Arrhythmias – Rapid Review
Causes of Arrhythmia
- Ischaemic heart disease, drugs, rheumatic heart disease, cardiomyopathy, thyrotoxicosis
- Oxygen, fluid or electrolyte disturbance (eg K+, Mg2+)
Differentiating SVT vs VT
- SVT: narrow QRS, usually regular, no/abnormal P waves, ⇓ with adenosine
- VT: broad QRS, may be irregular, P waves dissociated, inverted QRS, no adenosine response, fusion beats possible
Ventricular Ectopics (VE)
- Unifocal (same shape) or multifocal (different shapes); pulse irregular
- Danger: R-on-T ⇒ ventricular fibrillation
- Treat if VE:normal >1:6 OR multifocal OR new onset (may signal sepsis)
- Common precipitants: post-MI, hypoxia, hypokalaemia, hypomagnesaemia, digitalis toxicity, valvular/cardiomyopathic disease
Sinus Tachycardia
- Regular rhythm, ≤160 bpm (lower in elderly)
- Normal P morphology, gradual onset
- Always treat underlying cause (hypovolaemia, anaemia, PE, sepsis)
Paroxysmal Supraventricular Tachycardia (PSVT)
- Origin: SA node, atria or AV node
- P waves abnormal/hidden; QRS usually narrow (may widen with BBB)
- Rate 150!–!250bpm; regular
- May cause ST depression (ischaemia)
- Acute therapy: adenosine (diagnostic & therapeutic)
- Ongoing control: verapamil, digoxin or β-blocker (avoid β-blocker + verapamil)
Atrial Fibrillation (AF)
- Irregularly irregular; ventricular rate 100!–!180bpm
- Post-op triggers: hypovolaemia, hypoxia, hypoK+/hypoMg2+
- Serious adverse signs (hypotension, shock, chest pain, failure, LOC, rate >140): urgent DC cardioversion or IV amiodarone
- Without adverse signs: correct triggers ± digoxin or amiodarone
- Long-standing AF may worsen if regular meds omitted
Atrial Flutter
- Saw-tooth flutter waves at ≈300min−1; QRS normal with variable AV block
- Often coexists with AF; linked to structural heart disease
- Adenosine can transiently reveal/flutter or terminate
- Management: cardioversion, digoxin, verapamil