Control of Gene Expression
Controlling gene expression is often accomplished by controlling transcription initiation.
Gene expression is often controlled by regulatory proteins binding to specific DNA sequences.
Regulatory proteins bind to DNA to either block or stimulate transcription, depending on how they interact with RNA polymerase.
Regulatory proteins gain access to the bases of DNA at the major groove.
Regulatory proteins possess DNA-binding motifs
Prokaryotic Regulation
Prokaryotic organisms regulate gene expression in response to their environment.
Eukaryotic cells regulate gene expression to maintain homeostasis in the organism.
Sometimes regulation means that a cell induces apoptosis
Programmed cell death
Prokaryotes use operons
Sequence of DNA containing a cluster of genes under the control of a single promoter
Control of transcription initiation can be:
Positive control: increases transcription when activators bind DNA
Negative control: reduces transcription when repressors bind to DNA regulatory regions called operators
The promoters positions and orients the polymerase correctly
The operator is where the repressor binds
RNA polymerase binds to a region that is immediately upstream from the region of DNA that codes for a protein. The binding region is termed the operator. The promoter acts to position the polymerase correctly, so that the molecules can then begin to move along the DNA, interpreting the genetic information as it moves along.
Operons can regulate genes negatively or positively
→ Negative regulation
Uses a repressor protein
Can be inducible (EX: lac operon)
Can be repressible (EX: trp operon)
→ Positive regulation
Uses an activator protein
Activators: glucose, cAMP, and CAP
The trp operon
Encondes genes for the biosynthesis of tryptophan
The operon is not expressed when the cell contains sufficient amounts of tryptophan
The operon is expressed when levels of tryptophan are low
It is negatively regulated by the trp repressor protein
The presence of tryptophan causes the activation of the repressor, the repressor stops the production of tryptophan = repressed
→ trp repressor binds to the operator to block transcription
→ Binding the repressor to the operator required a co-repressor which is tryptophan
→ low levels of tryptophan prevent the repressor from binding to the operator
The lac operon
Contains genes to breakdown lactose as an energy source
Regulatory regions of the operon include the CAP binding site, promoter, and the operator
The lac operator is negatively regulated by a repressor protein
The action is induction
→ the lac repressor binds to the operator to block transcription in the presence of lactose, an inducer molecule binds to the repressor protein, so the repressor can no longer bind to the operator
→ now transcription can proceed
If glucose is present
RNA pol poorly binds to the lac operon promoter
The operon needs extra help from catabolite activators protein (CAP)
If no glucose is around, cAMP goes up
cAMP binds with and activates CAP
CAP binds to a region of DNA just before the lac operon promoter and helps RNA pol attach to the promoter, driving high levels of transcription
Glucose, because the CAP site activation increases operon activity end, because it promotes the activity in response to the CAP/cAMP activations, it is said to be positively regulated. CAP/cAMP is an activator
Positive control - the control of lac by cAMP and CRP
Eukaryotic regulation
Controlling the expression of eukaryotic genes requires transcription factors
General transcription factors are required for transcription initiation and required for proper binding of RNA pol to the DNA
Specific transcription factors increase transcription in certain cells or in response to signals
Coactivators and mediators are also required for the function of transcription factors
Coactivators and mediators bind to transcription factors and bind to other parts of the transcription apparatus
The entire thing together is the TRANSCRIPTION COMPLEX
General transcription factors bind to the promoter region of the gene
RNA pol II then binds to the promoter to begin transcription at the start site (+1)
Enhancers are DNA sequences to which specific transcription factors (activators) bind to increase the rate of transcription
Are often far upstream of the gene. When bound, it will fold DNA until it is close to the gene causing the activation of transcription and increasing it beyond basal levels.
Eukaryotic Chromosome Structure
Methylation (addition of CH3) of DNA or histone proteins is associated with the control of gene expression
Clusters of methylated cytosine nucleotides binds to a protein that prevents activators from binding to DNA
Methylated histone proteins are associated with inactive regions of chromatin.
Acetylated histones make DNA more accessible for transcription
Chromatin-remodeling complexes
Modify histones, DNA, and chromatin
→ ATP dependent remodeling factors
→ nucleosome sliding
→ Remodeled nucleosome
→ nucleosome displacement
→ histone replacement
Posttranscriptional Regulation
Gene expression can be controlled after transcription, with mechanisms such as:
Alternative splicing
RNA editing
mRNA degradation
RNA interference RNAi
Alternative splicing
Introns are spliced out of pre-mRNAs to produce the mature mRNA that is translated
Alternative splicing recognized different splice sites in different tissue types
The mature mRNAs in each tissue possess different exons, resulting in different polypeptide products from the same gene
RNA editing creates mature mRNA that are not truly encoded by the genome
Ex: apolipoprotein B exists in 2 isoforms
→ one isoform is produced by editing the mRNA to create a stop codon
→ this RNA editing is tissue-specific
Other means of control:
Transportation: movement of the mRNA may be interfered with
Translation Repressor Proteins: interfere with translation
Degradation of mRNA: loss of poly A tail
→ mature mRNA molecules have various half lives depending on the gene and the location (tissue) of expression
→ the amount of polypeptide produced from a particular gene can be influenced by the half life of the mRNA molecules
Small RNAs act after transcription has occurred
miRNA (micro RNA)
→ 22 nucleotides in length
→ involved in repression of a gene
siRNA (small interfering RNA)
→ dsRNA
→ 20-24 nucleotides
circRNA (small circular RNA)
→ degrade miRNA and siRNA
snRNA (small nuclear RNA)
snoRNA (small nucleolar RNA)
piRNA (piwi-interacting RNA)
→ largest non-coding RNAs
→ help to form RNA-protein complexes
→ many sub-types
RNA interference (RNAi): a biological process in which RNA molecules inhibit gene expression or translation, by neutralizing targeted mRNA molecules
Two types of small RNA molecules - miRNA and siRNA - are central to RNAi
CRISPR
Clustered regularly interspaced short palindromic repeats
Development of the genome editing technique was discovered by Emmanuelle Charpentier and Jennifer Doudna (2020 Nobel prize recipients)
DNA sequences found in the genomes of prokaryotes
Derived form DNA fragments of bacteriophages that had previously infected the cell
Foreign DNA recognized by a cell
Cas enzymes (such as Cas9 - CRISPR associated protein) and RNA become precision-guided weapons
Together, the viral RNA and the Cas enzymes drift through the cell
If they encounter genetic material from a virus that matches the CRISPR RNA, the RNA latches on tightly
The Cas enzymes then chop the DNA in two, preventing the virus from replicating
Protein Degradation
Proteins are produced and degraded continually in the cell
Proteins to be degraded are tagged with ubiquitin
Degradation of proteins marked with ubiquitin occurs at the proteasome