Acid-Base
Define and recognize acid/base disorders
Arterial pH usually 7.35 - 7.45
less acidosis and more alkalosis
disturbances affect several organ systems and etiology can be drug related changing drug therapy and therapeutic plan
Physiology HCO3- + H+ ⇆ H2CO3 ⇆ CO2 + H2O
HCO3- basic retain in kidneys more basic/alkalotic
CO2 - acidic retain in lungs more acidic/acidosis
Disorder | pH | PaCO2 | HCO3- | Causes |
Normal | 7.35-7.45 | 35-45 mmHg | 22-26 mEq/L | |
Metabolic Acidosis | lower | lower (compensate) | lower | elevated AG M.U.D.P.I.L.E.S. normal AG G.R.E.E.D |
Metabolic Alkalosis | higher | higher (compensate) | higher | D.C. N.E.V.E.R |
Respiratory Acidosis | lower | higher | higher (compensate) | hard or difficulty breathing |
Respiratory Alkalosis | higher | lower | lower (compensate) | Huffed & P.U.F.T |
pH matches underlying disorder telling clinically
Same metabolic Different respiratory
compensation is the one that does not match disorder
younger can compensate for longer elderly cannot
Metabolic Acidosis low pH low HCO3-
Reduced excretion of H+ and exogenous sources of Cl-
Anion Gap secondary marker to help tell cause of acidosis
AG = Na+ - (Cl- + HCO3-) usually 7-16
Treatment - correct underlying cause maybe Sodium Bicarbonate controversial
ADR hypernatremia, volume overload, over-alkalinization, hypokalemia
CKD chronic bicarb tablets to offset disease
Dialysis
Elevated AG Metabolic Acidosis
Causes - M.U.D.P.I.L.E.S
methanol, metformin
uremia
diabetic ketoacidosis —> glucose and ketones
propylene glycol
ischemia,intoxication
lactic acidosis —> lactate
ethanol,ethylene glycol —> SCr/BUN
salicylates,starvation —>plasma osmolal gap
Normal AG Metabolic Acidosis
Causes - G.R.E.E.D
GI loss (diarrhea)
renal loss (renal tubular acidosis)
endocrine (hypoaldosteronism)
excess Cl-
drug (acetazolamide)
Metabolic Alkalosis high pH high HCO3-
loss of H+/Cl- and net increase in HCO3- a
Causes - D.C N.E.V.E.R
diuretics
contraction (intravascular volume)
nasogastric suctioning
endocrine (hyperaldosteronism, mineralocorticoids)
vomiting
excess alkali
refeeding (hypokalemia)
Treatment - correct underlying cause
fix fluid deficits and replace Cl - and/or K+ as appropriate
acetazolamide (carbonic anhydrase inhibitor) dumps the bicarb
hydrochloric acid replaces the Cl-
Respiratory Acidosis low pH high CO2
respiratory failure leading to reduced CO2 excretion
Causes - things that make you stop or hard breathing
Pulmonary - COPD, Asthma, Aspiration, Mechanical ventilation
Cardiopulmonary - Cardiac arrest , Edema, Embolism
Neurologic - CNS depressants, cerebral vascular accident, seizure
Neuromuscular - multiple sclerosis, GBS, myasthenia gravis
Treatment - correct underlying cause and respiratory support
Respiratory Alkalosis high pH low CO2
hyperventilation leading to increased CO2 excretion
Causes - Huffed & P.U.F.T
hyperventilation,hypoxia
pregnancy, pneumonia
uncus (CNS pathology)
fever, heart failure
trauma
Treatment - correct underlying cause and rebreathe expelled air
Mixed Acid-base disorders
mixed metabolic and respiratory disorders
identify primary disorder and access compensation