Drug Use, Drug Addiction, and the Brain’s Reward Circuits

Chapter 15: Drug Use, Drug Addiction, and the Brain’s Reward Circuits: Chemicals That Harm with Pleasure

Learning Objectives

  • 15.1 Compare the various routes of drug administration.
    • Understand different methods by which drugs enter the body, including ingestion, injection, inhalation, and absorption.

  • 15.2 Explain the ways in which drugs can influence the nervous system and how they are eliminated from the body.
    • Explore the mechanisms by which drugs affect neural activity and brain function.
    • Understand the metabolic pathways for drug elimination.

  • 15.3 Describe how the body becomes tolerant to drugs and the process of drug withdrawal. Explain what it means to be physically dependent on a drug.
    • Detail the concept of drug tolerance, mechanisms involved, and characteristics of withdrawal symptoms.
    • Definition of physical dependence, where the body adapts to a drug resulting in withdrawal symptoms when the drug is discontinued.

  • 15.4 Define drug addiction.
    • Definition of addiction; emphasize that it involves compulsive drug use despite known adverse consequences.

  • 15.5 Explain contingent drug tolerance.
    • Describe the principle that tolerance can develop in relation to the specific circumstances or environments associated with drug intake.

  • 15.6 Describe conditioned drug tolerance and conditioned compensatory responses.
    • Explain the concept of conditioned drug tolerance which refers to increased tolerance related to environmental contexts associated with drug use.
    • Discuss conditioned compensatory responses, which are physiological changes triggered by drug-related cues.

  • 15.7 Describe the health hazards associated with nicotine consumption.
    • Identify multiple health risks linked to nicotine, including its addictive nature and consequences for physical health.

  • 15.8 Describe the health hazards associated with alcohol consumption and the various stages of the full-blown alcohol withdrawal syndrome.
    • Understand the health impacts of excessive alcohol consumption, highlighted stages of withdrawal including symptoms such as tremors, anxiety, seizures, and delirium tremens.

  • 15.9 Explain the health effects of marijuana and the mechanism of action of THC.
    • Discuss the biological effects of THC bound to cannabinoid receptors and the subsequent health implications.

  • 15.10 Describe the health hazards associated with the consumption of cocaine and other stimulants.
    • Outline the risks associated with stimulant consumption, including cardiovascular problems and addictive tendencies.

  • 15.11 Describe the health hazards associated with the consumption of opioids and the opioid withdrawal syndrome.
    • Discuss the dangers of opioid use including addiction potential and severe withdrawal syndrome.

  • 15.12 Explain why it is difficult to determine causality in studies of the health hazards of drugs.
    • Address complexities in establishing cause-and-effect relationships due to confounding variables in studies.

  • 15.13 Compare the direct health hazards of nicotine, alcohol, marijuana, cocaine, and heroin.
    • Compare and contrast the health risks posed by these substances based on their physiological effects and societal impacts.

  • 15.14 Explain the physical-dependence and positive-incentive perspectives of addiction.
    • Physical-dependence perspective focuses on withdrawal avoidance as a motivator for continued use.
    • Positive-incentive perspective focuses on the pleasurable effects of drugs that drive use.

  • 15.15 Describe the intracranial self-stimulation (ICSS) paradigm.
    • Explain this method used to measure the rewarding effects of stimulation in specific brain circuits.

  • 15.16 Describe two methods for measuring the rewarding effects of drugs.
    • Detail behavioral paradigms including drug self-administration and conditioned place preference.

  • 15.17 Explain the role of the nucleus accumbens in drug addiction.
    • Discuss the critical role of the nucleus accumbens in mediating the effects of addictive drugs and the experience of reward.

  • 15.18 Describe the three stages in the development of a drug addiction.
    • Outline progression from initial drug use to habitual use, then to addiction characterized by cravings and relapse.

  • 15.19 Describe two sets of findings that have challenged the relevance of drug self-administration studies.
    • Identification of environmental factors and potential moderating variables influencing outcomes of drug self-administration studies.

  • 15.20 Explain the significance of the case of Sigmund Freud.
    • Discuss Freud’s experiences with cocaine and its eventual implications for his health, particularly oral cancer.

Drug Administration, Absorption, and Penetration of the Central Nervous System

  • Routes of Administration:
    Ingestion: Oral intake of drugs.
    Injection: Direct entry into the bloodstream via different techniques (intravenous, intramuscular, subcutaneous).
    Inhalation: Direct respiratory intake leading to rapid absorption into the bloodstream.
    Absorption: Drug contact with mucous membranes, including nasal membranes.

Drug Action, Metabolism, and Elimination

  • Drug Penetration of CNS:
    • Varied ability of substances to penetrate the blood-brain barrier, affecting the onset and intensity of drug action.

  • Mechanisms of Drug Actions:
    • Drugs can mimic neurotransmitters or interfere with their action, leading to altered neurological activity.

  • Drug Metabolism and Elimination:
    • Metabolism through liver enzymes (e.g., cytochrome P450 family) and variations in individual metabolic rates affect how long a drug stays active in the body.

Drug Tolerance, Drug Withdrawal Effects, and Physical Dependence

  • Drug Tolerance:
    • Defined as a reduced response to a drug after repeated use, requiring increased doses for the same effect.
    How Measured: Tolerance can be quantified through dose-response curves, demonstrating rightward shifts in response thresholds over time after regular administration.
    Types of Tolerance:
    Metabolic Tolerance: Body metabolizes the drug more efficiently.
    Functional Tolerance: Neurological adaptations reduce the effects of the drug.

