have found that benzodiazepines such as Valium (diazepam) and Xanax (alprazolam) are highly addictive with long-term use and often have severe withdrawal complications requiring medical detoxification.
Barbiturates act quickly, decreasing neurotransmitter activity and depressing the CNS; they are particularly dangerous when combined with other CNS depressants like alcohol.
Nonbarbiturates include drugs such as Quaalude (methaqualone) that have a similar physiological profile to barbiturates.
Opioids
Opioids include both natural (opiates) and synthetic drugs, with examples including codeine, morphine, and heroin, as well as synthetic versions like OxyContin and fentanyl.
Opioids are often abused for their dramatic effects on the brain’s reward pathway.
The increase in opioid use has escalated substantially over the past two decades; among young adults aged 18 to 25, heroin use has doubled.
The increased rate of use and addiction correlates with high levels of opioid prescribing.
High-prescribing rates remain in small cities, large towns, and areas with higher proportions of White residents.
Medication-assisted therapy, such as methadone, is frequently used in the treatment of opioid addiction.
Stimulants
Stimulants are substances that increase brain activity primarily through the neurotransmitters dopamine and norepinephrine, involved in the reinforcement of rewards.
Prescription stimulants are used for ADHD and narcolepsy, aimed at boosting alertness and energy levels.
Misuse can lead to anger, paranoia, and psychosis; stimulant overdose can result in symptoms such as tremors, confusion, and irregular heartbeat.
Withdrawal can bring fatigue, depression, or sleep issues.
Nicotine
Nicotine is a stimulant found in tobacco products.
Associated dangerous health risks: increased cancers, heart disease, stroke, diabetes, and chronic respiratory illnesses.
Smoking and tobacco addiction lead to over six million deaths globally each year.
Treatment for nicotine addiction often includes medications like bupropion and varenicline.
Ephedrine, Amphetamines, and Amphetamine-Like Medications
Used in ancient Chinese medicine for over 5,000 years; legal use peaked in the U.S. after 1930.
Misuse of amphetamines can lead to severe side effects, including psychosis and death.
Methamphetamine is a popular variant that can be administered in various forms and is known for its highly addictive properties.
Cocaine
Historically, cocaine was used in medical practice starting in 1884, but epidemics of addiction led to its prohibition through the Harrison Act of 1914.
The rapid inhalation of cocaine results in intense cravings, making it addictive within weeks to months.
Cannabinoids
Marijuana is one of the most abused federally illicit drugs in the U.S., despite its growing decriminalization.
Tolerance, withdrawal, and dependence can develop with chronic use, though less intense than with other substances.
Negative effects include decreased lung capacity and psychosis complications.
Hallucinogens and Other Psychedelics
Examples include LSD, psilocybin, and MDMA. While hallucinogens can alter perceptions, they do not have physically addicting properties like tolerance or withdrawal symptoms.
Neurobiology and Physiology of Addiction
The biological basis of addiction involves activation of the reward pathways in the brain, including the ventral tegmental area (VTA), nucleus accumbens (NAc), and prefrontal cortex. Dopamine is a key neurotransmitter in this process, along with others like GABA and glutamate.
The addiction cycle consists of three phases:
- Phase 1: Binge/Intoxication
- Phase 2: Negative Affect/Withdrawal
- Phase 3: Preoccupation/Anticipation
Chapter 3: Addictions Classification
Addictions can be classified as either ingestive or process addictions. Process addictions encompass behaviors producing euphoria without mood-altering substances.
Research indicates that physical dependence is not always necessary to diagnose an addiction.
**