ch 5/6 hypo/hyper kinesia
Hypokinetic Movement Disorders
Definition of Hypokinesia:
Refers to too little voluntary movement.
Definition of Hyperkinesia:
Refers to too much involuntary movement.
Key Functions Benefiting from Non-English Movements:
Posture enhancement
Maintenance of normal tone
Initiation of volitional movement
Learning of new movements
Impact of Basal Ganglia Dysfunction
Symptoms Associated with Dysfunction:
Abnormal tone
Abnormal posture
Extraneous motor movements
Importance of Basal Ganglia:
Vital for regulating movement smoothly
Involvement in hypokinesia and hyperkinesia due to its regulatory role
Potential coexistence of both conditions
Parkison's Disease
Overview of Parkinson's Disease:
Most common disease affecting the basal ganglia
Related to the degeneration of substantia nigra which produces dopamine.
Consequences of Dopamine Deficiency:
Leads to basal ganglia dysfunction, manifesting as hypokinesia and possibly tremors.
Medication Treatment:
Use of Levodopa, commercial name Sinemet
Initial effectiveness, followed by reduced efficacy over time.
Patient presentation varies by medication cycle leading to changing symptoms between hypokinesia and hyperkinesia.
Visual Diagnosis:
Notable cases like Michael J. Fox showcase the on-off effects related to medication.
Clinical Presentation of Symptoms
Gait in Parkinson's Disease:
Reduced range of motion and stiff posture
Lack of arm movement when walking
Tremor at Rest:
Typical characteristic (4-6 Hz)
Quick tremors usually observed in hands, fingers, and potentially vocal cords.
Rigidity:
Cogwheel Rigidity:
Contrast with spasticity; people with rigidity don't resist movement as spastics do but are tight and stiff in motion.
Speech Implications:
Hypokinetic Dysarthria Characteristics:
Monopitch, monoloudness, reduced stress with appropriate silences
Short rushes of speech
Breathiness in voice
Symptoms lead to difficulty in:
Initiating speech (bradykinesia)
Achieving proper articulation and clarity.
Patients may describe their voice as weak or quiet.
Impact on Daily Life and Functioning
Social Interaction Effects:
Reduced facial affect known as masked facial affect
Lack of animation and reduced expressiveness can affect social dynamics.
Writing Changes:
Micrographia: progressively smaller handwriting as disease advances.
Deterioration in repetitive motor tasks with decreasing speed and amplitude.
Awareness and Perception:
Patients often lack insight about reduced animation or expressiveness.
Family members often observe changes first, noting they are less animated.
Diagnosis and Evaluation
Oral Motor Examination:
Visual assessment may not reveal hypokinesia until movement is elicited.
Connected speech assessments help reveal subtle deficits not apparent at rest.
Assessment Techniques:
Use of sustained vowel tasks to draw out vocal fold issues
Examination of repetition and elicitation of speech tasks to gauge ability.
Therapeutic Approaches
Working on Intent in Therapy:
Encouraging patients to speak with intent which can enhance strength, loudness, and clarity.
Involves altering motor plans to ensure that the basal ganglia's impact accommodates for motor planning.
Feedback and Adjustment:
Recording sessions to assist patients in understanding perceived loudness vs. actual loudness in their speech patterns.
Hyperkinetic Dysarthria
Definitions and Characteristics:
Hyperkinetic dysarthria arises from extra involuntary movements (hyperkinesias).
Observed through involuntary movements affecting speech.
Common Types of Hyperkinesia:
Tardive dyskinesia: long-term medication effects
Myoclonus: quick involuntary jerks in body parts
Chorea: significant and noticeable dance-like movements affecting speech
Description of Dysarthria due to Hyperkinesia:
Characterized by pauses and interruptions in speech as movements impact fluency.
Conclusion
Understanding basal ganglia functionality is crucial in recognizing and managing both hypokinetic and hyperkinetic dysarthrias. Observational assessments alongside therapeutic management are vital to improving patient outcomes.