Synaptic Transmission Notes
Synaptic Transmission
Lecture Objectives
Explain the different types of synapses (chemical, electrical).
Describe in detail how an electrical signal (action potential) is transmitted from one neuron to another by a chemical synapse.
Explain how a synaptic potential may generate an action potential in the postsynaptic neuron, and that this may be an Excitatory (EPSP) or Inhibitory Post-Synaptic Potential (IPSP).
Outline
Electrical vs. chemical synapses.
Mechanisms of chemical synaptic transmission.
Types of post-synaptic potentials.
Synaptic Transmission
A synapse is the junction between neurons or between a neuron and an effector.
Electrical Synapse: Gap junctions connect cells and allow the transfer of information to synchronize the activity of a group of cells.
Chemical Synapse: One-way transfer of information from a presynaptic neuron to a postsynaptic neuron. The lecture focuses on chemical synapses.
Neural Pathways and Synapses
Specific brain pathways are activated (e.g., dorsal column pathway, corticospinal pathway).
These pathways are composed of a series of neurons.
Neurons communicate via specialized sites called synapses.
Information transfer to and from the brain occurs through these pathways.
Communication Between Neurons
Communication between neurons is referred to as synaptic transmission.
Synaptic transmission may be either:
Electrical via connexons.
Chemical, mediated by neurotransmitters.
Electrical Synapse Details
Gap junctions connect the cytoplasm of two neurons.
Ions can flow directly through connexon channels.
Connexons are made of connexin proteins.
Electrical synapses synchronize activity of interconnected neurons.
Distance between membranes: 3.5 nm
Gap junction channel diameter: 20 nm
Chemical Synapse Details
Chemical synapses use neurotransmitters to transmit signals from one neuron to another.
Types of connections:
Axoaxonic (axon to axon)
Axodendritic (axon to dendrite)
Axosomatic (axon to cell body)
Mechanisms of Chemical Synaptic Transmission
Transmitter Synthesis and Storage:
Neurotransmitter is synthesized and then stored in vesicles.
Action Potential Arrival:
An action potential invades the presynaptic terminal.
Depolarization and Calcium Influx:
Depolarization of the presynaptic terminal causes opening of voltage-gated Ca^{2+} channels.
Calcium's Role:
Influx of Ca^{2+} through channels.
Ca^{2+} causes vesicles to fuse with the presynaptic membrane.
Neurotransmitter Release:
Transmitter is released into the synaptic cleft via exocytosis.
Receptor Binding:
Transmitter binds to receptor molecules in the postsynaptic membrane.
Postsynaptic Current:
Opening or closing of postsynaptic channels.
Postsynaptic current flow.
Postsynaptic Potential:
Postsynaptic current causes excitatory or inhibitory postsynaptic potential that changes the excitability of the postsynaptic cell.
Neurotransmitter Removal:
Removal of neurotransmitter by glial uptake or enzymatic degradation.
Events at the Chemical Synapse: Synaptic Transmission
AP Arrives: Action potential arrives at the synaptic bouton.
Depolarization: Depolarizes synaptic bouton.
Calcium Channels Open: Depolarization opens voltage-gated Ca^{2+} channels. The intracellular/extracellular Ca^{2+} concentration gradient means Ca^{2+} moves into the terminal.
Vesicle Fusion: Ca^{2+} influx triggers synaptic vesicle (SV) fusion. Ca^{2+} sensing proteins are activated on the SV membrane and presynaptic neuron membrane, resulting in fusion.
Neurotransmitter Release: Neurotransmitter is released from synaptic vesicles.
Receptor Activation: Activates receptors on the postsynaptic cell.
Neurotransmitter Clearance: Neurotransmitter diffuses or is cleared from the synaptic cleft.
Key Components at the Synapse
Synaptic vesicles contain neurotransmitter.
Synaptic vesicles are localized to the active zone.
Voltage-gated Ca^{2+} channels are present in the presynaptic terminal.
There is a space between the presynaptic terminal and postsynaptic cell called the synaptic cleft.
Receptors are localized to the postsynaptic density of the postsynaptic neuron.
Tetrodotoxin (TTX)
Puffer fish contain tetrodotoxin (TTX).
TTX blocks sodium channels, so no action potential is generated.
Synaptic Inputs onto Dendrites
Dendrites contain microtubule-associated protein 2 (MAP2).
Synapses contain synaptic vesicle protein called synaptotagmin.
Communication is unidirectional.
There can be 1000’s of synapses, both excitatory and inhibitory, on a single neuron.
Neurotransmitters Regulate Ion Channels
Directly (Ligand-Gated Ion Channel Receptors):
The receptor is part of the ion channel complex.
Neurotransmitter binding changes the permeability of the ion channel.
Allows fast postsynaptic potentials (10-100 ms duration).
Indirectly (G Protein-Coupled Ion Channel Receptors):
The receptor complex is separate from the ion channel.
Allows slow postsynaptic potentials (100ms - 2s).
Plays a major role in overall modulation.
One Neurotransmitter, Different Effects
One neurotransmitter can act on different receptors/channels.
The receptor type determines the response of the cell.
Example: Acetylcholine (ACh)
Nicotinic receptors: e.g., skeletal muscle cells
Directly ligand-gated channel.
Opened by ACh binding.
Receptor permeable to Na^{+} (influx) and K^{+} (efflux).
Main effect is increased Na^{+} influx.
Fast Excitatory Post Synaptic Potential (EPSP) results.
Muscarinic receptors: e.g., smooth muscle cells
Indirectly gated channel linked to a K^{+} leak channel that is open to K^{+} at rest.
Closed by ACh binding.
G protein closes K^{+} channel and decreases permeability to K^{+}.
Main effect is decreased K^{+} efflux.
Slow EPSP results.
Receptor (Synaptic) Potentials
Receptor Potentials are triggered by synaptic transmission
Excitatory postsynaptic potentials (EPSP): A depolarizing postsynaptic potential.
Inhibitory postsynaptic potentials (IPSP): A hyperpolarizing postsynaptic potential.
A postsynaptic neuron can receive many signals at once.
Additional Resources
Lisman JE, Raghavachari S, Tsien RW. The sequence of events that underlie quantal transmission at central glutamatergic synapses. Nature Review Neuroscience (2007), 8: 451-465.
Ryan TJ & Grant SGN. The origin and evolution of synapses. Nature Review Neuroscience (2009), 10: 701-712.