  • Drug Withdrawal Effects:
    • Symptoms vary depending on the drug but generally include physical and psychological effects when drug use is abruptly stopped.

  • Physical Dependence:
    • Characterized by the body's adaptation to a substance leading to withdrawal symptoms in absence of use, reinforcing the cycle of addiction.

Drug Addiction: What Is It?

  • Defining Features of Addiction:
    • Persistent use of drugs despite harmful consequences; different from mere physical dependence which can occur without addictive behavior.
    • The context of addiction also encompasses behavioral addictions, beyond just chemical substances.

Contingent Drug Tolerance

  • Concept of Contingent Drug Tolerance:
    • Empirical research shows tolerance can develop related to specific cues and contexts surrounding drug use.
    Pinel Research on Ethanol and Convulsions: This highlighted that tolerance develops differently based on the timing of drug administration relative to stimulus or environmental cues.

Conditioned Drug Tolerance
  • Conditioned Drug Tolerance and Drug Predictive Stimuli:
    • Conditioned responses can be elicited by environmental cues involved in drug use, influencing tolerance levels and potentially leading to overdosing.
    Siegel's Conditioned Compensatory Response Theory: Highlights physiological adaptations to the conditioning environment leading to decreased drug efficacy over time.
    Conditioned Withdrawal Effects: Recovery processes that engage the body’s intrinsic responses can mimic withdrawal in environments associated with drug taking.

Five Commonly Used Drugs

  • Nicotine:
    • The primary psychoactive ingredient acting on nicotinic receptors.
    • Notably addictive and identified as a leading cause of preventable deaths.
    • Associated health conditions include Buerger’s disease and teratogenic effects.

  • Alcohol:
    • Known as a depressant, both water-soluble and fat-soluble.
    • Used extensively with notable tolerance and dependence issues, including alcohol withdrawal syndrome characterized by severe physical and psychological symptoms.
    • Risk of fetal alcohol syndrome when consumed during pregnancy.

  • Marijuana:
    • Comes from the cannabis plant, containing THC and other psychoactive elements.
    • Different routes of administration affect its impact; potential for addiction, tolerance, and withdrawal.
    • THC binds to cannabinoid receptors affecting mood and appetite, along with clinical benefits for various health conditions.

  • Cocaine and Other Stimulants:
    • Highly potent stimulants characterized by short-term euphoria and significant addiction potential.
    • Cocaine administration methods (snorting, intravenous, or smoking) influence its onset and intensity.
    • Mechanism of action involves dopamine receptors and significant side effects leading to addiction.

  • Opioids: Heroin and Morphine:
    • Regarded as strong analgesics, with a high potential for addiction and physiological tolerance.
    • Bind to opioid receptors, providing pain relief and euphoric effects while posing severe health risks and withdrawal challenges.
    • Treatment options include substitutive therapies like methadone and buprenorphine.

Interpreting Studies of the Health Hazards of Drugs

  • Observational Nature of Studies:
    • Most research is correlational, exploring relationships within highly addicted populations, which complicates establishing causality.

Comparison of the Hazards of Nicotine, Alcohol, Marijuana, Cocaine, and Heroin

  • Health Impact Rankings:
    • Tobacco and alcohol present more severe health risks compared to other drugs; with global death rates indicating a substantial public health challenge from these substances.

Physical-Dependence and Positive-Incentive Perspectives of Addiction

  • Understanding Variability in Addiction Theories:
    • Physical-dependence focuses on coping with withdrawal symptoms.
    • Positive-incentive reflects motivations behind drug intake based on pleasurable effects, encompassing social and psychological dimensions of addiction.

Intracranial Self-Stimulation and the Mesotelencephalic Dopamine System

  • Pioneering Research by Olds and Milner (1950s):
    • Identified pathways in the brain involved in reward processes, particularly the mesocorticolimbic pathway including the substantia nigra and ventral tegmental area.

  • Evidence Supporting the Dopaminergic Hypothesis:
    • Studies linking dopaminergic signaling to addiction behaviors and the reinforcing properties of drugs as stimuli activating these circuits.

Timeline of Addiction Theory

  • Historical Perspectives:
    • Evolution of understanding addiction, showcasing how approaches to behavioral and physiological mechanisms have been shaped over time by scientific discoveries.

Three Stages in the Development of an Addiction

  • Stages of Progression:
    Initial Drug Taking: First exposure, often experimental or recreational.
    Habitual Drug Taking: Characterized by an increase in use driven by positive incentive, sensitivity of dopamine system, and behavior reinforcement.
    Craving and Relapse: Triggered by environmental cues, stressful events, or drug primes leading to compulsive drug-seeking behaviors.

Ethical Considerations and Real-World Implications

  • Addiction Research:
    • Addressing potential biases in addiction studies, especially those surrounding models that focus heavily on stimulatory drugs and ignore environmental influences.

A Noteworthy Case of Addiction: Sigmund Freud

  • Freud's Experimentation with Cocaine:
    • Personal use and professional endorsement raised questions about the understanding of addiction and its health impacts, culminating in serious health decline due to oral cancer.