silent killer,
Cardiovascular System Overview
Delivered by Mrs. Krista K. Fowler, MSN, RN, NP-C
Email: ksklemm@ua.edu
Functions of the Cardiovascular System
Vital functions include:
Delivers vital oxygen and nutrients to cells
Removes waste products from the body
Transports fluids, electrolytes, and hormones
Arteries: Carry blood away from the heart
Composed of three layers:
Tunica intima: Inner layer made of endothelial cells; metabolically active and in contact with blood.
Tunica media: Middle layer of smooth muscle and nerve fibers.
Constriction (alpha-adrenergic stimulation)
Dilation (beta-adrenergic inhibition)
Tunica adventitia: Outer layer of supportive connective tissue.
Key for regulating blood flow under high volume and pressure.
Veins: Carry blood back to the heart
Composed of three layers similar to arteries.
More compliant than arteries.
Utilize valves and muscle contraction to promote venous return.
Capillaries: Site of exchange between blood and tissues.
Arterial Disorders
Arterial Structure:
Blood carried away from the heart via the aorta with three distinct wall layers.
Arterial Endothelium:
Acts as a semipermeable barrier between the vessel and tissue.
Injury leads to endothelial dysfunction which can result in:
Atherosclerosis
Coronary artery disease
Diabetes mellitus
Hypertension
Hypercholesterolemia
Smoking effects.
Cardiac Output (CO)
CO represents the volume of blood expelled from the left ventricle per minute.
It is considered a gold standard measure for the heart's effectiveness as a pump.
A normal CO value is approximately 5 L/min.
Determinants of CO:
Stroke volume (SV): Volume of blood expelled with each left ventricular contraction, averaging around 70 mL/contraction.
Ejection fraction: Percentage of diastolic volume ejected during systole.
Heart rate (HR): Number of times the left ventricle contracts in a minute.
Formula:
Factors Influencing Cardiac Output
Preload: Volume of blood in the left ventricle prior to contraction.
Afterload: Resistance the left ventricle must overcome to eject blood.
Contractility: The force of left ventricular contraction.
Blood Pressure (BP)
Defined as the force blood exerts on vessel walls.
BP is represented as a fraction:
Systolic: Measures cardiac contraction (normal <120 mmHg)
Diastolic: Measures cardiac relaxation (normal <80 mmHg)
Example: 132/82 mmHg indicated a systolic BP of 132 mmHg and diastolic BP of 82 mmHg.
Pulse pressure is around 40 mmHg, calculated as the difference between systolic and diastolic (SBP - DBP).
Peripheral Vascular Resistance (PVR): Resistance to blood flow in vessels.
Blood Pressure equation:
Mean Arterial Pressure (MAP)
Major indicator of cerebral perfusion; desired MAP > 60 mmHg.
Calculation formulas:
OR
Blood Pressure Regulation
Nervous System Regulators
Parasympathetic Nervous System (PNS)
Affects the heart via the vagus nerve leading to decreased heart rate but has no direct effect on vessels.
Sympathetic Nervous System (SNS)
Impacts both heart and vessels via spinal cord:
Causes vasoconstriction (alpha receptors) and increases heart rate and contractility.
Receptor Regulators
Baroreceptors: Sensitive to blood pressure changes; located in arteries and stimulate SNS response upon pressure drops.
Chemoreceptors: Monitor changes in blood chemistry affecting BP regulation.
Hormonal Regulators
RAAS System: Activates when BP or blood volume decreases, triggering renal reabsorption of water/sodium and potassium excretion.
Vasopressin (ADH): Causes vasoconstriction and reabsorption of water in kidneys.
ANP and BNP: Hormones released when BP or blood volume increases; promote natriuresis (excretion of water and sodium).
Hypertension
Defined as a prolonged elevation in blood pressure.
Major risk factors include excessive cardiac workload due to increased afterload and vasoconstriction.
Types
Primary Hypertension:
Most common form (90-95% of cases).
Develops gradually with no known cause.
Secondary Hypertension:
More sudden and severe; resulting from pathology in other systems/organs.
Both forms are significantly risk-laden for coronary artery disease (CAD), stroke, and myocardial infarction (MI).
Risk Factors
Advancing age, ethnicity (higher prevalence in African Americans), family history, obesity, diabetes, sedentary lifestyle, tobacco use, high sodium/low potassium/Vitamin D diet, excessive alcohol consumption, and stress.
Diagnosis
Involves medical history, physical examinations, multiple blood pressure readings across different times, with a waiting period of 5 minutes between assessments.
Laboratory tests also help determine organ damage presence.
Manifestations
Often termed the