Enzymatic Mechanisms
Lecture Overview
Title: Molecular Biology & Biochemistry Lecture 12: Enzyme Active Sites - Mechanism
Presenter: Dr. Emily Flack
Learning Outcomes
Understand key features of enzyme active sites.
Familiarity with experimental methods to study active sites and mechanisms.
Explain the mechanism of action for serine proteases and carbonic anhydrase, focusing on binding specificity and catalysis.
Analyze substrate binding and transition state formation in enzyme catalysis.
Enzyme Active Sites
Definition: The active site is crucial for substrate binding and catalysis.
Characteristics:
Small relative to the entire protein.
Three-dimensional cleft formed by amino acid residues from different parts of the primary structure.
Substrate binding is mediated by weak, non-covalent interactions (e.g., hydrogen bonds).
Mechanisms of Enzyme Catalysis
Types of Catalysis:
Covalent Catalysis (serine proteases)
Acid-base Catalysis (serine proteases and carbonic anhydrase)
Metal Ion Catalysis (carbonic anhydrase)
Proximity and Orientation Effects (various enzymes)
Electrostatic Catalysis (serine proteases)
Preferential Binding to Transition State (serine proteases)
Understanding Enzyme Mechanisms
Structural Biology Methods:
X-ray crystallography provides models of protein structure, useful for understanding enzyme mechanisms.
Substrate analogues bind to active site without being processed by the enzyme.
Mutations in key amino acids can be used to probe functions.
Proteases
Definition: Hydrolase enzymes that cleave peptide bonds.
Selectivity: Depends on the structure of the enzyme and substrate specificity.
Serine Proteases
Family of enzymes hydrolyzing proteins in the digestive system.
Roles in digestion, blood clotting, fertilization, and immune response.
Inactive forms are released from the pancreas as zymogens (proenzymes).
Example: Trypsin is activated from Trypsinogen.
Chymotrypsin Specificity
Substrate Preference: Favors bulky aromatic amino acids (e.g., tryptophan, phenylalanine).
Mechanism:
Active site includes a hydrophobic pocket accommodating aromatic residues.
Cleaves on the carbonyl side of aromatic residues.
Comparison of Serine Proteases
Trypsin: Cleaves after basic residues (Arg, Lys); contains Asp189 at the bottom of its pocket.
Elastase: Cleaves after small neutral residues; shallow pocket with bulky residues.
Catalytic Triad of Serine Proteases
Components: Serine, Histidine, Aspartate are key for enzymatic function.
Chymotrypsin Mechanism:
Serine oxygen is activated through interaction with Histidine.
Formation of a covalent bond between serine and substrate.
Stabilization of intermediates in the oxyanion hole.
Catalytic breakdown leading to product release.
Oxyanion Hole
Function: Stabilizes the transition state intermediate during catalysis.
Interactions help lower the energy barrier for the reaction.
Summary of Chymotrypsin Mechanism
Details on substrate specificity and catalytic steps including the role of serine as a nucleophile, involvement of histidine, and the stabilization of transition states.
Evolution of Serine Proteases
Divergent Evolution: Similar structures with different functions (e.g. Chymotrypsin, Factor X).
Convergent Evolution: Different folds with the same active site residues (e.g. Subtilisin).
Role of Metal Ions in Catalysis
Example: Carbonic Anhydrase (CA) utilizes zinc ions in catalysis.
Function of Zinc: Activates water, stabilizes hydroxide ion, and plays a role in rapid reactions.
Carbonic Anhydrase Functions
Catalyzes the hydration of CO2 for effective transport, pH regulation, and water balance.
Achieves high reaction rates (Kcat ≈ 10^6 s^-1) at neutral pH using metal ion-mediated catalysis.
Summary Points
Enzyme active sites are a small part of the protein's structure yet crucial for function.
Active sites involve residues and metal ions that contribute to catalysis and specificity.
Structural insights allow for understanding of enzyme mechanisms and their evolution.